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1 d with peptic ulcer disease and an increased bleeding tendency.
2 bosis, but its inhibition causes only a mild bleeding tendency.
3 ysfunction is a third possible mechanism for bleeding tendency.
4 terozygotes may display a distinct, if mild, bleeding tendency.
5 hould be evaluated carefully for symptoms of bleeding tendency.
6 ructure and function resulting in a variable bleeding tendency.
7 n severely reduced thrombin generation and a bleeding tendency.
8 er traumatic brain injury without increasing bleeding tendencies.
9                       History of hemorrhagic/bleeding tendencies (93%) was the most common reason for
10 e disorder in which oculocutaneous albinism, bleeding tendency and a ceroid-lipofuscin lysosomal stor
11 isorder that is characterized by a life-long bleeding tendency and complications in wound healing.
12 w that mutant mice lacking CD148 exhibited a bleeding tendency and defective arterial thrombosis.
13    Affected patients present with a moderate bleeding tendency and have Factor V and Factor VIII leve
14 nia and lack of natural killer (NK) cells, a bleeding tendency and neurologic abnormalities.
15 fected individuals, including differences in bleeding tendency and VWF quantity, activity, and multim
16 czema, frequency and severity of infections, bleeding tendency, and autoimmunity and reduction in dis
17 er characterized by oculocutaneous albinism, bleeding tendency, and lysosomal ceroid storage disease,
18                       Gingival inflammation, bleeding tendency, and supragingival plaque were clinica
19 leeding time measurements revealed no severe bleeding tendency as a consequence of GP VI deficiency.
20  effect of annexin V was not associated with bleeding tendency, as judged by the amount of blood abso
21 III or LAD-1/variant) present with increased bleeding tendency because of the lack or dysfunction of
22                              Analyses of the bleeding tendency by means of the bleeding score (BS) ha
23 onstitutive JAK2(V617F) expression exhibit a bleeding tendency combined with the accelerated formatio
24 emely challenging in patients with increased bleeding tendency, detection and aggressive treatment of
25                                 Importantly, bleeding tendency did not correlate with plasma VWF leve
26 of factor V which shed light on the variable bleeding tendencies in severe factor V deficiency.
27 ADP receptor gene (P2RY12) contribute to the bleeding tendency in 92 index cases enrolled in the Euro
28 ssociated with a thrombocytopathy inducing a bleeding tendency in the homozygous mutated patients.
29 de clear explanations for the thrombotic and bleeding tendency in these MPDs, suggesting the need for
30 bocytopathy, which likely contributes to the bleeding tendency in vWD-type 2B.
31                                              Bleeding tendency is associated with heterogeneous clini
32                           This suggests that bleeding tendencies may likewise vary among patients wit
33                     GPS is associated with a bleeding tendency, myelofibrosis, and splenomegaly.
34 individuals (n = 97), and between those with bleeding tendency (n = 50) and without (n = 24).
35                       There was no increased bleeding tendency, no neurologic abnormalities, and no i
36 e data suggest that a ketogenic diet-related bleeding tendency occurs in about one third of treated p
37                              The significant bleeding tendencies of individuals homozygous for prothr
38 l assessments (Plaque Index, Gingival Index, Bleeding Tendency, pocket depth, and coronal staining) w
39 causes of morbidity and mortality in FA is a bleeding tendency, resulting from low platelet counts.
40 y had significantly lower Gingival Index and Bleeding Tendency scores at the one-, three-, six-, and
41                      Measurement of gingival bleeding tendency should be an integral part of a compre
42 usually lead to integrin nonexpression and a bleeding tendency termed Glanzmann thrombasthenia.
43 h Factor XI (FXI) deficiency have a variable bleeding tendency that does not correlate with FXI:C lev
44             Patients with vWD-type 2B have a bleeding tendency that is linked to loss of vWF multimer
45                         Apart from increased bleeding tendency, the pathophysiological consequences o
46                          The severity of the bleeding tendency varies among patients and is related t
47 n ITP patients account for this variation in bleeding tendency, we conducted a single-center, cross-s

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