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1 division, are considered reliable markers of biological aging.
2 psychoactive medication do further impact on biological aging.
3 may be a key factor in HIV-related premature biological aging.
4           Telomere length may be a marker of biological aging.
5 ium surfaces, indicating a titanium-specific biological aging.
6 nditions of oxidative stress observed during biological aging.
7   Reduced telomere length may be a marker of biological aging.
8 ementia and mortality and may be a marker of biological aging.
9 ybrid rats, a well accepted animal model for biological aging.
10 on during development, oxidative stress, and biological aging.
11 ks support the applicability of the model to biological aging.
12 els under conditions of oxidative stress and biological aging.
13 chemical or photochemical modifications, not biological aging.
14  help us to better comprehend the process of biological aging.
15 onsequences contributing to disease risk and biological aging.
16 hronic environmental stressors to accelerate biological aging across multiple organ systems.
17      This suggests mechanisms of accelerated biological aging among the depressed, which can be indic
18 iency and discuss the actions of GH/IGF-1 on biological aging and lifespan.
19                     Telomere length reflects biological aging and may be influenced by environmental
20                Our results suggest that both biological aging and reduced regenerative capacity contr
21 ights into molecular processes that underlie biological aging and, perhaps more importantly, potentia
22 thelium that are known to be associated with biological aging, and cellular senescence markers in HIV
23          Telomere length (TL) is a marker of biological aging, and numerous studies have shown associ
24 e whether MDD is associated with accelerated biological aging, and whether depression characteristics
25 osclerosis is also associated with premature biological aging, as atherosclerotic plaques show eviden
26 ent diseases in the United States, caused by biological aging, autoimmune conditions, trauma, or iatr
27 no evidence for salient differences in neuro-biological aging between the two sensory regions, the ob
28     Modern programmed (adaptive) theories of biological aging contend that organisms including mammal
29  the same chronological age varied in their "biological aging" (declining integrity of multiple organ
30                                Senescence or biological aging impacts a vast variety of molecular and
31                                  The rate of biological aging in AD patients, which cannot be explain
32 nking internalizing disorders to accelerated biological aging in the first half of the life course, p
33                                     Measured biological aging in young adults can be used to identify
34           We applied these methods to assess biological aging in young humans who had not yet develop
35 ross-sectional data suggest that accelerated biological aging may be a mechanism through which sleep
36    Thus, the overproduction of noggin during biological aging may result in impaired osteoblast forma
37                       We show that increased biological aging (measured using epigenetic data from bl
38 S-mediated DNA damage results in accelerated biological aging of hVSMCs via 2 mechanisms: (1) Acute S
39 eloping MDD are characterized by accelerated biological aging, operationalized as shortened telomere
40 r the AHP amplitude is strictly dependent on biological aging or is modified by the training procedur
41                A series of chronological and biological aging parameters in XRCC1 heterozygous (HZ) m
42  oxidative modification of CaM during normal biological aging results in a reduced calcium sensitivit
43               Telomere length is a marker of biological aging that may provide a cellular memory of e
44                         During the course of biological aging, there is a gradual accumulation of dam

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