ライフサイエンスコーパス: bladder capacity

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1 e in micturition frequency and a decrease in bladder capacity.

2 0.1-50 mg kg(-1), i.v.) which also increased bladder capacity.

3 of vesicoureteral reflux, and posttransplant bladder capacity.

4 in the rat urothelium in vivo and increased bladder capacity.

5 isplayed bladder hyperactivity and decreased bladder capacity.

6 sets: nocturnal polyuria, storage or reduced bladder capacity, 24-h polyuria, and sleep-associated no

7 a distinct bladder volume threshold (74% of bladder capacity) above which flow-evoked bladder contra

8 One patient with decreased bladder capacity and increased PVR had a stroke and was

9 ected rats also showed significantly reduced bladder capacity and postvoid residual volume than diabe

10 bladders of SK3T/T had significantly greater bladder capacity, and urine output exceeded the infused

11 lter filling pressure, threshold pressure or bladder capacity, but micturition pressure was elevated

12 by decreased voiding frequency and increased bladder capacity, but normal bladder pressures.

13 1), i.v.) significantly (P < 0.05) increased bladder capacity during saline distension but not during

14 tivity and significantly (P <0.05) increased bladder capacity during slow infusion of saline or 0.25%

15 hable from that in age-matched controls, but bladder capacity in old animals was only approximately 5

16 At 26 weeks after subtotal cystectomy, bladder capacity in young animals was indistinguishable

17 ior cohort of four consecutive patients with bladder capacities < or =30 ml showed that three of four

18 e, girls and patients with reflux had larger bladder capacities (P < .01).

19 s is yet unknown, but may involve changes in bladder capacity rather than simple fluid management.

20 One patient with reflux and decreased bladder capacity refused treatment.

21 tivity and significantly (P < 0.001) reduced bladder capacity to 14.9 +/- 10.3% of the saline control

22 usion PNS significantly (P < 0.05) increased bladder capacity to 167.7 +/- 27.1% at 1T and 196.0 +/-

23 der overactivity and significantly increased bladder capacity to 68.0 +/- 31.3% at 1T (P < 0.05) and

24 Four patients with decreased bladder capacity underwent hydrodistention.

25 transplant bladder and 1 year posttransplant bladder capacity was 14.5% and 84% of expected, respecti

26 nnel antagonist Brilliant Blue FCF increased bladder capacity, whereas i.v. administration did not.

27 Clozapine increased the bladder capacity while reducing the micturition volume t

28 to increased voiding frequency and decreased bladder capacity with cystitis.

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