ライフサイエンスコーパス: biotype

1 in biotype; group 2 = 50 patients with thick biotype).

2 ) and those in which it was not (i.e., thick biotype).

3 hat only functions in the V. cholerae El Tor biotype.

4 specific activity compared to the classical biotype.

5 s nearly silent in V. cholerae of the El Tor biotype.

6 but not within those of a whitefly nonvector biotype.

7 ing certain characteristics of the classical biotype.

8 onization by V. cholerae O1 of the classical biotype.

9 seem to be influenced by the adjacent teeth biotype.

10 uces CT expression in the V. cholerae El Tor biotype.

11 by Vibrio cholerae O1 strains of the El Tor biotype.

12 at a fivefold higher level in the classical biotype.

13 s higher in the classical than in the El Tor biotype.

14 peptides, and iron was higher in the El Tor biotype.

15 e virulence gene expression in the classical biotype.

16 e genes were regulated by VieA in the El Tor biotype.

17 Tor that is not functional in the classical biotype.

18 type are controlled by VieA in the classical biotype.

19 k peri-implant biotype as compared to a thin biotype.

20 5-fold higher IC(50)(glyphosate) for the (R) biotype.

21 by Vibrio cholerae serogroup, serotype, and biotype.

22 ins expressing the mature EPSPS from the (S) biotype.

23 mechanism of glyphosate resistance in parent biotype.

24 olerae O1 serogroup strains of the classical biotype.

25 ility in Vibrio cholerae O1 of the classical biotype.

26 biotype in 9 sites which had an initial thin biotype.

27 inized tissue, and thickness of the gingival biotype.

28 tinized tissue and thickness of the gingival biotype.

29 zed gingiva (WKG) and assessment of gingival biotype.

30 otype in nine sites that had an initial thin biotype.

31 atinized gingiva, and assessment of gingival biotype.

32 on in individuals with a thick-flat gingival biotype.

33 see-accessed, glyphosate-resistant horseweed biotype.

34 ing phages for the Vibrio cholerae O1 El Tor biotype.

35 s NTHI strains using PCR capsule typing, and biotyped.

36 CYDV-RPV was determined in all genotypes and biotypes.

37 lly independent, field-collected S. graminum biotypes.

38 expressed at similar levels in both cholera biotypes.

39 nt increase in CT and TCP expression in both biotypes.

40 clustering based on IS1016, hmw or hia, and biotypes.

41 ype A) and sexually transmitted (serotype D) biotypes.

42 inically to distinguish classical and El Tor biotypes.

43 ic, low-pathogenicity, and highly pathogenic biotypes.

44 nd to be differentially expressed in the two biotypes.

45 in carbohydrate metabolism between these two biotypes.

46 ty (GMD) as an independent validator for the Biotypes.

47 erated just a small consensus of significant biotypes.

48 phosate-resistant and glyphosate-susceptible biotypes.

49 ipolar psychosis); this was not the case for biotypes.

50 rties of Rosalia, Montero and Leiva 1 lucuma biotypes.

51 he divergence between host plant specialized biotypes.

52 ed with glyphosate-resistant or -susceptible biotypes.

53 FGE), multilocus enzyme electrophoresis, and biotyping.

54 omain structure distinct from that of either biotype 1 or biotype 2.

55 e that encodes MARTX(Vv) in 40 V. vulnificus Biotype 1 strains and found four distinct variants of rt

56 Biotype 1 strains of V. vulnificus are most commonly ass

57 We examined 69 V. vulnificus biotype 1 strains that were genotyped by several methods

58 spect to pathogenicity, i.e., nonpathogenic (biotype 1A), low pathogenicity (biotypes 2 to 5), and hi

59 However, seven other biotype 1B strains sequenced did possess the domain.

60 ty (biotypes 2 to 5), and highly pathogenic (biotype 1B).

61 re distinct from that of either biotype 1 or biotype 2.

62 npathogenic (biotype 1A), low pathogenicity (biotypes 2 to 5), and highly pathogenic (biotype 1B).

63 d analysis of the MARTX toxin and found that biotype 3 MARTX toxin has an effector domain structure d

64 This is the first demonstration that the biotype 3 MARTX toxin is essential for virulence and tha

65 nctional autoprocessing RTX (MARTX) toxin of biotype 3 strains was shown to be an essential virulence

66 cognized variant of V. vulnificus designated biotype 3.

