ライフサイエンスコーパス: bleeding on probing

1 probing) and > or =3 mm diseased (featuring bleeding on probing).

2 nical parameters including probing depth and bleeding on probing.

3 tence of sites with probing depths >4 mm and bleeding on probing.

4 , buccal flap thickness, papilla height, and bleeding on probing.

5 e per-patient percentage of tooth sites with bleeding on probing.

6 ts, probing depths, plaque index scores, and bleeding on probing.

7 ng attachment level, gingival recession, and bleeding on probing.

8 probing depth reduction and the reduction in bleeding on probing.

9 ariables measured: PD, attachment level, and bleeding on probing.

10 robing depth was 2 mm (SE 0.02), and 17% had bleeding on probing.

11 ttachment level and proportion of sites with bleeding on probing.

12 as clinical attachment level, recession, and bleeding on probing.

13 robing depth, clinical attachment level, and bleeding on probing.

14 s included gingival index, plaque index, and bleeding on probing.

15 4) the infected sites had higher plaque and bleeding on probing 0.9 +/- 0.3, 73 +/- 12%, respectivel

16 Pill users had more sites with bleeding on probing (44.0% versus 31.1%; P = 0.017).

17 er percentage of sites with PD >/=4.5 mm and bleeding on probing (9.78% versus 12.69%; P = 0.052).

18 iodontal disease to be present in teeth with bleeding on probing and a probing depth >/=3 mm at one o

19 gnificantly associated with increased extent bleeding on probing and levels of microorganisms Porphyr

20 score (peak at induction minus baseline) for bleeding on probing and probing depth (PD), the patients

21 In contrast, in the experimental group, bleeding on probing and probing depths were significantl

22 mplete compliers tended to show reduction in bleeding on probing and reduction in plaque index compar

23 nine patients with 4 pockets > or = 5 mm and bleeding on probing and/or suppuration were randomized i

24 ngival sulcus: < or =3 healthy (featuring no bleeding on probing) and > or =3 mm diseased (featuring

25 ment loss, 13.1% (95% CI, 8.1% to 18.1%) for bleeding on probing, and 0.27 (95% CI, 0.17 to 0.37) for

26 g depth (PD), supragingival plaque, gingival bleeding on probing, and calculus.

27 of periodontal probing depth, plaque score, bleeding on probing, and clinical attachment level (CAL)

28 ere included according to the probing depth, bleeding on probing, and clinical attachment level.

29 ss (AL), the presence of gingival recession, bleeding on probing, and full-mouth radiographic surveys

30 -gingival margin distance (attachment loss), bleeding on probing, and furcation involvement.

31 ng probing depth, clinical attachment level, bleeding on probing, and gingival and plaque indices, we

32 attachment level (CAL), probing depth (PD), bleeding on probing, and gingival index change after tre

33 ded probing depth, clinical attachment loss, bleeding on probing, and gingival index.

34 th, clinical attachment level, plaque index, bleeding on probing, and gingival index.

35 ntly higher mean PD and CAL, more sites with bleeding on probing, and increased levels of CRP compare

36 bing depth, clinical attachment level (CAL), bleeding on probing, and percentage of visible plaque.

37 sessment of probing depth, attachment level, bleeding on probing, and plaque and calculus accumulatio

38 depth (PD), clinical attachment level (CAL), bleeding on probing, and plaque index were measured, and

39 depth (PD), clinical attachment level (CAL), bleeding on probing, and plaque index were selected as o

40 , clinical attachment level, gingival index, bleeding on probing, and plaque index) were recorded at

41 obing depth (PD), clinical attachment level, bleeding on probing, and plaque index.

42 Plaque index, bleeding on probing, and probing depth were recorded at

43 l status was assessed by probing depth (PD), bleeding on probing, and radiographic alveolar bone loss

44 s including probing depth, attachment level, bleeding on probing, and root caries remineralization we

45 s are superior to a single strip in reducing bleeding on probing, and that local delivery of tetracyc

46 baseline in probing depth, attachment level, bleeding on probing, and the Modified Gingival Index (MG

47 ved in both groups for: 1) PIss; 2) PDss; 3) bleeding on probing; and 4) relative CAL.

48 with at least 6 mm LOA; number of sites with bleeding on probing; and deepest probing depth per perso

49 tooth with > or =4 mm loss of attachment and bleeding on probing as an indicator of inflammation), or

50 robing depth, clinical attachment level, and bleeding on probing, as well as crevicular levels of int

51 attachment level, probing depth, plaque, and bleeding on probing at 6 sites per tooth.

