ライフサイエンスコーパス: blood flow

1 the role of blood vessel tone in regulating blood flow.

2 of reference region and changes in cerebral blood flow.

3 ake in the tumor and muscle and on hind limb blood flow.

4 icial effects on cardiovascular function and blood flow.

5 y weak gamma activity despite showing normal blood flow.

6 c interruption and exaggeration of pulmonary blood flow.

7 directional coupling of platelet motion with blood flow.

8 nzyme in the regulation of vascular tone and blood flow.

9 eracted the NAI-induced reduction in uterine blood flow.

10 mean arterial pressure to regional cerebral blood flow.

11 link between neuronal activity and cerebral blood flow.

12 DS in infants with duct-dependent pulmonary blood flow.

13 e-decorrelation algorithm was used to detect blood flow.

14 ET showed no significant changes in cerebral blood flow.

15 itically important for the autoregulation of blood flow.

16 reased renal tissue oxygenation and cortical blood flow.

17 single ventricle and very limited pulmonary blood flow.

18 plexus in a process triggered and shaped by blood flow.

19 es: the coupling between neural activity and blood flow.

20 by physiological extremes of O2 tension and blood flow.

21 response to changes in pressure or cerebral blood flow.

22 laser Doppler flowmetry was used to measure blood flow.

23 a phenotype that followed the initiation of blood flow.

24 ar stress, the key biophysical stimulus from blood flow.

25 e VWF A1 domain to GpIbalpha on platelets in blood flow.

26 e change from baseline in total renal artery blood flow.

27 leases heparin to prevent coagulation in the blood flow.

28 h increases intra-tumoral drug perfusion and blood-flow.

29 P=0.001) and presence of antegrade pulmonary blood flow (61% of PDA stents versus 38% of BT shunts; P

30 ine hemodynamics that reduces uteroplacental blood flow, a mechanism underlying maternal smoking-asso

31 ic microscopy enables dynamic measurement of blood flow across the network with capillary-level resol

32 for infants with ductal-dependent pulmonary blood flow adjusted for differences in patient factors,

33 w onset of blood flow and surgically-altered blood flow affects Tie2-GFP expression.

34 METHODS AND Recovery of blood flow after femoral artery ligation was impaired (>

35 es in vitro Initial studies monitoring renal blood flow after IRI did not find significant effects wi

36 oronary capillaries and reduce microvascular blood flow after ischaemia, despite re-opening of the cu

37 ber of brain regions than those for cerebral blood flow alone.

38 tions with Alzheimer's disease than cerebral blood flow alone.

39 Blood flow and (11)C-HED retention index (RI, an indirec

40 In contrast to BAT, cold stress reduces blood flow and (18)F-FDG uptake in subcutaneous WAT, ind

41 s from the blood by maintaining both resting blood flow and activity-evoked changes therein.

42 Hence, cerebral blood flow and cerebral oxygenation are important biomar

43 Resting coronary blood flow and coronary functional hyperaemia are reduce

44 jured vessels by sequestering platelets from blood flow and depositing them to collagen and other exp

45 plantation Cohort Study was to study hepatic blood flow and effect of portal flow modulation on graft

46 ciated with a mild increase in both cerebral blood flow and femoral blood flow (P<0.05 versus normoxi

47 racterized by progressive reduction in renal blood flow and glomerular filtration rate and showed evi

48 fluorodeoxyglucose, to map regional cerebral blood flow and glucose metabolism, and with [(11)C]-isoa

49 escent protein (n=5), improved ischemic limb blood flow and limb muscle histology and restored muscle

50 Moreover, we observed higher cerebral blood flow and lower oxygen saturation in females compar

51 Blood flow and mechanical forces in the ventricle are im

52 r survivors had significantly lower cerebral blood flow and metabolic activity in key brain areas com

53 , oxygen saturation, and indices of cerebral blood flow and metabolism.

54 ilities to measure maximal absolute coronary blood flow and minimal microcirculatory resistance.

55 ctly or indirectly regulate both hippocampal blood flow and neurogenesis.

56 smitters, and releasing factors to influence blood flow and neuronal activity.

