ライフサイエンスコーパス: blood group

1 o receive standard-issue red cells (standard-blood group).

2 d, consequently, manifesting the rare In(Lu) blood group.

3 confirm SMIM1 as the gene underlying the Vel blood group.

4 ule and stratified by centre and patient ABO blood group.

5 tion occurred in secretors regardless of ABO blood group.

6 he highest rate (51%) among secretors with O blood group.

7 be accounted for by the A1 allele of the ABO blood group.

8 nding to fucosylated ABH antigens of the ABO blood group.

9 pancreatic cancer include smoking and non-O blood groups.

10 ereas in erythrocytes it determines the Kidd blood groups.

11 selective force affecting the expression of blood groups.

12 cur preferentially in individuals of certain blood groups.

13 ed ABH were expressed on erythrocytes of all blood groups.

14 ce treatment with MHA mixed with whole human blood; group 6 (G6) implant samples were treated in the

15 3 (7%), alpha thalassaemia 438 (28%), type O blood group 621 (40%), and G6PD deficiency 72 (9%) in 76

16 functionalized with immunoglobulin G against blood group A (anti-A IgG) by forming a self-assembled m

17 The accuracy for detecting blood group A (n=12), B (n=13), AB (n=9), O (n=14), and

18 We show that mAb F77 can also bind to blood group A and B analogs but with lower intensities.

19 rase (GTB) catalyze the final step in ABO(H) blood group A and B antigen synthesis through sugar tran

20 lates gastrointestinal mucosal expression of blood group A and B antigens) (rs602662, P=3.4x10(-5)).

21 nal step of the biosynthesis of ABO(H) human blood group A and B antigens.

22 igands supported this preference over type 1 blood group A and B oligosaccharides.

23 cause in-hospital mortality in patients with blood group A and O who were transfused.

24 In analogy with histo-blood group A antigen, Forssman (Fs) antigen terminates

25 Acbeta3Galalpha4Galbeta4Glcbeta1Cer), i.e. a blood group A determinant on a type 4 core chain.

26 ulfated and methylated variants of the histo-blood group A epitope.

27 recombinant lectin, rCNL, agglutinates human blood group A erythrocytes and is specific for the uniqu

28 asite glycans structurally distinct from the blood group A oligosaccharides on the hemocyte surface m

29 that CvGal1 preferentially recognizes type 2 blood group A oligosaccharides.

30 Blood group A or B patients had lower antibody levels th

31 sitization, donor blood group O to recipient blood group A or B transfer, and ciclosporin treatment h

32 lood stored 7 days or less (in patients with blood group A or O: 0.92, 0.74-1.15, p=0.48; in all 0.90

33 blood product use history (in patients with blood group A or O: hazard ratio 0.94, 95% CI 0.73-1.20,

34 Blood group A subjects had higher levels of anti-pig IgM

35 osylceramide) were identified as well as the blood group A type 1 hexaosylceramide.

36 B6 wild-type (WT) mice were sensitized with blood group A-human erythrocytes; others received passiv

37 d no longer than 7 days, both in patients of blood groups A and O and all patients.

38 Blood groups A, AB and B significantly reduced IE bindin

39 ound sulfated disaccharides as well as human blood groups A, B, and H on both N-glycans and linear gl

40 ors for parasite invasion, such as the Duffy blood group, a receptor for Plasmodium vivax, and the Ge

41 Blood group ABH(O) carbohydrate antigens are carried by

42 , cytomegalovirus serostatus, HLA match, and blood group ABO match) and survival after transplantatio

43 (arylamine N-acetyltransferase) (NAT2), ABO blood group (ABO), and GRB2 associated binding protein 3

44 RBCs fixation on paper accurately detected blood groups (ABO and RhD) using ascending buffer for 10

45 Although blood group active O-glycans of the Lewis-type form the

46 Red cells lacking or having altered forms of blood group-active molecules are commonly found in regio

47 oth human adult and fetal globins as well Rh blood group Ag which persisted for 3 weeks and the reten

48 Comprehensive molecular blood group analysis using 3 genotyping platforms, nucle

49 t 90 days, 448 patients (37.0%) in the fresh-blood group and 430 patients (35.3%) in the standard-blo

50 ed summary odds ratio estimates according to blood group and by populations endemic versus nonendemic

