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1 rations in microbial communities relevant in blood pressure control.
2 gic system in salt and water homeostasis and blood pressure control.
3 These factors are associated with poor blood pressure control.
4 i-ischemic therapy, and aggressive lipid and blood pressure control.
5 l ENaC is critical for Na(+) homeostasis and blood pressure control.
6 nal C1 adrenergic neurons and is involved in blood pressure control.
7 echanism by which patient education improved blood pressure control.
8 ivate patients to achieve strict glucose and blood pressure control.
9 uation of the hypertensive patient with poor blood pressure control.
10 dication when evaluating a patient with poor blood pressure control.
11 are had room for improvement, especially for blood pressure control.
12 sport, contributing to Na(+) homeostasis and blood pressure control.
13 tment with more than 1 drug class to achieve blood pressure control.
14 overt CHF who are candidates for aggressive blood pressure control.
15 effective as enalapril in LVH regression and blood pressure control.
16 fering is an important mechanism in arterial blood pressure control.
17 ose associated with conventional therapy and blood pressure control.
18 plays a critical role in Na+ homeostasis and blood pressure control.
19 nd tubular function that, in turn, influence blood pressure control.
20 n-angiotensin system, a major participant in blood pressure control.
21 ve treatment still remain on medications for blood pressure control.
22 d felodipine, a calcium channel blocker, for blood pressure control.
23 s also received furosemide concomitantly for blood pressure control.
24 or insulin resistance, lipid metabolism, and blood pressure control.
25 ent reviews for people with poor glycemic or blood pressure control.
26 f adherence to antihypertensive treatment on blood pressure control.
27 can prevent glucose intolerance and improve blood pressure control.
28 equired for physiological vasoreactivity and blood pressure control.
29 nal inflammation and fibrosis independent of blood pressure control.
30 eration, angiogenesis, channel activity, and blood pressure control.
31 benefit from late CNI withdrawal by improved blood pressure control.
32 e to osmotic stress is important to systemic blood pressure control.
33 sels and is considered a major mechanism for blood pressure control.
34 (ENaC) is critical for Na(+) homeostasis and blood pressure control.
35 LV diastolic function and facilitated better blood pressure control.
36 ignificantly differ between strict and usual blood pressure control.
37 bitors and beta-blockers, and low for strict blood pressure control.
38 ial Na(+) absorption, a major contributor to blood pressure control.
39 ing bilateral adrenalectomy in childhood for blood pressure control.
40 ertensives may facilitate efforts to improve blood pressure control.
41 Renin is essential for blood pressure control.
42 ease to receive either intensive or standard blood-pressure control.
43 her risk of coronary events, despite similar blood-pressure control.
44 with antihypertensive medications have their blood pressure controlled.
45 e and 60% of treated hypertensive people had blood pressure controlled.
46 uestion in hypertension is: How is long-term blood pressure controlled?
47 (1.26, 1.08-1.47; p=0.0038), and have their blood pressure controlled (1.13, 1.00-1.28; p=0.0562) th
48 ed to the patient education group had better blood pressure control (138/75 mm Hg) than those in the
49 treated male hypertensive patients with good blood pressure control (139.6/85.7 mm Hg), young blacks,
50 y (1.53, 1.13-2.07; p=0.054), and have their blood pressure controlled (2.06, 1.69-2.50; p<0.0001) th
54 ith HF-preserved EF less frequently achieved blood pressure control (adjusted odds ratio, 0.44 versus
56 pertension were aware of the diagnosis, with blood pressure control among 32.5% of those being treate
57 ypertensive medication use and its impact on blood pressure control among US adults with hypertension
59 ed renal function had a beneficial effect on blood pressure control and a nondeleterious effect on re
60 a post hoc analysis of the Effect of Strict Blood Pressure Control and ACE Inhibition on Progression
61 medical therapy as a factor leading to poor blood pressure control and adverse outcomes remains a ke
63 rect targets and mechanisms linking FGF21 to blood pressure control and hypertension are still elusiv
64 udy identifies a new role for vascular MR in blood pressure control and in vascular aging and support
65 t protocols including intensive glycemic and blood pressure control and laser photocoagulation for ne
66 ions, in addition to meticulous attention to blood pressure control and lifestyle changes, have the p
67 egarding the importance of blood glucose and blood pressure control and may motivate patients to achi
69 stained-release- or atenolol-based strategy; blood pressure control and outcomes were equivalent.