57 lood viscosity, which contribute to impaired blood flow and other pathophysiological aspects of diabe

58 thesis rates that match the reduced hindlimb blood flow and oxygen consumption rates in IUGR fetal sh

59 otein synthesis rates match reduced hindlimb blood flow and oxygen consumption rates in the IUGR fetu

60 Prospective study measuring cerebral blood flow and oxygen extraction fraction using O-PET.

61 NT The human preterm brain commonly sustains blood flow and oxygenation disturbances that impair cere

62 While DCE-MRI assesses a combination of blood flow and permeability, ultrasound imaging of micro

63 rtension, and a progressive decline in renal blood flow and renal function.

64 endothelial cells perceive the direction of blood flow and respond through SMAD signaling to regulat

65 ration (pulse oximetry), and brachial artery blood flow and shear rate (ultrasound) were recorded bef

66 ransferred for thrombectomy, poor collateral blood flow and stroke clinical severity are the main det

67 e tumour vasculature, resulting in arrest of blood flow and subsequent collapse of tumours, similar t

68 n mediating the protective effect of laminar blood flow and suggest that Grx1 may be a potential ther

69 ent-reporter mice, we evaluated how onset of blood flow and surgically-altered blood flow affects Tie

70 evealed gradual restoration of the choroidal blood flow and unmasking of the big choroidal vessels.

71 d based on the macroscopic appearance, renal blood flow and urine output.

72 Blood flow and vascular shear stress patterns play a sig

73 Coronary blood flow and vasodilatory responses of coronary arteri

74 e combined with long periods of low cerebral blood flow and/or circulatory arrest.

75 by transcranial Doppler ultrasound (cerebral blood flow) and constant infusion thermodilution (femora

76 ofeeders are the result of impaired cerebral blood flow, and are thus a risk factor for further brain

77 peak, relative blood volume (rBV), relative blood flow, and blood flow velocity were quantified.

78 essment of coronary perfusion and myocardial blood flow, and discuss their application in distinct fo

79 The effects on fetoplacental blood flow, and hence nutrient transfer to the fetus, ar

80 P12 reduces fibrosis, reestablishes coronary blood flow, and improves ventricular function following

81 In addition, renal volume, perfusion, blood flow, and oxygenation were assessed.

82 progressive loss in renal volume, perfusion, blood flow, and oxygenation.

83 s because of the spatial acceleration of the blood flow, and the ejection jet is a single streamline

84 tities, which is a hallmark of microvascular blood flow, appears both in space and time, and that the

85 rentially in vascular regions with disturbed blood flow (arches, branches, and bifurcations).

86 Chronic reductions in carotid artery blood flow are associated with increased carotid body ch

87 ependence on the arterial duct for pulmonary blood flow are often palliated with a shunt usually betw

88 While laminar blood flows are known to maintain endothelial homeostasi

89 k pressure drops because of approximation of blood flow as a single streamline.

90 primarily based on the identification of the blood flow as spiral in the whole arterial system and is

91 and irregular fluctuations in uterine artery blood flow associated with cardiac arrhythmia and high m

92 protective mechanism that maintains cerebral blood flow at a relatively constant level despite fluctu

93 < 0.05) was accompanied by impaired coronary blood flow at rest and during exercise.

94 olism, subcutaneous abdominal adipose tissue blood flow (ATBF), and lipid metabolism in humans.

95 iac conditions with duct-dependent pulmonary blood flow between January 2012 and December 31, 2015.

96 Using ultrasound, we determined regional blood flow, blood vessel sizes, and distensibility in IU

97 reased blood pressure and decreased cerebral blood flow both linked to in vivo biomarkers and clinica

98 nse to ischemia and impaired the recovery of blood flow, both of which were reversed by ANG-(1-7) in

99 Between 2005 and 2012, we measured cerebral blood flow by 2-dimensional phase-contrast magnetic reso

100 n for 120 min increased total renal arterial blood flow by 65% (95% CI 40%, 95%; p < 0.001) from base

101 Augmentation of tissue blood flow by therapeutic ultrasound is thought to rely

102 ion and determination of absolute myocardial blood flow can be achieved noninvasively using dynamic i

103 Here, cerebral blood flow (CBF) and blood oxygen-level dependent (BOLD)

104 s in rat barrel cortex, measured by cerebral blood flow (CBF) and neurophysiological recordings (loca

105 e of pericytes in the regulation of cerebral blood flow (CBF) and neurovascular coupling remains, how

