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1 orhood income, soda consumption, and child's birth weight.
2 ion between endoscopy and gestational age or birth weight.
3 .9), respectively, in the probability of low birth weight.
4 Childhood BMI, change in BMI, and birth weight.
5 pregnancies complicated by stillbirth or low birth weight.
6 with adverse pregnancy outcomes such as low birth weight.
7 teries and not correlated significantly with birth weight.
8 ociations are age dependent or influenced by birth weight.
9 gnancy is very large, is associated with low birth weight.
10 here was no effect of vaccine on SGA or mean birth weight.
11 which was partly mediated by differences in birth weight.
12 or gestational age (SGA) or preterm and mean birth weight.
13 ence of the impact of DHA supplementation on birth weight.
14 statistically significant (P = 0.05) for low birth weight.
15 h maternal anemia, placental malaria, or low birth weight.
16 risk of adverse birth outcomes, such as low birth weight.
17 he great arteries had smaller HC relative to birth weight.
18 ren with a shorter gestational age and lower birth weight.
19 ts in previous GWAS and the CCNL1 locus with birth weight.
20 1 of 19 303) and 0.6% of infants <1000 grams birth weight.
21 irment in 11-year-old children with very low birth weight.
22 oncentrations were inversely associated with birth weight.
23 s, decreased vaccine response, and decreased birth weight.
24 n volumes at the time of MRI as well as with birth weights.
25 nonsignificant decreases in the risk of low birth weight (0.68; .29-1.57) and fetal or neonatal deat
26 rmance in adolescents (per 100-g increase in birth weight, -0.004 grade, 95% CI: -0.04, 0.04) using i
27 th years of schooling (per 100-g increase in birth weight, -0.006 years, 95% confidence interval (CI)
29 ale); and 254 children with trisomy 18 (mean birth weight, 1.8 [0.7] kg; 157 [61.8%] female), with fo
30 valuated by intent-to-treat analysis (median birth weight, 1066 g; mean gestational age, 28.4 weeks).
31 uded 174 children with trisomy 13 (mean [SD] birth weight, 2.5 [0.7] kg; 98 [56.3%] female); and 254
32 were consistent with the association between birth weight (21.1% mediation effect; P = 6.20 x 10-3) a
33 findings delivered babies with a lower mean birth weight (2960 vs 2867 g; mean difference, -93 g [95
34 g women with only subpatent infections (mean birth weight, 3013 g; mean difference, 54 [95% CI, -33-1
35 s and faecal samples were analysed for a low-birth weight (725 g) neonate EGA 25 weeks in intensive c
36 odds ratio, 1.15; 95% CI, 0.93 to 1.42), low birth weight (76 cases among 1768 exposed pregnancies an
37 iation] gestational age 25.7 [1.2] weeks and birth weight 813 [183] grams), 184 (24%) underwent ligat
40 atresia, while maximal absolute SB width by birth weight, age, PN duration, and remaining bowel leng
42 were born very preterm and/or with very low birth weight and 106 term-born control subjects from the
44 aternal glucose during pregnancy and newborn birth weight and adiposity demand fuller characterizatio
50 lic diseases increase offspring risk for low birth weight and cardiometabolic diseases in adulthood.