70 describe the role of pressure natriuresis in blood pressure control and outline the cascade of biophy
71 a complex but important factor in achieving blood pressure control and reducing adverse cardiovascul
72 procedures sometimes offer major benefits in blood pressure control and stabilization of renal functi
73 tional basis for the effects of estrogens on blood pressure control and suggest a mechanism for the m
75 ion to improve both medication adherence and blood pressure control and to reduce cardiovascular even
76 rtensive patients need two or more drugs for blood-pressure control and concomitant statin treatment
77 terol control, 1 in 3 persons still has poor blood pressure control, and 1 in 5 persons still has poo
79 inhibitors and ARBs have similar effects on blood pressure control, and that ACE inhibitors have hig
80 ange between the proportion of patients with blood pressure control/appropriate response for individu
81 directed toward earlier and more aggressive blood pressure control are likely to offer the greatest
83 e hoping that it would translate into better blood pressure control as well as incremental nephroprot
86 xide release, whereas for the same degree of blood pressure control, atenolol/bendrofluazide had no e
88 nts receiving antihypertensive therapy (with blood pressure controlled below target levels) had reduc
89 additional meaningful barriers to achieving blood pressure control beyond access to the monitor itse
90 he association between adherence and TI with blood pressure control (BP </= 140/90 at the clinic visi
91 ccurred in diagnosis, treatment and adequate blood pressure control (BP below 140/90 mmHg) since 2001
92 as effective in improving both adherence and blood pressure control, but it did not appear to improve
93 tics that could inform strategies to improve blood pressure control by decreasing untreated hypertens
94 tihypertensive drug utilization patterns and blood pressure control by insurance status, age, sex, an
95 ic hypertension (after an initial attempt at blood pressure control by weight reduction, if indicated
97 tegration of vestibular system pathways with blood pressure control centers in the ventrolateral medu
98 for aspirin or antithrombotic prescription, blood pressure control, cholesterol control, and smoking
99 S" (aspirin therapy in appropriate patients, blood pressure control, cholesterol management, and smok
100 scular disease, including lifestyle factors, blood pressure control, cholesterol-lowering, antithromb
101 vention including patient education improved blood pressure control compared with provider education
102 sure, we found a small reduction in systolic blood pressure control compared with usual care at 12 mo
104 t the importance of dietary sodium intake in blood pressure control, consideration of the most recent
105 sk factors for bleeding should be addressed (blood pressure control, discontinuing unnecessary medica
106 tarling mechanism contributes to compromised blood pressure control during simulated haemorrhage in h
107 r disease, achieved with the use of improved blood-pressure control during the trial, would be sustai
108 ion, skeletal muscle pump was found to drive blood pressure control (EMG --> SBP) as well as control
110 of intraventricular hemorrhage, and adequate blood pressure control for the optimization of cerebral
111 ngs for redefinition of future management of blood-pressure control for individuals at high risk of s
114 Patients randomized to placebo (moderate blood pressure control) had a mean blood pressure of 137
120 surgery represents an effective strategy for blood pressure control in a broad population of patients
121 on the contextual red flag, such as improved blood pressure control in a patient presenting with hype
122 there are cardiovascular benefits other than blood pressure control in blocking the renin system rema
123 association with use of these medicines and blood pressure control in countries at varying levels of
126 ports its role as a polygenic determinant of blood pressure control in humans, and results obtained f
128 f ischemic events, but the role of intensive blood pressure control in PAD has not been established.