51 The absence of an association between ABO blood group and platelet-bound P-selectin levels in an i

52 Previous genetic studies of blood group and serum markers suggested that Jewish grou

53 ed the effects of host age, hemoglobin type, blood group and severe malaria on levels of IE adhesion

54 risks of death and relapse between the cord-blood group and the two other unrelated-donor groups app

55 ample of the potential for basic research on blood groups and malaria to be translated into a vaccine

56 Studies over 5 decades have examined ABO blood groups and risk of pancreatic cancer in Western, A

57 s to investigate the association between ABO blood groups and the incidence of first and recurrent ve

58 e assigned to receive fresh red cells (fresh-blood group) and 1219 patients were assigned to receive

59 arison after adjustment for VWF:Ag, FVIII:C, blood group, and age.

60 en (VWF:Ag), factor VIII activity (FVIII:C), blood group, and age.

61 l trait (HbAS) sickle cell polymorphism, ABO blood group, and other hemoglobinopathies remain the few

62 haemoglobin C (HbC), alpha thalassaemia, ABO blood groups, and glucose-6-phosphate dehydrogenase (G6P

63 s, there were no changes in the viral or the blood group antibodies in these patients.

64 o P. marinus trophozoites, no binding of ABH blood group antibodies was observed.

65 nt non-donor specific HLA antibody, viral or blood group antibody rise was found.

66 onors for patients with low to moderate anti-blood group antibody significantly increases transplant

67 the first to examine the distribution of ABO blood group antibody titers in a population of pediatric

68 atible donors is allowed in cases where anti-blood group antibody titres are deemed amenable to remov

69 Techniques for quantifying blood group antibody-antigen interactions are also very

70 icle (VLP) antigenic relationships and histo-blood group antigen (HBGA) binding profiles with strains

71 nked immunosorbent assay (ELISA) and a histo-blood group antigen (HBGA) blocking assay using sera fro

72 s influenced by genetically determined histo-blood group antigen (HBGA) expression.

73 trains correlates with an individual's histo-blood group antigen (HBGA) profile, the biological basis

74 rus-like particle (VLP) binding to the histo-blood group antigen (HBGA) receptors.

75 e change, Q396R, is able to modify the histo-blood group antigen (HBGA) recognition pattern.

76 Histo-blood group antigen (HBGA) typing of saliva showed that

77 strain specifically recognizes A-type histo-blood group antigen (HBGA) using a glycan array screen c

78 a region topologically similar to the histo-blood group antigen (HBGA)-binding sites of the human no

79 on waveguides is applied to the detection of blood group antigen A on whole erythrocytes.

80 able to infect humans lacking the Duffy (Fy) blood group antigen because this receptor is critical fo

81 Expression of the Helicobacter pylori blood group antigen binding adhesin A (BabA) is more com

82 s in genogroups I and II interact with histo-blood group antigen carbohydrates, bovine noroviruses (g

83 Previous studies utilized SPR for blood group antigen detection, however, showed poor rege

84 The Blood Group Antigen Gene Mutation Database (BGMUT) is an

85 The Vel blood group antigen is expressed on the red blood cells

86 genes, which encode a serologically distinct blood group antigen known as Dantu.

87 is dependent on the expression of the Duffy blood group antigen on erythrocytes.

88 ysis suggested pathways related to the histo-blood group antigen production, and the regulation of io

89 inity to glycophorin A, a well-studied human blood group antigen that forms TM homodimers.

90 P. vivax binds the Duffy blood group antigen through its Duffy-binding protein 1

91 throcyte proteins, we have found that the Ok blood group antigen, basigin, is a receptor for PfRh5, a

92 nsfected with either vector expressed the P1 blood group antigen, which was absent from untransfected

93 pylori outer membrane proteins, such as the blood group antigen-binding adhesin (BabA), are associat

94 re to the human gastric epithelium using the blood group antigen-binding adhesin (BabA).

95 e intestinal tract, recognize and kill human blood group antigen-expressing Escherichia coli while fa

96 ty binding of Helicobacter pylori to the ABO blood group antigen-glycosylated gastric mucosa.

97 In comparison to blood group antigen-specific IA, nonantigen-specific IA

98 d cost of nonantigen-specific as compared to blood group antigen-specific IA.

99 cell's surface receptor, known as the Duffy blood-group antigen (Fy).