52 was used to analyze the association between birth weight and cognitive ability in a baseline model a
53 ster maternal plasma PFAS concentrations and birth weight and cord blood concentrations of leptin and
54 esults show that the association between low birth weight and decreased cognitive ability has decline
56 aminations by an ophthalmologist), CHOP-ROP (birth weight and gestational age, with weekly weight gai
59 in maternal height and education and infant birth weight and length were associated with greater rel
67 ference (SD) in cognitive scores between low-birth-weight and normal-birth-weight children was large
68 as 1.86 between children with low and normal birth weight, and 1.66 between children of smoking and n
69 aternal height and education and infant sex, birth weight, and birth length, which suggest that key d
70 al level, maternal smoking during gestation, birth weight, and breastfeeding duration, girls carrying
71 onal adjustment for gestational weight gain, birth weight, and children's insulin concentrations.Our
80 ropriately growing fetuses (e.g., singleton, birth weight appropriate for their gestational age, and
81 weeks of gestation, including neonates with birth weights appropriate, small, and large for age, wit
83 hort, despite a higher proportion of the low-birth-weight babies having a very low birth weight (<1,5
84 operations associated with 1 additional low birth weight baby, every 25 operations associated with 1
85 ODS AND A cohort study of 58 FGR (defined as birth weight below 10th centile) and 94 normally grown f
86 nal plasma betaine was associated with lower birth weight (beta: -57.6 g; 95% CI: -109.9, -5.3 g), sh
88 ased CVD risk when compared to subjects with birth weight between 2500-3999 g (OR 2.47, 95%CI, 1.07-5
90 lent and has been associated with both lower birth weight (birth weight <2,500 g) and preterm birth (
91 , neural tube defects, preterm birth and low birth weight, birth asphyxia, and intracranial hemorrhag
92 identified associations between AA and sex, birth weight, birth by caesarean section and several mat
93 livery, gestational age (for preterm birth), birth weight, birth weight in relation to gestational ag
94 Preconception alcohol-exposed offsprings' birth weight, body growth, stress response, anxiety-like
98 between maternal B12 levels in pregnancy and birth weight, but B12 deficiency (<148 pmol/L) was assoc
105 nopathy of Prematurity (CHOP ROP) model uses birth weight (BW), gestational age at birth (GA), and we
107 Exposure during periods 1, 2, and 4 reduced birth weight by approximately 10% compared with FA, and
109 were born very preterm and/or with very low birth weight, cBF volumes were significantly reduced com
110 egression was used, including adjustment for birth weight, center, breastfeeding, illness severity, a
111 %CI 0.60, 0.62]) compared with noncustomised birth weight centiles (AUROC 0.62 [95%CI 0.60, 0.63]) an
113 tality between non- and partially customised birth weight centiles was compared using area under the
118 0.6-6.3 mum; P = .02) thinner than in normal-birth-weight children after adjustment for all variables
119 e scores between low-birth-weight and normal-birth-weight children was large in the NCDS [-0.37 SD, 9
120 methylation status, and both correlated with birth weight, circulating IGF-I, and total and abdominal
121 single nucleotide polymorphisms determining birth weight combined with results from the Social Scien
122 laining differences of more than 50 grams in birth weight compared to white infants were: gestational
123 Such a group comprises premature birth, low-birth-weight, congenital anomalies, perinatal asphyxia,
124 their mid-20s was similar to that of normal-birth-weight controls (>2500g), there was uncertainty as
126 tion number but positively with weaning AGD, birth weight, dam AGD and percentage of males in the lit
128 l characteristics, and pregnancy conditions, birth weight decreased by 2.20 g per year (P < 0.0001).
130 2009-2012 in the United States, we examined birth weight differences among 14 races and ethnicities
131 from the lowest and highest quintiles of the birth weight distribution (irrespective of their mode of
135 ion is associated with preterm delivery, low birth weight, fetal growth retardation and developmental
137 ely sampled to be discordant on sex-specific birth weight for gestational age (BW/GA) in order to min
139 on, by trimester, and by toxicity influences birth weight, gestational length, or birth abnormalities
142 hy FT infants (>/=37 weeks' gestational age; birth weight >2499 g; born at the Royal Women's Hospital
143 r 1-mmol/L increase) and risk of macrosomia (birth weight >4000 g) (RR = 1.21; 95% CI: 1.07, 1.38 per
147 th maternal smoking during pregnancy and low birth weight have been implicated in impaired developmen
148 med to determine whether temporal changes in birth weight have occurred amongst 2.3 million neonates
149 ad not smoked after correction for age, sex, birth weight, height, body weight, Tanner stage of puber
150 ounting for sex, parity, breastfeeding, term birth weight, household income, maternal education, deli
151 PFAS exposure has been associated with lower birth weight; however, impacts on body composition and f
152 sed on the gestation period and not based on birth weight; however, this is done in several studies f
153 e), wasting (low weight-for-height), and low birth weight in children aged between 0 and 59 mo at the
154 ndelian randomization to clarify the role of birth weight in ischemic heart disease (IHD) and lipids.
155 /L) was associated with a higher risk of low birth weight in newborns (adjusted risk ratio = 1.15, 95
156 ional age (for preterm birth), birth weight, birth weight in relation to gestational age, 5-minute Ap
157 ample (Vietnam Era Twin Study of Aging), but birth weight in the child sample had an effect on cortic
158 actors associated with preterm birth and low birth weight included treatment with chemotherapy and a
159 that mice lacking Snord116 globally have low birth weight, increased body weight gain, energy expendi
161 port by nurses have higher rates of very low birth weight infants discharged home on human milk.