106 Cerebral blood flow (CBF) and oxygen extraction fraction (OEF) ar

107 This pilot study aims to evaluate cerebral blood flow (CBF) and oxygen metabolism (CMRO2) intraoper

108 Resting-state regional cerebral blood flow (CBF) can be measured noninvasively with magn

109 tudy to show regional reductions in cerebral blood flow (CBF) in response to decreased oxygen supply

110 has previously been shown to evoke cerebral blood flow (CBF) increases via the release of the vasodi

111 Cerebral blood flow (CBF) is controlled by arterial blood pressur

112 Cessation of cerebral blood flow (CBF) leads to cell death in the infarct core

113 Changes in cerebral blood flow (CBF) may occur with acute exposure to high a

114 ach may be influenced by changes in cerebral blood flow (CBF) or radiotracer clearance.

115 Cerebral blood flow (CBF) was measured at the internal carotid ar

116 g this training period, hippocampal cerebral blood flow (CBF) was measured by functional magnetic res

117 rs: (1) global and regional resting cerebral blood flow (CBF), (2) oxygen extraction fraction (OEF),

118 d to study structural connectivity, cerebral blood flow (CBF), and corticospinal excitability, respec

119 her brain blood density, lower mean cerebral blood flow (CBF), and significant cerebral circulatory d

120 brain tissue (Pbto2), and regional cerebral blood flow (CBF).

121 rlie variance in medication-induced regional blood flow changes has not been directly tested.

122 resistive index and increased renal cortical blood flow compared to mice with normal TLR4.

123 relaxation along resistance arteries during blood flow control.

124 ricting systems that could finely tune local blood flow depending on neuronal activity changes at the

125 Local sweat rate and/or forearm blood flow differed as a function of menstrual phase and

126 to both non-demented groups, but no cerebral blood flow differences between non-demented amyloid-posi

127 However, increased blood flow does not necessarily mean increased oxygen te

128 Interestingly, using fully developed steady blood flow does not result in a representative number of

129 In healthy subjects, we measured forearm blood flow (Doppler ultrasound) and calculated changes i

130 vein (CV) in Ncx1 (-/-) mutants, which lack blood flow due to a deficiency in a sodium calcium ion e

131 on emission tomography to determine striatal blood flow during active atypical antipsychotic medicati

132 ore pronounced increases observed in femoral blood flow during exercise (P<0.05 versus rest) in propo

133 caused by loss and subsequent restoration of blood flow during organ procurement, and prolonged ische

134 giogenesis and ensure maintenance of uterine blood flow during pregnancy.

135 dy may help further understand the different blood flow dynamics pattern in MAs.

136 , brain tissue, inflammatory mechanisms, and blood flow dynamics.

137 The difference in performance between blood flow, expressed as relative to remote myocardium,

138 shed inferior parietal and temporal cerebral blood flow for patients with Alzheimer's disease (n = 33

139 ngiopoietins is required in combination with blood flow for the formation of a functional vascular ne

140 At later stages of blood flow, Foxc2 and calcineurin-Nfat signaling are eac

141 statin resulted in a progressive increase in blood flow from baseline (both P < 0.001).

142 confirmed that these reductions in cerebral blood flow from hypoxia were related to vasoconstriction

143 plifying physiological processes that govern blood flow, hemostasis, inflammation, and angiogenesis.

144 -salt diet, Tmem27(Y/-) mice had lower renal blood flow, higher abundance of renal sodium-hydrogen an

145 the physical structure to form conduits for blood flow; however, the involvement of ECs in the proce

146 scatter bias for accurate cardiac myocardial blood flow imaging were 3-14 MBq/kg, 1.5-4.0, 22-64 Mcps

147 ether Doppler endoscopic probe monitoring of blood flow improves risk stratification and outcomes in

148 to suppress projection artifacts and resolve blood flow in 3 distinct parafoveal plexuses.

149 To study choriocapillaris blood flow in age-related macular degeneration (AMD) usi

150 neous coronary intervention (PCI) to restore blood flow in an infarct-related coronary artery improve

151 sonance imaging to measure regional cerebral blood flow in brain regions susceptible to ageing and Al

152 However, the measurement of retinal blood flow in clinical studies has been challenging.

153 Nonpulsatile pulmonary blood flow in Fontan circulation results in pulmonary va

154 uting of recipient capillaries reestablished blood flow in grafted islets.

155 correspondingly, greater reductions in skin blood flow in HTN.