52 erdental bleeding index, probing depths, and bleeding on probing at interdental sites and underwent a

53 asuring attachment level, probing depth, and bleeding on probing at monthly examinations at qualifyin

54 logistic regression models, with tooth-level bleeding on probing at sites with attachment loss<or=2 m

55 1) health, all probing depth (PD) <3 mm and bleeding on probing (BOP) <10%; 2) gingivitis, all PD <3

56 : P = 0.002; 6 and 9 months: P = 0.001), and bleeding on probing (BOP) (3 months: P <0.01; 6 months:

57 ment loss (AL), and percentage of sites with bleeding on probing (BOP) and clinical AL >/=5 mm.

58 severe periodontitis, and the combination of bleeding on probing (BOP) and clinical attachment loss (

59 ontal outcomes were percentage of teeth with bleeding on probing (BOP) and combination of BOP and att

60 bular quadrants were evaluated for calculus, bleeding on probing (BOP) and loss of gingival attachmen

61 dontal probing depth (PD) and assessments of bleeding on probing (BOP) and radiographic alveolar bone

62 ng explorer-detectable supragingival plaque, bleeding on probing (BOP) and relative clinical attachme

63 In each patient four pockets > 5 mm with bleeding on probing (BOP) and/or suppuration were studie

64 (PD), clinical attachment levels (CAL), and bleeding on probing (BOP) as well as gingival crevicular

65 t one site with probing depth (PD) >4 mm and bleeding on probing (BOP) at 12 months post-therapy.

66 of sites with a probing depth (PD) >4 mm and bleeding on probing (BOP) at the 3-month reevaluation.

67 ed into two groups based on probing depth or bleeding on probing (BOP) at the site of collection of t

68 rs, including visible plaque index (VPI) and bleeding on probing (BOP) from six sites per tooth.

69 Participants with bleeding on probing (BOP) in <10% of sites were classifi

70 aque (PI), calculus (CI), gingival (GI), and bleeding on probing (BOP) indices were used to assess gi

71 The absence or presence of bleeding on probing (BOP) is a sign of periodontal healt

72 Bleeding on probing (BOP) is widely interpreted as a sig

73 bing pocket depth (PPD), plaque indices, and bleeding on probing (BOP) measured at baseline, intermed

74 r of sites with probing depth (PD) >4 mm and bleeding on probing (BOP) per patient was the primary ou

75 = 0.31 to 0.96) and lower odds of increased bleeding on probing (BOP) percentage values (OR = 0.62,

76 ) reduction, clinical attachment level gain, bleeding on probing (BOP) reduction, radiographic bone f

77 were defined using probing depths (PDs) and bleeding on probing (BOP) scores.

78 clinical attachment level (CAL), and reduced bleeding on probing (BOP) to a greater extent than SRP a

79 CB samples from those patients with adequate bleeding on probing (BOP) were collected on special bloo

80 leeding index (GBI), probing depth (PD), and bleeding on probing (BOP) were measured in implants and

81 gingival index (GI), probing depth (PD), and bleeding on probing (BOP) were measured, and gingival cr

82 h (PD), clinical attachment level (CAL), and bleeding on probing (BOP) were observed both short and l

83 with probing depths (PD) of > or =5 mm with bleeding on probing (BOP) were randomized into the test

84 vel (CAL), modified gingival index (GI), and bleeding on probing (BOP) were recorded at baseline and

85 h (PD), clinical attachment level (CAL), and bleeding on probing (BOP) were recorded.

86 teeth, restorations, probing depth (PD) and bleeding on probing (BOP) were recorded.

87 al attachment level, and percentage of sites bleeding on probing (BOP) were significantly higher in t

88 depth (PD), clinical attachment level (CAL), bleeding on probing (BOP), and gingival index (GI) at ba

89 ingival recession (GR), gingival index (GI), bleeding on probing (BOP), and horizontal and vertical b

90 depth (PD), clinical attachment level (CAL), bleeding on probing (BOP), and plaque index were measure

91 cal attachment level, furcation involvement, bleeding on probing (BOP), and suppuration.

