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1 lopment of high throughput methods for whole blood folate.
2 with the Lactobacillus casei assay for whole blood folate.
3 )]pABA as IS and m/z 318 for pABA from whole blood folate.
4 e requirement with 5-CH3THF, the predominant blood folate.
5 C1) is an important route by which the major blood folate, 5-methyltetrahydrofolate, is transported i
6                We describe an improved whole blood folate analysis method that facilitates increased
7 lar disease, presumably because of increased blood folate and decreased blood homocysteine in the pop
8 coding folate-metabolizing enzymes influence blood folate and homocysteine concentrations, but the ef
9 re was a nonlinear relationship between cord blood folate and sensitization, with folate levels <50 n
10 ck of confidence in results of current whole blood folate assays have limited its popularity for asse
11 embryonic folate concentration, not maternal blood folate concentration.
12 ssociation between MTHFR C677T genotypes and blood folate concentrations among healthy women aged 12-
13 id intake is the most important predictor of blood folate concentrations among nonpregnant women, but
14 l and clinical studies, where accurate whole blood folate concentrations are needed.
15                                        Lower blood folate concentrations associated with this polymor
16 s for estimates of percentage differences in blood folate concentrations between genotypes.
17                                     However, blood folate concentrations have decreased from their po
18 ntakes during pregnancy on maternal and cord blood folate concentrations have not been well establish
19                                      Average blood folate concentrations increased and homocysteine c
20             The effect of reporting error on blood folate concentrations is important in interpreting
21 ry measures for other folate indicators (eg, blood folate concentrations) and asthma or allergy-relat
22 pacity, which has been associated with lower blood folate concentrations.
23 fect of reporting error of supplement use on blood folate concentrations.
24 ave adequate folate nutriture as assessed by blood folate concentrations.
25 late restriction results in reduced maternal blood folate, elevated plasma homocysteine and reduced e
26                                Neonatal cord blood folate, homocysteine and vitamin B12 levels were m
27 conversion-based homogeneous assay for whole blood folate is introduced based on resonance energy tra
28                                        Whole blood folate level is a superior indicator of folate nut
29 dietary folates, resulting in relatively low blood folate levels and consequent hyperhomocysteinemia.
30 tion of dietary folates that precipitate low blood folate levels and hyperhomocysteinemia have not be
31                                          Low blood folate levels result in hyperhomocysteinemia, whic
32 have previously been associated with reduced blood folate levels, especially among patients with low-
33 sity may depend on nutritional status (e.g., blood folate levels, intake of vitamins) or on the genot
34  dietary folates is the chief determinant of blood folate levels.
35  provides a simple and fast method for whole blood folate measurement.
36 al assay rather than alternative methods for blood folate measurement.
37 ient range (serum folate: <6.8 nmol/L), cord blood folate status (median: 40.2 nmol/L) was similar to
38  of pregnancy can increase maternal and cord blood folate status and prevent the increase in homocyst
39 nts for folate are paramount, such that cord blood folate status is maintained, even when maternal st
40 d samples were obtained for determination of blood folates, vitamin B(12), homocysteine, DNA methylat
41                                         Cord blood folate was significantly higher in the FA group th

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