67 y regulates gene expression in the classical biotype; 401 genes (10.3% of the genome), including thos

68 ter rodentium (formally Citrobacter freundii biotype 4280) is a highly infectious pathogen that cause

69 Clostridium perfringens biotype A strains are the causative agents of gas-gangre

70 ed with vinifera-based rootstocks, known as "biotype A".

71 into cytopathic (cp) and noncytopathic (ncp) biotypes according to their effect on cultured cells.

72 pathogenic strains, including H. influenzae biotype aegyptius and Brazilian purpuric fever isolates.

73 se sequence comparisons suggest that a novel biotype aegyptius AT arose by swapping an unrelated sequ

74 We carried out mutant screens of both biotypes, aiming to identify classical V. cholerae mutan

75 cataloguing the profiles based on the known biotypes, all the employed RNA-Seq methods generated jus

76 from assembling the unmapped reads pooled by biotype allowed us to recover some divergent genomic reg

77 als and evaluate potential correlations with biotype, along with other clinical parameters.

78 ontrast brain anatomy characteristics across Biotypes alongside conventional diagnoses, examining gra

79 The gingival biotype also showed a thick biotype in 9 sites which had

80 The gingival biotype also showed a thick biotype in nine sites that h

81 Both classical and El Tor biotypes alternated and persisted between 1966 and 1988;

82 tient and site characteristics, e.g., tissue biotype and buccal plate thickness.

83 There was an excellent correlation of biotype and genotype: S. bovis biotype II/2 isolates for

84 and in PBMCs by V. cholerae varies with the biotype and is mediated by both NLRP3-dependent and -ind

85 bolites and functional properties related to biotype and ripeness stage.

86 e factors that drove emergence of the El Tor biotype and the displacement of the classical biotype ar

87 relatives, and healthy subjects organized by Biotype and then by DSM-IV-TR diagnosis (n = 1409) using

88 IIP in thick biotype and with immediate provisionalization had less M

89 s in which the probe was visible (i.e., thin biotype) and those in which it was not (i.e., thick biot

90 are colonized on average with five commensal biotypes, and it is widely thought that the commensals s

91 expressed in the classical versus the El Tor biotype are controlled by VieA in the classical biotype.

92 s, we conclude that the strains of classical biotype are likely defective compared to those of El Tor

93 iotype and the displacement of the classical biotype are unknown.

94 iation in pathogenesis and whether different biotypes are associated with specific nonhuman hosts are

95 oted in the presence of a thick peri-implant biotype as compared to a thin biotype.

96 uccal plate thickness between thick and thin biotypes as determined by probe visibility.

97 Most genogroups had consistent biotypes (as determined with the RapID ANA II system); h

98 ype were significantly greater than the thin biotype at MT, MI, and DT (P<0.05).

99 iated with only AXR#1 rootstock, defined as "biotype B", and another group associated with vinifera-b

100 roteins differentially expressed between the biotypes before and after glyphosate treatment.

101 or biotype compared to that of the classical biotype both as a global cause of cholera and as an envi

102 sequently displaced strains of the classical biotype both in the environment and as a cause of choler

103 old standard to discriminate thick from thin biotype but is prone to subjective interpretation.

104 ior foregut or cibarium of a whitefly vector biotype but not within those of a whitefly nonvector bio

105 olera pandemics were caused by the classical biotype, but El Tor has subsequently spread globally and

106 slightly less in IIP with thick versus thin biotypes, but not statistically significantly different

107 We determined the VieA regulon in both biotypes by transcriptome comparison of wild-type and vi

108 Biotyping by MALDI-TOF-MS will prove effective in situat

109 orption ionization (MALDI) mass spectrometry biotyping can be used for measurement of cellular potenc

110 These biotypes cannot be differentiated solely on the basis of

111 However, the evolution of new soybean aphid biotypes capable of defeating host-plant resistance conf

112 Although strains of the El Tor biotype caused sporadic infections and cholera epidemics

113 th whole live M. mycoides subsp. mycoides SC biotype cells, indicating cellular proliferation.

