ライフサイエンスコーパス: bisphenol

1 cations and the limits of detection for each bisphenol.

2 eceptor, which are two known main targets of bisphenols.

3 ylhexyl phthalate (DEHP) and polychlorinated bisphenol 153 (PCB153), were detected in adult dog teste

4 tly with K-Ras and that Rheb weakly binds to bisphenol A (10) and 4,4'-biphenol derivatives.

5 We show for the first time that bisphenol A (10) has the capacity to interact directly w

6 Our results propose a new mode of action for bisphenol A (10) that advocates a reduced exposure to th

7 Bisphenol A (2,2-bis[4-hydroxyphenyl]propane, BPA), the

8 By combination with an antibody directed to bisphenol A (alphaBPA), this conducting polymer-based bi

9 Environmental exposure to bisphenol A (BPA) affects mammary gland development in r

10 hinylestradiol (EE2)] and two xenoestrogens [bisphenol A (BPA) and 4-n-nonyl-phenol (NP)] in surface

11 Bisphenol A (BPA) and bisphenol A diglycidyl ether (BADG

12 Bisphenol A (BPA) and its brominated derivative tetrabro

13 The health effects related to bisphenol A (BPA) and its exposure sources have undergon

14 ence regarding associations for exposures to bisphenol A (BPA) and phthalates with type 2 diabetes (T

15 is growing concern that prenatal exposure to bisphenol A (BPA) and phthalates, which are widely used

16 The endocrine disruptor bisphenol A (BPA) and the pharmaceutical 17alpha-ethinyl

17 Phthalates and bisphenol A (BPA) are endocrine disruptors, and previous

18 Phthalates and bisphenol A (BPA) are widely used industrial chemicals t

19 Increasing concern over bisphenol A (BPA) as an endocrine-disrupting chemical an

20 cale sequencing batch reactors (SBRs), using bisphenol A (BPA) as the TOrC.

21 erns, variability, and predictors of urinary bisphenol A (BPA) concentrations in 337 children from th

22 n expert panel reviewed associations between bisphenol A (BPA) exposure and reproductive health outco

23 We used in utero bisphenol A (BPA) exposure as a model environmental expo

24 Bisphenol A (BPA) exposure early in life results in orga

25 Prenatal bisphenol A (BPA) exposure has been associated with adve

26 Gestational phthalate and bisphenol A (BPA) exposure may increase the risk of adve

27 ticipants previously randomly selected for a bisphenol A (BPA) exposure study who had unusually high

28 Human health risks due to bisphenol A (BPA) exposure through canned food consumpti

29 As concern regarding the toxic effects of bisphenol A (BPA) grows, BPA in many consumer products i

30 As the evidence of the toxic effects of bisphenol A (BPA) grows, its application in commercial p

31 Early-life exposure to bisphenol A (BPA) has been implicated to play a role in

32 Exposure to bisphenol A (BPA) has been reported to alter global gene

33 Prenatal exposure to nonylphenol (NP) and/or bisphenol A (BPA) has been reported to be associated wit

34 Widespread environmental contamination by bisphenol A (BPA) has created the need to fully define i

35 Pressures to ban bisphenol A (BPA) has led to the use of alternate chemic

36 We report on an innovative heterogeneous bisphenol A (BPA) immunoassay based on an electrolyte-ga

37 try, has been developed for determination of bisphenol A (BPA) in canned tomatoes.

38 ead use of the endocrine disrupting chemical bisphenol A (BPA) in consumer products has resulted in n

39 The determination of bisphenol A (BPA) in foods and beverages sold in Turkish

40 s increasingly been used as a substitute for bisphenol A (BPA) in some "BPA-free" consumer goods and

41 ectrochemical biosensor for the detection of Bisphenol A (BPA) in water has been developed by immobil

42 y bottles, little attention has been paid to bisphenol A (BPA) intake from packaged water consumption

