ライフサイエンスコーパス: bipolar affective disorder

1 Lithium is a first-line therapy for bipolar affective disorder.

2 (95% CI, 1.6-4.5) times more likely to have bipolar affective disorder.

3 (95% CI, 2.7-20.6) times more likely to have bipolar affective disorder.

4 ctive disorder (N=26 380), of which 1928 had bipolar affective disorder.

5 ived neurotrophic factor in the aetiology of bipolar affective disorder.

6 s being overrepresented in the patients with bipolar affective disorder.

7 may help define more homogeneous subtypes of bipolar affective disorder.

8 everity and course, is a familial feature of bipolar affective disorder.

9 some neuropsychiatric conditions, including bipolar affective disorder.

10 ociated gene expression in schizophrenia and bipolar affective disorder.

11 re effective mood-stabilizing treatments for bipolar affective disorder.

12 es with exclusively maternal transmission of bipolar affective disorder.

13 ts and lymphocytes of euthymic patients with bipolar affective disorder.

14 ion of lithium and in the pathophysiology of bipolar affective disorder.

15 ng are associated with functional decline in bipolar affective disorder.

16 cludes schizophrenia as well as unipolar and bipolar affective disorders.

17 is relation is present for both unipolar and bipolar affective disorders.

18 rated for men and women and for unipolar and bipolar affective disorders.

19 of foreign residence had increased IRRs for bipolar affective disorder, affective disorders, persona

20 typing was carried out in 1099 patients with bipolar affective disorder and 1152 healthy comparator i

21 and lymphocytes of 44 euthymic patients with bipolar affective disorder and 27 matched comparison sub

22 f the standard antimanic treatments used for bipolar affective disorder and 38% of the treatments use

23 nd D21S171, a region which includes loci for bipolar affective disorder and a recessive form of deafn

24 set including 67 males and 113 females with bipolar affective disorder and a similar number of match

25 ty at illness onset is a familial feature of bipolar affective disorder and is associated with import

26 e results suggest a molecular basis for both bipolar affective disorder and its treatment.

27 A possible link between bipolar affective disorder and schizophrenia remains arg

28 nosis, implicating dopamine dysregulation in bipolar affective disorder and schizophrenia, in line wi

29 In the study of bipolar affective disorder and schizophrenia, there is s

30 familial spastic paraplegia, schizophrenia, bipolar affective disorder and spinocerebellar ataxia ty

31 gene for schizophrenia, was associated with bipolar affective disorder and tested this hypothesis us

32 previous reports of high Gs alpha levels in bipolar affective disorder and, furthermore, suggest tha

33 llection, consisting of 15 schizophrenia, 15 bipolar affective disorder, and 15 control brains.

34 in three or more generations, an absence of bipolar affective disorder, and a single progenitor sour

35 chiatric disorders, including schizophrenia, bipolar affective disorder, and borderline personality d

36 are likely to contain genes contributing to bipolar affective disorder are also relevant to schizoph

37 Molecular genetic studies of bipolar affective disorder are beginning to show some po

38 Schizophrenia and bipolar affective disorder are chronic, disabling illnes

39 thesis that lithium's therapeutic effects in bipolar affective disorder are mediated by alterations i

40 An increased rate has also been reported in bipolar affective disorder (BD).

41 usly, we demonstrated evidence of linkage to bipolar affective disorder (BP) in a single large, multi

42 Bipolar affective disorder (BP) is a common, highly heri

43 Bipolar affective disorder (BP) is a major neuropsychiat

44 ), have been implicated in susceptibility to bipolar affective disorder (BP) through genome-wide asso

45 in 110 parent-offspring trios affected with bipolar affective disorder (BP).

46 clarify the issue of genetic linkage between bipolar affective disorder (BPAD) and chromosome 18q, co

47 Bipolar affective disorder (BPAD) and schizophrenia (SZ)

48 Bipolar affective disorder (BPAD) is a complex disease w

49 Decades of longitudinal research on bipolar affective disorder (BPAD) revealed cosegregation

50 have reported evidence suggesting linkage of bipolar affective disorder (BPAD) to chromosome 18.

51 Bipolar affective disorder (BPAD; manic-depressive illne

52 Twin studies of bipolar affective disorder (BPD) have either been small

53 pletion may be the way that lithium works in bipolar affective disorder, but others have suggested th

54 and RFLP at the monoamine oxidase A locus in bipolar affective disorder cases and controls in the UK

55 hospitalization with nonaffective psychosis, bipolar affective disorder, depressive disorder, eating

56 Patients with bipolar affective disorder differed from those with unip

57 There is also linkage evidence for bipolar affective disorder, epilepsy and autism in this

58 s, a significant proportion of patients with bipolar affective disorder experience frequent relapses.

59 rate of lithium monotherapy for treatment of bipolar affective disorder (from 84% to 43%) and schizoa

60 the actual treatment of schizoaffective and bipolar affective disorders had changed in light of rece

61 , and SAP102 in subjects with schizophrenia, bipolar affective disorder I, and a comparison group.

62 olecules in hippocampus in schizophrenia and bipolar affective disorder I.

63 earby locus contributes to susceptibility to bipolar affective disorder in some families.

64 bipolar affective disorder, suggesting that bipolar affective disorder in the Old Order Amish is inh

65 The main clinical feature of bipolar affective disorder is a change of mood to depres

66 Bipolar affective disorder is a common neuropsychiatric

67 cifically designed for relapse prevention in bipolar affective disorder is a useful tool in conjuncti

68 Bipolar affective disorder is clinically heterogeneous,

69 Their involvement in bipolar affective disorder is much less prominent.

70 Rapid cycling among patients with bipolar affective disorders is important because of its

71 peutic efficacy in manic-depressive illness (bipolar affective disorder) is the inositol depletion hy

72 chizophrenia, schizoaffective disorders, and bipolar affective disorders) is well described, but litt

73 The most characteristic features of bipolar affective disorder (manic-depressive illness) ar

74 Bipolar affective disorder (manic-depressive illness) is

75 Molecular genetic research in bipolar affective disorder may lead to the development o

76 polygenic diseases, including schizophrenia, bipolar affective disorder, non-insulin-dependent diabet

77 the apparent excess maternal transmission of bipolar affective disorder observed in some families.

78 The results demonstrate that bipolar affective disorder occurs across all of the majo

79 Reported linkages of bipolar affective disorder on chromosomes 11, 18, 21 and

80 unilineal multiplex families segregating the bipolar affective disorder phenotype.

81 f dementia, hereditary ataxia, Parkinsonism, bipolar affective disorder, schizophrenia and autism.

82 pecifically designed to prevent relapses for bipolar affective disorder showed encouraging results wh

83 onsidered a good candidate to investigate in bipolar affective disorder since this enzyme plays an im

84 rs and impulsivity), DRD3 (schizophrenia and bipolar affective disorder), SLC6A3 (susceptibility to c

85 6, 13 and 15 harbour susceptibility loci for bipolar affective disorder, suggesting that bipolar affe

86 confirm or refute previous reports that link bipolar affective disorder to polymorphic DNA markers at

87 s (average age = 34 +/- 16.5) diagnosed with Bipolar Affective Disorder to three patient groups all d

88 cation of candidate genes that predispose to bipolar affective disorder, to the completion of the seq

89 idual with an atypical movement disorder and bipolar affective disorder type II contains 46 triplets,

90 Those with bipolar affective disorder were more likely to receive s

91 ene may play a role in the susceptibility to bipolar affective disorder, which underscores a potentia