92 nt level (CAL), recession (REC), presence of bleeding on probing (BOP), and supragingival plaque (PL)

93 attachment level (RAL), probing depths (PD), bleeding on probing (BOP), and the full-mouth plaque sco

94 Periodontal probing depth (PD), gingival bleeding on probing (BOP), clinical attachment level (CA

95 depth (PD), clinical attachment level (CAL), bleeding on probing (BOP), gingival index (GI), and peri

96 depth (PD), clinical attachment level (CAL), bleeding on probing (BOP), gingival index (GI), and plaq

97 depth (PD), clinical attachment level (CAL), bleeding on probing (BOP), gingival index (GI), plaque i

98 depth (PD), clinical attachment level (CAL), bleeding on probing (BOP), gingival index (GI), plaque i

99 Clinical periodontal parameters included bleeding on probing (BOP), mean probing depth (PD), and

100 uding probing depth (PD), plaque index (PI), bleeding on probing (BOP), mucosal redness (MR), suppura

101 an plaque indices (PI), probing depths (PD), bleeding on probing (BOP), or relative clinical attachme

102 tes of a novel lateral-flow immunoassay with bleeding on probing (BOP), oral hygiene, and periodontal

103 Plaque index (PI), GI, bleeding on probing (BOP), PD, and attachment gain were

104 were plaque index (PI), gingival index (GI), bleeding on probing (BOP), PD, gingival crevicular fluid

105 , baseline periodontal parameters, including bleeding on probing (BOP), periodontal probing depths (P

106 Positive correlations were found between bleeding on probing (BOP), plaque index (PI) scores, and

107 gingivitis demonstrated significantly higher bleeding on probing (BOP), plaque index (PI), and gingiv

108 ded probing depth, clinical attachment loss, bleeding on probing (BOP), plaque index (PI), and tooth

109 with CAL loss or gain > or =2 mm, changes in bleeding on probing (BOP), plaque index, and mobility.

110 Full-mouth plaque index (PI), bleeding on probing (BOP), probing depth (PD) >/=4 mm, a

111 tal parameters, including plaque index (PI), bleeding on probing (BOP), probing depth (PD) >3 mm, cli

112 Plaque index (PI), bleeding on probing (BOP), probing depth (PD), and attac

113 ta, including approximal plaque index (API), bleeding on probing (BOP), probing depth (PD), and clini

114 ull-mouth periodontal examination, including bleeding on probing (BOP), probing depth (PD), and clini

115 uth periodontal examination with a record of bleeding on probing (BOP), probing depth (PD), and clini

116 Periodontal examinations of bleeding on probing (BOP), probing depth (PD), and clini

117 Demographic and biologic data, bleeding on probing (BOP), probing depth (PD), and clini

118 Plaque index (PI), bleeding on probing (BOP), probing depth (PD), and clini

119 essed included plaque index, gingival index, bleeding on probing (BOP), probing depth (PD), and clini

120 ect: plaque index (PI), gingival index (GI), bleeding on probing (BOP), probing depth (PD), clinical

121 Bleeding on probing (BOP), probing depth (PD), relative

122 ewly diagnosed PCOS had increased sites with bleeding on probing (BOP), probing depth, clinical attac

123 hment level (CAL), gingival recession (REC), bleeding on probing (BOP), suppuration (SUP), and suprag

124 uded CAL, probing depth, gingival recession, bleeding on probing (BOP), visible plaque, supragingival

125 h (PD), clinical attachment level (CAL), and bleeding on probing (BOP), were performed, and subgingiv

126 pth (PD), clinical attachment loss (AL), and bleeding on probing (BOP).

127 grouped based on adjacent probing depth and bleeding on probing (BOP).

128 achment level (CAL), probing depth (PD), and bleeding on probing (BOP).

129 h (PD), clinical attachment level (CAL), and bleeding on probing (BOP).

130 proximal pocket during PMT with a history of bleeding on probing (BOP).

131 e attachment level (RAL), probing depth, and bleeding on probing (BOP).

132 h (PD), clinical attachment level (CAL), and bleeding on probing (BOP).

133 s of > 2 mm, probing depth (PD) >/=5 mm with bleeding on probing (BOP).

134 h (PD), clinical attachment level (CAL), and bleeding on probing (BOP).

135 ites with a probing depth (PD) >/= 5 mm with bleeding on probing (BOP).

136 2 mm, probing pocket depth (PD) >/=5 mm with bleeding on probing (BOP).

137 indices, including: 1) plaque index (PI); 2) bleeding on probing (BOP); 3) probing depth (PD); and 4)

138 ding: 1) plaque index; 2) gingival index; 3) bleeding on probing (BOP); 4) probing depth; and 5) atta

139 with: 1) periodontal probing depth (PD); 2) bleeding on probing (BOP); and 3) attachment loss (AL).

140 < 0.01), gingival index (G]; P < 0.01), and bleeding on probing (BOP; P < 0.05) scores increased ove

141 (PI), gingival index (GI), and percentage of bleeding on probing (%BOP) were observed in placebo-trea

142 recession, mobility, plaque index [PI], and bleeding on probing [BOP]) and defect (vertical and hori

143 th [PD], clinical attachment loss [CAL], and bleeding on probing [BOP]) were performed.