43 Bisphenol A (BPA) is a common chemical used in the manuf

44 Bisphenol A (BPA) is a high-production-volume chemical a

45 Bisphenol A (BPA) is a synthetic chemical widely used in

46 Bisphenol A (BPA) is a ubiquitous compound that is emerg

47 Bisphenol A (BPA) is a well-known endocrine disruptor th

48 Bisphenol A (BPA) is an endocrine disrupting compound th

49 Bisphenol A (BPA) is an endocrine disruptor and potentia

50 Bisphenol A (BPA) is an endocrine disruptor frequently d

51 Bisphenol A (BPA) is an endocrine-disrupting compound wi

52 Bisphenol A (BPA) is an estrogenic endocrine disruptor w

53 The plastic additive bisphenol A (BPA) is commonly found in landfill leachate

54 Bisphenol A (BPA) is found in polycarbonate plastic and

55 ure to the environmental endocrine disruptor bisphenol A (BPA) is ubiquitous and associated with the

56 Bisphenol A (BPA) is used widely to manufacture food con

57 The endocrine-disrupting chemical bisphenol A (BPA) is widely used in food and beverage pa

58 Bisphenol A (BPA) is widely used in the manufacture of p

59 Bisphenol A (BPA) is widely used in the manufacture of p

60 products from store shelves after reports of bisphenol A (BPA) leaching from baby bottles, reusable d

61 oprobe is designed for the quantification of bisphenol A (BPA) levels in the blood after human exposu

62 rly-life exposure to the endocrine disruptor bisphenol A (BPA) may contribute to the development of o

63 tory actions and public concerns, the use of bisphenol A (BPA) may decrease, while the use of BPA alt

64 Early-life exposure to bisphenol A (BPA) may increase childhood obesity risk, b

65 1.0 microM) and a response time of 250 s for bisphenol A (BPA) monitoring.

66 Effects of bisphenol A (BPA) on ovarian transcript profiles as well

67 Certain phthalates and bisphenol A (BPA) show reproductive effects in animal st

68 Humans are exposed to low-dose bisphenol A (BPA) through plastic consumer products and

69 ion on the homogeneous Fenton degradation of bisphenol A (BPA) used as a model pollutant, was investi

70 The xenoestrogen bisphenol A (BPA) used in the manufacturing of various p

71 However, bisphenol A (BPA) was identified by chemical:gene covari

72 The electrochemistry of bisphenol A (BPA) was studied by voltammetric methods at

73 scharge of the endocrine disrupting compound bisphenol A (BPA) with wastewater treatment plant (WWTP)

74 sed on the MS results these were assigned to bisphenol A (BPA), 2,4-di-tert-butylphenol, and a possib

75 Bisphenol A (BPA), a chemical incorporated into plastics

76 Studies have implicated exposure to bisphenol A (BPA), a commonly used chemical, in the deve

77 t chronic exposure of synthetic xenoestrogen bisphenol A (BPA), a component of consumer plastic produ

78 Bisphenol A (BPA), a component of some dialysis membrane

79 Bisphenol A (BPA), a high volume production chemical com

80 Bisphenol A (BPA), a plastics component, has been descri

81 Bisphenol A (BPA), a prevalent endocrine-disrupting chem

82 Bisphenol A (BPA), a ubiquitous endocrine disruptor that

83 al models suggests that prenatal exposure to bisphenol A (BPA), a ubiquitous endocrine-disrupting che

84 Bisphenol A (BPA), a widely used endocrine-disrupting ch

85 Exposure of humans to bisphenol A (BPA), a widely used industrial chemical, is

86 S (BPS) are increasingly used to substitute bisphenol A (BPA), a widespread environmental endocrine

87 Bisphenol A (BPA), a xenoestrogenic endocrine-disrupting

88 Bisphenol A (BPA), an endocrine-disrupting chemical that

89 Bisphenol A (BPA), an endocrine-disrupting compound with

90 Bisphenol A (BPA), an environmental contaminant with wea

91 to investigate the carcinogenic property of Bisphenol A (BPA), an environmental estrogen, by long-te

92 tions have been reported between phthalates, bisphenol A (BPA), and child behavior.

