ライフサイエンスコーパス: blood alcohol

1 a-III excretion correlating with the rise in blood alcohol.

2 by ethanol (EtOH) at concentrations seen in blood alcohol after binge drinking, and diminished ALDH1

3 Blood alcohol concentration (BAC) and rate of alcohol el

4 a significant difference in relation to the blood alcohol concentration (BAC) as a function of gende

5 ragastrically with the ethanol solution, the blood alcohol concentration (BAC) was found to peak late

6 hat it is a criminal offense to drive with a blood alcohol concentration above the state's legal limi

7 ect to demographic factors, injury type, and blood alcohol concentration and urine toxicology results

8 cle crashes was lower when laws specifying a blood alcohol concentration of 0.08 g/dl per se (laws st

9 usly administered 6% v/v ethanol to a target blood alcohol concentration of 0.08% or placebo between

10 tar rats exposed to alcohol vapor had a mean blood alcohol concentration of 176.2 mg/dl during the la

11 se decreased as both the child's age and the blood alcohol concentration of the child's driver increa

12 tion in the laboratory, the maximum achieved blood alcohol concentration was influenced in an allele-

13 ubation controls, (3) 3 g/kg/day of alcohol (blood alcohol concentration {BAC}=139.9 mg/dl), (4) 4 g/

14 dramine (50 mg), alcohol (approximately 0.1% blood alcohol concentration), or placebo, given at weekl

15 and peer norms) on alcohol expenditure, peak blood alcohol concentration, alcohol dependence, and acc

16 Patients were screened using a blood alcohol concentration, gamma glutamyl transpeptida

17 al aldehyde dehydrogenase, in part determine blood alcohol concentration, thereby contributing to the

18 ol concentrations equivalent to 0.05 to 0.08 blood-alcohol concentration-levels that would be mildly

19 C, CeMPV and EW was significantly related to blood alcohol concentrations (BAC).

20 binge alcohol drinking (intake resulting in blood alcohol concentrations (BACs) >/=80 mg% within a 2

21 Elevated blood alcohol concentrations (BACs) are found in about o

22 Of 302 injury deaths, blood alcohol concentrations (BACs) were available for 2

23 cantly reduced their alcohol consumption and blood alcohol concentrations achieved.

24 quate to intoxicating through to supralethal blood alcohol concentrations in humans and provides an i

25 We found that, at equivalent blood alcohol concentrations, HDs reported lower subject

26 f the non-motor-carrier drivers had positive blood alcohol concentrations.

27 Native American heritage, despite equivalent blood alcohol concentrations.

28 ol drinking access that achieved significant blood alcohol concentrations.

29 had blood alcohol measured were intoxicated (blood alcohol content >22 mmol/L [100 mg/dL]).

30 Breathalyzers estimate Blood Alcohol Content (BAC) from the concentration of et

31 ale (P < 0.0001), Black (P < 0.0001), have a blood alcohol content above 80 mg/dL (P < 0.0001), and s

32 Male sex, Black race, and elevated blood alcohol content and penetrating injury are associa

33 ld) and cortex (up to 50-fold), depending on blood alcohol content.

34 es (P<.001) during the ascending limb of the blood alcohol curve and remained significantly above bas

35 (P<.001) and descending (P<.001) limb of the blood alcohol curve.

36 mal enhancement at 16 mM, close to the legal blood alcohol driving limit in most U.S. states (17.4 mM

37 to test the hypothesis that the presence of blood alcohol has a clinically significant impact on GCS

38 To address the hypothesis that elevated blood alcohol increases systemic oxidant stress, we meas

39 concentration six times lower than the legal blood-alcohol intoxication (driving) limit in most state

40 ng methamphetamine had a significantly lower blood alcohol level compared with the control group.

41 The average peak blood alcohol level in the treatment group was 40 +/- 4

42 ven at concentrations as low as 10 mm (legal blood alcohol level in the United States is below 0.08 g

43 as relevant and comparable to that seen at a blood alcohol level of 0.05%, the legal limit in many co

44 ary seatbelt laws, maximum speed limit laws, blood alcohol level of 0.08, and administrative license

45 ethanol in utero (average maternal and fetal blood alcohol level of 25 mg/dl) promotes premature tang

46 Binge drinking, defined as reaching a blood alcohol level of 80 mg%, carries a risk of negativ

47 Blood alcohol level of more than 100 mg/dl (OR=9.43, P=0

48 hemselves and others despite having a normal blood alcohol level.

49 Among decedents who had a negative blood-alcohol level (52 in residences and 26 in vehicles

50 ing; whereas, among those who had a negative blood-alcohol level and most likely were awake at the ti

51 A blood-alcohol level greater than 0.01% was present in 56

52 rom the HS/Npt line) to achieve intoxicating blood alcohol levels (BALs) after binge-like drinking.

53 no significant differences between groups in blood alcohol levels (BALs), these results are not due t

54 gnificantly shorter sleeping time and higher blood alcohol levels after regaining reflex than adult r

55 olerance during adolescence and suggest that blood alcohol levels alone do not fully explain ethanol-

56 cts of intoxication, despite greater initial blood alcohol levels and ethanol hypothermia in the form

57 Blood alcohol levels and task performance were similar a

58 concentrations comparable with or less than blood alcohol levels associated with intoxication and at

59 Blood alcohol levels did not differ significantly in wom

60 Whisky promoted highest blood alcohol levels in both sexes.

61 enzymes were associated with 30-fold higher blood alcohol levels in KO mice.

62 of the distilled drinks promoted a spike in blood alcohol levels more quickly than ingestion of the

63 Peak blood alcohol levels of 21 mmol/L were attained in each

64 f 1-2 alcoholic beverages in humans leads to blood alcohol levels of approximately 10 mM.

65 ere significantly correlated with changes in blood alcohol levels on both the ascending (P<.001) and

66 In women, beer promoted the lowest blood alcohol levels over the 6h of the experiment.

67 Blood alcohol levels were determined, and the retinas an

68 3 g/kg b.wt., i.p.) resulted in elevation of blood alcohol levels, ACTH and corticosterone in all gro

69 d the effects of four alcoholic beverages on blood alcohol levels, plasma insulin concentrations and

70 Chronic-binge ethanol feeding leads to high blood alcohol levels; thus, this simple model will be ve

71 Binge drinking (blood-alcohol levels >/= 0.08 g% in a 2-h period), is a

72 me alone, and 69 (53%) of 130 adults who had blood alcohol measured were intoxicated (blood alcohol c

73 tine were currently drug dependent, 11.7% of blood alcohol-negative and 3.9% of drug-negative patient

74 While 54.3% of blood alcohol-positive patients were currently alcohol d

75 I (Head Abbreviated Injury Score [AIS]), and blood alcohol testing status were known, were included.

76 e frequently monitored with random urine and blood alcohol tests; patients found positive were exclud

77 A transient elevation of blood alcohol to approximately 120 mg/dL was sufficient