ライフサイエンスコーパス: bisphenol A

1 micals such as phthalates and phenols (e.g., bisphenol A).

2 co-crystallized with an alkyl derivative of bisphenol A.

3 ates, diethylhexyl adipate, alkylphenols and bisphenol A.

4 her than the corresponding concentrations of bisphenol A.

5 ase compounds of potential toxicity, such as bisphenol A.

6 ndensation reaction of phenol and acetone to bisphenol A.

7 mixture of 4,4'-dichlorodiphenyl sulfone and bisphenol A.

8 ol, 2',3',4', 5'-tetrachlorobiphenyl-ol, and bisphenol A.

9 ahistidine peptide for the quantification of bisphenol A.

10 ethinylestradiol (0.1 microg/kg per day) and bisphenol A (10 microg/kg per day), which are doses belo

11 tly with K-Ras and that Rheb weakly binds to bisphenol A (10) and 4,4'-biphenol derivatives.

12 We show for the first time that bisphenol A (10) has the capacity to interact directly w

13 Our results propose a new mode of action for bisphenol A (10) that advocates a reduced exposure to th

14 Bisphenol A (2,2-bis[4-hydroxyphenyl]propane, BPA), the

15 trations of BADGE and its three derivatives, bisphenol A (2,3-dihydroxypropyl) glycidyl ether [BADGE.

16 water consumption was associated with higher bisphenol A, 2,4-dichlorophenol, and 2,5-dichlorophenol

17 Ethyl-paraben, bisphenol A, 2,5-dichlorophenol, and DIDP tended to be a

18 e concentrations of 8 phthalate metabolites, bisphenol A, 25 polychlorinated biphenyls (PCBs), 6 orga

19 hydroxypropyl) glycidyl ether [BADGE.H(2)O], bisphenol A (3-chloro-2-hydroxypropyl) (2,3-dihydroxypro

20 ne of nine environmental phenolic compounds: Bisphenol A; 4-tert-octylphenol; o-phenylphenol; 2,4-dic

21 insight to the potential adverse effects of bisphenol A, a functionally similar and ubiquitous endoc

22 an environmentally relevant concentration of bisphenol A, a nearly ubiquitous environmental contamina

23 We investigated whether bisphenol-A, a prominent contaminant with endocrine-disr

24 Upon bisphenol A addition, the competitive dissociation of th

25 By combination with an antibody directed to bisphenol A (alphaBPA), this conducting polymer-based bi

26 ite dental restoration materials may release bisphenol-A, an endocrine-disrupting chemical.

27 PA), 2,4-di-tert-butylphenol, and a possible bisphenol A analog.

28 in the condensation of acetone and phenol to bisphenol A and 14 times more active in the condensation

29 n sample age groups for case study chemicals bisphenol A and 2,5-dichlorophenol.

30 Bisphenol A and adiposity in an inner-city birth cohort.

31 not identified for the nonantimicrobial EDCs bisphenol A and benzophenone-3 (P > .2).

32 ime polymerase chain reaction indicated that bisphenol A and bisphenol AF consistently activated endo

33 e strain CBDB1 completely converted TBBPA to bisphenol A and BPB to phenol red with a stepwise remova

34 With the exceptions of bisphenol A and DEET, all TOrCs that were detected in th

35 ansgenic reporter demonstrated that the EEDs bisphenol A and genistein preferentially activate estrog

36 Combined effects of high-dose bisphenol A and oxidizing agent (KBrO3) on cellular micr

37 Human exposure to phenols, including bisphenol A and parabens, is widespread.

38 Urinary concentrations of bisphenol A and phthalate metabolites measured during pr

39 Recent fast food consumption and bisphenol A and phthalates exposures among the U.S. popu

40 g and toxic pollutants, many of which (e.g., bisphenol A and substituted phenols) are known to be ins

41 ants of emerging concern were performed with bisphenol A and sulfamethoxazole.

42 id/thiol paired catalyst in the synthesis of bisphenol A and Z, and this increase in activity does no

43 environmental contaminants (benzo[a]pyrene, bisphenol A, and acrolein) in human serum via a competit

44 al smoking/nicotine, organotins, phthalates, bisphenol A, and pesticides.