93 Phthalate esters (PEs), bisphenol A (BPA), and parabens (PBs), which are used in

94 crine-disrupting chemicals (EDCs), including bisphenol A (BPA), are environmental ubiquitous pollutan

95 We examined the association of bisphenol A (BPA), benzophenone-3 (BP-3), triclosan (TCS

96 phenoxyethanol (2-PE), 4-nonylphenol (4-NP), bisphenol A (BPA), benzylbuthyl phthalate (BBP) and dime

97 al samples were analyzed for the presence of bisphenol A (BPA), bis (2-ethylhexyl) phthalate (DEHP),

98 like activity is attributed to EDCs, such as bisphenol A (BPA), bisphenol AF (BPAF), and zearalenone

99 ion of bisphenol F diglycidyl ether (BFDGE), bisphenol A (BPA), bisphenol B (BPB), bisphenol F (BPF)

100 Three selected chemicals, bisphenol A (BPA), diethylhexyl phthalate (DEHP) and min

101 ironment of some of these chemicals, such as bisphenol A (BPA), external contamination during handlin

102 One such exposure, bisphenol A (BPA), has been associated with obesity and

103 e effect of one suspicious food contaminant, bisphenol A (BPA), in vivo.

104 of environmental contaminants, particularly bisphenol A (BPA), is understudied with regard to PTB.

105 vestigated the direct effects of exposure to bisphenol A (BPA), mono-n-butyl phthalate (Pht), and pol

106 With the pressure to ban or limit the use of bisphenol A (BPA), production of alternatives such as bi

107 hlorophenol (25-DCP), benzophenone-3 (BP-3), bisphenol A (BPA), triclosan (TCS), butyl paraben (B-PB)

108 Analytes included parabens, phthalates, bisphenol A (BPA), triclosan, ethanolamines, alkylphenol

109 s of a single endocrine disrupting compound, bisphenol A (BPA), was developed.

110 sh register receipts, contain high levels of bisphenol A (BPA).

111 e electrochemical biosensor for detection of bisphenol A (BPA).

112 te (ICC: 0.55), whereas the ICC was 0.39 for bisphenol A (BPA).

113 oplasia from early-life exposure to low-dose bisphenol A (BPA).

114 for label-free and single step detection of Bisphenol A (BPA).

115 Upon bisphenol A addition, the competitive dissociation of th

116 PA), 2,4-di-tert-butylphenol, and a possible bisphenol A analog.

117 n sample age groups for case study chemicals bisphenol A and 2,5-dichlorophenol.

118 Bisphenol A and adiposity in an inner-city birth cohort.

119 not identified for the nonantimicrobial EDCs bisphenol A and benzophenone-3 (P > .2).

120 ime polymerase chain reaction indicated that bisphenol A and bisphenol AF consistently activated endo

121 e strain CBDB1 completely converted TBBPA to bisphenol A and BPB to phenol red with a stepwise remova

122 With the exceptions of bisphenol A and DEET, all TOrCs that were detected in th

123 ansgenic reporter demonstrated that the EEDs bisphenol A and genistein preferentially activate estrog

124 Combined effects of high-dose bisphenol A and oxidizing agent (KBrO3) on cellular micr

125 Human exposure to phenols, including bisphenol A and parabens, is widespread.

126 Urinary concentrations of bisphenol A and phthalate metabolites measured during pr

127 Recent fast food consumption and bisphenol A and phthalates exposures among the U.S. popu

128 g and toxic pollutants, many of which (e.g., bisphenol A and substituted phenols) are known to be ins

129 ants of emerging concern were performed with bisphenol A and sulfamethoxazole.

130 l molecules, as we demonstrated with a novel bisphenol A aptamer.