144 th presenting probing depth [PD] >/=5 mm and bleeding on probing [BOP]) were selected and randomly al

145 ual pockets (probing depth [PD] >/=5 mm with bleeding on probing [BOP]).

146 PD], plaque index [PI], gingival index [GI], bleeding on probing [BOP], and clinical attachment level

147 one diseased site (probing depth [PD] >4 mm, bleeding on probing [BOP], and clinical attachment level

148 ve teeth with probing depth [PD] > or =5 mm, bleeding on probing [BOP], and clinical attachment or ra

149 t soft tissue parameters (plaque index [PI], bleeding on probing [BOP], and probing depth [PD] >/=4 m

150 st two diseased interproximal papillae (with bleeding on probing [BOP], probing depth [PD] > or =4 mm

151 Periodontal parameters (plaque index [PI], bleeding on probing [BOP], probing depth [PD], clinical

152 Periodontal parameters (plaque index [PI], bleeding on probing [BOP], probing depth [PD], clinical

153 inflammatory conditions (plaque index [PI], bleeding on probing [BOP], probing depth [PD], marginal

154 depth > 2 mm, filled and decayed teeth, and bleeding on probing by smoking history were not signific

155 laque index, probing depth, attachment loss, bleeding on probing, calculus index, and furcation invol

156 oth loss; dental caries; periodontal status (bleeding on probing, calculus, and attachment loss); and

157 ge in probing depth was the primary outcome, bleeding on probing, clinical attachment level, gingival

158 Probing depth, gingival bleeding on probing, clinical attachment loss (CAL), and

159 0.78 +/- 0.30) had a significant decrease in bleeding on probing compared to baseline.

160 Sites with subgingival calculus and bleeding on probing demonstrated more LCAL and PD, and s

161 Plaque index and bleeding on probing did not change.

162 measurements, including probing depth (PD), bleeding on probing, gingival index, and plaque index (P

163 Probing depth, bleeding on probing, gingival index, and plaque index cl

164 ed probing depth, clinical attachment level, bleeding on probing, gingival index, and plaque index.

165 ing pocket depths, clinical attachment loss, bleeding on probing, gingival index, fasting glucose lev

166 uated were plaque (full-mouth plaque score), bleeding on probing, gingival recession, probing depth (

167 redictor of periodontal disease progression, bleeding on probing has low sensitivity owing to a high

168 days moderately increased the appearance of bleeding on probing in a population that had > or = 20%

169 PTX3 levels correlated with plaque index and bleeding on probing in the CP group (P <0.05).

170 According to gingival and bleeding on probing indices, 84 children were classified

171 ng clinical outcomes were tested: plaque and bleeding on probing indices, pocket probing depth (PD),

172 Clinical parameters, including bleeding on probing, mobility, suppuration, mucosal rece

173 However, comparisons of other parameters (bleeding on probing, modified bleeding index, GI, probin

174 l attachment level (P <0.05), and sites with bleeding on probing (not significant).

175 depth, clinical attachment level (CAL), and bleeding on probing on all teeth except third molars and

176 healthy (no clinical attachment loss [AL] or bleeding on probing) or as having early periodontitis (c

177 bone loss, gingival inflammation measured as bleeding on probing, or a combination of these measures.

178 robing depth, percentage of sites exhibiting bleeding on probing, or plaque and calculus accumulation

179 t transparency, mucosal recession, mobility, bleeding on probing, or suppuration (n = 40) at 48 month

180 ons tended to show a reduction in plaque and bleeding on probing over time.

181 ent level; blood glucose was related only to bleeding on probing (P <0.05).

182 0.006), probing depth 4 to 6 mm (P = 0.016), bleeding on probing (P = 0.001), and radiographic bone l

183 with high probing depth (P = 0.02) and high bleeding on probing (P = 0.008).

184 cases than controls had plaque (P = 0.004), bleeding on probing (P = 0.013), and probing depths of >

185 higher levels of plaque index (P = 0.01) and bleeding on probing (P = 0.03) and higher severity of pe

186 multiple strip group significantly decreased bleeding on probing (P = 0.05) compared to all other tre

187 olar teeth with probing depth > or =5 mm and bleeding on probing participated in this split-mouth tri

188 buted 198 gingival papillae: 158 'diseased' (bleeding-on-probing, PD > 4 mm, and AL >/= 3 mm) and 40

189 mination measuring probing depth, recession, bleeding on probing, plaque index (PI), and GE.

