ライフサイエンスコーパス: blood cell

1 hose diameter is comparable to that of a red blood cell.

2 ncentrations of other metabolites in the red blood cell.

3 ayer-by-layer assembly technique using human blood cells.

4 ne expression in brain as well as peripheral blood cells.

5 platform for probing the biomechanics of red blood cells.

6 al for terminal maturation of definitive red blood cells.

7 horesis to remove magnetically-labeled white blood cells.

8 d detect only scattered extravasation of red blood cells.

9 at are present at high concentrations in red blood cells.

10 ies to a protein crucial for invasion of red blood cells.

11 nals for engraftment of normal and malignant blood cells.

12 ng and metabolic flux analysis in stored red blood cells.

13 routes for purification of manufactured red blood cells.

14 erential DNA methylation at numerous loci in blood cells.

15 H3K27me3 from extended regions in erythroid blood cells.

16 adjacent unaffected adrenal tissue and white blood cells.

17 /red blood cells; and rodent fibroblasts/red blood cells.

18 disseminated cells with platelets and white blood cells.

19 ding into membrane vesicles derived from red blood cells.

20 to various types of glial, endothelial, and blood cells.

21 (neg) cells and spared normal umbilical cord blood cells.

22 % and greater than five-log depletion of red blood cells.

23 s, patients exposed to TXA received less red blood cell (3 [0, 6] vs 4 [1, 7] P = 0.003) and frozen p

24 d an immunopurification method to enrich red blood cell AChE (RBC AChE) as a biomarker of exposure.

25 is, transport of nutrients, and migration of blood cells across the barrier.

26 chanism of local sensory neurons controlling blood cell adaptation invites evolutionary parallels wit

27 Red blood cell Ae1-mediated anion-exchange activity and surf

28 sed the inflammatory potential of peripheral blood cells after lunch.Compared with 3 meals/d, meal sk

29 k reports on the spatial distribution of red blood cell aggregates in a T-shaped bifurcation on the s

30 flows are often considered to be free of red blood cell aggregates, however, recent studies have demo

31 , we observed significant reduction of white blood cell and absolute lymphocyte count up to 1 year po

32 White blood cell and absolute lymphocyte count were significan

33 immunocompetent hosts with normal CSF white blood cell and protein levels (</=5 cells/mm(3) and </=5

34 onship between loss of Y chromosome (LOY) in blood cells and a higher risk of cancer and mortality.

35 rotoma (Lh), a generalist wasp, kills larval blood cells and actively suppresses immune responses.

36 to detect the presence of CHIP in peripheral-blood cells and associated such presence with coronary h

37 ascular accumulation of both parasitized red blood cells and CD8(+) T cells to the brain, and an incr

38 y due to reduced membrane deformation of red blood cells and decreased level of reactive oxygen speci

39 ated the expression of FcepsilonRII on human blood cells and found that it was primarily expressed on

40 ion and that hemoglobin diffusion in the red blood cells and in solutions at similar concentration ar

41 ted DNA hypomethylation has been observed in blood cells and linked to lung cancer risk.

42 To do so, 5-HT was added to red blood cells and lipid membranes bearing different degree

43 Because of the binding of (123)I-CLINDE to blood cells and peripheral tissues, SUV is not a suffici

44 microm-50 microm) used to first deplete red blood cells and platelets.

45 of CD47 in normal phagocytosis of aging red blood cells and results reported in the original study.

46 or for T. gondii in primary human peripheral blood cells and to define an upstream regulator of its a

47 s expressing IFN-induced genes in peripheral blood cells and with TLRs promoting type I IFNs and auto

48 ilitates this reversible reaction inside red blood cells, and band 3 [anion exchanger 1 (AE1)] provid

49 nvestigation by imaging live HeLa cells, red blood cells, and neurons.

50 live and dead yeast; human cancer cells/red blood cells; and rodent fibroblasts/red blood cells.