162 een the dependent variable (rate of very low birth weight infants discharged on "any human milk") and
163 -blind randomized clinical trial in very low-birth-weight infants (birth weight <1500 g) admitted to
164 participants who were able to provide their birth weight information and were followed from 2002 to
168 g pregnancy with preterm birth (PTB) and low birth weight (LBW) but disagree over which time frames a
169 ia, and although this has been linked to low birth weight (LBW) in these populations, the relation be
170 the effectiveness of IPTp-SP at reducing low birth weight (LBW) were assessed among human immunodefic
171 hand smoke (SHS) during pregnancy causes low birth weight (LBW), but its mechanism remains unknown.
172 tors according to presence or absence of low birth weight (LBW, birth weight <2500 g), adjusted CVD r
173 igated sex-specific associations between low birth weight (LBW; <2.5 kg) and adult-onset diabetes in
174 dies have reported that individuals with low birth weights (LBW, <2500 g) have a lower intelligence q
176 e at birth, but finds no evidence of smaller birth weight, length, or head circumference among whites
177 age at diet diary, sex, total energy intake, birth weight/length, and rate of prior growth and cluste
179 onates younger than 30 weeks' gestation with birth weight less than 1250 g receiving mechanical venti
180 he e-ROP Study were premature infants with a birth weight less than 1251 g and a known ROP outcome en
182 ber 31, 2013, in 1257 premature infants with birth weights less than 1251 g in 13 neonatal units in N
184 ded into cases (109 mothers of newborns with birth weight < 2,500 g) and controls (263 mothers of new
186 oser surveillance for those with a predicted birth weight </=25th or >/=85th centile may reduce adver
187 large for gestational age (LGA), defined as birth weight <10(th) or >90(th) centile respectively for
188 ion criteria were gestational age <32 weeks, birth weight <1000 g, known immunodeficiency or no Danis
189 tive observational study of preterm infants (birth weight <1500 g and/or gestational age <32 weeks) w
190 ical trial in very low-birth-weight infants (birth weight <1500 g) admitted to 1 of 6 neonatal intens
191 stational weeks), small for gestational age (birth weight <2 SDs below the mean for gestational age),
192 een associated with both lower birth weight (birth weight <2,500 g) and preterm birth (length of gest
193 groups, but the incidence of newborns with a birth weight <2.4 kg (weight-for-age z score <-2) was hi
194 sting for confounding factors, subjects with birth weight <2500 g were at a significantly increased C
195 resence or absence of low birth weight (LBW, birth weight <2500 g), adjusted CVD risk was significant
197 he low-birth-weight babies having a very low birth weight (<1,500 g) in the more recent birth cohort.
198 irth independently increases the risk of low birth weight (<2,500 g) and preterm birth (<37 weeks' ge
199 me measures included induction of labor, low birth weight (<2500 g), cesarean section, Apgar score <7
200 ssion models controlled for gestational age, birth weight, maternal age, parity, prepregnancy body ma
201 ntrolling for known modifiers, including low birth weight, maternal education, seizure disorder, kidn
203 findings delivered babies with a lower mean birth weight (mean difference, -94 g [95% CI, -31 to -15
206 h weight (VLBW, 1000-1499 g), moderately low birth weight (MLBW, 1500-2499 g) and NBW individuals wer
207 ne associations of prenatal OP exposure with birth weight (n = 1,169), length (n = 1,152), and head c
208 parental season of birth predicted offspring birth weight (n = 2097) or length (n = 1172), height-for
210 scue treatment was associated with decreased birth weight (odds ratio [OR], -0.007; P = 0.04) and age
212 WE and WWOE was associated with a mean lower birth weight of 110 and 136g, respectively, as compared
213 growth of very preterm infants with a median birth weight of 1200 g, who achieved near-fetal growth r
216 , born in the 28(th) week of gestation, with birth weight of 950 grams, who was born in an ambulance
217 with a gestational age of at least 35 weeks, birth weight of at least 2.5 kg, and toleration of full
218 were gestational age of less than 32 weeks, birth weight of less than 1000 g, known immunodeficiency
219 lationship between THM and HAA exposures and birth weight of up to 7,438 singleton term babies using
