ライフサイエンスコーパス: blood monocyte

1 situ, but require constant replenishment by blood monocytes.

2 d CD4-mediated macrophage differentiation of blood monocytes.

3 3 in cell-mediated immunity using peripheral blood monocytes.

4 intact in the absence of circulating LyC6(-) blood monocytes.

5 flammatory profile and expression of CD36 on blood monocytes.

6 of human monocytic cell lines and peripheral blood monocytes.

7 subpopulation of AAMvarphis originates from blood monocytes.

8 e both taken up by the same peripheral human blood monocytes.

9 through 7 days of infection in both AMs and blood monocytes.

10 r cells having the morphologic appearance of blood monocytes.

11 K in aortic endothelial cells and peripheral blood monocytes.

12 nc transporter gene expression in peripheral blood monocytes.

13 te cytokine production from human peripheral blood monocytes.

14 lize on the cell surface of human peripheral blood monocytes.

15 in contrast to other tissue macrophages and blood monocytes.

16 nd represent a newly derived population from blood monocytes.

17 cytokine gene expression in human peripheral blood monocytes.

18 ished prenatally and can be replenished from blood monocytes.

19 nd growth factors by normal human peripheral blood monocytes.

20 ent on human B cells, T cells and peripheral blood monocytes.

21 d on cultured macrophages but not peripheral blood monocytes.

22 erentiated macrophages or control peripheral blood monocytes.

23 uced expression of CXCL8 in human peripheral blood monocytes.

24 udies with purified endotoxin aggregates and blood monocytes.

25 hibited during LC differentiation from human blood monocytes.

26 the functional effect of hyperlipidaemia on blood monocytes.

27 a rather than from recruitment of peripheral blood monocytes.

28 yzed the putative functions of stabilin-1 in blood monocytes.

29 y to stimulate the release of TNF-alpha from blood monocytes.

30 abolished by deletion of IL-1beta in CCR2(+) blood monocytes.

31 ion equal to pDC from blood and greater than blood monocytes.

32 a wide range of antiviral responses in cord blood monocytes across the population.

33 significantly lower viraemia (p<0.0001) and blood monocyte-activation patterns (p=0.0233).

34 microglia and/or macrophages, and CX3CR1 on blood monocytes) allowed us to distinguish animals with

35 tes the biology of target primary peripheral blood monocytes, allowing HCMV to use monocytes as "vehi

36 ine alveolar macrophages, but not peripheral blood monocytes, also formed METs in response to M. haem

37 ional macrophages are derived primarily from blood monocytes, although recent research has shown that

38 rom lipopolysaccharide-stimulated peripheral blood monocytes and 30% lower monocyte surface Toll-like

39 s on the adhesive interactions of peripheral blood monocytes and activated endothelial cell monolayer

40 Viral DNA isolated from blood monocytes and alveolar macrophages (but not T cell

41 We cultured cord blood monocytes and assessed IFNG and CCL5 mRNA producti

42 toring innate immune cells in BM, increasing blood monocytes and blood/liver bacteria clearance, and

43 multiple markers (CD44v6, CCR2, and CCR5 on blood monocytes and brain microglia and/or macrophages,

44 We report using human peripheral blood monocytes and Chinese hamster ovary cells transfec

45 nature that is closer to nontumor "resident" blood monocytes and embryonic macrophages than TAMs, sug

46 PS-stimulated inflammatory response in human blood monocytes and explored potential mechanisms of vit

47 e control of processing and release in human blood monocytes and human monocyte-derived macrophages.

48 n-like protein 4 (Angptl4) in adherent human blood monocytes and in human lung microvascular endothel

49 DNA samples derived from peripheral blood monocytes and in vitro differentiated dendritic ce

50 re protein (HCVc) engages TLR2 on peripheral blood monocytes and induces production of multiple infla

51 e-4 trimethylation (H3K4Me3), and H3K9Me2 in blood monocytes and lymphocytes obtained from 30 DCCT co

52 for frequencies and functional parameters of blood monocytes and macrophages in duodenal mucosa.

53 Human peripheral blood monocytes and macrophages were incubated in the pr

54 iption stimulated by LPS in human peripheral blood monocytes and macrophages.

55 Neonatal cord and adult peripheral blood monocytes and MoDCs were cultured in autologous pl

56 ell-activating cells can express TL1A, fresh blood monocytes and monocyte-derived dendritic cells wer

57 y expressed in membranes of human peripheral blood monocytes and monocytoid cells.

