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1 entified using a combination of clinical and biologic markers.
2 the scoring systems through addition of new biologic markers.
3 cal activity indices in CD and with numerous biologic markers.
4 ctious complications, and prognostic role of biologic markers.
5 ed with specific clinical manifestations and biologic markers and may be a useful addition to the bio
7 a decrease in profibrotic and proangiogenic biologic markers, and upregulation of lung CXCL11 mRNA a
12 rapy and tested microbiologic, clinical, and biologic markers as prognostic indicators for clinical s
14 We recommend that treatment response and biologic markers be incorporated into the American Joint
17 led to more pronounced beneficial effects in biologic marker expressions in pulmonary acute respirato
18 uggest that toenail samples provide a useful biologic marker for quantifying low-level arsenic exposu
19 inducible hsp70 in the model could provide a biologic marker for the prediction of clinical acute and
20 othelial progenitor cells may be a surrogate biologic marker for vascular function and cumulative car
22 tments, and the advent of valid and reliable biologic markers hold promise in the coming decade for d
23 We suggest that the use of such surrogate biologic markers in conjunction with F-cell numbers may
24 use of a combination of clinical factors and biologic markers is a promising strategy that needs to b
25 ntal diagnostic value over other imaging and biologic markers makes them easier to implement by other
28 by PTSD is corroborated using an independent biologic marker of immune response to infection, CMV-a t
32 was to review known clinical predictors and biologic markers of adverse clinical outcomes in acute l
34 dy adjusted for C-reactive protein (CRP) and biologic markers of inflammation such as CRP, interleuki
38 t appropriate statistical methods to analyze biologic markers of stress, such as salivary cortisol, r
40 fferent high-risk groups, screening for both biologic markers provided better prognostic information
41 al response was correlated with amelioration biologic markers such as cytokine production (IFN-gamma,
45 aggressive lymphoma but clinically validated biologic markers that can predict natural history to tai
46 There are literally dozens of clinical and biologic markers that have been proposed as being predic
47 uantitative Western analysis, is a sensitive biologic marker to assess the short-term responsiveness
48 nfluence this variation and that addition of biologic markers to AJCC staging improves determination
49 suggesting a potential to develop surrogate biologic markers to identify the onset and progression o
51 e were calculated for clinical variables and biologic markers using the Cox model for multivariate an
53 injury occurs in scleroderma and provides a biologic marker whose relationship to disease activity a
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