129 ed statistically significant improvements in blood pressure control in patients with diabetes balance
130 [95% CI, 5.2 to 26.3 percentage points]) and blood pressure control in patients with hypertension (im
131 eplerenone and spironolactone, in improving blood pressure control in patients with resistant hypert
132 ommendations issued by scientific societies, blood pressure control in RTRs is far from the recommend
134 p; the time-to-event experience of achieving blood pressure control in the intervention versus contro
135 s for regulation of catecholamine action and blood pressure control in this widely studied model of h
136 GI-alpha oxidation in the EDHF mechanism and blood pressure control in vivo, we generated a knock-in
138 ther vasopressors may contribute to improved blood-pressure control in CAPD in contrast to hemodialys
139 ms of Na-K-ATPase inhibitors might relate to blood-pressure control in hemodialysis (N = six ultrafil
140 findings emphasise the importance of prompt blood-pressure control in hypertensive patients at high
141 py with an ACE inhibitor was associated with blood-pressure control in most patients with ADPKD and s
142 ere may be differential effects of intensive blood-pressure control in patients with and those withou
144 adherence and TI, the odds (OR) of achieving blood pressure control increased by 28% and 55%, respect
149 s, angiotensin-converting enzyme inhibitors, blood pressure control, lipid control, diabetic glycemic
151 ntemporary approach of antiplatelet therapy, blood pressure control, low-density lipoprotein reductio
152 in cholesterol <70 mg/dL or statin therapy), blood pressure control (<140 mm Hg systolic, <90 mm Hg d
154 cemic control, avoidance of smoking and good blood pressure control may be helpful in preventing or d
155 Particularly in black women, weight and blood pressure control may be important community health
157 ntrol, smoking cessation and prevention, and blood pressure control may help to reduce the risk of ha
158 12 patients with stage 2 and 3 CKD with good blood pressure control (mean daytime ambulatory blood pr
160 k of complications, but it appears that good blood-pressure control must be continued if the benefits
161 to prevent the development of HF, especially blood pressure control, must be a priority if mortality
162 2% to 9.7%], P = .001 for interaction term), blood pressure control (no comorbidities: 9.7% vs 4.3%,
163 e effect of the procedure on renal function, blood pressure control, number of antihypertensive medic
164 ifferences between fatty acid metabolism and blood pressure control of humans and laboratory animals,
167 or combined incentives, resulted in greater blood pressure control or appropriate response to uncont
168 A) and stenting indicate that improvement in blood pressure control or renal function is not a predic
169 ess than 9.0% (OR, 0.96 [95% CI, 0.74-1.2]); blood pressure control (OR, 0.99 [95% CI, 0.69-1.4]); LD
170 iated with a 25% increased odds of achieving blood pressure control (OR, 1.24; 95% CI, 1.21-1.27).
171 y of DN resulting from improved glycemia and blood pressure control, or whether there were overestima
172 study was designed to assess improvement in blood pressure control over a 10-year period in a large
173 ight loss, smoking cessation, and aggressive blood pressure control, particularly in African-American
174 renal dysfunction could beneficially affect blood pressure control, preserve or prevent deterioratio
175 In this simulation study, intensive systolic blood-pressure control prevented cardiovascular disease
179 Yet few trials have tested whether intensive blood-pressure control retards the progression of chroni
180 lic acidosis and anemia, achieving excellent blood pressure control, reversing cardiovascular complic
182 ts, Mr R faces several barriers to effective blood pressure control: societal, health system, individ
183 ed established lifestyle recommendations for blood pressure control (sodium reduction, weight loss, a
185 mphocytes in peripheral tissues important in blood pressure control, such as the kidney and vasculatu
186 wide-association studies broaden our view of blood pressure control, suggesting that renal sympatheti
188 ular disease who received intensive systolic blood-pressure control (target, <120 mm Hg) had signific
189 iometabolic events and death, independent of blood pressure control, than for patients with essential
193 ial cells, consistent with a primary role in blood pressure control through modulation of vascular to
194 factor for stroke, a national guideline for blood-pressure control to reduce the incidence of stroke
195 tatistically significant, ranging from 0.01 (blood pressure control) to -0.21 (cholesterol control in
196 Patient outcomes (glycemic, cholesterol, and blood pressure control, urgent care visits, emergency de
197 ned to test the hypothesis that for the same blood-pressure control, valsartan would reduce cardiac m
199 to 80+/-11 mm Hg, respectively; P<0.05), and blood pressure control was more facile in approximately
202 th standard blood-pressure control, rigorous blood-pressure control was associated with a slower incr
203 To test this hypothesis and its relevance to blood pressure control, we determined whether RGS2 funct
204 enin ratio (ARR), serum potassium level, and blood pressure control were assessed at 3 months and at
205 Significant positive predictors of adequate blood pressure control were CHD and antihypertensive med
207 eceiving tight, as compared with less tight, blood-pressure control were not sustained during the pos
209 affordability to use of these medicines and blood pressure control with multilevel mixed-effects log
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