131 We demonstrate that bisphenol A as the template in combination with phthaloy

132 ained from horse radish peroxidase (HRP) and bisphenol A assays were 12.5 ng/ml (2.84x10(-10) M ) and

133 ata values, with diethyl hexyl phthalate and bisphenol A being the most ubiquitous compounds detected

134 esorcinol bis(diphenylphosphate) (RBDPP) and bisphenol A bis(diphenylphosphate) (BPA-BDPP) are two ha

135 fication and temporal variability in urinary bisphenol A concentrations among couples in Utah-the HOP

136 e were no measured chlorinated byproducts of bisphenol A despite occasionally high concentrations of

137 We present here a proof-of-concept for bisphenol A detection; the device could be readily adapt

138 Despite reports of the occurrence of bisphenol A diglycidyl ether (BADGE) and its derivatives

139 Epoxy resins incorporating bisphenol A diglycidyl ether (BADGE) and novolac glycidy

140 A), bisphenol B (BPB), bisphenol F (BPF) and bisphenol A diglycidyl ether (BADGE) down to 0.50ng/mL;

141 Bisphenol A (BPA) and bisphenol A diglycidyl ether (BADGE), used in manufactur

142 Bisphenol A diglycidyl ether (BADGE)- and bisphenol F di

143 ly lacquered with epoxy resins (ER) based on bisphenol A diglycidyl ether (BADGE).

144 PPARgamma antagonists (bisphenol A diglycidyl ether or GW9662) blunted the effe

145 cerol dimethacrylate (PMGDM) and ethoxylated bisphenol A dimethacrylate (EBPADMA) at mass ratio of 1:

146 xanediol dimethacrylate (HDDMA), ethoxylated bisphenol A dimethacrylate (EBPADMA), and urethane dimet

147 lectivity towards closely related compounds (bisphenol A dimethacrylate, and dibutyl phthalate).

148 d respiratory health, with ethyl-paraben and bisphenol A exhibiting some consistency across respirato

149 ne the multigenerational effects of maternal bisphenol A exposure on mouse pancreatic islets.

150 which factor, the dimethacrylate component, bisphenol A glycerolate dimethacrylate (BisGMA) or photo

151 2-hydroxyethyl methacrylate (HEMA) and 5% of bisphenol A glycidyl dimethacrylate (BisGMA) (PEHB).

152 The monomer mixture consisted of bisphenol A glycidyl methacrylate (BisGMA), hexanediol d

153 als composed of a trimethacrylate monomer or bisphenol A glycidyl methacrylate, which is a monomer st

154 Bisphenol A has been reported to be a ubiquitous contami

155 The change in the amount of bisphenol A in all of these food, based on expiration da

156 compounds 2,4-dichlorophenol, triclosan, and bisphenol A in chlorinated waters containing I(-).

157 see that in these kind of food the amount of bisphenol A increases with an increase in the amount of

158 Finally, we observe that bisphenol A induces rapid and parallel modulation of 5S

159 visualization of dose-dependent induction of Bisphenol A inducible genes showed a weak gene activatio

160 Bisphenol A is widely used in food and drinks packaging.

161 Bisphenol A led to a wide range of stress responses, and

162 method was applied for the determination of bisphenol A migrated from some polycarbonate plastic pro

163 The binding of bisphenol A onto the polymer film was successfully chara

164 The trends between bisphenol A pregnancy level and increased asthma and bro

165 , the UV filter benzophenone-2 (BP2) and the bisphenol A substitute bisphenol S (BPS) was deciphered

166 w and comparison of the hormonal activity of bisphenol A substitutes.

167 intramolecular glycosylations using a rigid bisphenol A template supplemented with linkers of variou

168 ith FEV1% (beta=-0.59; 95% CI: -1.24, 0.05); bisphenol A tended to be associated with increased rates

169 free chlorine alone, while for triclosan and bisphenol A the free iodine mediated transformations wer

170 at levels ranging from 0.6 to 4.6ngg(-1) and bisphenol A was detected in all the samples at concentra

171 lutants) and nonpersistent (some phthalates, bisphenol A) environmental pollutants, and exposure to a

172 for rapid detection of estrogenic substance (bisphenol A) has been proposed.

173 f other xenoestrogens (e.g., alkylphenols or bisphenol A) that are sometimes found in surface water.

174 micals such as phthalates and phenols (e.g., bisphenol A).