114 A and motility to the pathogenesis of El Tor biotype cholera.

115 s, whereas a previous pandemic strain of the biotype Classical is polymyxin-sensitive.

116 In Vibrio cholerae, two natural epidemic biotypes, classical and El Tor, exhibit distinct phenoty

117 rae strains of the O1 serogroup exist as two biotypes, classical and El Tor.

118 arrheal disease cholera, is divided into two biotypes: classical and El Tor.

119 tely 2- to 4-fold greater than the sensitive biotype collected from the same region.

120 olutionary fitness of the V. cholerae El Tor biotype compared to that of the classical biotype both a

121 nique brain structure characteristics within Biotypes, consistent with their cognitive and sensorimot

122 rine substitution at position 106 in the (R) biotype, corresponds to a substitution previously identi

123 We hypothesized that commensal biotypes could exert colonization resistance by consumin

124 lthough some differences were detected among biotypes, data indicate that biotype does not play a fun

125 ided into four neurophysiological subtypes ('biotypes') defined by distinct patterns of dysfunctional

126 Analysis of a defined mutation in the El Tor biotype demonstrated that msbB is required for resistanc

127 One gene from the resistant biotype, designated PPX2L, contained a codon deletion th

128 Bone width and soft-tissue biotype did not influence the incidence of gingival papi

129 egion near the N terminus (L8P) in both BVDV biotypes did not antagonize IFN-alpha/beta production, c

130 rsisted between 1966 and 1988; the classical biotype disappeared by 1988, and the O139 serogroup firs

131 ize the variants, including an assessment of biotype-distinguishing characteristics.

132 can be concluded that bone width and tissue biotype do not have an effect on the incidence and heigh

133 detected among biotypes, data indicate that biotype does not play a fundamental role in influencing

134 Current pandemic O1 Vibrio cholerae biotype El Tor is resistant to polymyxins, whereas a pre

135 rt, cases of V. cholerae O1 (serotype Ogawa, biotype El Tor) were confirmed in all 10 administrative

136 rly as 1910, it was not until 1961 that this biotype emerged to cause the 7th pandemic, eventually re

137 Toxigenic strains of the El Tor biotype emerged to cause the seventh pandemic of cholera

138 This capacity was found in all biotypes examined and could extend into sepals and corol

139 ressing the mature EPSPS enzyme from the (R) biotype exhibited an approximately 3-fold increase in gl

140 avian-adapted S. enterica serovar Gallinarum biotypes Gallinarum and Pullorum, and the most frequent,

141 ted, along with its possible relationship to biotype, genotype, and resistance to metronidazole and b

142 quenced the genome of a UK clinical isolate (biotype gravis strain NCTC13129), representative of the

143 ed the strain as nontoxigenic C. diphtheriae biotype Gravis.

144 ional study (group 1 = 50 patients with thin biotype; group 2 = 50 patients with thick biotype).

145 In V. cholerae strains of the classical biotype, H-NS has been reported to silence virulence gen

146 Tissue biotypes have been linked to the outcomes of periodontal

147 Periodontal biotype, horizontal and vertical peri-implant bone defec

148 y on plants previously attacked by the other biotype, however, on their respective noninfested contro

149 ins were more often biotype V (P < 0.001) or biotype I (P = 0.04) than IS1016-negative NTHI strains,

150 Two of the S. bovis biotype I cases were associated with colon cancer.

151 The biotype I strain is much more frequently isolated from p

152 nt of a PCR test which can identify S. bovis biotype I strains among S. bovis clinical isolates.

153 We describe here the isolation of biotype I-specific DNA sequences and the development of

154 ype II/1, and that of cluster 2a as S. bovis biotype I.

155 enterococci were shown to represent S. bovis biotypes I (11 isolates) and II/2 (1 isolate), S. saliva

156 s variant (sometimes referred to as S. bovis biotypes I and II, respectively) are phenotypically simi

157 The majority of IS1016-positive NTHi were biotypes I and V and showed some genetic relatedness by

158 S. bovis is divided into two biotypes: I and II.

159 Whether S. bovis biotype II or other organisms closely related to and his

160 negative NTHI strains, which were most often biotype II.

161 nus, previously known as Streptococcus bovis biotype II.2, is an uncommon pathogen in neonates.