100 Fy is an important minor blood-group antigen that has two immunologically distinc

101 oth norovirus and rotavirus, involving histo-blood group antigenic associations, which may also prove

102 vealed the involvement of type A and B histo-blood group antigens (HBGA) in TV infection.

103 ibit both strains of VLPs' bindings to histo-blood group antigens (HBGA) receptors on human cells at

104 block the interaction of VLPs with ABH histo-blood group antigens (HBGA), suggesting that multiple an

105 ns have shown the association of human histo-blood group antigens (HBGAs) and susceptibility to infec

106 Histo-blood group antigens (HBGAs) are a source of antigenic v

107 Histo-blood group antigens (HBGAs) are norovirus binding ligan

108 worldwide, and interactions with human histo-blood group antigens (HBGAs) are thought to play a criti

109 taviruses are known to recognize human histo-blood group antigens (HBGAs) as a host ligand that is be

110 NoVs use histo-blood group antigens (HBGAs) as attachment factors.

111 oviruses (NoVs) are known to recognize histo-blood group antigens (HBGAs) as attachment factors.

112 The discovery of human histo-blood group antigens (HBGAs) as receptors or ligands of

113 Histo-blood group antigens (HBGAs) expressed on enterocytes ar

114 ing of the RV surface protein (VP4) to histo-blood group antigens (HBGAs) in an RV genotype-dependent

115 Histo-blood group antigens (HBGAs) in saliva and gut recognize

116 GI VLPs bound to histo-blood group antigens (HBGAs) including fucose, Lewis, an

117 rface carbohydrate structures known as histo-blood group antigens (HBGAs) prior to internalization, a

118 Noroviruses bind to gut histo-blood group antigens (HBGAs), but only 70%-80% of indivi

119 nsialylated glycoconjugates, including histo-blood group antigens (HBGAs), in the infectivity of huma

120 the genus Norovirus and recognizes the histo-blood group antigens (HBGAs), similarly to human norovir

121 had no binding affinity for synthetic histo-blood group antigens (HBGAs), suggesting that PSVs could

122 ic gastroenteritis, is associated with histo-blood group antigens (HBGAs), which are also cell attach

123 olled secretor-dependent expression of histo-blood group antigens (HBGAs), which are also critical fo

124 rocess have been identified, including human blood group antigens (HBGAs).

125 otype-dependent binding to nonsecretor histo-blood group antigens (HBGAs).

126 (P[4], P[6], and P[8]) recognize human histo-blood group antigens (HBGAs).

127 human RVs interacts with the secretor histo-blood group antigens (HBGAs).

128 n cores 2, 4, and 6, as well as type 1 histo-blood group antigens (HBGAs).

129 The presence of histo-blood group antigens (secretor, ABO, and Lewis type) was

130 On the other hand, histo-blood group antigens also present in the cell surface ca

131 he interaction between malaria parasites and blood group antigens and discusses how the knowledge gle

132 FUT2 participates in the production of histo-blood group antigens and has previously been implicated

133 ligosaccharides, on the basis of human histo-blood group antigens and milk oligosaccharides, against

134 e determines the secretor phenotype in which blood group antigens are found in non-blood body fluids.

135 Analogous to human leukocyte antigens, blood group antigens are surface markers on the erythroc

136 y charged, but neutral glycans such as histo-blood group antigens can also function as receptors.

137 The expression of ABO(H) blood group antigens causes deletion of cells that gener

138 As expected, IgG and IgM antibody signals to blood group antigens correlated strongly with blood type

139 ting enzyme effects more complete removal of blood group antigens from cell surfaces, demonstrating t

140 the molecular bases of almost all the major blood group antigens have been determined.

141 nonsecretors and are unable to express histo-blood group antigens in secretions and on mucosal surfac

142 ess the BabA adhesin, which binds to ABO/Leb blood group antigens on gastric mucin and epithelial cel

143 g made in understanding the effects of other blood group antigens on malaria.

144 pean descent affects the expression of histo-blood group antigens on the mucosal epithelia of human r

145 in particular terminal fucosylated Lewis and blood group antigens present on N- and O-glycans.

