ライフサイエンスコーパス: bladder dysfunction

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1 inary bladder and exhibit varying degrees of bladder dysfunction.

2 multiple sclerosis, spinal cord injury, and bladder dysfunction.

3 procedure for pediatric patients with severe bladder dysfunction.

4 n urinary bladder neurotrophic factors after bladder dysfunction.

5 in patients with multiple system atrophy and bladder dysfunction.

6 or vesicoureteral reflux grade and bowel and bladder dysfunction.

7 h ketamine + COX-2 inhibitor prevented these bladder dysfunctions.

8 ked immunosorbent assay and displayed marked bladder dysfunction 12 weeks after STZ injection.

9 These changes may contribute to urinary bladder dysfunction after SCI.

10 nent in the pathogenesis of diabetes-induced bladder dysfunction, although the molecular mechanisms h

11 instay of surgical treatment for neuropathic bladder dysfunction and detrusor instability.

12 spinal cord level, BDNF is a key mediator of bladder dysfunction and pain during cystitis, it is pres

13 mmatory mediator provides novel insight into bladder dysfunction and supports new avenues for therape

14 e study on placebo or antibiotics, bowel and bladder dysfunction, and renal scarring.

15 cations, plane of surgery, 30-day mortality, bladder dysfunction, and sexual dysfunction, none showed

16 the precise mechanisms of TGF-beta mediating bladder dysfunction are not yet known.

17 icles that have been published on neurogenic bladder dysfunction as well as on posterior urethral val

18 disease characterized by congenital urinary bladder dysfunction, associated with a significant risk

19 Subjects had to have urodynamic evidence of bladder dysfunction, be ambulatory, and not have dementi

20 Bladder dysfunction characterized by abnormal bladder sm

21 ly impaired locomotor function and prolonged bladder dysfunction compared with wild-type (WT) litterm

22 te deficits, consisting of paresis and bowel/bladder dysfunction, completely recovered neurologic fun

23 Diabetic bladder dysfunction (DBD) is a common urological complic

24 Diabetic bladder dysfunction (DBD) is common and affects 80% of d

25 as a target for new therapy for BPH-induced bladder dysfunction in aging men.

26 This review covers bladder dysfunction in children.

27 activated K2P channels may underlie myogenic bladder dysfunction in humans.

28 loop for inflammatory responses involved in bladder dysfunction in MS.

29 of candidates for intervention, but ongoing bladder dysfunction in patients after valve ablation rem

30 ond to nicotine despite the absence of major bladder dysfunction in vivo.

31 on which characterizes many types of urinary bladder dysfunctions including urinary incontinence.

32 n Continence Society discussed the issues of bladder dysfunction, including enuresis, and this review

33 Bladder dysfunction is common in Multiple Sclerosis (MS)

34 Here, we demonstrate that a prevalent bladder dysfunction, produced by partial obstruction in

35 Neuropathic pain and bladder dysfunction represent significant quality-of-lif

36 ced cardiovascular fitness, ataxia, fatigue, bladder dysfunction, spasticity, pain, cognitive deficit

37 turition center may contribute to the severe bladder dysfunction that characterizes multiple system a

38 Because of bladder dysfunction, there were 8 infectious events of t

39 These bladder dysfunctions were accompanied by increases in th

40 ere disorders with endpoints of irreversible bladder dysfunction with vesicoureteral reflux, urinary

41 gulation of Rho A signaling and reversed the bladder dysfunction, without affecting hyperglycemia.

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