190 odontal attachment loss (AL), probing depth, bleeding on probing, plaque index (PI), and gingival ind

191 th probing depth, clinical attachment level, bleeding on probing, plaque index scores, and keratinize

192 l recession; and 4) percentage of sites with bleeding on probing, plaque, PD >/=5 mm, and CAL >/=3 mm

193 e newly forming peri-implant sulcus depth or bleeding on probing prevalence.

194 h visits, gingival index (GI), plaque index, bleeding on probing, probing depth (PD), and attachment

195 The waist/hip ratio (WHR), plaque index, bleeding on probing, probing depth (PD), and clinical at

196 Recordings of plaque, bleeding on probing, probing depth (PD), and clinical at

197 Recordings of plaque, bleeding on probing, probing depth (PD), and clinical at

198 Plaque and gingival indices, bleeding on probing, probing depth, and clinical attachm

199 ere positively associated with the extent of bleeding on probing, probing depth, and clinical attachm

200 six sites on each premolar included plaque, bleeding on probing, probing depth, and relative attachm

201 evels, periodontal parameters (plaque index, bleeding on probing, probing depth, attachment loss, and

202 e following clinical periodontal parameters: bleeding on probing, probing depth, clinical attachment

203 dontal disease severity was measured through bleeding on probing, probing depth, clinical attachment

204 Periodontal parameters (plaque index, bleeding on probing, probing depth, clinical attachment

205 ing parameters were evaluated: plaque index, bleeding on probing, probing depth, gingival recession,

206 eters measured included plaque accumulation, bleeding on probing, probing depths (PDs), and clinical

207 Recordings of bleeding on probing, probing depths, and clinical attach

208 'diseased' gingival papillae (n = 241; with bleeding-on-probing, probing depth >/= 4 mm, and clinica

209 n available, a 'healthy' papilla (n = 69; no bleeding-on-probing, probing depth </= 4 mm, and clinica

210 Salivary-ProCT levels were correlated with bleeding-on-probing (r = 0.45, p = 0.05), as well as wit

211 eduction (PDR), attachment level gain (ALG), bleeding on probing reduction (BOP), and plaque index we

212 gain was 1.65 mm (95% CI: 1.17-2.13 mm), and bleeding on probing reduction was 45.8% (95% CI: 38.5%-5

213 h reduction, clinical attachment level gain, bleeding on probing reduction, and mucosal recession.

214 graphic, and behavioral risk variables, only bleeding on probing remained significantly associated wi

215 evel, probing depth, plaque, gingivitis, and bleeding on probing scores) and significant decreases in

216 1) and attachment loss (P = 0.006) and fewer bleeding on probing sites (P = 0.001).

217 sulcus depth: 1 to 3 mm (normal), 3 mm with bleeding on probing (slight disease), 3 to 6 mm (moderat

218 Plaque, gingivitis, bleeding on probing, suppuration, probing depth, and cli

219 .002); and for every 10 sites that exhibited bleeding on probing, the clinical attachment gain was 0.

220 tal examination consisting of probing depth, bleeding on probing, tooth mobility, gingival index, and

221 aque index versus no reduction, reduction in bleeding on probing versus no reduction, reduction in th

222 ers measured at six sites per tooth included bleeding on probing, visible plaque, probing depth, and

223 D), clinical attachment level (CAL), plaque, bleeding on probing, visual gingival inflammation, and c

224 Bleeding on probing was a prerequisite for target sites

225 depth, gingival recession, plaque index, and bleeding on probing was performed in 75 Indonesians with

226 1.38 mm (compared to 1.01 mm by SRP alone), bleeding on probing was reduced by 25.2% (compared to 13

227 Bleeding on probing was significantly associated with TG

228 sensitivity and specificity of the test for bleeding on probing were 71.8% and 77.5%, respectively (

229 the clinical parameters of probing depth and bleeding on probing were compared in 15 patients with mo

230 Probing depth (PD) and bleeding on probing were measured at six sites per tooth

231 on cLCAL/cPD, while subgingival calculus and bleeding on probing were negatively associated with cLCA

232 s with 4 periodontal pockets > or = 5 mm and bleeding on probing were randomized into four groups of

233 ce of supragingival plaque accumulation, and bleeding on probing were recorded.

234 obing depth, clinical attachment levels, and bleeding on probing were used to determine the periodont

235 nt level [CAL], and percentage of sites with bleeding on probing) were evaluated.

236 evel [FST], papilla index, plaque index, and bleeding on probing) were measured, and periapical radio

237 ng periodontal probings and an assessment of bleeding on probing, were made using an NIDR probe at 3

238 achment loss were positively associated with bleeding on probing, with stronger associations among me

239 No significant differences were found for bleeding on probing (WMD = 10.77; 95% CI = -3.43 to 24.9