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1 mer (division occurs after a fixed time from birth).
2 al genetic diseases that can be diagnosed at birth.
3 cell progenitors, but is downregulated after birth.
4 systemic GBS infection and rates of preterm birth.
5 ch crippling muscle weakness is evident from birth.
6 ar metabolism and transcriptomic profiles at birth.
7 mice than in wild-type mice at 21 days after birth.
8 ive outcomes such as infertility and preterm birth.
9 SThma study were followed prospectively from birth.
10 ium following a dynamic priming period after birth.
11 childhood asthma is already demonstrable at birth.
12 sed by quarterly questionnaires beginning at birth.
13 rth, and at 3, 6, 9, 12, and 18 months after birth.
14 cardiovascular disease among those small at birth.
15 itions are suspected to be causes of preterm birth.
16 reduced brain preparations and breathing at birth.
17 f economic conditions on selection into live birth.
18 he successful transition to air-breathing at birth.
19 velopment through dietary fatty acids before birth.
20 ued to mature until approximately 3 wk after birth.
21 heart disease (CHD) affects up to 1% of live births.
22 ity in childhood and occurs in 1 in 500 live births.
23 d RASopathies, affecting about 1/1,000 human births.
25 sociated with reductions in rates of preterm birth (-3.77% [95% CI -6.37 to -1.16]; ten studies, 27 5
28 BS disease in infants was 0.49 per 1000 live births (95% confidence interval [CI], .43-.56), and was
29 e To compare the probability of a first live birth, age at time of birth, and time between diagnosis/
30 ly with 10 people without MS by sex, year of birth, age/vital status at MS diagnosis, and region of r
32 than 95% probability that life expectancy at birth among men in South Korea, Australia, and Switzerla
36 between ASD and residential location both at birth and at age 6 years (i.e., closer to average age at
37 elivered at term, women with a preterm first birth and at least 1 later preterm birth had a HR of CVD
38 xposures included cat and dog ownership from birth and cat and dog allergen levels in bedding at age
40 m colonization and shows how factors such as birth and feeding modes could influence this acquisition
43 each of LHWs in accessing newborn infants at birth and in the early postnatal period underscores the
46 role of environmental factors in regulating birth and mortality rates can lead to erroneous demograp
47 the afferent imbalance, which is present at birth and nonprogressive, indicating that sensory neuron
50 hip between variations in DNA methylation at birth and the development of allergic disease, we examin
52 enting the food of dams (for 3 weeks between birth and weaning) with ezetimibe, which is secreted int
54 oman in pregnancy, within 72 h of her baby's birth, and at 3, 6, 9, 12, and 18 months after birth.
55 from age 15 to 60 years, life expectancy at birth, and cause-specific and age-specific mortality by
56 enetic variation that can be detected before birth, and for discovering more about maternofetal and p
57 ose the ability to proliferate shortly after birth, and further increase in cardiac mass is achieved
59 bility of a first live birth, age at time of birth, and time between diagnosis/referent date and birt
60 gestations, no previous spontaneous preterm births, and cervical lengths of 25 mm or less at 18 week
61 tnatal mastery of suckling at 4 months after birth; and (2) thereafter, from preparation to the maste
63 variants associated with later age at first birth are associated with a longer maternal life span (P
66 during pregnancy was associated with preterm birth (ARR, 1.16) but not with small for gestational age
69 sk based on lifetime breast cancer risk from birth, as estimated by BOADICEA (Breast and Ovarian Anal
70 ; and five of neonatal morbidity, comprising birth asphyxia (n=3), septicaemia (n=1), and neonatal co
71 tial colonisation of i) intrauterine/vaginal birth associated taxa, ii) skin derived taxa and other t
74 on-based cohort study of 1441623 live single births at 22 or more completed gestational weeks in Swed
75 4,556 infants that were at least 2,000 g at birth, at least 35 weeks of gestation, and with no signs
78 cular impairment in baboons who were IUGR at birth because of moderate maternal nutrient reduction.
79 ere was a strong association between preterm birth before 32 weeks of gestation and HF in childhood a
80 eterm birth before 37 weeks and with preterm birth before 34 weeks were characterized by an initial r
81 associations of arginine intake with preterm birth before 37 weeks and with preterm birth before 34 w
82 verage from the Alabama Medicaid Agency from birth between 2008 and 2012, adjusting for demographics,
83 and time between diagnosis/referent date and birth between childhood and adolescent cancer survivors
84 ic IgE measured before conception than after birth [BHR: aRRR = 2.96 (95% CI: 1.92, 4.57) and 1.40 (1
86 pregnancies and no prior spontaneous preterm birth but with short cervical length on transvaginal ult
87 ZIKV-exposed infants who are asymptomatic at birth but, we assume, may develop problems later in life
88 role in brain injury associated with preterm birth, but little is known about the microglial response
89 H levels, we disrupted its synthesis both at birth by breeding a Gclc loxP mouse with a thy1-cre mous
94 or 940 children from the prospective Swedish birth cohort BAMSE (Children, Allergy, Milieu, Stockholm
95 nts and 2,922 controls from the 1958 British birth cohort identified an additional 14 regions associa
96 d transient CMV infections, in a prospective birth cohort of 30 highly exposed CMV-uninfected infants
103 6 weeks and 6 months postdelivery in the Ulm Birth Cohort Study (n=720 and n=454, respectively).
