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1 of respiratory processes elicited increased biofilm formation.
2 mple, in host colonization, persistence, and biofilm formation.
3 acillus subtilis but is essential for robust biofilm formation.
4 re associated with decreases in motility and biofilm formation.
5 e proteins in membrane stress, motility, and biofilm formation.
6 nificantly (P < 0.01) enhance its endogenous biofilm formation.
7 titer of phosphorylated SaeR is required for biofilm formation.
8 for early, intermediate, and late stages of biofilm formation.
9 variety of virulence factors and to promote biofilm formation.
10 ect the expression of cytolytic toxicity and biofilm formation.
11 PIs in P. mirabilis, and control crystalline biofilm formation.
12 ere tested for antibiotic susceptibility and biofilm formation.
13 ulence factor involved in immune evasion and biofilm formation.
14 bacterial attachment to surfaces but reduces biofilm formation.
15 se of cytosolic DNA contributed to increased biofilm formation.
16 ess tolerance, virulence, phage defense, and biofilm formation.
17 tachment to biomaterials, the first stage of biofilm formation.
18 eeded for pilus assembly, coaggregation, and biofilm formation.
19 eased SepA production, and in turn augmented biofilm formation.
20 ing sporulation to a derived role regulating biofilm formation.
21 s of the TFN membranes, resulting in limited biofilm formation.
22 ety of environment-specific cues to regulate biofilm formation.
23 that the Acinetobacter T1SS is required for biofilm formation.
24 rs, including virulence factor secretion and biofilm formation.
25 genes of unknown functions are critical for biofilm formation.
26 the bacterial infections are associated with biofilm formation.
27 ability to determine swarming initiation and biofilm formation.
28 % of bacterial infections are accompanied by biofilm formation.
29 ulence factors (eg, adhesins and toxins) and biofilm formation.
30 a, and are involved in cell colonization and biofilm formation.
31 ponsible for virulence factor production and biofilm formation.
32 the importance of Fap1 glycosylation in the biofilm formation.
33 the relationship between quorum sensing and biofilm formation.
34 binding partners, as well as for early stage biofilm formation.
35 essenger that controls cellular motility and biofilm formation.
36 rogrammed cell death with quorum sensing and biofilm formation.
37 l-acquired infections, with a propensity for biofilm formation.
38 portant for cyclic-di-GMP production and for biofilm formation.
39 that curli contributes most significantly to biofilm formation.
40 sly unidentified targets to combat bacterial biofilm formation.
41 ted by A. actinomycetemcomitans enhanced the biofilm formation.
42 erons promote robust secondary P. aeruginosa biofilm formation.
43 to delayed swarming initiation and increased biofilm formation.
44 ress responses, cell division, motility, and biofilm formation.
45 ts was associated with Psl EPS expression or biofilm formation.
46 derstood cell signaling pathways involved in biofilm formation.
47 , the complete removal of H2 O2 impaired the biofilm formation.
48 teractions play a critical role in promoting biofilm formation.
49 genes with previously unappreciated roles in biofilm formation.
50 including DNA competence, immune evasion and biofilm formation.
51 bution of fibrin and staphylokinase (Sak) to biofilm formation.
52 opted as a model organism to study bacterial biofilm formation.
53 ial motility and promote autoaggregation and biofilm formation.
54 s and is required for bacterial adhesion and biofilm formation.
55 design of inhibitors that target K pneumonia biofilm formation.
56 l, and protein was an influential factor for biofilm formation.
57 ever, little is known about its mechanism of biofilm formation.
58 nce bacterial adhesion, and propagation, and biofilm formation.
59 that argB was essential for ciaR to regulate biofilm formation.
60 ls) for prevention of bacterial adhesion and biofilm formation.
61 tion-dependent manner and were proficient in biofilm formation.
62 es, including the ability to alter bacterial biofilm formation.
63 cy in infections associated with significant biofilm formation.
64 role in surface-sensing during swarming and biofilm formation.
65 genously supplied homoagmatine also restored biofilm formation.
66 tical for host cell adherence, invasion, and biofilm formation.
67 th PelB are also essential for PEL-dependent biofilm formation.
68 spermidine, but not homospermidine, restored biofilm formation.
69 t or more exposed during the early stages of biofilm formation.
70 increasing expression of AtlA and FnBPA and biofilm formation.