51 and (ii) that high glutathione levels in red blood cells are associated with the emergence of visible

52 Blood cells are derived from a common set of hematopoiet

53 cellular barriers that protect primary human blood cells are likely to be important in protection aga

54 Mice transfused with these red blood cells are resistant to highly lethal doses of BoNT

55 We found that blood cells are uniquely resistant to infection with FeL

56 genetically engineered and to unmodified red blood cells as a means of inducing antigen-specific tole

57 ed homogeneous data with composition of some blood cells available and compared them with the estimat

58 C and -MC), and binding of the tracer to red blood cells (+BB and -BB) was evaluated.

59 cell-cycle indicators in single CD34(+) cord blood cells before and up to 2 hours after their stimula

60 Additionally, we need to ensure the blood cell biocompatibility of developed devices prior t

61 neurotoxin A (BoNT/A) on the surface of red blood cells by expressing chimeric proteins of VHHs with

62 In the US, red blood cells can be stored for up to 6 weeks, but randomi

63 fDNA to the proportion of donor DNA in white blood cells can differentiate between relapse and the on

64 stion on how hemoglobin diffusion in the red blood cells can help the oxygen capture at the cell leve

65 Red blood cells can synthesize H2S but, lacking organelles,

66 Mice whose red blood cells carry the chimeric proteins exhibit resistan

67 t (96.6% white blood cell yield, 0.0059% red blood cell carryover) by processing whole blood at 3 mL/

68 ve of FeLV replication, innate resistance of blood cells could be critical in protecting against cros

69 to 109 per liter, multiply by 0.001); white blood cell count >/=15000/microL, 27% (95% CI, 18% to 36

70 tive chronic myelomonocytic leukaemia (white blood cell count <13 000/muL), and had anaemia with or w

71 Optimal thresholds for white blood cell count (11600/microL), absolute neutrophil cou

72 [29%]), anaemia (26 [29%]), decreased white blood cell count (17 [19%]), and decreased lymphocyte co

73 , leucopenia (27 [10%]), and decreased white blood cell count (21 [8%]).

74 (31 [10%] vs 41 [13%]), and decreased white blood cell count (39 [13%] vs 33 [11%]).

75 (hazard ratio [HR], 3.299; P < .001), white blood cell count (HR, 1.910; P = .017), platelet count (

76 Model for End-Stage Liver Disease and white blood cell count (OR, 4.68; 95% CI, 1.80-12.17; P = .001

77 flammation, fluid, appendicoliths, and white blood cell count (WBC) were significantly correlated wit

78 White blood cell count analysis after alpha-radioimmunotherapy

79 waist-hip ratio, alanine transaminase, white blood cell count and lower high-density lipoprotein chol

80 piratory, cardiac, and liver function, white blood cell count at least 3 x 10(9) cells per L, platele

81 his large cohort of APL patients, high white blood cell count emerged as an independent predictor of

82 sterol in men, and with higher BMI and white blood cell count in women (differences 0.03-0.06 standar

83 cclusion, n=1 each; placebo: vomiting, white blood cell count increased, n=1 each).

84 blood cell count parameter thresholds: white blood cell count less than 5000/microL, 10% (95% CI, 4%

85 -2 and 5.20 (95% CI, 2.70-10.02) for a white blood cell count of >/=20 000/muL vs <20 000/muL.

86 ins vs. late immunotherapy), and a low white blood cell count on the first cerebrospinal examination

87 positivity, and inflammatory markers (white blood cell count or cytokine level).

88 No complete blood cell count parameter at commonly used or optimal t

89 Sensitivities were low for common complete blood cell count parameter thresholds: white blood cell

90 and/or bacterial meningitis, using complete blood cell count parameters.

91 on day 1) added to high-risk patients (white blood cell count, >10 x 10(9)/L), as well as low-risk pa

92 We tested the accuracy of the white blood cell count, absolute neutrophil count, and platele

93 t of patients with FLT3-ITD, only age, white blood cell count, and < 4-log reduction in PB-MRD, but n

94 oxicity analysis included body weight, white blood cell count, and hematocrit.

95 es, physical triggers, BAT results, complete blood cell count, C-reactive protein levels, thyroid-sti

96 r outcome (CSF culture positivity, CSF white blood cell count, hemoglobin, Glasgow Coma Scale, and pu

97 sing tricuspid regurgitation velocity, white blood cell count, history of acute chest syndrome, and h

98 abnormal blood cells, or reduced peripheral blood cell count.

99 ge, creatinine clearance, haemoglobin, white-blood-cell count, and previous spontaneous bleeding) sho

100 bition of mTORC1 significantly increased red blood cell counts and hemoglobin content in the blood, i

101 SPIROMICS baseline visit data with complete blood cell counts and, in a subset, acceptable sputum co

102 Raised white blood cell counts as well as peaks of serum levels of C-

103 All other blood cell counts in Tpm4-deficient mice were normal.

104 sterone), hepatobiliary enzyme levels, white blood cell counts, and iron homeostasis.