156 lls (MPC) form vascular networks and restore blood flow in ischemic skeletal muscle, and whether host

157 + MPC delivery could be used to reestablish blood flow in ischemic tissues, and this may be enhanced

158 magnetic nanoparticles to reversibly occlude blood flow in microvessels.

159 ad as well, thereby improving subendocardial blood flow in patients with HF.

160 ia in mice for >24 hours and increase muscle blood flow in patients with sickle cell disease.

161 ct numerical simulation of 3D cellular-scale blood flow in physiologically realistic microvascular ne

162 y that provides a direct measure of cerebral blood flow in response to cognitive activity.

163 brinolysis, platelet activation, and forearm blood flow in response to intra-arterial infusions of en

164 endothelial (MVE) mechanosensors that sense blood flow in response to mechanical and chemical stimul

165 ranscranial Doppler, measurement of cerebral blood flow in response to transient changes in arterial

166 Abnormal sickle-shaped erythrocytes disrupt blood flow in small vessels, and this vaso-occlusion lea

167 nowledge, these novel findings are absent in blood flow in straight tubes, and they underscore the im

168 tification of mechanisms governing capillary blood flow in the CNS and how they are altered in diseas

169 The functions of blood flow in the morphogenesis of mammalian arteries an

170 tomographic angiography (OCTA) have measured blood flow in the retinal capillary layers in children w

171 centile and absent or reversed end-diastolic blood flow in the umbilical artery on Doppler velocimetr

172 ndividuals showed increased resting cerebral blood flow in the ventral striatum and ventromedial pref

173 Cerebral blood flow in these brain regions correlated with the de

174 intracranial thrombus that impedes ascending blood flow) in the context of acute stroke.

175 ust be playing hand in hand, namely cerebral blood flow increase and microvascular flow homogenizatio

176 Similar to seizures, cerebral blood flow increases in patients with PDs to compensate

177 Concomitant analysis of brain venous blood flow indicated that CSF and venous flux act as clo

178 elevated ratio of blood pressure to cerebral blood flow, indicative of cerebrovascular resistance, wo

179 In a culture model of blood flow-induced shear stress, human coronary artery e

180 Blood flow influences atherosclerosis by generating wall

181 ssion and endothelial cell migration against blood flow into developing arteries.

182 elial cells transduce mechanical forces from blood flow into intracellular signals required for vascu

183 Blood flow into the brain is dynamically regulated to sa

184 ses upstream arteriolar dilation, increasing blood flow into the capillary bed.

185 ucing FA resistance that otherwise restricts blood flow into the microcirculation.

186 Adequate blood flow is essential to brain function, and its disru

187 ounts for the cross-sectional profile of the blood flow is proposed and adapted to both cardiovascula

188 Overall, blood flow is the best independent predictor of (11)C-HE

189 ired oxygen delivery due to reduced cerebral blood flow is the hallmark of delayed cerebral ischemia

190 Infants with ductal-dependent pulmonary blood flow may undergo palliation with either a patent d

191 d that the integration of maximal myocardial blood flow (MBF) and coronary flow reserve (CFR), termed

192 Myocardial blood flow (MBF) and myocardial flow reserve (MFR) measu

193 Myocardial blood flow (MBF) is the critical determinant of cardiac

194 2 (PETco2) increases cerebral and myocardial blood flow (MBF), suggesting that it may be a suitable a

195 We propose a molecular mechanism by which blood flow mediates DA and CV morphogenesis, by regulati

196 ucoma and normal eyes and correlations among blood flow metrics, VF thresholds, and clinical optical

197 or (18)F-GP1 was investigated by an in vitro blood flow model.

198 ying perfusion and to screen the efficacy of blood flow-modifying drugs for use as adjuvants to exist

199 e ; P < 0.01) and greater reductions in skin blood flow (NTN: -16 +/- 2%baseline vs. HTN: -28 +/- 3%b

200 with anti-Gr-1 antibody blocked the improved blood flow observed with ECFC + MPC and reduced ECFC and

201 up-regulated by the anti-atherogenic laminar blood flow often seen in straight or unbranched regions

202 ed tool for the quantification of myocardial blood flow, other modalities, including single-photon em

203 ruction and their relationship with cerebral blood flow, oxygen delivery, and carbon dioxide reactivi

204 ease in both cerebral blood flow and femoral blood flow (P<0.05 versus normoxia) with further, more p

205 ectively) with increasing amount of cerebral blood flow (P<0.05).

