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1 good agreement with in-vitro measurements of blood viscosity.
2 on blood flow and the estimates of effective blood viscosity.
3 ions with both fibrinogen concentrations and blood viscosity.
4 e posed an experimental challenge to measure blood viscosity.
5  cell measures is probably mediated by whole blood viscosity.
6         Hematocrit is a determinant of whole blood viscosity.
7  predicted a reduction in local and apparent blood viscosity after isovolemic hemodilution.
8  us to investigate the variation of apparent blood viscosity along the cross-section of a vessel.
9 e, we will review hemorheology, particularly blood viscosity along with other hemorheological factors
10                          Therefore, elevated blood viscosity and hematocrit deserve attention as emer
11 nown predictors of diabetes, estimated whole blood viscosity and hematocrit predicted incident type 2
12          These findings suggest that in men, blood viscosity and its major determinants are associate
13 ing hyperlipidemia is secondary to increased blood viscosity and not abnormal coronary vasomotion.
14 iated with hyperlipidemia is due to increase blood viscosity and not abnormal vascular function.
15 may be "downregulated." This may lower whole blood viscosity and partially reduce DBP without comprom
16 s a significant negative association between blood viscosity and the mean flow velocity in the middle
17 comes, including oxidative stress, increased blood viscosity, and impaired systemic response to infla
18 iscosity (P< or =.01), fibrinogen, corrected blood viscosity, and plasma viscosity (all P< or =.05).
19 uclear neutrophil; PMN) deformability, whole-blood viscosity, and platelet-neutrophil interactions on
20                  Other major determinants of blood viscosity are hematocrit, level of inflammatory pr
21 r and tissue pressures and/or an increase in blood viscosity as a result of increased hematocrit.
22 an additional mechanism of increased in vivo blood viscosity as compared to that determined in vitro.
23              Its width affects the effective blood viscosity as well as the scavenging and production
24 , we investigated the feasibility to monitor blood viscosity by electrical bioimpedance in 10 patient
25                     Instead, the increase in blood viscosity causes capillary resistance to rise, whi
26  data (immunoglobulin level, hematocrit, and blood viscosity), clinical examination results, and OCT
27 sure, decreased cardiac output and increased blood viscosity combine to diminish venous return.
28 monstrates the proof of principle to monitor blood viscosity continuously in the human right atrium b
29    The M-protein and the related increase in blood viscosity could be a novel etiologic discovery for
30                                              Blood viscosity decreases with shear stress, a property
31                                 Furthermore, blood viscosity, fibrinogen (both P< or =.01), and plasm
32  bulk modulus of the ruptured wall material, blood viscosity, flow rate and mass density of the monoc
33 r the non-linear regression model estimating blood viscosity from its major determinants hematocrit a
34 quartiles, adults in the highest quartile of blood viscosity (hazard ratio = 1.68, 95% confidence int
35                                   Changes in blood viscosity in vivo are associated with changes in f
36                                              Blood viscosity increased significantly as temperature d
37     Hemorheologic analyses revealed enhanced blood viscosity, increased aggregation, and disaggregati
38                                              Blood viscosity is an important determinant of microvasc
39 of evidence support the notion that elevated blood viscosity may predispose to insulin resistance and
40                                              Blood viscosity, or the resistance of flow, can be alter
41                                      In men, blood viscosity (P< or =.001) and its major determinants
42 s distribution (1.05 mm) for SD increases in blood viscosity (P< or =.01), fibrinogen, corrected bloo
43                                        Whole blood viscosity, plasma free hemoglobin, TR jet, and FMD
44                                        Whole blood viscosity, plasma viscosity, hematocrit, and fibri
45 nse relationship with fibrinogen, plasma and blood viscosity, platelet count, coagulation factors VII
46                             Considering that blood viscosity plays an important role in cerebral bloo
47                                              Blood viscosity provides the rheological basis to elucid
48  noted between serum triglyceride levels and blood viscosity (r=0.82).
49                      Radial distributions in blood viscosity, shear stress, and shear rate are obtain
50                                          The blood viscosity-shear rate relationship was obtained fro
51 This model explains 89.2% (R(2)=.892) of the blood viscosity-shear rate relationship.
52 t than DM patients and had an elevated whole blood viscosity that correlated with plasma glucose (p =
53  stimulates platelet function, and increases blood viscosity through hemoconcentration.
54                                   Correcting blood viscosity to a standard hematocrit of 45% had litt
55                                 Reduction of blood viscosity via gata1a morpholino oligonucleotides (
56 n the following equation to estimate in-vivo blood viscosity (Viscosity(imp)) from plasma resistance
57 l configuration, the experimentally acquired blood viscosity was compared with a vacuum-driven capill
58                                        Whole blood viscosity was estimated by using a validated formu
59                       At baseline, estimated blood viscosity was independently associated with severa
60 dynamics, myocardial blood flow (MBF), whole blood viscosity (WBeta), erythrocyte charge (EC) and mob
61                        To determine if whole blood viscosity (WBV), a rheologic variable contributing
62 with reduced cell deformability and elevated blood viscosity, which contribute to impaired blood flow
63 in Experiment 1 is associated with increased blood viscosity, which is an important factor affecting
64 ry high hemoglobin concentrations cause high blood viscosity, which results in both compromised oxyge
65 pression by nitrate may thus act to decrease blood viscosity while matching oxygen supply to demand,

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