162 nus, previously known as Streptococcus bovis biotype II.2, is known to cause multiple infectious comp

163 biotype II/2, those of cluster 1 as S. bovis biotype II/1, and that of cluster 2a as S. bovis biotype

164 orrelation of biotype and genotype: S. bovis biotype II/2 isolates form a separate genospecies distin

165 ingitis caused by S. bovis variant (S. bovis biotype II/2) and review the literature.

166 enotypically reported as Streptococcus bovis biotype II/2, 16S rRNA sequencing revealed S. gallolytic

167 dentified isolates of cluster 2b as S. bovis biotype II/2, those of cluster 1 as S. bovis biotype II/

168 was identified as nontypeable H. influenzae, biotype III.

169 ing analysis of weedy rice (Oryza sativa L.) biotypes illuminates distinct evolutionary paths and out

170 The gingival biotype also showed a thick biotype in 9 sites which had an initial thin biotype.

171 the enhanced survival capacity of the El Tor biotype in environmental reservoirs.

172 The gingival biotype also showed a thick biotype in nine sites that had an initial thin biotype.

173 y spread globally and replaced the classical biotype in the current pandemic.

174 ssical (O395) and El Tor (C6706) V. cholerae biotypes in growth and biochemical assays.

175 The commensal biotypes included E. coli HS, which is known to successf

176 ntial against M. mycoides subsp. mycoides SC biotype-induced mycoplasmemia.

177 y of recession during tooth movement in thin biotype is high to justify gingival augmentation when th

178 The classical biotype is susceptible to CAMPs, but current pandemic El

179 sate-resistant horseweed (Conyza canadensis) biotypes is an example of how unrelenting use of a singl

180 ptible to CAMPs, but current pandemic El Tor biotype isolates gain CAMP resistance by altering the ne

181 microscopy showed no surface pili on the two biotype IV H. influenzae isolates examined; strain 4162

182 Thus, although biotype IV H. influenzae isolates of the cryptic genospe

183 of nontypeable Haemophilus influenzae (NTHI) biotype IV isolates from the human genital tract or from

184 None of the six biotype IV NTHI isolates tested agglutinated human red b

185 In enzyme-linked immunosorbent assays, biotype IV strains adhered to 16HBE14o(-) and HEp-2 cell

186 Haemophilus biotype IV strains belonging to the recently recognized

187 s was assessed, the binding of H. influenzae biotype IV strains to human epithelial cells was charact

188 HifE-possessing pili in the adhesion of NTHI biotype IV strains to human epithelial cells was determi

189 receptor analog, did not inhibit binding of biotype IV strains to ME180, HEp-2, or HeLa cells, and G

190 ifA and hifE and their gene products in NTHI biotype IV strains was assessed, the binding of H. influ

191 isolates in serogroup O1 (consisting of two biotypes known as 'classical' and 'El Tor') and the deri

192 We sequenced two isolates from the remaining biotype, LGV, a long-term laboratory passaged strain and

193 Thirty healthy patients with thin gingival biotype (<1 mm) and history of periodontal disease recei

194 and better PH maintenance in IIP with thick biotype (MFR: MD -0.478, P <0.001; cumulative PH: MD -0.

195 Recently, a glyphosate-resistant biotype of goosegrass (Eleusine indica) was identified i

196 mediators of resistance against an avirulent biotype of Hyaloperonospora parasitica.

197 ese effects are dynamic and dependent on the biotype of TcpA and the haplotypes of the host.

198 characterized genome, and RNAi in the sexual biotype of the planarian Schmidtea mediterranea to test

199 root-knot nematodes (Meloidogyne spp.) and a biotype of the potato aphid (Macrosiphum euphorbiae).

200 Tissue biotypes of 22 fresh cadaver heads were assessed clinica

201 ip performed best, correctly classifying the biotypes of 371 of 380 (97.6%) different challenge strai

202 sequences of this bacterium from the B and Q biotypes of B. tabaci.

203 Various biotypes of endogenous small RNAs (sRNAs) have been dete

204 The biotypes of Haemophilus influenzae and Haemophilus parai

205 The El Tor and classical biotypes of O1 V. cholerae show striking differences in

206 iations between disease states and different biotypes of S. bovis are apparent.

207 i of both the classical (Cl) and El Tor (ET) biotypes of TCP.

