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1 ative angiography and Doppler measurement of blood flow velocity.
2 between arterial blood pressure and cerebral blood flow velocity.
3 ndependently of beta-cell expansion or islet blood flow velocity.
4 a frame count method was used to quantitate blood flow velocity.
5 hy scan and rapidly increasing mean cerebral blood flow velocities.
7 a substantially larger increment in coronary blood flow velocity (0.51 versus 0.14 m/s, P <0.001).
8 4% +/- 24 [adenosine]; P = .80) and coronary blood flow velocity (21% +/- 16 [hypercapnia] vs 26% +/-
9 age reductions of mean (+/- s.e.m.) cerebral blood flow velocity after 10 min upright tilt were small
11 Restriction of vessel diameter increased blood flow velocity and caused reduction in vascular ACE
12 on depth information regarding blood volume, blood flow velocity and direction, vascular architecture
14 emia: NE evoked similar decreases in femoral blood flow velocity and femoral vascular conductance (FV
16 olor Doppler imaging, suggest that decreased blood flow velocity and increased vascular resistance ar
17 ([Cl] 98 mmol/L, Baxter Healthcare) on renal blood flow velocity and perfusion in humans using magnet
20 althcare, Thetford, United Kingdom] on renal blood flow velocity and renal cortical tissue perfusion
21 f 0.9% saline results in reductions in renal blood flow velocity and renal cortical tissue perfusion.
22 poral sequence of changes of cerebral artery blood flow velocity and systemic arterial pressure in 15
23 In addition, quantitative measurements of blood flow velocity and tissue perfusion will be feasibl
24 e measured as the product of the transaortic blood flow velocity and transesophageal echocardiographi
26 t changes in retinal and ONH morphology, ONH blood flow velocity, and retinal and cortical pattern-de
28 , the systolic (PSV), end-diastolic and mean blood flow velocities as well as pulsatility and resista
29 Lower peak-systolic, end-diastolic, and mean blood flow velocities at baseline were associated with h
30 se apparent viscosity significantly, slowing blood flow velocity at arterial oxygen tension even with
31 recordings of ascending aortic pressure and blood flow velocity at rest and with supine bicycle exer
32 udy was to assess serial changes in coronary blood flow velocity before and after Rotablator atherect
33 ntified the coupling between BP and cerebral blood flow velocity (BFV) using transfer function analys
37 e of ticagrelor versus prasugrel on coronary blood flow velocity (CBFV) during increasing doses of in
38 changes in blood pressure (BP) and cerebral blood flow velocity (CBFV) in 13 healthy subjects (30 +/
39 nutes after indomethacin ingestion, cerebral blood flow velocity (CBFV) in the middle cerebral artery
41 ts indicated a morning reduction in cerebral blood flow velocity (CBFV) relative to values from the p
45 anial Doppler ultrasound was used to measure blood flow velocity (CFV) in the middle cerebral artery
46 nteric artery revealed a twofold increase in blood flow velocity compared with tumor-free mice (P < 0
49 R) was assessed intermittently from brachial blood flow velocity (Doppler ultrasound) divided by mean
51 lood pressure (Finapres) divided by brachial blood flow velocity (Doppler) and cardiac responses from
52 baboons there was increased carotid arterial blood flow velocity during late systole and diastole.
54 tions, one optimized for arterial and venous blood flow (velocity encoding range, +/-50 cm/sec) and t
58 methodologies for simultaneously quantifying blood flow (velocity, flux, hematocrit and shear rate) i
59 in children with elevated cerebral arterial blood flow velocity, further study is required to determ
60 c vasospasm, the mean time for mean cerebral blood flow velocities >120 cm/sec was 7.0 +/- 3 days (p
62 gnetic resonance imaging measured in vivo 3D blood flow velocities in 60 AF patients and 15 controls.
63 d was better at detecting high mean cerebral blood flow velocities in patients with symptomatic vasos
68 FR) was calculated as the ratio of pulmonary blood flow velocity in response to Ach relative to basel
69 Doppler ultrasonography to demonstrate that blood flow velocity in the anterior cerebral artery is h
70 transcranial Doppler sonography showed that blood flow velocity in the anterior cerebral artery is s
71 the ratio of hyperemic to baseline coronary blood flow velocity in the left anterior descending coro
75 dies that showed that the time-averaged mean blood-flow velocity in the internal carotid or middle ce
76 f mean arterial pressure and cerebral artery blood flow velocity, in the very low- (0.02-0.07 Hz), lo
78 r-mediated, whereas the increase in coronary blood flow velocity is due to activation of A2 adenosine
79 blood volume without altering microvascular blood flow velocity, leading to a significantly increase
80 -beat measurements of middle cerebral artery blood flow velocity (MCAv; transcranial Doppler), heart
81 nts, we recorded middle cerebral artery mean blood flow velocity (MCAVm) using transcranial Doppler u
82 lectrocardiogram) and middle cerebral artery blood flow velocities (mean, total, mean/RR interval; Go
83 rast analysis were compared with the retinal blood flow velocities measured by video fluorescein angi
85 by intravascular ultrasound), renal Doppler blood flow velocity (measured by a Doppler flow wire in
87 rovascular blood volume (MBV), microvascular blood flow velocity (MFV), and microvascular blood flow
88 nal arterial replacement is clear, the lower blood flow velocities of small-diameter arteries like th
89 ured rcSO2 using near-infrared spectroscopy, blood flow velocities of the middle cerebral artery, and
90 01 compared with pretreatment angiogram) and blood flow velocity (p < 0.01 compared with pretreatment
91 determine whether changes of cerebral artery blood flow velocity precede or follow reductions of arte
93 action and relaxation, it increases coronary blood flow velocity, predominantly by increasing the dom
96 ance: This study suggests that a low retinal blood flow velocity reflects in a visually distinct cont
99 ed significantly (P<0.0001) lower myocardial blood flow velocity reserve in vascular territories subt
100 epicardial cross-sectional area and coronary blood flow velocity, resulting in an increase in absolut
101 ity and precise, simultaneous calculation of blood flow velocity, shear stress and drug distribution.
102 tion, and 8 with old myocardial infarction), blood flow velocities through the LVOT were recorded usi
105 ood flow was calculated from measurements of blood flow velocity using intracoronary Doppler and coro
106 acute (3-7 d) ischemia-induced increases in blood flow velocity, vessel lumen diameter, and red bloo
107 t impairment of microhemodynamics, including blood flow velocity, volumetric blood flow, and function
108 smaller than SV luminal diameters (P=0.029), blood flow velocity was greater in RA than SV (P=0.008),
111 adenosine; however, the increase in coronary blood flow velocity was not significantly affected.
112 er treatment, Doppler sonography showed that blood flow velocity was preserved in AdCOX-1-treated art
113 interval, chest pain severity, and coronary blood flow velocity were made before and after low-dose
114 transcranial Doppler middle cerebral artery blood flow velocity were measured during 5 min of sponta
117 volume [rBV], relative blood flow [rBF], and blood flow velocity) were measured on both 3D and 2D dat
118 ssessed by measuring the change in pulmonary blood flow velocity with a Doppler-tipped wire and the m
119 ured with intravascular ultrasound and renal blood flow velocity with the aid of an intravascular Dop
120 aditionally required measurement of coronary blood flow velocity with the Doppler wire and, more rece
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