45 hyl paraben, propyl paraben, benzophenone-3, bisphenol A, and triclosan in urine collected during 199

46 unds that contain a phenolic ring (parabens, bisphenol A, and triclosan) were completely transformed

47 lphenol, octylphenol, ibuprofen, diclofenac, bisphenol A, and triclosan.

48 l molecules, as we demonstrated with a novel bisphenol A aptamer.

49 wn plasticizers such as phthalate esters and bisphenol A as common laboratory contaminants.

50 We demonstrate that bisphenol A as the template in combination with phthaloy

51 ractitioners will learn about phthalates and Bisphenol-A as endocrine disruptors.

52 hemical detection of the endocrine disruptor bisphenol A, as well as the capture of living nonadheren

53 ained from horse radish peroxidase (HRP) and bisphenol A assays were 12.5 ng/ml (2.84x10(-10) M ) and

54 ere used to examine the sorption affinity of bisphenol A, atrazine, and diuron to five soils of varyi

55 hetic brominated fire retardants, brominated bisphenol A (BBPA), and polybrominated diphenyl ethers (

56 ata values, with diethyl hexyl phthalate and bisphenol A being the most ubiquitous compounds detected

57 Exposure to environmental phenols (e.g., bisphenol A, benzophenone-3, and triclosan) and parabens

58 erum testosterone concentrations and urinary bisphenol A, benzophenone-3, triclosan, and paraben leve

59 ihydroxypropyl) ether [BADGE.HCl.H(2)O], and bisphenol A bis (2,3-dihydroxypropyl) ether [BADGE.2H(2)

60 Bisphenol A bis(2,3-dihydroxypropyl) ether and bisphenol

61 esorcinol bis(diphenylphosphate) (RBDPP) and bisphenol A bis(diphenylphosphate) (BPA-BDPP) are two ha

62 ure of this overlayer would allow release of bisphenol A, BisGMA, and the decomposition products ther

63 Bisphenol A, bisphenol AF, and 2-2-bis(p-hydroxyphenyl)-

64 Bisphenol A, bisphenol S, and 4-hydroxyphenyl 4-isoproox

65 Environmental exposure to bisphenol A (BPA) affects mammary gland development in r

66 hinylestradiol (EE2)] and two xenoestrogens [bisphenol A (BPA) and 4-n-nonyl-phenol (NP)] in surface

67 Bisphenol A (BPA) and alkylphenols (APs) are widely used

68 Bisphenol A (BPA) and bisphenol A diglycidyl ether (BADG

69 Bisphenol A (BPA) and its brominated derivative tetrabro

70 The health effects related to bisphenol A (BPA) and its exposure sources have undergon

71 cerns exist regarding children's exposure to bisphenol A (BPA) and other phenols because of the highe

72 ence regarding associations for exposures to bisphenol A (BPA) and phthalates with type 2 diabetes (T

73 is growing concern that prenatal exposure to bisphenol A (BPA) and phthalates, which are widely used

74 The endocrine disruptor bisphenol A (BPA) and the pharmaceutical 17alpha-ethinyl

75 Phthalates and bisphenol A (BPA) are endocrine disruptors, and previous

76 Phthalates and bisphenol A (BPA) are widely used industrial chemicals t

77 Increasing concern over bisphenol A (BPA) as an endocrine-disrupting chemical an

78 cale sequencing batch reactors (SBRs), using bisphenol A (BPA) as the TOrC.

79 m of p-isopropenylphenol (IPP) synthesis via bisphenol A (BPA) cleavage in HTW.