146 The interplay between malaria parasites and blood group antigens remains a fascinating subject with

147 These histo-blood group antigens serve as host receptor sites necess

148 ve calculated the immunogenicities of common blood group antigens using data collected on clinically

149 cluding high mannose structures, fucosylated blood group antigens, and glycans with terminal 6-sulfat

150 NVs also were identified in genes regulating blood group antigens, coat color, fecundity, lactation,

151 CR1 contains the Knops blood group antigens, including the antithetical pairs S

152 Both viruses recognize and bind to histo-blood group antigens, which are expressed by the fucosyl

153 up IV and VI viruses can interact with histo-blood group antigens.

154 and binds glycans, including the Lewis histo-blood group antigens.

155 ve immunity toward pathogens bearing cognate blood group antigens.

156 r innate proteins that could recognize human blood group antigens.

157 ecificity of B cell tolerance to donor/graft blood group antigens.

158 ynthesis enzymes, cell-cycle regulators, and blood group antigens.

159 ith their capsid-protruding domains to histo-blood-group antigens (HBGAs), an interaction thought to

160 Kidney recipients in the O blood group are at a disadvantage in kidney exchange pro

161 mean (+/-SD) of 6.1+/-4.9 days in the fresh-blood group as compared with 22.0+/-8.4 days in the stan

162 sis, the hazard ratio for death in the fresh-blood group, as compared with the standard-blood group,

163 ng kidneys from A2 donors into patients with blood group B and O recipients has been used to alleviat

164 d to a series of oligomannose compounds, the blood group B antigen, and a fragment of beta-glucan rev

165 nhibitor for the donor binding site of human blood group B galactosyltransferase (GTB).

166 e, particularly for patients with unknown or blood group B or AB types.

167 The fucose moiety of the blood group B trisaccharide Galalpha1-3(Fucalpha1-2)Gal

168 Moreover, we found that the genetic-inferred blood group B was associated with a decreased risk (OR =

169 explanation for contrasts in associations of blood groups B and AB between CagA-endemic and -nonendem

170 despite smaller body size and more uncommon blood groups (B, AB) among Indo-Asians.

171 orption and could be transplanted across the blood group barrier.

172 nstrate that the African-specific Duffy-null blood group-believed to confer resistance against Plasmo

173 pylori adhesin BabA binds mucosal ABO/Le(b) blood group (bg) carbohydrates.

174 nor substrate specificity and utilization in blood group biosynthesis, which can very likely be explo

175 ogical processes including cell adhesion and blood group biosynthesis.

176 er in the HLA-matched group than in the cord-blood group but not significantly so (hazard ratio, 1.69

177 e regions, HLA-G, HLA-DQA2, HLA-A, and Duffy blood group, chemokine receptor (DARC), reached genome-w

178 We conclude that x2 fulfills blood group criteria and is synthesized by UDP-N-acetylg

179 ilized monolayer and allows for quantitative blood group detection.

180 a conformation similar to that of the Le(x) blood group determinant, bringing several sulfate groups

181 plasma-VWF, platelet-VWF does not express AB blood group determinants, although precursor H antigen e

182 also made based on subject age, gender, ABO blood group, diet, and history of vaccination.

183 cipient and donor characteristics, including blood group distribution, level of recipient's sensitiza

184 ecause they can only receive an organ from O blood group donors.

185 m malaria has been clarified, with the non-O blood groups emerging as significant risk factors for li

186 is healthy population of blood donors, non-O blood groups explain >30% of venous thromboembolic event

187 , and sensitive assay for rapid detection of blood group for point-of-care applications.

188 of human blood groups has been debated since blood group frequencies were shown to differ between pop

189 l data, initially for human populations from blood-group gene frequencies.

190 was recently identified in an RNAi screen of blood group genes as a host factor for invasion, and we

191 lopment of DNA-based methods for determining blood group genotype.

192 In addition, the genetic-inferred ABO blood group genotypes were associated with sE-selectin c

193 w the bacteria discriminately bind different blood group glycans.

194 ral information of a dual specificity cis-AB blood group glycosyltransferase in complex with a synthe

195 zymological information for a representative blood group GT.

196 ostate cancers is a consequence of increased blood group H expression together with up-regulated bran

197 esults reveal F77 antigen to be expressed on blood group H on a 6-linked branch of a poly-N-acetyllac

198 oup and 430 patients (35.3%) in the standard-blood group had died (absolute risk difference, 1.7 perc

199 ection to the observed distribution of human blood groups has been debated since blood group frequenc

200 ABO blood groups have been shown to be associated with incre

201 two unrelated-donor groups than in the cord-blood group (hazard ratio in the HLA-mismatched group, 3

202 in the HLA-mismatched group than in the cord-blood group (hazard ratio, 2.92; 95% confidence interval

203 nd cross-match tests were performed with all blood group identical kidney offers.

