ライフサイエンスコーパス: blastocyst stage

1 ks such as H3K27me3 is achieved by the early blastocyst stage.

2 ODC did not block normal development to the blastocyst stage.

3 e embryos lacking Eomesodermin arrest at the blastocyst stage.

4 sult induces a high rate of apoptosis at the blastocyst stage.

5 o development and reduces progression to the blastocyst stage.

6 nd are expressed early in development at the blastocyst stage.

7 mplantation development was normal until the blastocyst stage.

8 sis of ZO-1alpha+ was only apparent from the blastocyst stage.

9 tage and was zonular just prior to the early blastocyst stage.

10 h isoforms to be phosphorylated at the early blastocyst stage.

11 tus could remain coupled ionically up to the blastocyst stage.

12 tivate both X chromosomes and die before the blastocyst stage.

13 on and in vitro embryonic development to the blastocyst stage.

14 but only if the host embryos are at the pre-blastocyst stage.

15 NANOG and reduced expression of CDX2 at the blastocyst stage.

16 ylase inhibitors, promote development to the blastocyst stage.

17 the ability to regenerate TE up to the early blastocyst stage.

18 es to be activated from the four-cell to the blastocyst stage.

19 tic null embryos that develop until the late blastocyst stage.

20 ge, and becomes spatially restricted by late blastocyst stage.

21 ts in defects in TE specification before the blastocyst stage.

22 3.3 KD embryos and allows development to the blastocyst stage.

23 os fail to correctly specify lineages at the blastocyst stage.

24 ergenic and become hypermethylated after the blastocyst stage.

25 y passive loss that reaches a minimum at the blastocyst stage.

26 o arrest of embryonic development around the blastocyst stage.

27 e on the expression of specific miRNA at the blastocyst stage.

28 the resultant triploid cells develop to the blastocyst stage.

29 n living mouse oocytes and embryos up to the blastocyst stage.

30 mics of all ICM cells from the early to late blastocyst stage.

31 dependent in the majority of spindles by the blastocyst stage.

32 neration of a stable outer epithelium by the blastocyst stage.

33 ivisions of all cells from the 2- to 32-cell blastocyst stage.

34 loped slower and largely failed to reach the blastocyst stage.

35 cell adhesiveness and a rounded shape at the blastocyst stage.

36 tage embryos were thawed and cultured to the blastocyst stage.

37 en it increases, becoming predominant at the blastocyst stage.

38 early human embryonic development beyond the blastocyst stage.

39 al and functional changes from the morula to blastocyst stage.

40 d the percentage of embryos that reached the blastocyst stage.

41 mplantation development and rarely reach the blastocyst stage.

42 an be traced back to polarity present at the blastocyst stage.

43 ccurs during a 4- to 8-h period at the early blastocyst stage.

44 s at the four-cell, eight-cell, 16-cell, and blastocyst stages.

45 yos but restored again in compact morula and blastocyst stages.

46 onic diapause) occurs when the embryo at the blastocyst stage achieves a state of suspended animation

47 rapidly elevated in embryos at the 4-cell to blastocyst stages after exposure to 10 nM calcitonin.

48 er, these uniparental embryos develop to the blastocyst stage, allowing the derivation of ES cell lin

49 , genic silencing initiates at the morula-to-blastocyst stage and absolutely requires Xist.

50 f developmental pluripotency, develop to the blastocyst stage and also die after implantation, becaus

51 normal, Klf5 mutant embryos arrested at the blastocyst stage and failed to hatch due to defective TE

52 ) were compared between embryos reaching the blastocyst stage and growth-arrested embryos (degenerate

53 d that zygotic Eset expression begins at the blastocyst stage and is ubiquitous during postimplantati

54 ects at 3.5 d.p.c. (days post-coitus) morula/blastocyst stage and lethality before 8.5 d.p.c.

55 die early in development, subsequent to the blastocyst stage and prior to 7.5 days p.c.

56 expressed in pre-implantation embryos at the blastocyst stage and show that NAT1 is also expressed in

57 he oocyte, these aneuploidies persist at the blastocyst stage and the reasons for the high incidence

58 ted in the oocyte compared to 1-cell through blastocyst stages and may reflect the oocyte's response

59 ted oocytes developed in vitro to the morula/blastocyst stage, and 8% of these embryos developed to l

60 stage, reached a maximal level at the early blastocyst stage, and decreased subsequent to blastocyst

61 d not reduce developmental efficiency to the blastocyst stage, and genome integrity was maintained pr

62 mpetent to become fertilized, advance to the blastocyst stage, and give rise to live young than their

63 VIgmyc allele is unmethylated at the day-3.5 blastocyst stage, and the final, adult methylation patte

64 of monkey (Macaca fascicularis) eggs to the blastocyst stage, and their use to create a pluripotent

65 cell lines, whereas those that have achieved blastocyst stage are a robust source of normal hES cells

66 curs first in extraembryonic lineages at the blastocyst stage around the time of implantation before

67 ence of pyruvate and develop normally to the blastocyst stage as well as produce normal viable offspr

68 er insemination decreased development to the blastocyst stage at day 7 and reduced numbers of trophec

69 nt of mouse embryos from the morula to early blastocyst stage, based on 4D confocal image volumes.

70 yos failed to proceed from the morula to the blastocyst stage because of defects in the molecular arc

71 the hypothesis that increased apoptosis at a blastocyst stage because of maternal hyperglycemia may r

72 At a blastocyst stage, both PI3K and Akt exhibited an apical

73 sfer from somatic cell nuclei develop to the blastocyst stage but die after implantation.