222 protein restriction was used to generate low-birth-weight offspring that underwent accelerated postna
223 ilarly obtained an estimate of the effect of birth weight on academic performance in 4,067 adolescent
224 nterpretation under specific assumptions, of birth weight on educational attainment using instrumenta
226 onship between advanced maternal age and low birth weight or preterm birth is statistically and subst
227 ndependently associated with the risk of low birth weight or preterm delivery among mothers who have
229 CI, 1.25-1.94; 14 studies; I2, 39%) and low birth weight (OR, 1.96; 95% CI, 1.24-3.10; 8 studies; I2
230 ds ratio [OR], 2.45; 95% CI, 1.36-4.40), low birth weight (OR, 3.41; 95% CI, 1.61-7.26), and use of v
233 ractive error (>/= 3 dioptres), astigmatism, birth weight percentile, gestational age, retinopathy of
235 ce ratio [PR], 1.52; 95% CI, 1.34-1.71), low birth weight (PR, 1.59; 95% CI, 1.38-1.83), and cesarean
239 r adjusting for sex, parental education, low birth weight, preterm birth, parental social class, mate
241 rticipants were extremely premature infants (birth weight range, 401-1000 g; gestational age, 22-27 w
242 as 3381 g, while for other races/ethnicities birth weight ranged from being 289 g smaller in Japanese
243 tal courses were similar in both groups, and birth weights ranged from 580 to 1495 g in the lower-pro
247 , stroke, stillbirth, preterm birth, and low birth weight; screening and risk prediction test perform
248 justment for the covariates gestational age, birth weight, sex, difference in age at diet-diary compl
249 There were no significant differences in low birth weight, small for gestational age, birth length, h
253 e an increased risk of preterm birth and low birth weight, suggesting that additional surveillance of
254 of patent infections at enrollment with low birth weight suggests the importance of preventing P. fa
255 eported that the transition of extremely low-birth-weight survivors (</=1000 g) in their mid-20s was
256 In the fourth decade of life, extremely low-birth-weight survivors achieved similar educational leve
257 ipants included 100 (39 males) extremely low-birth-weight survivors and 89 (33 males) normal-birth-we
260 tical intervention where no infants were low birth weight, the adjusted controlled direct effect of p
261 a 62.9-g decrease (95% CI: -111.6, -14.2) in birth weight, though the association was null in the oth
263 thus, reduction in the variability of piglet birth weight to improve the sow prolificacy is possible
266 nts born very preterm (VPT) or with very low birth weight (VLBW) is necessary to guide clinical manag
267 y low birth weight (ELBW, <1000 g), very low birth weight (VLBW, 1000-1499 g), moderately low birth w
268 tibiotic use among all hospitalized very low-birth-weight (VLBW) infants across Canada and the associ
269 from January 2010 to February 2014, very low-birth-weight (VLBW, </=1500 g) infants, within 5 days of
271 ajority of infants (89%) were term; the mean birth weight was 2577 +/- 260 g, and the mean head circu
274 infants had 4-week outcomes completed; mean birth weight was 709 g and mean gestational age was 24.9
283 were born very preterm and/or with very low birth weight was specifically associated with both neona
284 eeding in addition to standard care improves birth weight.We performed a multicenter, open-label rand
285 maternal smoking during uterine life and low birth weight were independently associated with having a
288 rome is an imprinting disorder involving low birth weight with complex genetics and diagnostics.
289 of maternal smoking during pregnancy and low birth weight with retinal nerve fiber layer (RNFL) thick
290 in life and are driven largely by height and birth weight, without any comparable influence of BMI or
291 od leptin levels in models that adjusted for birth weight z score but not in models that did not adju
293 oncentrations were inversely associated with birth weight z score, though the null value was included
295 er in the low-GI group than in the HF group (birth weight z score: 0.2 +/- 0.2 compared with 0.7 +/-
296 m the population mean head circumference and birth weight z scores were reduced by up to 66% with adj
298 ween-group difference in the median neonatal birth-weight z score (0.05 in the metformin group [inter
299 tcome was a reduction in the median neonatal birth-weight z score by 0.3 SD (equivalent to a 50% redu
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