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1                                Glucosides of biochanin A (4'-O-methylgenistein) and pratensein (3'-hy
2 orogenic acid (15.4 +/- 0.05 mug mg(-1)) and biochanin A (9.6 +/- 0.06 mug GAE mg(-1)), while minor c
3   From a panel of phytoestrogen isoflavones, biochanin A (BCA) was identified as the most potent indu
4 n (genistein 7-sulfate) and 2 metabolites of biochanin A (genistein and genistein 7-sulfate) were det
5 n, isoformononetin glycoside and malonylated biochanin A glycoside the major compounds.
6 timulated release of PSA, presumably because biochanin A increased UDPGT and increased the intracellu
7                                Additionally, biochanin A markedly decreased prostate specific antigen
8                    Intact cells treated with biochanin A produced testosterone-glucuronide from testo
9                                              Biochanin A significantly decreased the testosterone-sti
10 nap2 mutants and show that the phytoestrogen biochanin A specifically reverses the mutant behavioral
11 f genistein 7-sulfate from genistein or with biochanin A sulfate, genistein 7-sulfate, or genistein f
12 ted methylated form of each isoflavone and a biochanin A sulfate.
13                     Of the compounds tested, biochanin A was the most potent, with increased activity
14                              Neoponcirin and biochanin A were identified for the first time in the Me
15                                  Chrysin and biochanin A were the most potent BCRP inhibitors, produc
16  flavonoids, isoflavonoids (formononetin and biochanin A) and flavones (7,4'-dihydroxyflavone), respe
17 flavones (genistein, genistin, daidzein, and biochanin A) and soy phytochemical concentrate exhibit d
18 oybean isoflavones (genistein, daidzein, and biochanin A) are ERbeta-selective agonists of transcript
19 isoflavones (genistein, daidzein, glycitein, biochanin A, and formononetin), lignans (secoisolaricire
20 ding the flavonol kaempferol, the isoflavone biochanin A, and the chalcone isoliquiritigenin.
21 ch are aglycons, namely daidzein, genistein, biochanin A, and two of which, daidzin and genistin, are
22                 Environmental half-lives for biochanin A, genistein, and equol are expected to vary o
23                                              Biochanin A, genistein, and equol degraded relatively sl
24 s of the isoflavones daidzein, formononetin, biochanin A, genistein, and equol were studied under sim
25 expressed in LNCaP cells and was enhanced in biochanin A-treated LNCaP cells.
26 fate, genistein 7-sulfate, or genistein from biochanin A.
27 cubated with [4-(14)C]genistein and [4-(14)C]biochanin A.
28  cell lines by the isoflavones genistein and biochanin A.
29 ein, genistein, glycitein, formononetin, and biochanin-A and their mammalian metabolites equol and O-
30 avonoid metabolites such as formononetin and biochanin-A that peaked at 12 to 18 h following elicitat

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