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1 fate, genistein 7-sulfate, or genistein from biochanin A.
2 cubated with [4-(14)C]genistein and [4-(14)C]biochanin A.
3 cell lines by the isoflavones genistein and biochanin A.
5 orogenic acid (15.4 +/- 0.05 mug mg(-1)) and biochanin A (9.6 +/- 0.06 mug GAE mg(-1)), while minor c
6 ein, genistein, glycitein, formononetin, and biochanin-A and their mammalian metabolites equol and O-
7 flavonoids, isoflavonoids (formononetin and biochanin A) and flavones (7,4'-dihydroxyflavone), respe
8 flavones (genistein, genistin, daidzein, and biochanin A) and soy phytochemical concentrate exhibit d
9 isoflavones (genistein, daidzein, glycitein, biochanin A, and formononetin), lignans (secoisolaricire
11 ch are aglycons, namely daidzein, genistein, biochanin A, and two of which, daidzin and genistin, are
12 oybean isoflavones (genistein, daidzein, and biochanin A) are ERbeta-selective agonists of transcript
13 From a panel of phytoestrogen isoflavones, biochanin A (BCA) was identified as the most potent indu
14 n (genistein 7-sulfate) and 2 metabolites of biochanin A (genistein and genistein 7-sulfate) were det
17 s of the isoflavones daidzein, formononetin, biochanin A, genistein, and equol were studied under sim
19 timulated release of PSA, presumably because biochanin A increased UDPGT and increased the intracellu
23 nap2 mutants and show that the phytoestrogen biochanin A specifically reverses the mutant behavioral
24 f genistein 7-sulfate from genistein or with biochanin A sulfate, genistein 7-sulfate, or genistein f
26 avonoid metabolites such as formononetin and biochanin-A that peaked at 12 to 18 h following elicitat
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