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1 sis of RNA degradome data suggested that the binding of Arabidopsis ARGONAUTE7 to a noncleavable targ
2 pac1 is thought to exert its effects through binding of cAMP leading to a conformational change in Ep
5 -2 in a substrate-selective manner, with the binding of inhibitor to a single monomer sufficient to i
6 luciferase reporter assays revealed a direct binding of NANOG to a transcriptionally active site in a
10 at this blocking activity may originate from binding of the peptide to a sequence of SdrC involved in
11 s 1-101) pull downs showed sixfold-increased binding of vinculin and, to a lesser extent, alpha-actin
14 This analysis highlights that the strong binding of myosin Ic to actin is dominated by the ADP st
15 that Prep1 normally acts by restricting DNA binding of transcription factors to adipogenic enhancers
17 ed with Dectin-1 revealed that the forces of binding of Dectin-1 to all of the strains were similar,
20 op was not required for fibronectin-mediated binding of gp120 to alpha4beta7, nor did V2-specific ant
21 analysis revealed that OA-NO2 increased the binding of Nrf2 to an antioxidant response element in th
22 and enzyme activity are regulated in part by binding of purine nucleotides to an allosteric regulator
25 ecovery phase from a hypoxic injury and that binding of neuronal uPA to astrocytic uPAR induces astro
29 thic helix H10 is responsible for peripheral binding of dimeric tubulin to biomimetic "mitochondrial"
30 r, our studies suggest that PML mediates the binding of PER2 to BMAL1 in the BMAL1/CLOCK heterodimer
35 f Reb A-receptor interaction, as a result of binding of Reb A to BSA, which may ultimately lead to mo
36 affinity than Ca(2+)-free CaM (apoCaM); the binding of CaMKII peptide to CaM in return increases the
37 2 were present in human carotid plaques, and binding of MBL to CC was confirmed in vivo by immunohist
38 emokines, the compounds did not activate G12 Binding of J113863 to CCR2 or CCR5 also induced the recr
41 in MDA-MB-231 than MCF-7 potentially due to binding of hyaluronic acid to CD44 expressed by MDA-MB-2
42 e mutant FHR-1 protein strongly competed the binding of FH to cell surfaces, impairing complement reg
46 t, we show that direct, cell-cycle-regulated binding of M18BP1 to CENP-A nucleosomes recruits the Mis
47 in, or phosphorylation site causes excessive binding of RAD51 to CFS loci and impairs CFS expression.
48 ght be advantageous for entropically favored binding of unfolded OMPs to chaperones and, by facilitat
50 heat shock protein 90 (HSP90) and stabilizes binding of this chaperone to CMA substrates as they bind
51 ort a model of HCV infection influencing the binding of transcription factors to cognate sites in the
53 anges could be explained by predicted direct binding of miRNAs to corresponding mRNAs, suggesting tha
55 tudies disclosed the molecular bases for the binding of 19 to CXCR4 and for its improved potency comp
57 n the context of dynein alone, we found that binding of LIS1 to DDB does not strongly disrupt process
58 ated Atf1 phosphorylation does not stimulate binding of Atf1 to DNA but, rather, establishes a platfo
61 ic accessory subunit (POLG2), which enhances binding of Pol gamma to DNA and promotes processivity of
64 The vast majority of mutations do not affect binding of dynein to dynactin and BICD2 and are therefor
65 hat ENb-TRAIL blocks EGFR signalling via the binding of ENb to EGFR which in turn induces DR5 cluster
66 e previously reported that sterols stimulate binding of UBIAD1 to endoplasmic reticulum (ER)-localize
69 of band 3 (cdb3-PO4) with Kd = 14 nM; (iii) binding of cdb3-PO4 to erythrocyte membranes is inhibite
72 ise that stereospecific but necessarily weak binding of tropomyosin to F-actin is required for effect
76 The aim of this study was to evaluate the binding of IgE to five recombinant beta-conglutin isofor
79 ing site provide the molecular mechanism for binding of dectin-2 to fungal mannans and also to bacter
84 Separation-of-function mutations leave the binding of TPX2 to gamma-TuRC intact, whereas branching
86 nes to form nucleosomes, which restricts the binding of transcription factors to gene regulatory sequ
87 Glycan array analysis confirmed selective binding of the CRD to glycans that contain Manalpha1-2Ma
93 f Rad51 in an HDAC4-dependent manner and the binding of Ubc9 directly to HDAC4, which led to Ubc9 ace
94 the effect of N-linked glycosylation on the binding of Herceptin to HER2 protein in breast cancer an
97 than murine (m) PD-1 interactions, stronger binding of hPD-1 to hPD-L2 than hPD-L1, and comparable b
105 These results likely reflect insufficient binding of mAgrin to hypoglycosylated alpha-DG on muscle
108 site within DARC is key to the preferential binding of DBP to immature reticulocytes, which is the p
111 inium-labeling of AtRALF1 indicated that the binding of AtRALF1 to intact roots is CML38-dependent.
