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1 th the cell membrane are critical within the blood coagulation cascade.
2 tein complexes formed, and regulation of the blood coagulation cascade.
3 se inhibitors and the major inhibitor of the blood coagulation cascade.
4 rokinase, which have opposing effects on the blood coagulation cascade.
5 d fibrin monomers in the final stages of the blood coagulation cascade.
6 al intravascular thrombus development in the blood coagulation cascade.
7 ated protein C in the down-regulation of the blood coagulation cascade.
8 , without inhibiting serine proteases in the blood coagulation cascade.
9 VIIa complexes on cell surfaces triggers the blood coagulation cascade.
10 receptor tissue factor (TF) to initiate the blood coagulation cascade.
11 ieved to be the physiologic initiator of the blood coagulation cascade.
12 R, maintain vitamin K levels and sustain the blood coagulation cascade.
13 re primarily restricted to components of the blood coagulation cascade.
16 ice partially restored the activation of the blood coagulation cascade and accumulation of platelets.
17 as a key enzyme for the intervention of the blood coagulation cascade and for the development of new
18 he initiator of the extrinsic pathway of the blood coagulation cascade and normally released from dam
19 serine protease that plays a key role in the blood coagulation cascade and represents an attractive t
20 inhibition of factor Xa, a proteinase of the blood coagulation cascade, and is typically achieved wit
22 or VIIa is involved in the first step of the blood coagulation cascade, as a membrane-associated enzy
23 ntly showed that polyphosphate modulates the blood coagulation cascade at 3 steps: it triggers the co
24 by tissue factor-initiated activation of the blood coagulation cascade at the feto-maternal interface
26 flammatory protease that is regulated by the blood coagulation cascade, exerts similar effects depend
27 (TF), the cellular initiator of the protease blood coagulation cascade, has been shown to be expresse
28 d serine proteases, which participate in the blood coagulation cascade, have focused attention on the
31 elet-CTC interactions inherently rely on the blood coagulation cascade including platelet activation.
32 rombin, and its activation at the end of the blood coagulation cascade results in the formation of th
33 tamic acid domain-containing proteins of the blood coagulation cascade, the two PRGPs are expressed i
35 al modification of several proenzymes of the blood coagulation cascade, using either collisions or in
36 hich participates in the middle stage of the blood coagulation cascade was cloned from a rat liver cD
37 cular understanding of a key reaction of the blood coagulation cascade where cofactor Va enhances act
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