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1 had improved, with shrinkage of the enlarged blind spot.
2 o a stimulus presented inside or outside the blind spot.
3  to massive enlargement of the physiological blind spot.
4  degrees to 0.8 degrees from the edge of the blind spot.
5 f primary visual cortex corresponding to the blind spot.
6  in the visual location corresponding to the blind spot and its surround.
7                                              blind spot and kohtalo are not required for cell prolife
8   We have identified mutations in two genes, blind spot and kohtalo, that encode Drosophila homologue
9 cotoma then elongated toward the physiologic blind spot and spread toward the nasal periphery, sparin
10 ardwired, in which receptive fields span the blind spot and support fine orientation discriminations.
11 field defect demonstrated bilateral enlarged blind spots and altitudinal defects.
12 l, invariant to shifting and scaling, has no blind spots and has a sample-size-free interpretation.
13 a regular pattern, and the 'filling in' of a blind spot, are dramatic manifestations of the way conte
14 lation zone over several weeks, reducing the blind spot at scotopic and photopic luminances.
15 esholds were measured over the physiological blind spot at the optic nerve head and over equally ecce
16 us has been used to concurrently investigate blind spot awareness and blind spot filling-in.
17 within the blind spot) was used to determine blind spot awareness and filling-in for five subjects.
18                                              Blind spot awareness was achieved by stimulating 43% to
19                                              Blind spot awareness was reported for bar extensions bey
20  lineage had remained hidden as a taxonomic 'blind spot' because of mismatches in the primers commonl
21 cts located to the front (targets), causing "blind spots." Because the second harmonic is beamed more
22 mary visual cortex (V1) corresponding to the blind spot (BS) in the unpatched eye, and tested whether
23 hat the functional size of the physiological blind spot can be shrunk through training to distinguish
24                                              Blind spot dimensions were quantified, after which an in
25                         For 11 locations the blind spot edge estimates fit plausibly with the general
26  could be used to estimate the nature of the blind spot edge.
27 s fit plausibly with the general form of the blind spot (edge orientation within 90 degrees of expect
28 es included enlargement and expansion of the blind spot extending into large pericentral or other typ
29 lation of the ipsilateral eye to that of the blind-spot eye.
30                            Partial and total blind spot filling-in were reported between 1.1 degrees
31 rrently investigate blind spot awareness and blind spot filling-in.
32  of extremely low diversity, which represent blind spots for studies of natural variation and complex
33 erceptually dominant and suppressed when the blind-spot grating became dominant.
34                              Training on one blind spot, however, did not transfer to the blind spot
35 ht a stem cell/cancer link...and a potential blind spot in large-scale cancer genome sequencing proje
36 t white dot syndrome (MEWDS) and an enlarged blind spot in response to 30 degree diameter blue flashe
37  acuity but optic disc edema and an enlarged blind spot in the right eye (oculus dexter, OD).
38 blind spot, however, did not transfer to the blind spot in the untrained eye, ruling out mediation vi
39 entral nervous system pathologies, including blind spots in the retina.
40 leotide and that initiation does not have a "blind spot." In assembled initiation complexes, the cap
41 We study the spatial distribution of natural blind spot location (NBSL) and its impact on perimetry.
42 tually equivalent ones presented outside the blind-spots, looking for a Gabor stimulus without a smal
43  subspecialization have created a conceptual blind spot, namely, the inability to appreciate the endo
44 ed by large scotomata at or connected to the blind spot), ocular findings (paucity of pigmentary chan
45                        In every subject, the blind spot of the contralateral eye was conspicuous as a
46                                         This blind spot of the immune system and the redirection of r
47 e retinal area corresponding to the enlarged blind spot of the patient with MEWDS.
48                               Loss of either blind spot or kohtalo has identical effects on the devel
49 ning to distinguish direction signals at the blind spot periphery.
50                            The physiological blind spot refers to a zone of functional blindness all
51                                          The blind spot region in the primary visual cortex was label
52                                              Blind spots remain, especially at intermediate length sc
53                            Surprisingly, the blind spot representation of the open eye was shrunken b
54 thin profiles in layer 4C radiating from the blind spot representation.
55 dicted by interocular rivalry, the monocular blind-spot representation was activated when the ipsilat
56          Partial and total filling-in of the blind spot required between 78% and 83% and more than 85
57                   Twenty-seven of the 52 non-blind spot SAP locations exhibited a correlation greater
58  behaved opposite to optimal, preferring the blind-spot stimulus as the better example of a collinear
59 hresholds were better over the physiological blind spot than over equally eccentric temporal retina (
60 with predominantly arcuate loss and enlarged blind spots that require formal perimetry for detection.
61 artially overlap, or abut, the physiological blind spot, thereby enhancing sensitivity to weak signal
62 of filling-in occurring at the physiological blind-spots to compare partially inferred and veridical
63 rkably, the late response was diminished for blind-spot trials.
64                        The nasal edge of the blind spot was mapped in seven normal subjects with a 2
65 arcuate visual field defect with an enlarged blind spot was the most common perimetric finding.
66 ar bar of varying length centered within the blind spot) was used to determine blind spot awareness a
67                   To address this remarkable blind spot, we have measured the kinetics for racemizati
68 d cortical representations of the monocular "blind spot." We also activated area V1 preferentially (r
69 ing endoscopists to view behind folds and in blind spots, which might increase dysplasia detection.

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