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1 mp CABG (with or without valve surgery) with blood cardioplegia.
2 anced in the coronary microcirculation after blood cardioplegia.
3 ulmonary bypass and randomized to either all-blood cardioplegia (AB group) or dilute blood cardiopleg
8 s C) antegrade potassium all-blood or dilute blood cardioplegia and maintained with tepid retrograde
9 vested from patients (n=15) before and after blood cardioplegia and short-term reperfusion under cond
10 this, early death (6% crystalloid versus 4% blood cardioplegia) and late death (24% crystalloid vers
13 tested the hypothesis that ADO-supplemented blood cardioplegia (BCP) or ADO administered during repe
14 of the intraoperative administration of cold blood cardioplegia, blood cardioplegia containing 500 mi
17 e administration of cold blood cardioplegia, blood cardioplegia containing 500 microM adenosine, and
19 ditioning activation adjuvant to hypothermic blood cardioplegia enhances postischemic contractile rec
20 loid cardioplegia (iC-CCP; n=8) or with cold blood cardioplegia (iC-BCP; n=6) administered intermitte
21 ety and tolerance of adenosine when added to blood cardioplegia in increasing doses to enhance myocar
23 oid cardioplegia (iW-CCP) (n=8) or with warm blood cardioplegia (iW-BCP) (n=8) administered intermitt
24 atrial segment exposed to cold, hyperkalemic blood cardioplegia (mean, 60 minutes) and a brief period
25 o determine the effect, if any, of adenosine blood cardioplegia on blood component usage after heart
27 ) and late death (24% crystalloid versus 21% blood cardioplegia) statistics were not significantly di
28 stalloid cardioplegia versus those receiving blood cardioplegia were found to have significantly more
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