206 Infants with ductal-dependent pulmonary blood flow palliated with either a PDA stent or a BT shu

207 changes in artery sizes, distensibility, and blood flow pattern in young adult IUGR baboons, which ma

208 pplied to the data to provide information on blood flow patterns in major organs.

209 sults clearly revealed the complex nature of blood-flow patterns in the embryo with fine-temporal and

210 understood, however previous work indicates blood flow per se can affect CB function.

211 ained with oxygen-15 water regional cerebral blood flow PET in 39 healthy women genotyped for BDNF Va

212 Alterations in ocular blood flow play an important role in the pathogenesis an

213 Changes to capillary blood flow precede other anatomical and functional hallm

214 Myocardial contractility and blood flow provide essential mechanical cues for the mor

215 ely vessel diameter (D), blood velocity (V), blood flow (Q), wall shear rate (WSR), and wall shear st

216 Thus, we identified a mechanism by which blood flow quality balances artery growth and maturation

217 1), and rBV (r = 0.71, P = .01) and relative blood flow (r = 0.78, P = .005) correlated well with per

218 ng functional information from scans such as blood flow rate and oxygen consumption provides new pers

219 To determine whether blood flow rate influences circuit life in continuous re

220 cid and p-cresyl glucuronide, an increase in blood flow rate is advantageous.

221 Continuous renal replacement therapy using blood flow rate set at 250 mL/min was not more likely to

222 These adaptations resulted in hindlimb blood flow rates in IUGR that were similar to control fe

223 no difference in circuit life whether using blood flow rates of 250 or 150 mL/min during continuous

224 tients were randomized to receive one of two blood flow rates: 150 or 250 mL/min.

225 clerotic renal artery stenosis reduces renal blood flow (RBF) and amplifies stenotic kidney hypoxia.

226 tic renovascular disease (RVD) reduces renal blood flow (RBF) and GFR and accelerates poststenotic ki

227 age and diagnosis on glutamate and cerebral blood flow (rCBF) in adults with SZ and healthy controls

228 We measured regional cerebral blood flow (rCBF) using pseudo-continuous arterial spin

229 viously validated measure (relative cerebral blood flow [rCBF], <30%), thrombectomy patients had a sm

230 imbs injected with ECFC + MPC showed greater blood flow recovery compared with ECFC, MPC, or vehicle.

231 of IL-12 promotes angiogenesis and increases blood flow recovery in obese type 2 diabetic mice by an

232 The mechanism of blood flow recovery involves an increase in capillary/ar

233 However, after 12 weeks of an HFD, blood flow recovery was compromised in WT mice, whereas

234 Blood flow recovery was fully restored in 2 to 3 weeks a

235 f an HFD, the femoral artery was ligated and blood flow recovery was measured every week for 4 weeks.

236 duced mobilization of LSK cells and impaired blood flow recovery, which was associated with decreased

237 s process and can be precipitated by chronic blood flow reduction.

238 rts a potential role of intravascular ATP in blood flow regulation during exercise in humans.

239 rosclerosis impacts arteries where disturbed blood flow renders the endothelium susceptible to inflam

240 fness, and eliminated impairment of coronary blood flow responses and endothelium-dependent vasodilat

241 st this, we measured NBG, BOLD, and cerebral blood flow responses to stimuli that either correlate or

242 eveal a role for hypoxia driven by capillary blood flow restriction in ictal neurodegeneration.

243 utrient supply to the fetus affects hindlimb blood flow, substrate uptake and protein accretion rates

244 odels of cirrhosis without changes in portal blood flow, suggesting a reduction in IHVR.

245 the biochemical pathways that control local blood flow supply in the cerebellum are unclear.

246 Blood flow supply to the CB region was decreased only in

247 rated that BDG patients had greater cerebral blood flow than did Fontan patients and that an inverse

248 G patients had significantly higher cerebral blood flow than did Fontan patients.

249 , allowing for a rapid increase in pulmonary blood flow that is essential for efficient gas exchange.