105 stion of microvascular function: how are red blood cells delivered at the same rate to each micro-ves

106 ric mice with selective deficiency of APP in blood cells, developed much larger thrombi than control

107 including genes involved in oncogenesis and blood cell development.

108 iseases and blood malignancies and influence blood cell development.

109 stem cells to populate the liver and promote blood cell differentiation.

110 topoietic stem cells (HSCs) to treat genetic blood cell diseases has become a clinical standard but i

111 halassemias are a heterogeneous group of red blood cell disorders, considered a major cause of morbid

112 One white blood cell displayed the average esterolytic activity of

113 fixed cells, such as red blood cells, white blood cells, DU-145 prostate cancer cells, MCF-7 breast

114 how that pressure feedbacks created when red blood cells enter the finest vessels of the trunk act to

115 d throughout parasite development within red blood cells, even during a period coincident with extens

116 It is interesting to note that cord blood cells exhibited extremely low HLA class I expressi

117 c approach, we demonstrate that red drum red blood cells express 7 and 5 Hbalpha and Hbbeta isoforms,

118 Transfusion of red blood cells expressing self-antigen epitopes can allevia

119 We demonstrate that engineered red blood cells expressing VHHs can provide prolonged prophy

120 sections of treated tumors have detected red blood cell extravasation within tumors treated with temp

121 ltration of lymphocytes and neutrophils, red blood cell extravasation, and plumping endothelium were

122 ormation and dynamics of each individual red blood cell flowing through the networks with high fideli

123 , 1.30; 95% CI, 1.12-1.52; P < .001) and red blood cell folate (OR, 1.28; 95% CI, 1.10-1.49; P = .001

124 1.27; 95% CI, 1.10-1.47; P = .001), and red blood cell folate levels (OR, 1.25; 95% CI, 1.08-1.44; P

125 Serum and red blood cell folate levels are independent predictors of H

126 bserved regarding cobalamin, folate, and red blood cell folate.

127 o of Gr1(+) granulocytes in peripheral white blood cells following bacteremia.

128 r mechanism by which TH functions during red blood cell formation, results that are potentially usefu

129 moglobin concentration observed in human red blood cells ([Formula: see text]330 g.L (-1)) correspond

130 d ubiquitination activity in vitro and white blood cells from affected individuals exhibited signific

131 t embryos had reduced numbers of circulating blood cells from E9.5 onwards, with the development of s

132 Transfusion of red blood cells from female donors has been associated with

133 We collected peripheral blood cells from Framingham Heart Study participants and

134 ed improved enrichment and recovery of white blood cells from human blood.

135 e mutations have recently been identified in blood cells from normal, healthy elderly individuals wit

136 icantly reduced GLUT1 expression only on red blood cells from patients with GLUT1-DS (23 patients; 78

137 entration, a process critical for normal red blood cell function and survival.

138 Primitive erythroblasts are the first blood cells generated during embryonic hematopoiesis.

139 Margination of stiffened red blood cells has been implicated in many vascular disease

140 uidic devices, the detection and analysis of blood cells has remained an outstanding challenge.

141 To withstand these conditions, blood cells have evolved a special morphology and a spec

142 that while previous studies done largely in blood cells have led to important investigations into HI

143 of the peripheral nervous system (PNS), and blood cells (hemocytes) require the PNS for their surviv

144 rom precursors, how progenitors generate red blood cells, how hemoglobin synthesis is regulated, and

145 CyTOF datasets generated from primary immune blood cells: (i) 14 subjects with a history of infection

146 derived from genome-wide profiling in white blood cells, identifying 26 expression probes and 92 CpG

147 demonstrate that accurate estimates of white blood cell images can be recovered from extremely noisy

148 is the most common enzymatic disorder of red blood cells in human subjects, causing hemolytic anemia

149 erns of gene expression in circulating white blood cells in response to infection.

150 We simulate deformable red blood cells in the microcirculation using the immersed b

151 ptors, retinal pigment epithelial cells, and blood cells in the retinal vasculature, other important

152 od, there is much interest in generating red blood cells in vitro as an alternative clinical product.