80 erns, variability, and predictors of urinary bisphenol A (BPA) concentrations in 337 children from th

81 ry method for the determination of different bisphenol A (BPA) derivatives such as bisphenol A diglyc

82 n expert panel reviewed associations between bisphenol A (BPA) exposure and reproductive health outco

83 We used in utero bisphenol A (BPA) exposure as a model environmental expo

84 Bisphenol A (BPA) exposure early in life results in orga

85 Prenatal bisphenol A (BPA) exposure has been associated with adve

86 Gestational phthalate and bisphenol A (BPA) exposure may increase the risk of adve

87 ticipants previously randomly selected for a bisphenol A (BPA) exposure study who had unusually high

88 Human health risks due to bisphenol A (BPA) exposure through canned food consumpti

89 As concern regarding the toxic effects of bisphenol A (BPA) grows, BPA in many consumer products i

90 As the evidence of the toxic effects of bisphenol A (BPA) grows, its application in commercial p

91 Early-life exposure to bisphenol A (BPA) has been implicated to play a role in

92 Exposure to bisphenol A (BPA) has been reported to alter global gene

93 Prenatal exposure to nonylphenol (NP) and/or bisphenol A (BPA) has been reported to be associated wit

94 Widespread environmental contamination by bisphenol A (BPA) has created the need to fully define i

95 Pressures to ban bisphenol A (BPA) has led to the use of alternate chemic

96 We report on an innovative heterogeneous bisphenol A (BPA) immunoassay based on an electrolyte-ga

97 try, has been developed for determination of bisphenol A (BPA) in canned tomatoes.

98 ead use of the endocrine disrupting chemical bisphenol A (BPA) in consumer products has resulted in n

99 The determination of bisphenol A (BPA) in foods and beverages sold in Turkish

100 orted the occurrence of compounds other than bisphenol A (BPA) in sediment.

101 s increasingly been used as a substitute for bisphenol A (BPA) in some "BPA-free" consumer goods and

102 ectrochemical biosensor for the detection of Bisphenol A (BPA) in water has been developed by immobil

103 y bottles, little attention has been paid to bisphenol A (BPA) intake from packaged water consumption

104 Bisphenol A (BPA) is a base chemical used extensively in

105 Bisphenol A (BPA) is a common chemical used in the manuf

106 Bisphenol A (BPA) is a high production volume chemical u

107 Bisphenol A (BPA) is a high volume chemical used in a wi

108 Bisphenol A (BPA) is a high-production-volume chemical a

109 Bisphenol A (BPA) is a highly prevalent constituent of p

110 Bisphenol A (BPA) is a synthetic chemical widely used in

111 Bisphenol A (BPA) is a ubiquitous compound that is emerg

112 Bisphenol A (BPA) is a well-known endocrine disruptor th

113 Bisphenol A (BPA) is a widely used chemical that has bee

114 Bisphenol A (BPA) is an endocrine disrupting compound th

115 Bisphenol A (BPA) is an endocrine disruptor and potentia

116 Bisphenol A (BPA) is an endocrine disruptor frequently d

117 Bisphenol A (BPA) is an endocrine-disrupting compound wi

118 Bisphenol A (BPA) is an estrogenic endocrine disruptor w

119 Bisphenol A (BPA) is an industrial compound and a well k

120 The plastic additive bisphenol A (BPA) is commonly found in landfill leachate

121 Bisphenol A (BPA) is found in polycarbonate plastic and

122 ure to the environmental endocrine disruptor bisphenol A (BPA) is ubiquitous and associated with the

123 Bisphenol A (BPA) is used widely to manufacture food con

124 Bisphenol A (BPA) is widely used in epoxy resins lining

125 The endocrine-disrupting chemical bisphenol A (BPA) is widely used in food and beverage pa

126 Bisphenol A (BPA) is widely used in the manufacture of p

127 Bisphenol A (BPA) is widely used in the manufacture of p

128 products from store shelves after reports of bisphenol A (BPA) leaching from baby bottles, reusable d

129 oprobe is designed for the quantification of bisphenol A (BPA) levels in the blood after human exposu

130 rly-life exposure to the endocrine disruptor bisphenol A (BPA) may contribute to the development of o

131 tory actions and public concerns, the use of bisphenol A (BPA) may decrease, while the use of BPA alt

132 Early-life exposure to bisphenol A (BPA) may increase childhood obesity risk, b

133 In this report, the calcium switch and the bisphenol A (BPA) models were used to induce junction re

134 1.0 microM) and a response time of 250 s for bisphenol A (BPA) monitoring.