58 4-stimulated normal and sepsis-adapted human blood monocytes and mouse splenocytes.

59 o-photon and epifluorescence microscopy that blood monocytes and neutrophils crawling along and adher

60 unds induced transient changes in peripheral blood monocytes and neutrophils, and activated T lymphoc

61 rs may alter epigenetic states of peripheral blood monocytes and resident tissue macrophages.

62 duce production of TNF from human peripheral blood monocytes and rheumatoid synovial membrane culture

63 been gained into how the properties of human blood monocytes and rodent macrophages are influenced by

64 toneally injected Abeta could be detected in blood monocytes and some peripheral tissues (liver, sple

65 els of both Gr-1(low) and Gr-1(intermediate) blood monocytes and splenic macrophages and, in a paracr

66 DT treatment reduced peripheral blood monocytes and tissue macrophages and inhibited mac

67 e expression of stabilin-1 was diminished on blood monocytes and tissue macrophages under proinflamma

68 d immature DCs from healthy donor peripheral blood monocytes and treated them with plasma from uninfe

69 an cytomegalovirus (HCMV) infects peripheral blood monocytes and triggers biological changes that pro

70 rentiated from macaque and baboon peripheral blood monocytes and used to compare viral replication an

71 ated protein kinase phosphatase 1 (MKP-1) in blood monocytes, and hematopoietic MKP-1-deficiency in a

72 f transfected HEK293 cells, human peripheral blood monocytes, and mouse peritoneal exudate cells in a

73 etected in isolated brain mononuclear cells, blood monocytes, and peritoneal macrophages, suggesting

74 pression on adult monocytes compared to cord blood monocytes, and that CD300c and CD300e-mediated act

75 ated cells including bone marrow precursors, blood monocytes, and tissue macrophages.

76 pathways, and with gene expression in whole blood, monocytes, and myocardial tissue.

77 Two subsets of blood monocytes are commonly described in mice and human

78 Old reports suggest that blood monocytes are distributed into circulating and mar

79 Blood monocytes are heterogeneous effector cells of the

80 ial requirements for HIV-1 entry, peripheral blood monocytes are infrequently infected with HIV-1 in

81 Human peripheral blood monocytes are known to promote Ag presentation and

82 Blood monocytes are produced by bone marrow and splenic

83 Infected peripheral blood monocytes are proposed to play a key role in the h

84 ent as resident cells in adipose tissue, and blood monocytes are recruited in increased numbers to si

85 Primary peripheral blood monocytes are responsible for the hematogenous dis

86 that IL-32 induces differentiation of human blood monocytes as well as THP-1 leukemic cells into mac

87 Circulating blood monocytes belong to the first line of defense agai

88 Deletion of IL-1beta in blood monocytes blunts the production of IL-22 by ILC3 a

89 on of human THP-1 macrophages and peripheral blood monocytes but does not occur during infection of m

90 d LN had similar gene expression profiles to blood monocytes but elevated transcripts of a limited nu

91 or (EGFR) on the surface of human peripheral blood monocytes but not on other blood leukocyte populat

92 oduced are most closely similar to the major blood monocyte (CD14(+), CD16(low), CD163(+)).

93 In contrast to the peripheral blood monocytes, CD86 was still expressed by part of the

94 We show that CLA inhibits human peripheral blood monocyte cell adhesion to activated endothelial ce

95 mplete year of follow-up data for peripheral blood monocyte cell specimens were also analyzed.

96 ted from conjunctiva, spleen, and peripheral blood monocyte cells (PBMC) of HSV-1-infected and uninfe

97 metry and by Western blot in both peripheral blood monocyte cells and CD3+ T cells of renal transplan

98 We obtained peripheral blood monocyte cells from 29 living donor-recipient pair

99 ft and subcutaneous injections of peripheral blood monocyte cells from an antigen-positive donor.

100 ve animal after immunization with peripheral blood monocyte cells.

101 s were co-cultured with recipient peripheral blood monocyte cells.