105 (BCG) vaccination and childhood asthma in a birth cohort using administrative databases, and we dete
106 population-representative Hong Kong Chinese birth cohort, "Children of 1997," to examine the adjuste
108 s and 1970s birth cohorts and the 2000--2002 birth cohort, despite a higher proportion of the low-bir
109 At age 22 years, 262 members of a Faroese birth cohort, established in 1986-1987, underwent a grad
115 ity has declined between the 1950s and 1970s birth cohorts and the 2000--2002 birth cohort, despite a
117 ivery and asthma in children from 9 European birth cohorts that enrolled participants between 1996 an
118 Findings from two population-representative birth cohorts totaling more than 3,000 individuals and b
121 yses, adjusting for maternal age, ethnicity, birth country and weight, as well as infant gender, birt
126 Congenital heart disease is the most common birth defect, and because of major advances in medical a
127 periconceptional period), with no reports of birth defects among fetuses or infants with prenatal exp
130 ort study using data from the North Carolina Birth Defects Monitoring Program, North Carolina Departm
131 nd hypospadias, using data from the National Birth Defects Prevention Study, a population-based case-
135 palate-one of the most common of congenital birth defects-treatment following surgery is prolonged o
141 ilar for race, age at menarche, age at first birth, family history, alcohol consumption, and smoking
142 h nationwide registers on all women who gave birth for the first time between January 1, 1993, and De
143 mber 7, 2012, MLPT infants were recruited at birth from the neonatal unit and postnatal wards of the
144 from the Swedish Medical Birth Registry (for births from 1992 through 2011) with those from the Swedi
150 f interest were perinatal mortality, preterm birth, hospital attendance for asthma exacerbations, and
151 4R, STAT4 and TBET) in umbilical cord DNA at birth in a cohort of infants from the Southampton Women'
153 and then assessed the timing of infection at birth in children with ALL and age, gender, and ethnicit
157 sal screening to predict spontaneous preterm birth in nulliparous women using serial measurements of
161 th) were rare in all periods (5.2 per 100000 births in the baseline period, 1.9 per 100000 births in
162 s in the learning period, and 5.3 per 100000 births in the EOS calculator period) and did not differ
163 irths in the baseline period, 1.9 per 100000 births in the learning period, and 5.3 per 100000 births
164 pectrum disorder (ASD) now affects one in 68 births in the United States and is the fastest growing n
165 pe 1 (NF1), a common genetic disorder with a birth incidence of 1:2,000-3,000, is characterized by a
166 To test whether advanced maternal age at birth independently increases the risk of low birth weig
167 early postpartum stress, gestational age at birth, infant sex, and postnatal age at magnetic resonan
168 idual susceptibility to injury after preterm birth.Inflammation mediated by microglia plays a key rol
170 finding that small-for-gestational-age (SGA) birth is associated with increased adiposity in childhoo
173 states with increasing time since beta-cell birth, leading to functional and proliferative heterogen
175 tigator groups on the association of preterm birth (<37 weeks' gestation) and maternal GBS colonizati
176 k of low birth weight (<2,500 g) and preterm birth (<37 weeks' gestation), we compared between-family
180 s compared with unexposed offspring (preterm birth odds ratio [OR], 1.47 [95% CI, 1.40-1.55]; small f
181 d fraction of the genome, driven both by the birth of new hypomethylated regions and by extensive wid
182 ng: 2016 marked the 125th anniversary of the birth of the physical chemist Michael Polanyi, as well a
186 ressant exposure was associated with preterm birth (OR, 1.34 [95% CI, 1.18-1.52]) but not with small
189 iations between macroeconomic conditions and birth outcomes differed during and after (compared with
190 to 2010 and estimated PTBs and other adverse birth outcomes for infants borne by non-Hispanic black m
191 al area, showing that an increase in adverse birth outcomes is observed with very high levels of pest
193 he relationship between the macroeconomy and birth outcomes, in part due to the methodological challe
194 esticide use has been associated with poorer birth outcomes, neurodevelopment, and respiratory functi
195 ing in explaining black-white differences in birth outcomes, the individual contribution of PM2.5 is
196 e not had sufficient impact on pregnancy and birth outcomes, which suggests that the focus for interv
200 family socioeconomic status (SES) at time of birth (P = 0.001), but not parental age nor maternal ges
203 ignificantly increased prevalence of preterm birth (prevalence ratio [PR], 1.52; 95% CI, 1.34-1.71),
204 has high potential for prediction of preterm birth (PTB), a problem affecting 15 million newborns ann
205 Molecular mechanisms regulating preterm birth (PTB)-associated cervical remodeling remain unclea
207 etrics, gestational weight gain, and preterm birth rate, but not in maternal age, parity, socioeconom
208 cidence in women aged 15-49 years to 2010-15 birth rates to estimate infections during pregnancy.
