ライフサイエンスコーパス: biological half-life

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1 sms are with bioavailable concentrations and biological half-life.

2 etary manganese affected 54Mn absorption and biological half-life.

3 finity for its receptor and by extending its biological half-life.

4 be used to predict PFAS binding strength and biological half-life.

5 healing, but its use is hampered by a short biological half-life and lack of tissue selectivity.

6 c polypeptide (GIP) is hindered by its short biological half-life and rapid renal clearance.

7 a cell-permeable mimetic that increases the biological half-life and therefore enhances the biologic

8 [Carbonyl-11C]4a cleared the brain with a biological half-life averaging 41 min.

9 radioactivity was eliminated via the urine (biological half-life ca. 4 h), the urinary bladder wall

10 addiction treatment is limited by its short biological half-life (e.g., 8 h or shorter in rats).

11 like an antibody, has a considerably longer biological half-life (e.g., approximately 107 h in rats)

12 old increase in clotting activity, and has a biological half-life equivalent to recombinant wild-type

13 poor ribonuclease (RNase) resistance, short biological half-life, lack of tissue targeting, ineffici

14 ing from multivalency along with an improved biological half-life makes the tetravalent construct an

15 affinity are reflected by differences in the biological half-life, not the absolute uptake.

16 se holoenzyme activity was eliminated with a biological half-life of 1.3 hours, and the MEGC-PEG-rMET

17 PLGA based optimized formulation exhibited a biological half-life of 18.64 h, which was ~4.5 times lo

18 efflux from the whole brain, clearing with a biological half-life of 35 min.

19 25-30% of the total 123I administered with a biological half-life of 45 h.

20 -PEG-rMETase apoenzyme was eliminated with a biological half-life of 90 hours, an approximately 36-fo

21 Given the general observation that the biological half-life of a protein drug is significantly

22 inding protein, which naturally prolongs the biological half-life of growth hormone.

23 its physical half-life (78.41 h) matches the biological half-life of IgG antibodies.

24 ide a mechanistic explanation for the longer biological half-life of PFOS in humans, compared to PFOA

25 The biological half-life of scleral proteoglycans was signif

26 iation dose to the tumor (r = 0.9), with the biological half-life of the antibody in the tumor being

27 rganism OBT is a slow process with a tritium biological half-life on the order of months.

28 including controlled drug release, improved biological half-life, reduced toxicity and targeted deli

29 Biological half-life was longest when subjects with high

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