135 Effects of bisphenol A (BPA) on ovarian transcript profiles as well

136 Certain phthalates and bisphenol A (BPA) show reproductive effects in animal st

137 Humans are exposed to low-dose bisphenol A (BPA) through plastic consumer products and

138 ion on the homogeneous Fenton degradation of bisphenol A (BPA) used as a model pollutant, was investi

139 The xenoestrogen bisphenol A (BPA) used in the manufacturing of various p

140 However, bisphenol A (BPA) was identified by chemical:gene covari

141 The electrochemistry of bisphenol A (BPA) was studied by voltammetric methods at

142 scharge of the endocrine disrupting compound bisphenol A (BPA) with wastewater treatment plant (WWTP)

143 sed on the MS results these were assigned to bisphenol A (BPA), 2,4-di-tert-butylphenol, and a possib

144 Bisphenol A (BPA), a chemical incorporated into plastics

145 Studies have implicated exposure to bisphenol A (BPA), a commonly used chemical, in the deve

146 t chronic exposure of synthetic xenoestrogen bisphenol A (BPA), a component of consumer plastic produ

147 Bisphenol A (BPA), a component of some dialysis membrane

148 Bisphenol A (BPA), a high volume production chemical com

149 In utero or neonatal exposure to bisphenol A (BPA), a high-production-volume chemical use

150 Bisphenol A (BPA), a manufactured chemical, is found in

151 Bisphenol A (BPA), a plastics component, has been descri

152 Bisphenol A (BPA), a prevalent endocrine-disrupting chem

153 umans are routinely exposed to low levels of bisphenol A (BPA), a synthetic xenoestrogen widely used

154 Bisphenol A (BPA), a ubiquitous endocrine disruptor that

155 al models suggests that prenatal exposure to bisphenol A (BPA), a ubiquitous endocrine-disrupting che

156 Bisphenol A (BPA), a widely used endocrine-disrupting ch

157 Exposure of humans to bisphenol A (BPA), a widely used industrial chemical, is

158 S (BPS) are increasingly used to substitute bisphenol A (BPA), a widespread environmental endocrine

159 Bisphenol A (BPA), a xenoestrogen, is a common component

160 Bisphenol A (BPA), a xenoestrogenic endocrine-disrupting

161 shown previously that a known xenoestrogen, bisphenol A (BPA), activates a tumor-derived AR mutant (

162 perimental laboratory evidence suggests that bisphenol A (BPA), an endocrine disruptor, is a neurodev

163 Bisphenol A (BPA), an endocrine-disrupting chemical that

164 Bisphenol A (BPA), an endocrine-disrupting compound with

165 Bisphenol A (BPA), an environmental contaminant with wea

166 to investigate the carcinogenic property of Bisphenol A (BPA), an environmental estrogen, by long-te

167 tions have been reported between phthalates, bisphenol A (BPA), and child behavior.

168 Phthalate esters (PEs), bisphenol A (BPA), and parabens (PBs), which are used in

169 crine-disrupting chemicals (EDCs), including bisphenol A (BPA), are environmental ubiquitous pollutan

170 We examined the association of bisphenol A (BPA), benzophenone-3 (BP-3), triclosan (TCS

171 phenoxyethanol (2-PE), 4-nonylphenol (4-NP), bisphenol A (BPA), benzylbuthyl phthalate (BBP) and dime

172 al samples were analyzed for the presence of bisphenol A (BPA), bis (2-ethylhexyl) phthalate (DEHP),

173 like activity is attributed to EDCs, such as bisphenol A (BPA), bisphenol AF (BPAF), and zearalenone

174 ion of bisphenol F diglycidyl ether (BFDGE), bisphenol A (BPA), bisphenol B (BPB), bisphenol F (BPF)

175 Three selected chemicals, bisphenol A (BPA), diethylhexyl phthalate (DEHP) and min

176 ironment of some of these chemicals, such as bisphenol A (BPA), external contamination during handlin

177 One such exposure, bisphenol A (BPA), has been associated with obesity and

178 ate copolymers prepared from the reaction of bisphenol A (BPA), hydroquinone (HQ), and resorcinol (RS

179 e effect of one suspicious food contaminant, bisphenol A (BPA), in vivo.

