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1 hemselves and others despite having a normal blood alcohol level.
3 3 g/kg b.wt., i.p.) resulted in elevation of blood alcohol levels, ACTH and corticosterone in all gro
4 gnificantly shorter sleeping time and higher blood alcohol levels after regaining reflex than adult r
5 olerance during adolescence and suggest that blood alcohol levels alone do not fully explain ethanol-
6 cts of intoxication, despite greater initial blood alcohol levels and ethanol hypothermia in the form
8 ing; whereas, among those who had a negative blood-alcohol level and most likely were awake at the ti
9 concentrations comparable with or less than blood alcohol levels associated with intoxication and at
10 rom the HS/Npt line) to achieve intoxicating blood alcohol levels (BALs) after binge-like drinking.
11 no significant differences between groups in blood alcohol levels (BALs), these results are not due t
12 ng methamphetamine had a significantly lower blood alcohol level compared with the control group.
17 ven at concentrations as low as 10 mm (legal blood alcohol level in the United States is below 0.08 g
20 of the distilled drinks promoted a spike in blood alcohol levels more quickly than ingestion of the
21 as relevant and comparable to that seen at a blood alcohol level of 0.05%, the legal limit in many co
22 ary seatbelt laws, maximum speed limit laws, blood alcohol level of 0.08, and administrative license
23 ethanol in utero (average maternal and fetal blood alcohol level of 25 mg/dl) promotes premature tang
28 ere significantly correlated with changes in blood alcohol levels on both the ascending (P<.001) and
30 d the effects of four alcoholic beverages on blood alcohol levels, plasma insulin concentrations and
31 Chronic-binge ethanol feeding leads to high blood alcohol levels; thus, this simple model will be ve
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