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1 une-modulatory agents, cytostatic drugs, and biological response modifiers.
2 Interferons (IFNs) and retinoids are potent biological response modifiers.
3 ased therapies, including growth factors and biological response modifiers.
4 Interferons (IFNs) and retinoids are potent biological response modifiers.
5 uct with either leukocyte allo-antibodies or biological response modifiers.
6 eening of anticancer therapeutics, including biological response modifiers.
7 the xenotransplantation subcommittee of the Biological Response Modifiers Advisory Committee to disc
8 onundrum, and recent prospective trials with biological response modifiers aimed at the inflammatory
9 scular beds, as well as through secretion of biological response modifiers and recruitment of immune
10 rotein member of the TGF-beta superfamily of biological-response modifiers, causes regression of the
13 increase in immunogenicity provided by this biological response modifier in an otherwise well-tolera
16 Issues concerning the safety of the use of biological response modifiers in inducing sarcoidosis ne
17 syl-(1-3)-beta-glucopyranose (PGG) glucan, a biological-response modifier, in protecting against mort
22 ulation, indicating that Thy-1 is a critical biological response modifier that protects against fibro
25 RA) represent a growing class of pleiotropic biological response modifiers with demonstrable efficacy
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