180 of environmental contaminants, particularly bisphenol A (BPA), is understudied with regard to PTB.

181 docrine disrupting compounds (EDCs), such as bisphenol A (BPA), may cause adverse health effects in w

182 vestigated the direct effects of exposure to bisphenol A (BPA), mono-n-butyl phthalate (Pht), and pol

183 With the pressure to ban or limit the use of bisphenol A (BPA), production of alternatives such as bi

184 hlorophenol (25-DCP), benzophenone-3 (BP-3), bisphenol A (BPA), triclosan (TCS), butyl paraben (B-PB)

185 Analytes included parabens, phthalates, bisphenol A (BPA), triclosan, ethanolamines, alkylphenol

186 s of a single endocrine disrupting compound, bisphenol A (BPA), was developed.

187 the presence of the ubiquitous xenoestrogen, bisphenol A (BPA), we used nonmalignant random periareol

188 e electrochemical biosensor for detection of bisphenol A (BPA).

189 oplasia from early-life exposure to low-dose bisphenol A (BPA).

190 te (ICC: 0.55), whereas the ICC was 0.39 for bisphenol A (BPA).

191 of our animals to an environmental source of bisphenol A (BPA).

192 for label-free and single step detection of Bisphenol A (BPA).

193 sh register receipts, contain high levels of bisphenol A (BPA).

194 like estrogen, the well-studied xenoestrogen bisphenol A (BPA, a plastics monomer), induces strong br

195 A-seq in endometrial cancer cells exposed to bisphenol A (BPA; found in plastics), genistein (GEN; fo

196 ndocrine disrupting chemicals (EDCs) such as bisphenol-A (BPA) and diethylstilbestrol (DES).

197 health hazards related to persisting use of bisphenol-A (BPA) are well documented.

198 In contrast, Bisphenol-A (BPA) elicited a non-monotonic response.

199 The endocrine disruptor Bisphenol-A (BPA) has been shown to modulate estrogenic,

200 Bisphenol-A (BPA) is a nonsteroidal estrogen that is ubi

201 Bisphenol-A (BPA) is an endocrine disrupting chemical us

202 Bisphenol-A (BPA) is an environmentally ubiquitous estro

203 Bisphenol-A (BPA) is widely used in the manufacture of p

204 Exposure to Bisphenol-A (BPA), an endocrine disruptor used in plasti

205 lastic derived endocrine disruptor compounds bisphenol-A (BPA), bis(2-ethylhexyl)phthalate (DEHP) and

206 ny endocrine disrupting chemicals, including bisphenol-A (BPA), induce non-monotonic dose response (N

207 pared with synthetic estrogens, for example, bisphenol A (BPhA), nonylphenol (NPh), and diethylstilbe

208 s had overall false positive rates of </=5%, bisphenol A, caffeine, NP, OP, and triclosan had false p

209 ally active environmental estrogens, such as bisphenol A, can promote human diseases, including prost

210 tion of phenolic compounds including phenol, bisphenol A, catechol and cresols is reported.

211 ight organics such as cisplatin, daunomycin, bisphenol A, chlorinated phenols, and ethidium bromide.

212 fication and temporal variability in urinary bisphenol A concentrations among couples in Utah-the HOP

213 e patterns were observed on spin-coated poly(bisphenol A decane ether) (BA-C10) films prepared with c

214 e were no measured chlorinated byproducts of bisphenol A despite occasionally high concentrations of

215 We present here a proof-of-concept for bisphenol A detection; the device could be readily adapt

216 epoxy resin based on the diglycidyl ether of bisphenol A (DGEBA) was used as a reference material thr

217 Despite reports of the occurrence of bisphenol A diglycidyl ether (BADGE) and its derivatives

218 Epoxy resins incorporating bisphenol A diglycidyl ether (BADGE) and novolac glycidy

219 p-Hydroxybenzoic acid esters (parabens) and bisphenol A diglycidyl ether (BADGE) are widely present