102 After 6 weeks, we observed blood monocyte chimerism of 35.3+/-3.4%, whereas heart m

103 synovial fluid macrophages and RA peripheral blood monocytes compared with RA and normal peripheral b

104 Human blood monocytes comprise at least 3 subpopulations that

105 wild-type but not PDL-1-deficient peripheral blood monocytes containing apoptotic cell-associated OVA

106 conditioned media from Gd-exposed peripheral blood monocytes could induce a profibrotic phenotype in

107 igh-density lipoprotein cholesterol suppress blood monocyte counts and atherosclerosis.

108 High blood monocyte counts may represent a previously unrecog

109 educed high-density lipoprotein levels, high blood monocyte counts, and accelerated atherosclerosis.

110 most critical for the maintenance of normal blood monocyte counts.

111 each subject were assessed using peripheral blood monocytes cultured with 10% autologous serum (n =

112 candidate adjuvants, but human neonatal cord blood monocytes demonstrate impaired T(H)1-polarizing re

113 s completely abrogated by in vivo peripheral blood monocyte depletion by gadolinium chloride (GdCl3)

114 r activation by TLR agonists in inflammatory blood monocyte-derived DCs that express inducible NO syn

115 DCs) are the closest known equivalent of the blood monocyte-derived DCs that have been used for human

116 greatly downregulated in Mtb-infected human blood monocyte-derived DCs, indicating that reduction of

117 aB signaling pathway when porcine peripheral blood monocyte-derived dendritic cells (MoDCs) were trea

118 When human blood monocyte-derived dendritic cells were used, IL-38

119 apoptotic GECs to THP-1 cells or peripheral blood monocyte-derived macrophages as assayed by confoca

120 deficiency virus type 1 (HIV-1) infection of blood monocyte-derived macrophages that expressed IFN-la

121 vitro migratory response of human peripheral blood monocyte-derived macrophages to oxLDL.

122 lammation is primarily based on the study of blood monocyte-derived macrophages, cells that have neve

123 pro-inflammatory cytokine profile similar to blood monocyte-derived macrophages.

124 HIV reverse transcriptase activity in human blood monocyte-derived macrophages.

125 effect of BBI on HIV infection of peripheral blood monocyte-derived macrophages.

126 olk sac-derived Kupffer cells and peripheral blood monocyte-derived macrophages.

127 21 was noted in postefferocytotic peripheral blood monocyte-derived macrophages.

128 d CD16/32, and become indistinguishable from blood monocyte-derived Mphis.

129 id (GC) treatment during maturation of human blood monocyte-derived or murine bone marrow-derived mac

130 ivo engulfment of EMPs or cMPs by peripheral blood monocytes-derived macrophages was associated with

131 genome in either liver tissue or peripheral blood monocytes, despite constant negative results from

132 st that NUR77, which is required for LyC6(-) blood monocyte development, is expressed but not critica

133 ndometrium, endometrial cells and peripheral blood monocytes did not generate inflammatory responses

134 In atherosclerotic arteries, blood monocytes differentiate to macrophages in the pres

135 orphyromonas gingivalis, donor-matched human blood monocytes, differentiated DCs, and macrophages wer

136 onic inflammatory systemic disorder, CD14(+) blood monocytes display reduced miR-125b expression as c

137 CD11c, which increases on blood monocytes during hypercholesterolemia, plays an im

138 turnover rates that were similar to those of blood monocytes during steady-state homeostasis.

139 fferential contribution of Kupffer cells and blood monocytes during the development of NASH; Kupffer

140 ation from the bone marrow to the peripheral blood, monocytes enter tissues and differentiate into ma

141 ggests that in fibrotic lesions, a subset of blood monocytes enters the tissue and differentiates int

142 Blood monocytes exist in two primary subpopulations, cha

143 mitted bone marrow macrophage precursors and blood monocytes express EGFP in these animals.

144 4 (M2), we cultured primary human peripheral blood monocytes for 6 d.

145 ed TLR function and expression in peripheral blood monocytes from 159 subjects in 2 age categories, 2

146 Compared with normal subjects, peripheral blood monocytes from acne patients expressed significant

147 n vivo, as CD138-bound APRIL is expressed on blood monocytes from active SLE patients.

148 specific to all U12-type introns detected in blood monocytes from affected individuals.

149 Lung tissues and blood monocytes from asthma patients expressed significa

150 on was significantly decreased in peripheral blood monocytes from asthma patients.