74 rt normal preimplantation development to the blastocyst stage but failed to produce embryonic stem (E

75 at the morula stage; BHMT is abundant at the blastocyst stage but not other preimplantation stages, a

76 embryonic cells that becomes apparent at the blastocyst stage, but it is not known if the heterozygou

77 s embryos lacking Lias appear healthy at the blastocyst stage, but their development is retarded glob

78 , we find that success in progression to the blastocyst stage can be predicted with >93% sensitivity

79 Continued YY1 KD from oogenesis to the blastocyst stage caused further loss in DNA methylation

80 is increased in 16-cell embryos, and by the blastocyst stage cells fail to properly adopt a TE gene

81 the development of eight-cell embryos to the blastocyst stage compared to controls (no added immunogl

82 Interestingly, as embryos reach blastocyst stage, Dishevelled 2 becomes increasingly ass

83 ction between the trophectoderm cells of the blastocyst stage embryo and the endometrial cells of the

84 ithin the inner cell mass (ICM) of the mouse blastocyst stage embryo, are segregated into adjacent ti

85 of XEN cells, which are isolated from mouse blastocyst stage embryos and represent the PrE lineage.

86 t human embryonic stem cells and cleavage to blastocyst stage embryos, also identifies the adult mese

87 of embryonic and extra-embryonic lineages in blastocyst stage embryos, the formation of the three ger

88 elf-renewal of the pluripotent stem cells of blastocyst stage embryos.

89 Embryonic stem (ES) cells are derived from blastocyst-stage embryos and are thought to be functiona

90 ytes and single four-cell-, seven-cell-, and blastocyst-stage embryos are described.

91 embryonic stem cells (ESCs) are derived from blastocyst-stage embryos but have very different biologi

92 include the establishment of XEN cells from blastocyst-stage embryos in either standard embryonic or

93 toderm and primitive endoderm cells of human blastocyst-stage embryos.

94 med on three 5- to 10-cell biopsies from two blastocyst-stage embryos.

95 ges, prior to functional inactivation at the blastocyst stage, exclusively from X(P) in female embryo

96 d in embryos that are chronologically at the blastocyst stage following culture of 8-cell embryos in

97 Interestingly, at the blastocyst stage, Gata3 mRNA is selectively up-regulated

98 one significant nuclear reprogramming by the blastocyst stage; however, problems may occur during red

99 bly the second, persists intact to the early blastocyst stage in nearly two-thirds of mouse conceptus

100 latory gene as early as the pre-implantation blastocyst stage in the mouse.

101 x, a Bcl-2-like protein, is increased at the blastocyst stage in the presence of high concentrations

102 d in decreased development of embryos to the blastocyst stage in vitro and a reduction in inner cell

103 e development of the mouse embryo beyond the blastocyst stage in vitro.

104 is compatible with onward development to the blastocyst stage in vitro.

105 Embryo culture from two-cell to blastocyst stages in T6 medium either with or without a

106 nockout mice embryos are not viable past the blastocyst stage, indicating an essential role of Vav in

107 The mouse embryo at the blastocyst stage is differentiated into two cell types,

108 opment of zygotes of domestic species to the blastocyst stage is facilitated by culture in groups, su

109 tion of Cdx2 develop normally until the late blastocyst stage leading to the conclusion that Cdx2 is

110 We show that inhibition of aPKC from the mid blastocyst stage not only prevents sorting of PrE precur

111 a single-celled fertilized egg, through the blastocyst stage of a ball of cells and the embryonic st

112 evoid of Ron (Ron-/-) are viable through the blastocyst stage of development but fail to survive past

113 immature oocyte, mature egg, and through the blastocyst stage of embryonic development, where express

114 kt are expressed from the 1-cell through the blastocyst stage of murine preimplantation embryo develo

115 At the blastocyst stage of pre-implantation mouse development,

116 By the blastocyst stage only 5% of the transcripts are generate

117 expressed in the early mouse embryo from the blastocyst stage onwards and mediate Foxh1-dependent act

118 oid cells is progressively depleted from the blastocyst stage onwards.

119 By the late blastocyst stage, PECAM-1 expression is restricted to th

120 ch synchronized embryonic development to the blastocyst stage, preparation of the uterus for the rece

121 and fertilized in vitro can progress to the blastocyst stage, the developmental potential of blastoc

122 At the late blastocyst stage, the epithelial trophectoderm cells of

123 become committed to the trophectoderm at the blastocyst stage through Cdx2 activity, but here we show

124 on of trophoblast development from the early blastocyst stage through the onset of implantation appea

125 were able to progress from the morula/early blastocyst stage to more advanced stages of development.

126 opment of these fertilized oocytes up to the blastocyst stage was also similar to that registered in

127 The development of individual zygotes to the blastocyst stage was optimal when they were cultured 81-

128 ar rates and incidence of development to the blastocyst stage were observed.

129 Mouse embryos arrest at the late blastocyst stage when implantation is prevented.

130 Src-family PTK activity continued until the blastocyst stage when strong cortical activity became ev

131 ic lethality, null embryos survive until the blastocyst stage, which may be explained by the presence

132 l fate decision in development occurs at the blastocyst stage with establishment of the trophoblast a

133 This promotes efficient development to the blastocyst stage with no detectable effect on aneuploidy

134 a-A deficiency causes embryonic death at the blastocyst stage with pronounced cell proliferation fail