220 A), bisphenol B (BPB), bisphenol F (BPF) and bisphenol A diglycidyl ether (BADGE) down to 0.50ng/mL;

221 Bisphenol A diglycidyl ether (BADGE) is a ligand for PPA

222 ferent bisphenol A (BPA) derivatives such as bisphenol A diglycidyl ether (BADGE), bisphenol F diglyc

223 Bisphenol A (BPA) and bisphenol A diglycidyl ether (BADGE), used in manufactur

224 Bisphenol A diglycidyl ether (BADGE)- and bisphenol F di

225 ly lacquered with epoxy resins (ER) based on bisphenol A diglycidyl ether (BADGE).

226 PPARgamma antagonists (bisphenol A diglycidyl ether or GW9662) blunted the effe

227 Bisphenol A diglycidyl ether, a compound that binds to P

228 liferator-activated receptor-gamma inhibitor bisphenol A diglycidyl ether, which inhibits adipogenesi

229 nished by a PPAR gamma-selective antagonist, bisphenol A diglycidyl ether.

230 ied in a simplified overlayer model in which bisphenol A diglycidyl methacrylate (BisGMA) was covalen

231 Abbreviations: BisGMA, bisphenol A diglycidyl methacrylate; HPLC, high-performa

232 Fillers were mixed (70 wt%) with 4 bisphenol-A diglycidyl-ether-dimethacrylate (bis-GMA):tr

233 tor-activated receptor (PPAR)gamma inhibitor bisphenol-A-diglycidyl ether.

234 th were randomized to amalgam or composites (bisphenol-A-diglycidyl-dimethacrylate composite for perm

235 cerol dimethacrylate (PMGDM) and ethoxylated bisphenol A dimethacrylate (EBPADMA) at mass ratio of 1:

236 xanediol dimethacrylate (HDDMA), ethoxylated bisphenol A dimethacrylate (EBPADMA), and urethane dimet

237 lectivity towards closely related compounds (bisphenol A dimethacrylate, and dibutyl phthalate).

238 aining different compositions of ethoxylated bisphenol-A dimethacrylate (EBPADMA), 1,6-hexanediol dim

239 lutants) and nonpersistent (some phthalates, bisphenol A) environmental pollutants, and exposure to a

240 d respiratory health, with ethyl-paraben and bisphenol A exhibiting some consistency across respirato

241 ne the multigenerational effects of maternal bisphenol A exposure on mouse pancreatic islets.

242 at this site following neonatal estradiol or bisphenol A exposure resulted in continued, elevated PDE

243 trogen response after in utero xenoestrogen (bisphenol-A) exposure.

244 gel-filled, and water-filled (most labeled "bisphenol A-free"), were collected from the U.S. market

245 d disinfection byproducts of salicylic acid, bisphenol A, gemfibrozil, naproxen, diclofenac, technica

246 which factor, the dimethacrylate component, bisphenol A glycerolate dimethacrylate (BisGMA) or photo

247 2-hydroxyethyl methacrylate (HEMA) and 5% of bisphenol A glycidyl dimethacrylate (BisGMA) (PEHB).

248 rom bisphenol F diglycidyl ether (BFDGE) and Bisphenol A glycidyl methacrylate (BISGMA) after their e

249 The monomer mixture consisted of bisphenol A glycidyl methacrylate (BisGMA), hexanediol d

250 als composed of a trimethacrylate monomer or bisphenol A glycidyl methacrylate, which is a monomer st

251 Bisphenol A has been reported to be a ubiquitous contami

252 h should assess whether in utero exposure to bisphenol A has similar adverse effects.

253 for rapid detection of estrogenic substance (bisphenol A) has been proposed.

254 ens, including the environmental contaminant bisphenol A, have also been linked to reproductive probl

255 The change in the amount of bisphenol A in all of these food, based on expiration da

256 compounds 2,4-dichlorophenol, triclosan, and bisphenol A in chlorinated waters containing I(-).