151 septic lethality was diminished; and third, blood monocytes from both septic mice and patients with

152 t stress lowers the threshold of activation; blood monocytes from CAPS patients maintain the high lev

153 monstrate that macrophages from CGD mice and blood monocytes from CGD patients display minimal recrui

154 Blood monocytes from children with systemic lupus erythe

155 uantitate in vivo macrophage tracking (i.e., blood monocytes from donor mice) labeled ex vivo with fl

156 P on DC differentiation in vitro, peripheral blood monocytes from healthy donors were cultured in the

157 Using primary human blood monocytes from healthy donors, we identified ANXA1

158 iation was investigated in vitro using human blood monocytes from healthy subjects and patients with

159 Functionally, we found that peripheral blood monocytes from healthy subjects homozygous for the

160 of cytokines, in freshly isolated peripheral blood monocytes from LGMD2B patients and in the SJL dysf

161 Circulating blood monocytes from macFoxp1tg mice have reduced expres

162 Blood monocytes from patients affected by NLRP3-mediated

163 mic, functional, and mechanistic analysis of blood monocytes from patients during sepsis and after re

164 ynovial macrophages compared with peripheral blood monocytes from RA or healthy controls.

165 Blood monocytes from sepsis patients demonstrated an upr

166 rventions can reduce the number of available blood monocytes, from which macrophages and most DCs and

167 Recent studies have shown that blood monocytes harbor human immunodeficiency virus type

168 We found blood monocytes harboring viruses with multiple phenotyp

169 Compared with primary blood monocytes, high levels of IL-32 are constitutively

170 equally low levels in Ly6C(hi) and Ly6C(lo) blood monocytes, however expression is increased over 2-

171 The number of circulating blood monocytes impacts atherosclerotic lesion size, and

172 Activation of LILRA2 on peripheral blood monocytes impaired GM-CSF induced differentiation

173 tion of IL-1beta protein by human peripheral blood monocytes in a manner that requires neither the pr

174 acterise the role of regulatory receptors on blood monocytes in patients with sarcoidosis.

175 tered distribution of tissue macrophages and blood monocytes in the absence of Galpha(i2) but not Gal

176 eg), generated from cytokine-mobilized donor blood monocytes in vitamin D3 and IL-10, on renal allogr

177 nd growth factors by normal human peripheral blood monocytes in vitro and to examine whether conditio

178 n CD34(+) hematopoietic progenitor cells and blood monocytes in vitro.

179 early phase of NASH development by promoting blood monocyte infiltration through the production of IP

180 d the metabolic stress-induced conversion of blood monocytes into a proatherogenic phenotype.

181 Mo-DCs are recruited from blood monocytes into lymph nodes by lipopolysaccharide a

182 The differentiation of human blood monocytes into macrophages is a caspase-dependent

183 g in the differentiation of pro-inflammatory blood monocytes into noninflammatory intestinal macropha

184 e requirement of NOTCH1 for the migration of blood monocytes into the liver and subsequent M1 differe

185 -induced B7-2 expression on human peripheral blood monocytes is dependent on JNK signaling, indicatin

186 How LCs arise from blood monocytes is not fully understood.

187 cytes to microvascular endothelial cells and blood monocytes is significantly reduced relative to the

188 In conclusion, the recruitment of blood monocytes is the principle mechanism of Mphi accum

189 Normal human peripheral blood monocytes isolated by Ficoll-Hypaque gradient cent

190 Peripheral blood monocytes isolated from 10 AS patients and 10 heal

191 Blood monocytes isolated from diabetic mice showed a 55%

192 nse (within 4 h) in primary human peripheral blood monocytes isolated from healthy donors.

193 Peripheral blood monocytes isolated from individuals with polymorph

194 In peripheral blood monocytes isolated from patients with alcoholic he

195 Peripheral blood monocytes isolated from patients with bone metasta

196 nce believed to depend on the recruitment of blood monocytes, it is now clear that the accumulation o

197 hese mice had similar plasma cholesterol and blood monocyte levels but increased atherosclerosis comp

198 % at 24, 48, and 72 hours, with no effect on blood monocyte levels.

199 ata emphasise the functional consequences of blood monocyte lipid accumulation and reveal important i

200 A longitudinal analysis of blood monocyte markers revealed that as early as 28 days

201 at a pattern of surface marker expression in blood monocytes may serve as a marker for central nervou

202 the roles of Rho signaling in primary human blood monocytes migrating in chemotactic gradients and i

203 DNA-me was also profiled in blood monocytes (Monos) of the same patients obtained du

204 Blood monocytes, myeloid dendritic cells (mDC), and mono

205 LR2, TLR4, and CD86 expression on peripheral blood monocytes, natural killer (NK) cells, and NK T cel

206 major changes in macrophage polarization or blood monocyte numbers.