113 lating platelet activation that involves the binding of MMP-2 to integrin alphaIIbbeta3 and the subse
114 ation at Ser-1441/1443, leading to increased binding of Rac1 to IQGAP1 and thus higher levels of cort
115 ed an off-cycle pathway involving reversible binding of molecular oxygen to iridium, which contribute
117 nding specificities, why lipid increases the binding of apoE to its receptor, and why specific residu
119 structural and thermodynamic consequences of binding of each ligand to its allosterically coupled sit
125 is regularity is reflected in the consistent binding of dArmRP to (KR)-peptides, where affinities dep
127 single domain antibody mimics the effect of binding of HIV integrase to LEDGF which is crucial for H
128 aper describes an assay that can profile the binding of a protein to ligands and can rank the affinit
132 Thus, we speculate that if we induce the binding of platelets to lymphocytes, we will be able to
133 insic fluorescence enhancement observed upon binding of LM pectin to lysozyme was correlated with the
135 esults (at 15-25 A resolution) indicate that binding of ATP to MalK2 promotes an asymmetric, semi-clo
137 eceptors upon ligand binding, the sequential binding of receptors to membrane coupling proteins, and
141 ylation of CRMP-2, which results in elevated binding of CRMP-2 to microtubules and a concomitant incr
143 n the mutant CALR C terminus influences both binding of mutant CALR to MPL and activation of MPL sign
144 and its chaperone function are regulated by binding of ATP/ADP to mtHsp70's nucleotide-binding domai
147 ensal Bacteroides thetaiotaomicron decreased binding of E. coli to mucin and increased the attachment
148 show in vitro and ex vivo that CotE enables binding of spores to mucus by direct interaction with mu
150 eriments revealed that FGF13 potentiates the binding of calmodulin to NaV1.5 and that phosphomimetic
151 ow-coordinated Cu atoms and the weakening of binding of Cu to neighboring atoms help drive this proce
154 t that a relatively simple mechanism for the binding of ssDNA to non-specific SSBs may be at play, wh
155 ises questions about the possible off-target binding of this tracer to non-tau molecules associated w
161 We deploy this approach to show that the binding of cGMP to pacemaking ion channels is weakened b
162 of magnitude increase is due to preferential binding of PHF1 to partially unwrapped nucleosomes, and
165 eta(2)-O2)] is attributed to the enthalpy of binding of O2 to [Pd(IPr)2] (-14.5 +/- 1.0 kcal/mol) tha
168 hibitory monoclonal antibody to CyRPA blocks binding of CyRPA to PfRh5 and complex formation thus ill
169 n thus would be favorable for the postulated binding of E7 to phosphatidylethanolamine lipid headgrou
173 layers previously oxidized, and the covalent binding of amino-oligonucleotides to pure p-MBA monolaye
177 -AC), patient body motion (+MC and -MC), and binding of the tracer to red blood cells (+BB and -BB) w
184 tantly, pharmacological stabilization of the binding of calstabin2 to RyR2 channels, which prevents C
187 further demonstrate that MMP expression and binding of this complex to SBEs can either be enhanced b
190 influencing DNA binding, actually increased binding of Dax1 to Sf1 to further enhance transcriptiona
191 ranslational support to our findings, better binding of annexin A2 to sialostatin L2 in sera from 21
195 toplasmic relocalization of Cat L, decreased binding of CUX1 to Snail and the E-cadherin promoter, re
196 (APBA) to such vesicles results in specific binding of this reagent to some of the pendent cis-diol
198 study suggests the function of the membrane binding of INS is to stabilize pvLAAD structure and incr
199 tween Vn and lipid membranes, which leads to binding of autoinhibited Vn to supported lipid bilayers,
203 noprecipitation analyses reveal constitutive binding of CREB to target gene promoters in the absence
205 a quiescent state in CRC cells by disrupting binding of beta-catenin to TCF1 and TCF3 and regulation
206 ty acids was found to play a key role in the binding of a glycolipid to the carbohydrate recognition
211 tion of TFPI-2 with Ap-2alpha attenuated the binding of AP-2alpha to the MMP-2 promoter, therefore re