175 mmonly targeted EDC and their isobars (e.g., bisphenol A, (Z)- and (E)-diethylstilbestrol, hexestrol,

176 water consumption was associated with higher bisphenol A, 2,4-dichlorophenol, and 2,5-dichlorophenol

177 Ethyl-paraben, bisphenol A, 2,5-dichlorophenol, and DIDP tended to be a

178 e concentrations of 8 phthalate metabolites, bisphenol A, 25 polychlorinated biphenyls (PCBs), 6 orga

179 an environmentally relevant concentration of bisphenol A, a nearly ubiquitous environmental contamina

180 environmental contaminants (benzo[a]pyrene, bisphenol A, and acrolein) in human serum via a competit

181 al smoking/nicotine, organotins, phthalates, bisphenol A, and pesticides.

182 hyl paraben, propyl paraben, benzophenone-3, bisphenol A, and triclosan in urine collected during 199

183 unds that contain a phenolic ring (parabens, bisphenol A, and triclosan) were completely transformed

184 lphenol, octylphenol, ibuprofen, diclofenac, bisphenol A, and triclosan.

185 hemical detection of the endocrine disruptor bisphenol A, as well as the capture of living nonadheren

186 ere used to examine the sorption affinity of bisphenol A, atrazine, and diuron to five soils of varyi

187 Exposure to environmental phenols (e.g., bisphenol A, benzophenone-3, and triclosan) and parabens

188 erum testosterone concentrations and urinary bisphenol A, benzophenone-3, triclosan, and paraben leve

189 Bisphenol A, bisphenol AF, and 2-2-bis(p-hydroxyphenyl)-

190 Bisphenol A, bisphenol S, and 4-hydroxyphenyl 4-isoproox

191 s had overall false positive rates of </=5%, bisphenol A, caffeine, NP, OP, and triclosan had false p

192 tion of phenolic compounds including phenol, bisphenol A, catechol and cresols is reported.

193 d disinfection byproducts of salicylic acid, bisphenol A, gemfibrozil, naproxen, diclofenac, technica

194 ct on pbPPARG, while hexabromocyclododecane, bisphenol A, oxychlordane, and endosulfan were weak anta

195 ed for phthalates and to a lesser extent for bisphenol A, synthetic musks, some pesticides, and PAHs.

196 actone, genistein, daidzein, benzophenone-3, bisphenol A, the sum of parabens (methyl, ethyl, and pro

197 n significantly reduce the teratogenicity of bisphenol A, triclosan and 17alpha-ethinyl estradiol wit

198 pathways have been relatively well studied, bisphenol A, triclosan, and ibuprofen.

199 Other exposures (total parabens, bisphenol A, triclosan, benzophenone-3, total phthalates

200 oducts, namely methylparaben, propylparaben, bisphenol A, triclosan, galaxolide, and 4- methylbenzili

201 y diarylpropionitrile, genistein, equol, and bisphenol A, whereas its coactivation at the AP-1 site i

202 gel-filled, and water-filled (most labeled "bisphenol A-free"), were collected from the U.S. market

203 n estrogen receptor alpha (hERalpha), called bisphenol A-targeted receptor (BPA-R).

204 ahistidine peptide for the quantification of bisphenol A.

205 ates, diethylhexyl adipate, alkylphenols and bisphenol A.

206 her than the corresponding concentrations of bisphenol A.

207 co-crystallized with an alkyl derivative of bisphenol A.

208 utrition Examination Survey in which urinary bisphenol A; triclosan; benzophenone-3; propyl, methyl,

209 ndocrine disrupting chemicals (EDCs) such as bisphenol-A (BPA) and diethylstilbestrol (DES).

210 health hazards related to persisting use of bisphenol-A (BPA) are well documented.

211 In contrast, Bisphenol-A (BPA) elicited a non-monotonic response.

212 The endocrine disruptor Bisphenol-A (BPA) has been shown to modulate estrogenic,

213 Bisphenol-A (BPA) is an endocrine disrupting chemical us

214 Bisphenol-A (BPA) is an environmentally ubiquitous estro

215 Bisphenol-A (BPA) is widely used in the manufacture of p

216 lastic derived endocrine disruptor compounds bisphenol-A (BPA), bis(2-ethylhexyl)phthalate (DEHP) and

217 ny endocrine disrupting chemicals, including bisphenol-A (BPA), induce non-monotonic dose response (N

218 Gestational bisphenol-A in male mice primed macrophages in adulthood

219 that gestational, but not adult, exposure to bisphenol-A increased the development of experimental au

220 Early life exposure to bisphenol-A may represent an environmental consideration

221 factor blocked susceptibility to disease in bisphenol-A mice.