71 erall, sRNAs highlight different pathways to biofilm formation.
72 dine production, rhamnolipid production, and biofilm formation.
73 identify two mutually exclusive pathways for biofilm formation.
74 n metabolism, and DNA repair, as well as via biofilm formation.
75 QseB, the response regulator responsible for biofilm formation.
77 ded periods of exposure to bile salts led to biofilm formation, a conserved phenotype that we observe
78 r c-di-GMP (or cyclic diguanylate) regulates biofilm formation, a physiological adaptation process in
80 gtfP gene expression, glucan production and biofilm formation ability that was lost in DeltaciaR, in
81 s were determined in vitro by agar dilution, biofilm formation, adhesion, invasion and intracellular
82 ically significant reduction in human plaque biofilm formation after 72 hours of incubation in an exp
84 o and in vivo via antibiotic susceptibility, biofilm formation and adhesion, and invasion and intrace
85 ociated with "cured" and "not cured." Strong biofilm formation and aminoglycoside resistance were ass
89 tant/cleaner generator that limits undesired biofilm formation and as a washing agent in waterless ur
94 d treatment effectiveness is associated with biofilm formation and drug recalcitrance is thought to b
95 osphorylated PtsN as a negative regulator of biofilm formation and establish one of the first known r
96 asminogen activation that prevents S. aureus biofilm formation and induces detachment of existing bio
97 iscuss the role of antibiotics in modulating biofilm formation and interspecies communication in bact
98 s essential for survival, growth, virulence, biofilm formation and intra/interspecies interactions.
100 bility of raffinose to inhibit P. aeruginosa biofilm formation and its molecular mechanism opens new
104 tion in eukaryotes and archaea, to bacterial biofilm formation and specialized roles in natural produ
106 ted, inactivation of Synpcc7942_2071 enables biofilm formation and suppresses the planktonic growth o
108 molecular mechanisms that control S. aureus biofilm formation and the basis for the recalcitrance of
110 we observed an inverse relationship between biofilm formation and the magnitude of the applied poten
112 om secondary metabolism, ScmR also represses biofilm formation and transcriptionally activates ATP sy
113 omoserine lactone (3O-C12-HSL), that promote biofilm formation and virulence via interbacterial commu
114 fection of the bone/joint tissue and implant biofilm formation and were biocompatible with enhanced o
115 ve changes in liquid media growth, motility, biofilm formation, and acute insect virulence, but not i
116 s involved in mating, filamentous growth and biofilm formation, and also influences cAMP-regulated pr
117 d mitochondrial aconitase activity, impaired biofilm formation, and extenuated mouse organ colonizati
120 nspecific opsonophagocytic killing, enhanced biofilm formation, and increased adhesion to nasopharyng
121 s to SAPP, while monospecies and heterotypic biofilm formation, and invasion of oral epithelial cells
122 ective in the detection of initial stages of biofilm formation, and may be useful in the early preven
123 egulator of secondary metabolism, virulence, biofilm formation, and other stationary phase processes.
125 d by P. aeruginosa for host cell attachment, biofilm formation, and twitching motility, making this s
126 tes many natural phenotypes (e.q. virulence, biofilm formation, antibiotic resistance), and its compo
127 including communication via quorum sensing, biofilm formation, antibiotic resistance, and pathogenes
129 Chronic bacterial infections associated with biofilm formation are often difficult to resolve without
130 tures of spermidine required for B. subtilis biofilm formation are unknown and so are the molecular m
131 approaches in modulating quorum sensing and biofilm formation as a nonlethal method, as well as narr
132 vertheless, pili removal is not required for biofilm formation as evident by a piliated mutant of S.