153 significance of blasts, and of white and red blood cells, in CSF samples at diagnosis of acute lympho

154 id opening pressure of 28 mm H2O and 8 white blood cells, including 1 atypical plasma cell.

155 eas P cynomolgi merozoites invade monkey red blood cells indiscriminately in vitro, in humans, they a

156 To avoid clearance by the spleen, red blood cells infected with the human malaria parasite Pla

157 D50) of BoNT/A, and transfusion of these red blood cells into naive mice affords protection for up to

158 The adhesion of malaria infected red blood cells (iRBCs) to host endothelial receptors in the

159 AChE in red blood cells is a surrogate for AChE in the nervous syste

160 ises when a substantial proportion of mature blood cells is derived from a single dominant hematopoie

161 In contrast to CD45(pos) white blood cells isolated and processed by the identical appr

162 We find that red blood cell jamming at vascular bifurcations results in s

163 s that are not normally expressed in the red blood cell, leading to systemic effects that exacerbate

164 s for gravitational sedimentation of the red blood cells, leaving a layer of yellowish plasma at the

165 ion induced by integrin-dependent sickle-red blood cell-leukocyte adhesion.

166 agy is required for differentiation of other blood cell lineages, its function during granulopoiesis

167 o experiment-simulating an entire mammal red blood cell lipid bilayer and cytoskeleton as modeled by

168 cells/low-power field [lpf] and >/=25 white blood cells/lpf or a quality score [q-score] definition

169 urthermore, histone-induced increases in red blood cell lysis and splenic clearance may be a signific

170 For instance, nonmammalian red blood cells, mammalian erythroblasts, and platelets have

171 lood count (CBC) and inflammation-associated blood cell markers derived from them have been reported

172 The increase in red blood cell mass (polycythemia) due to the reduced oxygen

173 g delivery system that combines a robust red blood cell membrane-coated nanoparticle (RBCNP) with a u

174 We further observed that blood cells more efficiently promote the viability of no

175 We show that in human peripheral blood cells, more than 30% of Treg up-regulate CCR8 foll

176 Red blood cells must deform to squeeze through these narrow

177 B treatment can normalize hemoglobin and red blood cell numbers.

178 age on iPSC quality, we produced iPSCs from blood cells of 16 donors aged 21-100.

179 s (HSCs) in the bone marrow (BM) form mature blood cells of all lineages through expansion of lineage

180 h antibiotics associated with reduced ST2 in blood cells of both asthmatic and control children and r

181 -gamma ELISPOT for recognition in peripheral blood cells of CD patients and healthy controls carrying

182 files and drag forces imposed to trapped red blood cells of living zebrafish embryos.

183 blood group antigen is expressed on the red blood cells of most individuals.

184 tures were elevated in plasma and peripheral blood cells of patients with AT, Artemis deficiency, and

185 nutrient starvation and that the mature Red Blood Cells of some RTT patients retain mitochondria.

186 tem cell disorders characterized by abnormal blood cells, or reduced peripheral blood cell count.

187 the prioritization of available iron to red blood cells over all other tissues during negative iron

188 cell transit was highly constrained, and red blood cell oxygen saturation was low and inappropriately

189 archived blood smears and corresponding red blood cell pellets collected from asymptomatic children

190 al of 367 blood smears and corresponding red blood cell pellets, including 185 smears (50.4%) that we

191 ation for Plasmodium falciparum-infected red blood cells (Pf-iRBCs).

192 mbers of the pyruvate kinase, liver, and red blood cell (PKLR) gene as well as of the Fcgamma recepto

193 icting dynamic metabolic flux states for red blood cells, platelets, and Saccharomyces cerevisiae.

194 ion between RBCs, and between RBCs and white blood cells, platelets, and the endothelium.

195 he hypothesis that somatic TET2 mutations in blood cells play a causal role in atherosclerosis.

196 Common red blood cell polymorphisms (ie, hemoglobin S, glucose-6-ph

197 e the prevalence of 3 malaria-protective red blood cell polymorphisms in BWF cases vs both trial (non

198 cule or EpCAM) or size to separate them from blood cell populations.

199 utant (GPI-negative) HSPC-derived peripheral blood cell populations.