207 he effect of VacA on autophagy in peripheral blood monocytes obtained from subjects with different ge

208 pression in THP-1 cells and human peripheral blood monocytes occurred via the activation of PI3K, NAD

209 candidate genes was significantly greater in blood monocytes of diabetic patients relative to normal

210 uced by HIV-1 peptide-loaded DC derived from blood monocytes of HIV-1-negative adults and neonates (m

211 f inflammatory gene expression in peripheral blood monocytes of MZ twin pairs (n = 10 pairs) discorda

212 tion of efferocytic function was observed in blood monocytes of obese participants.

213 th solid tumor xenografts but not peripheral blood monocytes or inflammatory macrophages in animal mo

214 tosis did not trigger a respiratory burst in blood monocytes or monocyte-derived macrophages (MDM), a

215 Of note, in primary human blood monocytes, p300-c-Myc binding was strongly correla

216 e differential immunomodulatory responses of blood monocytes, particularly during pregnancy, on infec

217 Peripheral blood monocyte (PBMC) expression of mRNA for TLRs 2, 3,

218 nd Mf2, phenotypically similar to peripheral blood monocytes (PBMos), were largely replaced within 3

219 sured the expression of p66Shc in peripheral blood monocytes (PBMs) and renal biopsy tissues from DN

220 lated cell death in primary human peripheral blood monocytes (PBMs).

221 We now show that an M2-like blood monocyte phenotype is significantly associated wit

222 Peripheral blood monocytes play a role in sarcoidosis inflammation.

223 Blood monocytes play an important role in both processes

224 the infarct were again independent from the blood monocyte pool, returning to the steady-state situa

225 ugh which uric acid selectively lowers human blood monocyte production of the natural inhibitor IL-1

226 did not affect the number of bone marrow or blood monocytes, proliferation of precursors, or recruit

227 Our system of infected primary human blood monocytes provides us with an opportunity to answe

228 Furthermore, the numbers of peripheral blood monocytes rapidly increased between days 4 and 8 p

229 Blood monocytes recognize Gram-negative bacteria through

230 macrophages are continuously replenished by blood monocytes recruited to the peritoneal and pleural

231 from a resident prenatal population or from blood monocyte recruitment and that IL-4 can increase ma

232 Depleting Ly6C(hi) blood monocytes reduces DC accumulation and the skin cha

233 Here we demonstrate that human blood monocytes release processed IL-1beta after a one-t

234 TUNEL, suggesting a continuous transition of blood monocytes replacing IMs undergoing apoptosis.

235 d macrophage depletion in CD11b(DTR/+) mice, blood monocytes replenished heart macrophages.

236 These data suggest that HIV-1 circulating in blood monocytes represents diverse HIV-1 with multiple p

237 Thus the blood monocyte reservoir becomes the dominant presenting

238 6(+) monocyte subsets comprise 85 and 15% of blood monocytes, respectively, and are thought to repres

239 peritoneal macrophages and human peripheral blood monocytes, respectively, by flow cytometry.

240 le data sets for samples of human peripheral blood monocytes resulted in discovery of 3438 EERs in th

241 Here, the authors examine CD14+ blood monocyte's transcriptome and epigenome signatures

242 f 30-2000 pg/ml in both tumor and peripheral blood monocyte samples.

243 rum C-reactive protein levels and peripheral blood monocyte secretion of IL-6 and TNF-alpha.

244 Blood monocytes serve as the first line of host defense

245 thors generate transcriptome data from human blood monocytes stimulated with various immune stimuli a

246 The lineage relationship between blood monocyte subsets and the various phenotypes and fu

247 Selective labeling of blood monocyte subsets indicates that non-classical mono

248 c insights into CCR2-mediated recruitment of blood monocyte subsets into damaged tissue, the dynamics

249 airway monocytes were less inflammatory than blood monocytes, suggesting a more tolerant nature.