133 d closed states is important for V. cholerae biofilm formation, as RbmA variants with switches that a
134 ad to new insights into the basic biology of biofilm formation, as well as new strategies for therape
135 re model to study the formation of bacterial biofilm formation associated with the airway epithelium,
138 ved in processes such as twitching motility, biofilm formation, bacteriophage infection, surface atta
139 monstrates key differences in the process of biofilm formation between cyanobacteria and well-studied
140 compendium of c-di-GMP pathways connected to biofilm formation, biofilm-associated motilities, and ot
141 nd encoded for a number of traits related to biofilm formation (BopD), adherence (Epb pili), virulenc
142 bit altered exoprotein production or altered biofilm formation, but it was attenuated for survival up
143 oxin production and promotes pilus-dependent biofilm formation, but no specific biological functions
144 her, LL-37 significantly inhibited S. aureus biofilm formation, but no such effects were observed for
145 pipes, inhibiting culturable Legionella and biofilm formation, but promoted Legionella growth in pip
146 to C-methylation, which it is essential for biofilm formation, but that the length and symmetry of t
147 the genes ylbF, ymcA and yaaT are blocked in biofilm formation, but the mechanism by which they act h
148 ll adhesion during the accumulation phase of biofilm formation, but the molecular basis of this inter
149 ortant mechanism of cell accumulation during biofilm formation, but the underlying molecular mechanis
151 nd spermine in vitro Spermine also inhibited biofilm formation by altering the transcription of the v
158 ing mechanisms for bacterial persistence and biofilm formation by H. parainfluenzae and knowledge abo
160 hat the SaeRS TCRS also governs fermentative biofilm formation by positively influencing AtlA activit
162 l oral bacteria, and selectively inhibit the biofilm formation by S. mutans, indicative of its select
166 phenotypes, including cell shape, motility, biofilm formation, cell surface hydrophobicity, and sodi
167 the soil milieu, including the capacity for biofilm formation, cell surface modifications and cell a
168 C. albicans enhances filamentous growth and biofilm formation, colonization of the murine gastrointe
169 re deleted or added, were created to examine biofilm formation, colonization, and persistence in the
171 such as sporulation, competence, virulence, biofilm formation, conjugative plasmid transfer and anti
172 ound microenvironment, promotes adhesion and biofilm formation, decreases bacterial killing by neutro
173 tions in the lipid A structure, motility and biofilm formation defects, as well as changes in mannito
176 ole levels significantly decreased motility, biofilm formation, exopolysaccharide production and viru
177 mouse model of orthopedic implant-associated biofilm formation found that both SaeRS and SrrAB govern
178 for mutants with defective cytotoxicity and biofilm formation from 93 transposon insertion mutants p
179 fected autolysis, extracellular DNA release, biofilm formation, genetic competence, and bacteriocin p
181 hich is essential for virulence but inhibits biofilm formation, had been purged from the genome.
183 ans, causing acid-mediated cell death during biofilm formation; however, its complex structure limits
186 this work links for the first time increased biofilm formation in bacteria with a colonization advant
190 rrent study we report that oxygen influences biofilm formation in its capacity as a terminal electron
191 erococcus faecalis promotes Escherichia coli biofilm formation in low-iron conditions, thus facilitat
192 alian tissues that has been shown to enhance biofilm formation in multiple Gram-positive pathogens.
193 s at subinhibitory concentrations can impact biofilm formation in neighboring microbes, supporting th
195 The Cell wall-anchored protein Aap promotes biofilm formation in S. epidermidis, independently from
196 protein implicated in host colonization and biofilm formation in Staphylococcus aureus, provides ano
197 is required for Aap-dependent S. epidermidis biofilm formation in static and dynamic biofilm models.
199 ous polyamine content to the cell to control biofilm formation in the aquatic environment and within
201 obal regulators of the network that controls biofilm formation in the model bacterium Escherichia col
204 fcR translation and explaining the increased biofilm formation in this mutant compared to the wild ty
207 ype-independent anti-MrkA antibodies reduced biofilm formation in vitro and conferred protection in m
209 icant reductions in oxacillin resistance and biofilm formation in vitro and oxacillin persistence in
210 (ClfA), especially in combination, inhibited biofilm formation in vitro and the hematogenous implant-
213 TCRSs, SrrAB was the dominant system driving biofilm formation in vivo We propose a model wherein imp
215 is an essential structural component during biofilm formation, including initial bacterial adhesion,
217 logues to functionally replace spermidine in biofilm formation indicated that the aminopropyl moiety
218 This investigation of first stage and mature biofilm formation indicates that biofilm structure is st
219 dine-deficient DeltaspeD background restored biofilm formation, indicating that spermidine is require
220 innate immunity, adherence to host surfaces, biofilm formation, invasion into and growth within host
225 ure of a late stage biofilm, suggesting that biofilm formation is severely hampered in the natural en
229 by spermine supplementation, suggesting that biofilm formation may be regulated by variations in loca
230 in prominent biological processes, including biofilm formation, morphogenesis and cancer invasion.
