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1 g complex lactoside carbohydrates resembling blood group A.
2                                              Blood groups A, AB and B significantly reduced IE bindin
3 3)-N-acetylgalactosamine residues from human blood group A and AB mucin glycoproteins, Forssman hapte
4        We show that mAb F77 can also bind to blood group A and B analogs but with lower intensities.
5 rase (GTB) catalyze the final step in ABO(H) blood group A and B antigen synthesis through sugar tran
6   Recombinant E-ABase not only destroyed the blood group A and B antigenicity of human type A and B e
7                                        Histo-blood group A and B antigens are oligosaccharide antigen
8 stimulated to secrete substantial amounts of blood group A and B antigens linked to von Willebrand fa
9 lates gastrointestinal mucosal expression of blood group A and B antigens) (rs602662, P=3.4x10(-5)).
10 anti-blood group antibodies interacting with blood group A and B antigens.
11 e enzymes responsible for synthesis of histo-blood group A and B antigens.
12 nal step of the biosynthesis of ABO(H) human blood group A and B antigens.
13 glycosyltransferases that includes the histo blood group A and B enzymes.
14 glycosyltransferases that includes the histo-blood group A and B glycosyltransferases, Forssman glyco
15 ha1-->2)Gal) from glycoconjugates containing blood group A and B glycotopes, respectively.
16 igands supported this preference over type 1 blood group A and B oligosaccharides.
17                   Four of five patients were blood group A and had no significant comorbid conditions
18 nthesis of oligosaccharides similar to human blood group A and may participate in the synthesis of th
19 cause in-hospital mortality in patients with blood group A and O who were transfused.
20 wing immunocytochemical markers were tested: blood group A and precursors of blood antigens; laminin
21                   The carbohydrate antigens, blood groups A and B, and the alpha-gal epitope (Galalph
22 d no longer than 7 days, both in patients of blood groups A and O and all patients.
23 ytes, but also released A-Tri and B-Tri from blood group A(+)- and B(+)- containing glycoconjugates.
24 l for studying the structure and function of blood group A- and B-containing glycoconju-gates as well
25 functionalized with immunoglobulin G against blood group A (anti-A IgG) by forming a self-assembled m
26                        In analogy with histo-blood group A antigen, Forssman (Fs) antigen terminates
27 bodies against alpha-gustducin and the human blood group A antigen.
28 ncogene at codon 12 and all tumors expressed blood group A antigen.
29 monstrating its versatility for synthesis of blood group A antigens.
30 n for anitgen-negative donors, 6) to resolve blood group A, B, and D typing discrepancies, 7) to dete
31 ound sulfated disaccharides as well as human blood groups A, B, and H on both N-glycans and linear gl
32                 Incompatibility of the major blood groups A, B, and O has been an absolute contraindi
33 ythrocytes was insensitive to differences in blood groups A, B, O, or MN.
34 Acbeta3Galalpha4Galbeta4Glcbeta1Cer), i.e. a blood group A determinant on a type 4 core chain.
35 ulfated and methylated variants of the histo-blood group A epitope.
36 recombinant lectin, rCNL, agglutinates human blood group A erythrocytes and is specific for the uniqu
37  B6 wild-type (WT) mice were sensitized with blood group A-human erythrocytes; others received passiv
38                                    To cleave blood group A immunodeterminants from erythrocytes, we p
39 AB mucin glycoproteins, Forssman hapten, and blood group A lacto series glycolipids.
40 des by remodeling blood group B mimicry into blood group A mimicry.
41                   The accuracy for detecting blood group A (n=12), B (n=13), AB (n=9), O (n=14), and
42 Thr residues in the tandem repeat domains of blood group A-negative porcine submaxillary gland mucin.
43 asite glycans structurally distinct from the blood group A oligosaccharides on the hemocyte surface m
44 that CvGal1 preferentially recognizes type 2 blood group A oligosaccharides.
45 on-associated carbohydrate antigens, such as blood group A or B and the alpha-gal epitope (Gal(alpha)
46 pes of the same lines transfected with histo-blood group A or B genes.
47 zyme-linked immunosorbent assay (ELISA) with blood group A or B human red cell membranes, as solid ph
48                                              Blood group A or B patients had lower antibody levels th
49 sitization, donor blood group O to recipient blood group A or B transfer, and ciclosporin treatment h
50 lood stored 7 days or less (in patients with blood group A or O: 0.92, 0.74-1.15, p=0.48; in all 0.90
51  blood product use history (in patients with blood group A or O: hazard ratio 0.94, 95% CI 0.73-1.20,
52 al blood type donor (P = .01), recipient non-blood group A (P = .02), and donor cause of death other
53 ors for parasite invasion, such as the Duffy blood group, a receptor for Plasmodium vivax, and the Ge
54  for all B recipients, even shorter than for blood group A recipients (median waiting times of A2/A2B
55 lated specificity, such as B cells with anti-blood group A specificity.
56 ereus galectin shows particular affinity for blood group A structures.
57                                              Blood group A subjects had higher levels of anti-pig IgM
58  galectin for N-acetyllactosamine, the human blood group A tetrasaccharide and Galbeta1,3GalNAc relat
59                                        Histo-blood group A transferase produces A antigens and transf
60                 A bacterial version of human blood group A transferase was identified and found to be
61 osylceramide) were identified as well as the blood group A type 1 hexaosylceramide.
62                              Recipients with blood group A were also more likely to develop IEC-defin

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