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1 ce vaccine immunity against lethal pulmonary blastomycosis.
2 l development against experimental pulmonary blastomycosis.
3 port a potential role for antigen testing in blastomycosis.
4 retained pathogenicity in a murine model of blastomycosis.
5 role of the WI-1 adhesin in pathogenesis of blastomycosis.
6 y transfer protection in murine experimental blastomycosis.
7 mals from death due to progressive pulmonary blastomycosis.
8 s with histoplasmosis and, to lesser extent, blastomycosis.
9 with coccidioidomycosis, histoplasmosis, or blastomycosis.
10 ), 18 of 31 Chagas disease (58%), 7 of 28 SA blastomycosis (25%), and 14 of 83 leprosy sera (17%).
11 In Blastomyces dermatitidis, the agent of blastomycosis, a respiratory and disseminated mycosis of
12 Outdoor activities were not risk factors for blastomycosis among cluster case patients or when compar
13 eated with steroids, leading to disseminated blastomycosis and acute respiratory distress syndrome (A
17 osis, Blastomyces dermatitidis, the agent of blastomycosis, and Histoplasma capsulatum, the agent of
19 present them could modulate immunity during blastomycosis by selection and display of WI-1 peptides.
21 vaccine immunity against lethal experimental blastomycosis, compared with wild-type control mice.
22 tomyces dermatitidis, the etiologic agent of blastomycosis, has 2 genetic groups and shows varied cli
26 dimorphic fungus and the causative agent of blastomycosis, is widely considered an extracellular pat
29 ent mice were more resistant to experimental blastomycosis than were intact littermates, and passive
30 hown in a mouse model of vaccination against blastomycosis that antifungal memory CD8(+) T cells are
31 nvestigated an increase in reported cases of blastomycosis that occurred during 2009-2010 in Marathon
33 from 37 patients with a confirmed history of blastomycosis were tested for a response to WI-1 in prim
34 us diseases (leprosy and South American (SA) blastomycosis), which are prevalent in the same geograph
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