200 - or Plasmodium yoelii 17XNL-parasitized red blood cells (pRBCs) after transfusion into naive or acut

201 e in the aging of stored units of packed red blood cells (pRBCs) and subsequent lung inflammation aft

202 by Staphylococcus aureus, which target white blood cells preferentially and consist of an S- and an F

203 an iTregs generated in vitro from naive cord blood cells preferentially recruit Disc large homolog 1

204 opoietic hierarchy and are the origin of all blood cells produced throughout an individual's life.

205 A compartmental model of the dynamics of red blood cell production and destruction was designed to ch

206 emoglobin and haematocrit, and decreased red blood cell production rates.

207 tin (EPO) is the cytokine that regulates red blood cell production.

208 Invasion of the red blood cell (RBC) by the Plasmodium parasite defines the

209 , which itself causes reduced peripheral red blood cell (RBC) counts without altering relative abunda

210 en compared these intakes with serum and red blood cell (RBC) folate among 4878 men and nonpregnant,

211 quent interaction with the membrane of a red blood cell (RBC) is important to predict the altered mor

212 the temperature dependence of the human red blood cell (RBC) metabolic network between 4 and 37 degr

213 In this study, by using a two-component red blood cell (RBC) model and systematic parameter variatio

214 differences between donor and recipient red blood cell (RBC) survival times.

215 Red blood cell (RBC) transfusion poses significant risks to

216 nal bleeding is a leading indication for red blood cell (RBC) transfusion worldwide, although optimal

217 Red blood cell (RBC) transfusions are of vital importance in

218 ied over the number of Hb molecules in a red blood cell (RBC), the effect is detectable through motio

219 The mechanisms underlying red blood cell (RBC)-mediated hypoxic vasodilation remain co

220 determine the degree of agglutination of red blood cell (RBC).

221 complex genetic blood disorder in which red blood cells (RBC) exhibit heterogeneous morphology chang

222 e by comparing human antibody binding to red blood cells (RBC) isolated from knockout swine and to al

223 en supply, typically delivered by packed red blood cells (RBC).

224 ifferentiated by perfusate: (1) isolated red blood cells (RBCs) (current clinical standard for OCS);

225 ty is essential for redox equilibrium of red blood cells (RBCs) and, when compromised, the RBCs are m

226 al simulations with discrete tracking of red blood cells (RBCs) are performed in three realistic micr

227 sing high-resolution microscopy of human red blood cells (RBCs) as a case study.

228 Red blood cells (RBCs) demonstrate procoagulant properties i

229 t a physiologic ratio of 1 platelet to 9 red blood cells (RBCs) did not inhibit the in vitro developm

230 Vertebrate red blood cells (RBCs) display a range of sizes, spanning or

231 ld-type (WT)) and homozygous sickle (SS) red blood cells (RBCs) express a large number of surface rec

232 on ratio of fresh frozen plasma (FFP) to red blood cells (RBCs) has spread to other surgical and medi

233 Red blood cells (RBCs) have historically been considered pas

234 tality, among persons who receive stored red blood cells (RBCs) have recently garnered considerable a

235 A simple method to convert red blood cells (RBCs) into efficient microreactors is repor

236 Plasmodium falciparum, invasion of host red blood cells (RBCs) is essential.

237 Red blood cells (RBCs) of SCD patients have diverse shapes t

238 Transfusion of either platelets or red blood cells (RBCs) on days with bleeding was often not s

239 rials have addressed whether exposure to red blood cells (RBCs) stored longer than 35 days is associa

240 development of metabolic decay in human red blood cells (RBCs) under cold storage conditions.

241 or bee venom, were investigated on human red blood cells (RBCs) using quantitative phase imaging tech

242 r sized particles, either latex beads or red blood cells (RBCs), create aggregates that are easily de

243 measure accessible cholesterol in human red blood cells (RBCs).

244 st requires mechanisms to spread between red blood cells (RBCs).

245 glucose transporters on erythrocytes (or red blood cells, RBCs) membrane.

246 a history of pregnancy and mortality of red blood cell recipients.

247 ed migration, tumorigenesis, metastasis, red blood cell recruitment to tumors, and induced hepatosple

248 ly isolate CTC clusters from single CTCs and blood cells relies on the batch immobilization of cluste

249 ls in the adult mammalian bone marrow ensure blood cell renewal.