204 ot increase after transfusion of 2 fresh ABO blood group-identical platelet concentrates (therapeutic

205 MV, VZV and Anti-HBs IgG antibody levels and blood group IgG, IgM and IgA antibodies were quantified.

206 ificant comorbidities, including one case of blood group incompatibility.

207 Tailored desensitization in pediatric blood group incompatible kidney transplantation results

208 or 86% of the listed pairs and 52% were also blood group incompatible to their coregistered donor.

209 Blood group incompatible transplantation (ABOi) in child

210 Organ transplantation from ABO blood group-incompatible (ABOi) donors requires accurate

211 simulation modeling the effect of allocating blood group-incompatible deceased-donor kidneys to those

212 Acceptance of blood group-incompatible donors for patients with low to

213 Exclusion criteria were recipients were blood group-incompatible or crossmatch-positive or had C

214 Blood group-incompatible transplantation is one strategy

215 angeable platform capable of quantifying the blood group interactions between red blood cells (RBCs)

216 The accurate and reliable typing of blood groups is essential prior to blood transfusion.

217 Here we show that Lewis and blood group isomers, which have identical fragmentation

218 vide immunity against pathogens that express blood group-like antigens on their surface.

219 ) from the ARIC study, suggests that the ABO blood group may influence cleavage of the P-selectin pro

220 Although ABO blood groups may potentially be used with available pred

221 It occurs following ABO blood group mismatched solid organ and/or bone marrow tr

222 The occurrence of terminal blood group moieties on oyster dominin and on hemocyte s

223 s excellent reproducibility in that the same blood groups, namely A, AB, and O, were reported by usin

224 bited less human antibody binding than human blood group O allogeneic RBC in 22% of tested sera.

225 previously frozen plasma (four units each of blood group O and A), which can be issued immediately fo

226 nsplantation within 30 days for infants with blood group O and may benefit a broader range of transpl

227 to the Globo H hexaglycosylceramide, i.e. a blood group O determinant on a type 4 core chain, the ge

228 abA-expressing H. pylori strains bind to the blood group O determinants on type 1 core chains, i.e. t

229 mic equivalents [gEq]) for secretor-positive blood group O or A persons and 7.0 (approximately 2800 g

230 Twenty-one persons were infected (all blood group O or A), and 67% of those infected developed

231 In blood group O patients, levels of anti-A antibodies were

232 or B patients had lower antibody levels than blood group O patients.

233 ferase-deficient enzyme that encodes the ABO blood group O phenotype previously proposed to protect a

234 were isolated from the small intestine of a blood group O pig and characterized by mass spectrometry

235 Blood group O piglets received kidney allografts from gr

236 Previous ABO sensitization, donor blood group O to recipient blood group A or B transfer,

237 th VWF:RCo < 50 U/dL (< 40 for patients with blood group O) fulfilled the acquired von Willebrand syn

238 ible adult patients (aged >/= 18 years) with blood group O+, who required up to two whole blood unit

239 0.27-0.70) of type 2 diabetes compared with blood group O, adjusting for sE-selectin, sICAM-1, TNF-R

240 rmediate rate (30%) among secretors with non-blood group O, and the highest rate (51%) among secretor

241 Compared with blood group O, non-O blood groups were associated with h

242 el was associated with high SES, white race, blood group O, private insurance, and residence in regio

243 Ninety-one percent of donors were blood group O.

244 t cell wall oligosaccharides and human histo-blood group oligosaccharides H-type 2 and Lewis Y are bo

245 in carbohydrate structures, including histo-blood group oligosaccharides, resulting in anti-glycan a

246 fter years of controversy, the effect of ABO blood groups on falciparum malaria has been clarified, w

247 the simultaneous determination of ABO and Rh blood groups on the same device.

248 A rise is specific in contrast to the viral, blood group or third party antibodies post transplantati

249 cestry (OR = 1.8, 95% CI: 1.1, 3.1), and ABO blood group other than O (OR = 1.3, 95% CI: 1.0, 1.8).

