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1 d with peptic ulcer disease and an increased bleeding tendency.
2 bosis, but its inhibition causes only a mild bleeding tendency.
3 ysfunction is a third possible mechanism for bleeding tendency.
4 terozygotes may display a distinct, if mild, bleeding tendency.
5 hould be evaluated carefully for symptoms of bleeding tendency.
6 ructure and function resulting in a variable bleeding tendency.
7 n severely reduced thrombin generation and a bleeding tendency.
8 er traumatic brain injury without increasing bleeding tendencies.
10 e disorder in which oculocutaneous albinism, bleeding tendency and a ceroid-lipofuscin lysosomal stor
11 isorder that is characterized by a life-long bleeding tendency and complications in wound healing.
12 w that mutant mice lacking CD148 exhibited a bleeding tendency and defective arterial thrombosis.
13 Affected patients present with a moderate bleeding tendency and have Factor V and Factor VIII leve
15 fected individuals, including differences in bleeding tendency and VWF quantity, activity, and multim
16 czema, frequency and severity of infections, bleeding tendency, and autoimmunity and reduction in dis
17 er characterized by oculocutaneous albinism, bleeding tendency, and lysosomal ceroid storage disease,
19 leeding time measurements revealed no severe bleeding tendency as a consequence of GP VI deficiency.
20 effect of annexin V was not associated with bleeding tendency, as judged by the amount of blood abso
21 III or LAD-1/variant) present with increased bleeding tendency because of the lack or dysfunction of
23 onstitutive JAK2(V617F) expression exhibit a bleeding tendency combined with the accelerated formatio
24 emely challenging in patients with increased bleeding tendency, detection and aggressive treatment of
27 ADP receptor gene (P2RY12) contribute to the bleeding tendency in 92 index cases enrolled in the Euro
28 ssociated with a thrombocytopathy inducing a bleeding tendency in the homozygous mutated patients.
29 de clear explanations for the thrombotic and bleeding tendency in these MPDs, suggesting the need for
36 e data suggest that a ketogenic diet-related bleeding tendency occurs in about one third of treated p
38 l assessments (Plaque Index, Gingival Index, Bleeding Tendency, pocket depth, and coronal staining) w
39 causes of morbidity and mortality in FA is a bleeding tendency, resulting from low platelet counts.
40 y had significantly lower Gingival Index and Bleeding Tendency scores at the one-, three-, six-, and
43 h Factor XI (FXI) deficiency have a variable bleeding tendency that does not correlate with FXI:C lev
47 n ITP patients account for this variation in bleeding tendency, we conducted a single-center, cross-s
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