208 effects of the Ir gene associated with both biotypes of TcpA.

209 The two biotypes of V. cholerae O1 capable of causing cholera, c

210 ulence genes between the two disease-causing biotypes of V. cholerae, classical and El Tor.

211 Historically, the O1 El Tor and classical biotypes of Vibrio cholerae have been differentiated by

212 on patterns between the classical and El Tor biotypes of Vibrio cholerae O1 were determined under con

213 lence genes be-tween the two disease-causing biotypes of Vibrio cholerae, classical and El Tor, is pr

214 is being posed by the inexorable increase in biotypes of weeds that are resistant to herbicides.

215 pression between epidemic strains (i.e., the biotypes) of V. cholerae.

216 potential long-term influence of periodontal biotype on the volume maintenance of block grafts.

217 rference confers immunity to all Hessian fly biotypes on normally susceptible wheat genotypes.

218 had less MFR and better PH than IIP in thin biotype or with delayed restoration.

219 ce of neonatal infection caused by different biotypes or newer species of S. bovis.

220 s statistically associated with G. vaginalis biotype (P=.048) but not with genotype or resistance to

221 eSAB promoter by H-NS and HapR in the El Tor biotype prior to the cessation of exponential growth res

222 Both the classical and El Tor biotypes produce inactive ToxT protein when they are cul

223 Both biotypes produce the major virulence factors toxin-coreg

224 same species, and same susceptible-resistant biotype profile during any 7-day period) and uncontrolle

225 same species, and same susceptible-resistant biotype profile during any 7-day period) and uncontrolle

226 TG)-inducible M. mycoides subsp. mycoides SC biotype protein with a 28-kDa apparent molecular mass, i

227 enes from the M. mycoides subsp. mycoides SC biotype pyruvate dehydrogenase region, and two IPTG-inde

228 be significantly correlated with periodontal biotype (r = 0.325, P = 0.000) and post-suturing flap po

229 .2%) with thick-scalloped and thin-scalloped biotypes, respectively, had APE.

230 nificant anti-M. mycoides subsp. mycoides SC biotype responses were observed in mice vaccinated with

231 clinical isolate belonging to the O1 El Tor biotype responsible for the current cholera pandemic.

232 with same sensitivity-resistance profile and biotype results during any 7-day period) and uncontrolle

233 of a PPO-inhibitor-resistant A. tuberculatus biotype revealed that resistance was a (incompletely) do

234 red from the resistant (R) and sensitive (S) biotypes revealed an approximately 5-fold higher IC(50)(

235 PSPS mature protein coding regions from both biotypes revealed four single-nucleotide differences, tw

236 plasma mycoides subsp. mycoides small colony biotype (SC) is the high-consequence animal pathogen cau

237 and M. mycoides subsp. mycoides small colony biotype (SC), (ii) antibodies to specific peptide determ

238 All isolates were biotyped, serotyped by slide agglutination serotyping (S

239 istance to polymyxin B. msbB mutants of both biotypes showed decreased colonization of infant mice, w

240 Biotypes showed stronger between-group separation based

241 coli is a single species consisting of many biotypes, some of which are commensal colonizers of mamm

242 NA gene sequence analysis and recorded their biotypes, sources, and disease associations.

243 ion of HA/protease and motility in an El Tor biotype strain by constructing a Deltahns mutant.

244 This O1 biotype strain has 95 to 97% similarity with the classic

245 However, growth of El Tor biotype strain N16961 was enhanced due to its ability to

246 ain N16961 and the sixth-pandemic (classical biotype) strain 395 and found that the N16961 VPI is 41,

247 ared the VPI of the seventh-pandemic (El Tor biotype) strain N16961 and the sixth-pandemic (classical

248 lting in the global elimination of classical biotype strains as a cause of disease.

249 genotypic and phenotypic differences, El Tor biotype strains displaying classical biotype traits have

250 In contrast, El Tor biotype strains expressed negligible LeuO under identica

251 en grown with added carbohydrates, classical biotype strains generated a sharp decrease in medium pH,

252 El Tor biotype strains induced release of IL-1beta dependent on

253 e ToxRS-dependent expression of CT in El Tor biotype strains is related to the differences between cl

254 ion was observed in the classical and El Tor biotype strains of serogroup O1 that is most frequently

255 In El Tor biotype strains of toxigenic Vibrio cholerae, the CTXvar

256 wed the same defect in growth that classical biotype strains show in media rich in carbohydrates.