250 The arterial blood flow that underlies the stigmata rarely is monitor

251 analyses of CSF dynamics and cerebral venous blood flow, that is, in epidural veins at cervical level

252 lows the quantification of regional cerebral blood flow, the regional oxygen extraction fraction, and

253 yofiber VEGF to be necessary for maintaining blood flow through hippocampal regions independent of ex

254 MRI allowed the quantification of blood flow through the arteries, which was decreased in

255 nment of the FN pillars depends on pulsatile blood flow through the dorsal aortae.

256 However, resting blood flow through the hippocampal region was lower in V

257 rate [h(-1)], 52.6 for Kprest, 0.01 for the blood flow through the pericardial fluid [L/h], and 0.78

258 'inside-out' electrical signaling to direct blood flow to active brain regions.

259 locally constrict capillaries, which reduces blood flow to ischemic levels.

260 ce showing how a stimulus can increase local blood flow to the brain despite suppressing gamma power.

261 ques within the artery wall that can occlude blood flow to the heart and cause myocardial infarction.

262 (PCI) induced by repetitive interruptions of blood flow to the ischemic myocardial region immediately

263 After 10 h in hypoxia, decreased blood flow to the major nodes of the default mode networ

264 Sufficient blood flow to tissues relies on arterial blood vessels,

265 y therefore require treatments that modulate blood flow to utilize endogenous VEGF, in combination wi

266 effective method for measuring total retinal blood flow (TRBF) in the clinic.

267 Using treatments enabling blood flow tuning, we demonstrated that such a method is

268 hemostasis assisted by Doppler monitoring of blood flow under the stigmata (n = 72).

269 lication followed by measurement of cerebral blood flow using a combination of laser Doppler flowmetr

270 nvasive recordings of changes in the brain's blood flow using functional magnetic resonance imaging a

271 zolamide-induced change in regional cerebral blood flow using SPECT with (99m)Tc-labeled hexamethylpr

272 baboons there was increased carotid arterial blood flow velocity during late systole and diastole.

273 motion phases, contrast-agent dynamics, and blood flow velocity fields.

274 Peak aortic blood flow velocity measured at 24 hours was a good disc

275 blood volume (rBV), relative blood flow, and blood flow velocity were quantified.

276 High extracorporeal blood flow venovenous extracorporeal membrane oxygenatio

277 ontributes to IUGR by compromising umbilical blood flow via oxidative stress, reducing hydrogen sulfi

278 cells release ATP in response to changes in blood flow (via shear stress) or hypoxia, to act on P2 r

279 Purpose To estimate reference ranges for blood flow volume (BFV) in major splanchnic, thoracoabdo

280 was 9.7 g/dL (range, 6.9-12.9), and cerebral blood flow was 43 +/- 11 mL/100 g/min.

281 (15)O-H2O data showed that blood flow was decreased in AD compared with aMCI in cor

282 Blood flow was determined by transcranial Doppler ultras

283 vascular flow rate; based on this technique, blood flow was not significantly altered 30min after tre

284 relative to remote myocardium, and absolute blood flow was not statistically significant (areas unde

285 Blood flow was quantified by (15)O-H2O SUV.

286 The adequacy of leptomeningeal collateral blood flow was rated as no or poor, decreased, adequate,

287 Absolute hindlimb blood flow was reduced in IUGR (IUGR: 32.9 +/- 5.6 ml mi

288 Regional striatal blood flow was significantly higher during active treatm

289 tion-based assessment of volumetric coronary blood flow, we observed that intracoronary infusion of s

290 in vitro assay under shear forces mimicking blood flow, we observed that reduction of endothelial mi

291 ddition, (15)O-H2O scans to measure cerebral blood flow were acquired before each (11)C-erlotinib sca

292 cation-related increases in ventral striatal blood flow were associated with more robust amelioration

293 Quantitative perfusion parameters, such as blood flow, were calculated by parametric deconvolution

294 ypoxia causes an increase in global cerebral blood flow, which maintains global cerebral oxygen deliv

295 ere was a dose-dependent increase in forearm blood flow with all vasodilators (P<0.001), which was at

296 mbining nitric oxide (NO)-mediated increased blood flow with angiopoietin-1-Tie2 receptor signalling

297 We have demonstrated that acutely disturbed blood flow with increased retrograde shear stress furthe

298 mic imaging for quantification of myocardial blood flow with short-lived tracers, such as (82)Rb-chlo

299 iation exists of various indexes of cerebral blood flow with these brain lesions.

300 nd constant infusion thermodilution (femoral blood flow) with net exchange calculated via the Fick pr