250 ompared with 22.0+/-8.4 days in the standard-blood group (P<0.001).

251 , 0.63 [95% CI, 0.49-0.79; P < .001]) or the blood group (patient-level OR, 0.62 [95% CI, 0.46-0.83;

252 humans [X-linked Lu(a-b-) phenotype, In(Lu) blood group phenotype, hereditary persistence of foetal

253 ed on cells of the rare P/P1/P(k)-negative p blood group phenotype.

254 erythrocytes from individuals of all common blood group phenotypes and elevated on cells of the rare

255 The study of rare blood group phenotypes is a potent tool for discovery of

256 These studies explain the occurrence of rare blood group phenotypes with simultaneous altered express

257 his review describes the genetics of unusual blood group phenotypes, particularly those with altered

258 s and improved equity with regard to patient blood group, rareness of HLA type, and HLA homozygosity.

259 Compared with other recipients from the same blood group, recipients of A2 kidneys had significantly

260 The K blood group remains an important target in hemolytic dis

261 ed controls on panel reactive antibody, age, blood group, renal replacement time, prior kidney transp

262 These results demonstrate that the A and B blood groups result from a trans-species polymorphism am

263 Host ABO blood group, reticulocyte count, and parasitemia were no

264 lpha(1,2) fucosyltransferase that determines blood group secretor status.

265 age, we proposed that x2 joins P in the GLOB blood group system (ISBT 028) and is renamed PX2 (GLOB2)

266 Our results establish Augustine as a new blood group system and place SLC29A1 as a new candidate

267 PURPOSE OF REVIEW: The importance of ABO blood group system compatibility in platelet transfusion

268 Diets that are based on the ABO blood group system have been promoted over the past deca

269 opoietic tissue and the basis of a new histo-blood group system in man, FORS.

270 therapy-related drug resistance, and the new blood group system Langereis.

271 The Knops blood group system may also influence malaria susceptibi

272 altered expression of antigens from several blood group systems and illuminate the role of KLF1 in g

273 te also has general information on the human blood group systems and the genes responsible for them.

274 her are known to affect antigens of 30 human blood group systems.

275 that determine the antigens of various human blood group systems.

276 probability of relapse was lower in the cord-blood group than in either of the other groups.

277 The ABO histo-blood group, the critical determinant of transfusion inc

278 00, when Landsteiner first described the ABO blood groups, to the present, the methods used to charac

279 The direction of blood group transfer, from donor to recipient, was O Rh

280 Attachment to carbohydrates of the histo-blood group type of several human Rotavirus strains (RVA

281 , multi-functional platform for quantitative blood group typing via SPR detection is achieved by immo

282 nd extend the set of markers for genetic Vel blood group typing.

283 ariants, which are often missed by serologic blood group typing.

284 an interesting alternative for quantitative blood grouping using SPR analysis.

285 Blood group variants are characteristic of population gr

286 terest in the global prevalence of the Duffy blood group variants is multidisciplinary, but of partic

287 The blood group Vel was discovered 60 years ago, but the und

288 ccounted for Organ Procurement Organization, blood group, wait time, DR antigens, and prior offer his

289 incidence of graft failure in the peripheral-blood group was 3% (95% CI, 1 to 5), versus 9% (95% CI,

290 l survival rate at 2 years in the peripheral-blood group was 51% (95% confidence interval [CI], 45 to

291 of chronic GVHD at 2 years in the peripheral-blood group was 53% (95% CI, 45 to 61), as compared with

292 Donor blood group was insignificant on Cox regression for over

293 has excellent reproducibility where the same blood group was obtained in 26 samples assessed in 2 dif

294 d conditions, a rapid assay for detection of blood groups was developed by spotting blood to antibodi

295 h-blood group, as compared with the standard-blood group, was 1.1 (95% CI, 0.9 to 1.2; P=0.38).

296 ' disease, non-Hodgkin lymphoma, and various blood groups were accurate, as individuals manifesting t

297 Compared with blood group O, non-O blood groups were associated with higher incidence of bo

298 the simultaneous determination of ABO and Rh blood groups, which is promising for use in developing c

299 through atherosclerosis, except for the ABO blood groups, which show that A and B are associated wit

300 , CAV1, atypical chemokine receptor 1 [duffy blood group]) whose mRNA transcript levels in plasma exo