233 nted support a model wherein SrrAB-dependent biofilm formation occurs in response to the accumulation
234 te cell wall hydrolase genes and disrupt the biofilm formation of MRSA clearly indicated that Inh2-B1
235 eport that A. actinomycetemcomitans promoted biofilm formation of S. parasanguinis in vitro and in vi
236 ation greatly reduces bacterial adhesion and biofilm formation of two most common pathogens responsib
238 reduce the rate of P. mirabilis crystalline biofilm formation on catheters, and increase the time ta
239 a PPARgamma agonist, significantly inhibits biofilm formation on epithelial cells by a mechanism tha
240 logical characteristics of Vibrio sp. B2 and biofilm formation on nanofiltration (NF) membrane coupon
243 reconstructed the sequence of events during biofilm formation on plastic in the harbor environment a
246 ised of septic arthritis, osteomyelitis, and biofilm formation on the implants in the surgical legs c
247 . coli and S. aureus cells, and prevents the biofilm formation on the membrane surface, producing exc
249 occus mutans metabolizes sucrose to initiate biofilm formation on the tooth surface and consequently
250 The aim of the present study is to compare biofilm formation on zirconia and titanium implant surfa
251 cterial surface colonization, a precursor to biofilm formation, only when planktonic bacterial inocul
252 c-di-GMP levels that then regulate genes for biofilm formation or for swarming motility-the output ph
254 h as defense against phages, growth control, biofilm formation, persistence, and programmed cell deat
257 itory factor enhances Pseudomonas aeruginosa biofilm formation, potentially contributing to cystic fi
260 common ensuing cooperative behaviors include biofilm formation, quorum responses, development, and sw
261 hey act to mediate communal behavior such as biofilm formation, quorum sensing, and motility by influ
262 results suggest that CfcR is a key player in biofilm formation regulation by the Rsm proteins in P. p
266 on of these and many other genes and impacts biofilm formation, secondary metabolite production, and
267 Treatment with PodA disrupts P. aeruginosa biofilm formation similarly to DNase, suggesting interfe
268 onsistent with the spermidine requirement in biofilm formation, single-cell analysis of this mutant i
269 irects transcription of factors required for biofilm formation specifically by activating csgD (agfD)
272 lonization and attenuates persister cell and biofilm formation, suggesting that mesalamine aids in di
273 e-cell resolution provides a new window into biofilm formation that will prove invaluable to understa
274 edical devices, which provide a scaffold for biofilm formation, thereby reducing susceptibility to an
275 ruginosa, one of the best studied models for biofilm formation, this process is mediated by rhamnolip
276 s reveal a new role of c-di-AMP in mediating biofilm formation through a CabPA/VicR/GtfB signaling ne
277 tial virus (RSV) infection induces bacterial biofilm formation through a mechanism of dysregulated ir
278 that c-di-GMP can regulate both motility and biofilm formation through a single effector in this surf
279 r the reduction of C. albicans virulence and biofilm formation through the inhibition of hyphal forma
280 s an exogenous cue that inhibits V. cholerae biofilm formation through the NspS-MbaA signaling system
281 the levels of spermidine required to inhibit biofilm formation through this pathway are unlikely to b
285 increased C. jejuni's growth, motility, and biofilm formation under microaerobic (5% O2) conditions.
286 s endometritis was also developed to monitor biofilm formation, utilizing bioluminescence imaging wit
287 lk of alkanes and PAHs in the fuel; however, biofilm formation was incipient and phenanthrene was min
288 When DMAE was coated on substratum surfaces, biofilm formation was inhibited for prolonged periods of
289 c toxicity in combination with low levels of biofilm formation was predictive of an increased risk of
291 indings suggested that the genes involved in biofilm formation were under some form of carbon catabol
292 and ALS3, which are also involved in fungal biofilm formation, were upregulated in the presence of S
294 ample of coordinated behavior in bacteria is biofilm formation, where individual cells organize into
295 e, sucrose, and fructose were inhibitory for biofilm formation, whereas galactose, lactose, and low c
296 of most antimicrobials against P. aeruginosa biofilm formation, which in turn depends on the presence
297 pes of surfaces have been designed to combat biofilm formation, while attempting to maintain osseous
300 in S. alvi likely function in attachment and biofilm formation, with knockouts experiencing a competi
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