222 trogen response after in utero xenoestrogen (bisphenol-A) exposure.

223 We investigated whether bisphenol-A, a prominent contaminant with endocrine-disr

224 ite dental restoration materials may release bisphenol-A, an endocrine-disrupting chemical.

225 BPA, BPS, and BPF were the three major bisphenols, accounting for >98% of the total concentrati

226 A (BPA), production of alternatives such as bisphenol AF (BPAF) and bisphenol S (BPS) are increasing

227 such as BPA analogues bisphenol S (BPS) and bisphenol AF (BPAF) in production of consumer products;

228 tributed to EDCs, such as bisphenol A (BPA), bisphenol AF (BPAF), and zearalenone (Zea), but mechanis

229 s, bisphenol S (BPS), bisphenol F (BPF), and bisphenol AF (BPAF), in 616 archived samples collected f

230 hain reaction indicated that bisphenol A and bisphenol AF consistently activated endogenous ER target

231 Bisphenol A, bisphenol AF, and 2-2-bis(p-hydroxyphenyl)-1,1,1-trichlo

232 uture exposure trends to BPA and some of its bisphenol alternatives.

233 the urinary concentrations of BPA and three bisphenol analogs, bisphenol S (BPS), bisphenol F (BPF),

234 mined vertical profiles of concentrations of bisphenol analogues in sediment cores from the U.S. and

235 A series of triphenylethylene bisphenol analogues of the selective estrogen receptor m

236 study, concentrations and profiles of eight bisphenol analogues were determined using high-performan

237 Other bisphenol analogues were rare or not detected, with the

238 approximately 10-fold less amounts than were bisphenols and benzophenones.

239 silico evaluation of the interaction between bisphenols and ERs or other members of the nuclear hormo

240 ographic analysis of the interaction between bisphenols and ERs reveals two discrete binding modes, r

241 the chemical replacements, however, are also bisphenols and may have similar physiological effects in

242 ethylhexyl) tetrabromophthalate, halogenated bisphenols and phenols, and hydroxylated PBDEs were foun

243 Common environmental contaminants such as bisphenols and phthalates and persistent contaminants su

244 amount of the sum of nine bisphenols (Sigma9 bisphenols) and 5 benzophenones (Sigma5 benzophenones) l

245 enyl ethers (PBDEs), halogenated phenols and bisphenols, and their metabolites.

246 d a gradual decline in the concentrations of bisphenols as compared to the past decade.

247 ting readily available amines, anilines, and bisphenols as starting materials.

248 diglycidyl ether (BFDGE), bisphenol A (BPA), bisphenol B (BPB), bisphenol F (BPF) and bisphenol A dig

249 Parabens (p-hydroxybenzoic acid esters), bisphenols, benzophenone-type UV filters, triclosan, and

250 bisalkylsulfonyl fluorides (AA monomers) and bisphenol bis(t-butyldimethylsilyl) ethers (BB monomers)

251 Here we identify the bisphenols bithionol and hexachlorophene as potent, sAC-

252 the toxicity and teratogenic effects of the bisphenols BPA, BPS, BPF, and BPAF in zebrafish embryo-l

253 Among individual bisphenols, BPA and bisphenol F (BPF) were the predomina

254 BB monomers were obtained from silylation of bisphenols by t-butyldimethylsilyl chloride.

255 effects and potencies between the different bisphenol chemicals.

256 All bisphenol compounds induced estrogenic responses in Tg(E

257 nds, accounting for 64% and 30% of the total bisphenol concentrations in sediment.

258 , are uncommon antioxidants bearing isomeric bisphenol cores substituted with allyl functions.