257 Classical biotype strains, which do not produce either hemolysin o

258 ctxAB only on the large chromosome of El Tor biotype strains.

259 of CT, including levels typical of classical biotype strains.

260 regulator HapR, which is absent in classical biotype strains.

261 2) keratinized tissue width (KT); 3) tissue biotype (TB); and 4) plaque level.

262 onger discrimination for biologically driven biotypes than symptom-based diagnoses.

263 ts on adjacent teeth and in a thicker-tissue biotype that would be amenable to partial-thickness diss

264 d three neurobiologically distinct psychosis biotypes that did not respect clinical diagnosis boundar

265 S29 isoform was present in all genotypes or biotypes that efficiently transmit CYDV-RPV and more spe

266 Building on experimental constructs-Biotypes-that were previously developed from cognitive a

267 Even when adjacent teeth had a thin biotype, the transplanted sites maintained statistically

268 nses of the El Tor and classical V. cholerae biotypes to increased c-di-GMP concentrations were deter

269 e of Flight Mass Spectrometry (MALDI-TOF-MS) biotyping to deliver rapid and accurate strain separatio

270 om phenotypic analysis, such as serology and biotypes, to much-more-robust molecular genetic approach

271 MALDI should be accepted as a biotyping tool to complement and enhance standard molecu

272 holera toxin, lipopolysaccharide, and El Tor biotype traits can be confirmed.

273 El Tor biotype strains displaying classical biotype traits have been reported and subsequently were

274 a major regulator of genes in the classical biotype under virulence gene-inducing conditions.

275 red several properties with R2866: they were biotype V (indole and ornithine decarboxylase positive,

276 IS1016-positive NTHI strains were more often biotype V (P < 0.001) or biotype I (P = 0.04) than IS101

277 Multilocus sequence typing showed that most biotype V isolates belonged to the same phylogenetic clu

278 Restoring HapR expression in classical biotype V. cholerae repressed vieSAB transcription by bi

279 hapA) single and double mutants of an El Tor biotype V. cholerae strain.

280 in a ToxT-independent manner in a classical biotype V. cholerae, and that this activation requires t

281 We show that in classical biotype V. cholerae, LeuO cooperates with the nucleoid-a

282 A significant association of IS1016 with biotypes V and I and the presence of hia adhesins was fo

283 The V. cholerae classical biotype (V. cholerae(Cl)), which caused previous cholera

284 Probands grouped by Biotype versus healthy controls showed a stepwise patter

285 have constructed a relaxed mutant of El Tor biotype Vibrio cholerae strain C7258 by disruption of th

286 In El Tor biotype vibrios, transcription of vieSAB is repressed by

287 The influence of the peri-implant biotype was also examined.

288 a mycoides subsp. mycoides small colony (SC) biotype was cloned into lambda ZAP Express, and two stro

289 One biotype was common to several genogroups, with all of th

290 Biotype was determined by probe visibility through the t

291 In addition, the peri-implant biotype was evaluated and categorized as thick or thin.

292 e resequencing of seven additional horseweed biotypes was performed.

293 wed that lipid A of the msbB mutant for both biotypes was underacylated compared to lipid A of the wi

294 lants (horizontal distance), and soft-tissue biotype were assessed in 29 interimplant areas in the up

295 /=3 mm from the osseous crest and thick-flat biotype were associated with greater tissue rebound.

296 hermore, 34 (75.6%) patients with thick-flat biotype were diagnosed with APE, whereas 30 (31.3%) and

297 mensions of peri-implant mucosa in the thick biotype were significantly greater than the thin biotype

298 significant differences were detected among biotypes, whereas: 1) TT, 2) age, and 3) smoking habit w

299 of APE, clinical crown length, and gingival biotype, which was divided into three categories: thin-s

300 , and 4 = overfill), marginal tissue levels, biotype, width of keratinized tissue, and soft tissue th

301 Intraoral examination revealed a thick biotype with an adequate band of keratinized gingiva, Mi

302 interacted with the vieSAB promoter of both biotypes with similar affinities and protected overlappi