250 Circulating blood monocytes supply peripheral tissues with macrophag

251 ease in the expression of CD14 on peripheral blood monocytes that correlated with IL-23 expression an

252 h are believed to be derived from peripheral blood monocytes that have infiltrated the central nervou

253 Instead, it derives from blood monocytes that rapidly enter the PerC after stimul

254 y and reactivation in circulating peripheral blood monocytes that was developed in order to study the

255 n in the NF-kappaB signaling and function of blood monocytes, the exclusive source of intestinal macr

256 Blood monocytes then enter the inflamed peritoneum and d

257 Unlike human blood monocytes, these melanoma cells require no exogeno

258 ssion of CD44v6 on the 2 main populations of blood monocytes--those that express either low or high l

259 rrently based on the elevation of peripheral blood monocytes to >1 x 10(9)/L, measured for >/=3 month

260 the cell surface and diminished responses of blood monocytes to bacterial agonists.

261 In order for peripheral blood monocytes to facilitate viral dissemination, HCMV

262 sella infection, we exposed human peripheral blood monocytes to Francisella novicida and analyzed tra

263 ispensable for a potent cytokine response of blood monocytes to group B Streptococcus, although monoc

264 ctant protein-1 (MCP-1) directs migration of blood monocytes to inflamed tissues.

265 Exposure of peripheral blood monocytes to interleukin-4 can recapitulate the fu

266 s by airway (induced sputum) macrophages and blood monocytes to markers of monocyte programming, in v

267 land and non-New Zealand honeys to stimulate blood monocytes to release tumour necrosis factor (TNF)-

268 cally involved in directing the migration of blood monocytes to sites of inflammation.

269 short-term model system in human peripheral blood monocytes to study the immunological relevance of

270 taxis is involved in enhanced trafficking of blood monocytes to the gut.

271 ation of resident microglia and infiltrating blood monocytes toward an M2 or "alternatively" activate

272 ement of CCR2 and CCR6 in the recruitment of blood monocytes toward the lung upon exposure to DEPs.

273 Here we present the CD14+ blood monocyte transcriptome and epigenome signatures as

274 ils, airway epithelial cells, and peripheral blood monocytes treated with plasma purified A1AT protei

275 Human peripheral blood monocytes treated with the TLR2 agonist Pam2CSK4 s

276 e reported previously that a high peripheral blood monocyte turnover rate was predictive for the onse

277 +)] CD163(+)), suggesting that the increased blood monocyte turnover was required to rapidly replenis

278 he expression of ARHGEF1 by human peripheral blood monocytes varies between individuals and inversely

279 pression in either THP-1 cells or peripheral blood monocytes was reduced upon expression of microRNA-

280 Fresh human blood monocytes were compared with monocyte-derived macr

281 Human THP-1 monocytes and peripheral blood monocytes were differentiated into macrophages and

282 Peripheral blood monocytes were differentiated into macrophages, an

283 Peripheral blood monocytes were isolated and cultured with or witho

284 Peripheral blood monocytes were isolated from asymptomatic and symp

285 Blood monocytes were labeled by intravenous injection of

286 cA (WT-EcA), although nonleukemic peripheral blood monocytes were not affected.

287 Peripheral blood monocytes were treated with GM-CSF and IL-4 to yie

288 broblasts, small airway epithelial cells and blood monocytes were treated with IL-1beta or cigarette

289 ome studies argue that they are derived from blood monocytes, whereas others support a local origin f

290 which lack B7 molecules and FcgammaRs) or by blood monocytes (which express low levels of B7 molecule

291 lves antibody binding its cognate antigen on blood monocytes, which generates MIP-2 chemokine product

292 ellular responses in normal human peripheral blood monocytes, which may participate in the developmen

293 ELVs are taken up by peripheral blood monocytes, which then differentiate into activated

294 2) is highly expressed on a subpopulation of blood monocytes whose numbers are markedly decreased in

295 The depletion of blood monocytes with clodronate liposomes reduced neutro

296 rophages were derived by incubation of human blood monocytes with granulocyte-macrophage colony-stimu

297 on by natural killer cells, and expansion of blood monocytes with less proinflammatory cytokine respo

298 we demonstrate that activation of peripheral blood monocytes with P. acnes in vitro induced their dif

299 uman THP-1 monocytic cells and primary human blood monocytes with RvD2 and LPS to evaluate modulation

300 Proinflammatory blood monocytes with the phenotype CD14+,CD16+,HLA-DR++