250 Biogenesis of mammalian red blood cells requires nuclear expulsion by orthochromatic

251 ied with a reduced survival of Sox6(-/-) red blood cells, resulting in a compensated anemia.

252 Examination of peripheral red blood cells revealed an increase of reactive oxygen spec

253 Dynamic imaging of red blood cells revealed the regeneration of an extensive ne

254 Cytokine responses were measured in blood cell samples obtained at birth (cord blood) and ag

255 ared with available bone scintigraphy, white blood cell scintigraphy, and (18)F-FDG PET/CT results.

256 Abnormal sickle red blood cell (sRBC) biomechanics, including pathological d

257 of erythropoietin by the kidney; reduced red blood cell survival and iron deficiency; and mineral bon

258 Non-aggregating and aggregating human red blood cell suspensions were studied for a range of flow

259 d CD34+ culture system to engineer human red blood cells that express these chimeric proteins.

260 duce interacting epigenetic effects in white blood cells that influence immune function and health ou

261 g to C3 in freshly isolated human peripheral blood cells that was absent in corresponding cell lines.

262 runk act together to uniformly partition red blood cells through the microvasculature.

263 s (HSCs) ensure a balanced production of all blood cells throughout life.

264 ere the authors engineer mouse and human red blood cells to express VHHs against botulinum neurotoxin

265 We utilize red blood cells to prolong the circulatory half-life of VHHs

266 e ability of yolkin to stimulate human whole blood cells to release anti-inflammatory cytokines and n

267 oiesis, we performed association studies for blood cell traits in the population-based Estonian Bioba

268 nalyses identified 17 associations across 14 blood cell traits.

269 ergenic region, which is associated with red-blood-cell traits, including fetal hemoglobin levels.

270 To quantify the association between red blood cell transfusion from female donors with and witho

271 00/muL), and had anaemia with or without red blood cell transfusion support.

272 A patient with asplenia and multiple red blood cell transfusions acquired babesiosis infection wi

273 no-donor-mixture cohort (ie, either all red blood cell transfusions exclusively from male donors, or

274 Red blood cell transfusions from ever-pregnant or never-preg

275 er-pregnant female donors, compared with red blood cell transfusions from male donors.

276 for at least 28 days who either required red blood cell transfusions while on ruxolitinib or ruxoliti

277 Among female recipients of red blood cell transfusions, mortality rates for an ever-pre

278 Among patients who received red blood cell transfusions, receipt of a transfusion from a

279 nemia and frequently become dependent on red blood cell transfusions.

280 Red blood cell transit analysis revealed slow and stalled fl

281 ransfusion burden, defined as >/=4 vs <4 red blood cell units per 8 weeks); pre-study serum erythropo

282 The presence of CHIP in peripheral-blood cells was associated with nearly a doubling in the

283 Tenofovir-diphosphate (TFV-DP) in red blood cells was used to categorize participants into hig

284 White blood cell (WBC) analysis provides rich information in r

285 White blood cell (WBC) count appears to predict total mortalit

286 Elevated white blood cell (WBC) count is associated with increased majo

287 rijuana use and HIV disease markers or white blood cell (WBC) count were examined using mixed-effects

288 e the oncogenetic classifier, MRD, and white blood cell (WBC) count.

289 Circulating white blood cell (WBC) counts (neutrophils, monocytes, lymphoc

290 ammalian cells, more specifically from white blood cells (WBC).

291 Conventional inflammatory markers (white blood cell [WBC] count, erythrocyte sedimentation rate [

292 eponema pallidum 16S RNA in CSF or CSF white blood cells (WBCs) >20/uL or a reactive CSF-Venereal Dis

293 White blood cells (WBCs) play an important role in host immune

294 d mononuclear cells (PBMCs) and of CSF white blood cells (WBCs) that were activated monocytes (CD14+C

295 DNA methylation in white blood cells (WBCs) was associated with total IgE levels

296 nalysis of the tumor, non-involved lung, and blood cells, we provide a detailed immune cell atlas of

297 from Muc4(ko)/NDL tumors interact with fewer blood cells when injected directly into the vasculature

298 es continuous asexual replication within red blood cells, while its mosquito-borne transmission depen

299 rs for nonfixed and fixed cells, such as red blood cells, white blood cells, DU-145 prostate cancer c

300 produce an ultrapure buffy coat (96.6% white blood cell yield, 0.0059% red blood cell carryover) by p