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1 of ethanol ingestion than the measurement of blood ethanol .
2 at were positive, negative, or equivocal for blood ethanol.
3 that initially resembled the decay curve for blood ethanol.
4 l impairment by causing a rapid reduction in blood ethanol and acetaldehyde levels after acute ethano
6 rodent model of FASD, high doses of ethanol (blood ethanol concentration 80 mM) induces neuronal cell
7 en ethanol at a rate sufficient to cause the blood ethanol concentration to reach levels associated w
9 comotor activity, core body temperature, and blood ethanol concentration, as well as ethanol consumpt
10 ucing bacteria in NASH microbiomes, elevated blood-ethanol concentration in NASH patients, and the we
11 ly showed that heavy ethanol intake (average blood ethanol concentrations > 80 mg/dl) suppressed, whe
12 essed, whereas moderate alcohol consumption (blood ethanol concentrations < 50 mg/dl) enhanced, T and
18 intake was completed for 7 individuals whose blood ethanol level was elevated from 25 to 35 mmol/L.
19 nol drinking in C57BL/6J mice (that achieved blood ethanol levels >80 mg/dl in control conditions).
20 hanol-containing liquid diet that results in blood ethanol levels near the legal intoxication limit i
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