257 Gestational bisphenol-A in male mice primed macrophages in adulthood

258 that gestational, but not adult, exposure to bisphenol-A increased the development of experimental au

259 see that in these kind of food the amount of bisphenol A increases with an increase in the amount of

260 Finally, we observe that bisphenol A induces rapid and parallel modulation of 5S

261 visualization of dose-dependent induction of Bisphenol A inducible genes showed a weak gene activatio

262 Bisphenol A is widely used in food and drinks packaging.

263 Bisphenol A led to a wide range of stress responses, and

264 Early life exposure to bisphenol-A may represent an environmental consideration

265 factor blocked susceptibility to disease in bisphenol-A mice.

266 method was applied for the determination of bisphenol A migrated from some polycarbonate plastic pro

267 These observations were confirmed using the bisphenol A model in which the knockdown of Cx43 by RNAi

268 eral degradation products that contained the bisphenol A moiety from the overlayer into distilled wat

269 adation products, since they all contain the bisphenol A moiety.

270 Exposure to the estrogenic chemical bisphenol A occurs through food and beverages because of

271 The binding of bisphenol A onto the polymer film was successfully chara

272 ts to low, environmentally relevant doses of bisphenol A or estradiol increases prostate gland suscep

273 ct on pbPPARG, while hexabromocyclododecane, bisphenol A, oxychlordane, and endosulfan were weak anta

274 n catalysts used in synthesis of an aromatic bisphenol A polycarbonate.

275 The trends between bisphenol A pregnancy level and increased asthma and bro

276 ale mouse fetuses, both ethinylestradiol and bisphenol A produced an increase in the number and size

277 ized arylsulfonic acid/thiol catalyst in the bisphenol A reaction but exhibits greater activity and s

278 The absence of bisphenol A release from the overlayer reduces concerns

279 , the UV filter benzophenone-2 (BP2) and the bisphenol A substitute bisphenol S (BPS) was deciphered

280 w and comparison of the hormonal activity of bisphenol A substitutes.

281 ed for phthalates and to a lesser extent for bisphenol A, synthetic musks, some pesticides, and PAHs.

282 n estrogen receptor alpha (hERalpha), called bisphenol A-targeted receptor (BPA-R).

283 intramolecular glycosylations using a rigid bisphenol A template supplemented with linkers of variou

284 ith FEV1% (beta=-0.59; 95% CI: -1.24, 0.05); bisphenol A tended to be associated with increased rates

285 f other xenoestrogens (e.g., alkylphenols or bisphenol A) that are sometimes found in surface water.

286 free chlorine alone, while for triclosan and bisphenol A the free iodine mediated transformations wer

287 actone, genistein, daidzein, benzophenone-3, bisphenol A, the sum of parabens (methyl, ethyl, and pro

288 rders of magnitude from a low of 0.6 ppm for bisphenol A to a high of > 180,000 ppm for methyl parabe

289 n significantly reduce the teratogenicity of bisphenol A, triclosan and 17alpha-ethinyl estradiol wit

290 pathways have been relatively well studied, bisphenol A, triclosan, and ibuprofen.

291 Other exposures (total parabens, bisphenol A, triclosan, benzophenone-3, total phthalates

292 oducts, namely methylparaben, propylparaben, bisphenol A, triclosan, galaxolide, and 4- methylbenzili

293 utrition Examination Survey in which urinary bisphenol A; triclosan; benzophenone-3; propyl, methyl,

294 dence of associations between raised urinary bisphenol A (uBPA) and increased incidence of reported c

295 at levels ranging from 0.6 to 4.6ngg(-1) and bisphenol A was detected in all the samples at concentra

296 Bisphenol-A was judged by the Food and Drug Administrati

297 SPs of the xenoestrogens (e.g., genistein or bisphenol A) were clearly different from the TSP of 17be

298 y diarylpropionitrile, genistein, equol, and bisphenol A, whereas its coactivation at the AP-1 site i

299 mmonly targeted EDC and their isobars (e.g., bisphenol A, (Z)- and (E)-diethylstilbestrol, hexestrol,