259 BN was applied to concentrate bisphenol derivatives in spiked water samples and the co

260 se extraction method for the enrichment of 5 bisphenol derivatives using hexagonal boron nitride (BN)

261 Furthermore, bisphenol derivatives were analyzed in spiked and non-sp

262 FDGE), bisphenol A (BPA), bisphenol B (BPB), bisphenol F (BPF) and bisphenol A diglycidyl ether (BADG

263 Bisphenol F (BPF) and bisphenol S (BPS) are increasingly

264 Bisphenol S (BPS) and bisphenol F (BPF) are two such BPA substitutes.

265 to structurally similar compounds including bisphenol F (BPF) or bisphenol S (BPS) even at much high

266 Among individual bisphenols, BPA and bisphenol F (BPF) were the predominant compounds, accoun

267 three bisphenol analogs, bisphenol S (BPS), bisphenol F (BPF), and bisphenol AF (BPAF), in 616 archi

268 We evaluated tetramethyl bisphenol F (TMBPF) using in vitro and in vivo assays.

269 ery sensitive, allowing for determination of bisphenol F diglycidyl ether (BFDGE), bisphenol A (BPA),

270 Bisphenol A diglycidyl ether (BADGE)- and bisphenol F diglycidyl ether (BFDGE)-based epoxy resins

271 omatography-tandem mass spectrometry, BADGE, bisphenol F glycidyl ether (BFDGE), 3-ring NOGE, and eig

272 ssarily safer and support the removal of all bisphenols from consumer merchandise.

273 ethod for simultaneous determination of five bisphenols in canned energy drinks by UPLC with fluoresc

274 to determine contamination levels from these bisphenols in forty energy drinks of different brands, c

275 Continued biomonitoring of these bisphenols in populations and further investigations on

276 henols ( summation operatorBPs; sum of eight bisphenols) in dust were in the range of 0.026-111 mug/g

277 We assessed the potential EA of both a new bisphenol monomer used to synthesize polymeric coatings

278 his is the first report on the occurrence of bisphenols, other than BPA, in indoor dust.

279 ng agents such as 4,4'-dihydroxybiphenyl and bisphenol P.

280 le absorption of BPA or the BPA alternatives bisphenol S (BPS) and 4-hydroxyphenyl 4-isoprooxyphenyls

281 of alternate chemicals such as BPA analogues bisphenol S (BPS) and bisphenol AF (BPAF) in production

282 Bisphenol S (BPS) and bisphenol F (BPF) are two such BPA

283 alternatives such as bisphenol AF (BPAF) and bisphenol S (BPS) are increasing, but little is known on

284 Bisphenol F (BPF) and bisphenol S (BPS) are increasingly used to substitute bi

285 lar compounds including bisphenol F (BPF) or bisphenol S (BPS) even at much higher concentration.

286 Bisphenol S (BPS) has increasingly been used as a substi

287 Bisphenol S (BPS) is an alternative to BPA in plastic co

288 enone-2 (BP2) and the bisphenol A substitute bisphenol S (BPS) was deciphered in eight human and zebr

289 than the accepted human daily exposure) and bisphenol S (BPS), a common analog used in BPA-free prod

290 trations of BPA and three bisphenol analogs, bisphenol S (BPS), bisphenol F (BPF), and bisphenol AF (

291 placed with other related compounds, such as bisphenol S (BPS).

292 dually being replaced with compounds such as bisphenol S (BPS).

293 Bisphenol S and F: a systematic review and comparison of

294 Bisphenol A, bisphenol S, and 4-hydroxyphenyl 4-isoprooxyphenylsulfon

295 ed compounds including 2,6-dinitro-p-cresol, bisphenol S, clonixin, and triclopyr.

296 ompounds in surface water, like caffeine and bisphenol-S, five dihydroxydiphenylmethane isomers were

297 The total amount of the sum of nine bisphenols (Sigma9 bisphenols) and 5 benzophenones (Sigm

298 o other soil properties correlated well with bisphenol sorption.

299 The total concentrations of bisphenols ( summation operatorBPs; sum of eight bisphen

300 Metabolites identified for all three bisphenols support degradation pathways that include met

301 s of BPF were generally lower than for other bisphenols, the 95th percentile concentration of BPF was