209 hypothermia initiated at 6 to 24 hours after birth reduces the risk of death or disability at 18 mont
210 born at term and with appropriate weight at birth regarding their potential connection to psychiatri
211 study, linking data from the Swedish Medical Birth Registry (for births from 1992 through 2011) with
213 onstrates mediation of the phthalate-preterm birth relationship by oxidative stress, and the utility
214 head movements as early as 1-2 months after birth, relative to HR infants who showed more rapid deve
215 teorological parameters, to increase preterm birth risk has received significant attention worldwide,
217 uration of gestation and the risk of preterm birth, robust associations with genetic variants have no
222 height (m)2), gestational weight gain (GWG), birth size, and childhood growth factors with adult BMI
223 vision occurs after adding a fixed size from birth), sizer (division occurs after reaching a size thr
224 ity, we examined pregnancy outcomes (preterm birth, stillbirth, small for gestational age, or congeni
226 sponse, and implicated Vts1 in de novo gene "birth." TGA provided single-nucleotide resolution for ea
227 erm outcome had higher numbers of B cells at birth than those who developed LTI; no difference in B c
228 Here we demonstrate that in those deaf from birth the left and the right STC have altered and dissoc
229 the reason the woman was at risk of preterm birth, the gestational age at which magnesium sulphate t
230 who were exposed to vitamin A in utero or at birth.The aim of this study was to examine independent a
231 ents use some antenatal care for their first birth, they seek care later, make fewer visits during pr
232 dren continuously enrolled within a month of birth through 15 months who received at least 3 doses of
235 al multicentre cohort study of children from birth to 15 years of age undergoing elective or urgent a
237 e calculated the probabilities of death from birth to age 5 years and from age 15 to 60 years, life e
238 odemographic characteristics of women giving birth to an infected baby after primary and nonprimary i
240 om school age to adulthood and c-height from birth to school age were positively associated with hype
248 fficacy of UCB in MPS-I mice transplanted at birth, we first defined the features of murine UCB cells
249 disease during early childhood after preterm birth, we performed a prospective, longitudinal study of
251 ion criteria were gestational age <32 weeks, birth weight <1000 g, known immunodeficiency or no Danis
252 tive observational study of preterm infants (birth weight <1500 g and/or gestational age <32 weeks) w
253 he low-birth-weight babies having a very low birth weight (<1,500 g) in the more recent birth cohort.
254 irth independently increases the risk of low birth weight (<2,500 g) and preterm birth (<37 weeks' ge
258 ce ratio [PR], 1.52; 95% CI, 1.34-1.71), low birth weight (PR, 1.59; 95% CI, 1.38-1.83), and cesarean
259 esults show that the association between low birth weight and decreased cognitive ability has decline
260 aminations by an ophthalmologist), CHOP-ROP (birth weight and gestational age, with weekly weight gai
262 single nucleotide polymorphisms determining birth weight combined with results from the Social Scien
263 th maternal smoking during pregnancy and low birth weight have been implicated in impaired developmen
264 /L) was associated with a higher risk of low birth weight in newborns (adjusted risk ratio = 1.15, 95
265 actors associated with preterm birth and low birth weight included treatment with chemotherapy and a
267 , born in the 28(th) week of gestation, with birth weight of 950 grams, who was born in an ambulance
268 nterpretation under specific assumptions, of birth weight on educational attainment using instrumenta
269 as 3381 g, while for other races/ethnicities birth weight ranged from being 289 g smaller in Japanese
272 infants had 4-week outcomes completed; mean birth weight was 709 g and mean gestational age was 24.9
274 maternal smoking during uterine life and low birth weight were independently associated with having a
275 rmance in adolescents (per 100-g increase in birth weight, -0.004 grade, 95% CI: -0.04, 0.04) using i
276 between maternal B12 levels in pregnancy and birth weight, but B12 deficiency (<148 pmol/L) was assoc
277 methylation status, and both correlated with birth weight, circulating IGF-I, and total and abdominal
278 ion is associated with preterm delivery, low birth weight, fetal growth retardation and developmental
279 ad not smoked after correction for age, sex, birth weight, height, body weight, Tanner stage of puber
282 in life and are driven largely by height and birth weight, without any comparable influence of BMI or
284 ference (SD) in cognitive scores between low-birth-weight and normal-birth-weight children was large
285 hort, despite a higher proportion of the low-birth-weight babies having a very low birth weight (<1,5
287 0.6-6.3 mum; P = .02) thinner than in normal-birth-weight children after adjustment for all variables
288 e scores between low-birth-weight and normal-birth-weight children was large in the NCDS [-0.37 SD, 9
290 tal courses were similar in both groups, and birth weights ranged from 580 to 1495 g in the lower-pro
291 nerally increased with age, but responses at birth were poorly predictive for those at ages 1 and 3 y
294 matologic and neurologic complications after birth, whereas TCblR/CD320 gene defects appear to produc
296 adients for mortality and life expectancy at birth, with outcomes improving with increasing socioecon
297 is seen regardless of the reason for preterm birth, with similar effects across a range of preterm ge
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