212 ChIP experiments revealed a preferential binding of BRD4 to the Myog promoter during C2C12 myobla
213 e membranes; and (v) phosphorylation-induced binding of cdb3-PO4 to the membrane-spanning domain of b
214 sly described auto-inhibition mechanism, the binding of Chp2 to the H3K9me3 mark is not analogously r
216 hat neuronal activity induces a preferential binding of CRTC1 to the transcriptional complex in CRE/T
218 eristics of hemoglobin (Hb) changes with the binding of dioxygen (O2) to the heme prosthetic groups o
223 protein synthesis typically begins with the binding of eIF4F to the 7-methylguanylate (m(7)G) cap fo
224 y, we found that cAMP elevation caused rapid binding of Epac2A to the beta-cell plasma membrane, wher
225 and absorption studies support the multiple binding of F-groups to the C-center of CO2 (CF 3.190(9)-
226 Blockade of alphavbeta1 prevented direct binding of fibroblasts to the latency-associated peptide
231 The BabA adhesin mediates high-affinity binding of Helicobacter pylori to the ABO blood group an
232 ich were found to bind IL-15, to inhibit the binding of IL-15 to the IL-2Rbeta chain or the prolifera
235 iquitination by MDM2, a process initiated by binding of MDM2 to the intrinsically disordered transact
236 tinct domains designated S1(A) through S1(D) Binding of MERS-CoV to the cell surface entry receptor d
237 ence of the fluoroionophore is enhanced upon binding of Na(+)-ions to the highly selective aza-crown
241 ch-Rheb regulatory loop, in which the cyclic binding of Notch1 to the Notch-responsive elements (NREs
242 obably because these mutations decreased the binding of NS1 to the cleavage and polyadenylation speci
244 rther showed that the sdAb-Fcs do not affect binding of PCSK9 to the LDLR but rather block its induce
245 proteinuria through cGMP- and PKG-dependent binding of PPAR-gamma to the TRPC6 promoter, which inhib
247 mines the initial crosslinking of cisplatin, binding of proteins to the genome largely contributes to
248 d in this species, possibly due to a favored binding of Pt to the polysaccharide-rich cell wall of th
252 with BRG1 in differentiating melanocytes and binding of SOX10 to the Tyrp1 distal enhancer temporally
253 ty protein 1 (Sp1), which then inhibited the binding of Sp1 to the VEGF promoter to reduce VEGF expre
254 chanistic model in the field postulates that binding of ssDNA to the OB core induces the flexible, un
255 firmed that Lyn overexpression decreased the binding of STAT6 to the promoter region of Muc5ac in mic
260 these data suggested that the expression and binding of TFs contributed to the establishment of subse
261 he experiments and shows preferential strong binding of the AFP to the fast growing surfaces of the s
265 eparation effectively prevents the potential binding of the counteranion to the cationic metal center
268 8], which we find additionally required for binding of the MCC to the APC/C and for MCC disassembly.
269 d as a fusion to the relevant enzyme so that binding of the receptor to the immobilized ligand brings
273 ear magnetic resonance demonstrated specific binding of UM101 to the computer-aided drug design-targe
275 ion has previously been shown to inhibit the binding of VWF to the platelet surface receptor glycopro
280 was transcriptionally upregulated by direct binding of HOXA5 to their promoter sequences as demonstr
284 from an axonal injury express uPAR and that binding of uPA to this uPAR promotes axonal recovery by
286 leukemic cell differentiation revealed that binding of S100A9 to Toll-like receptor 4 (TLR4) promote
287 the capsid (Gly299Lys/Ala300Lys) altered the binding of the capsid to transferrin receptor type 1 (Tf
289 ease of intracellular calcium concentration, binding of calcium to troponin in the actin-containing t
291 ha electrostatically repels the DNA, and the binding of IkappaBalpha appears to twist the NFkappaB he
293 the Tup-family corepressors) suppress direct binding of Rst2 to UAS2, but this suppression is counter
294 ve mass spectra matching, the promiscuity of binding of cisplatin to ubiquitin is revealed, with 14 d
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