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1 good agreement with in-vitro measurements of blood viscosity.
2 on blood flow and the estimates of effective blood viscosity.
3 ions with both fibrinogen concentrations and blood viscosity.
4 e posed an experimental challenge to measure blood viscosity.
5 cell measures is probably mediated by whole blood viscosity.
6 Hematocrit is a determinant of whole blood viscosity.
9 e, we will review hemorheology, particularly blood viscosity along with other hemorheological factors
11 nown predictors of diabetes, estimated whole blood viscosity and hematocrit predicted incident type 2
13 ing hyperlipidemia is secondary to increased blood viscosity and not abnormal coronary vasomotion.
15 may be "downregulated." This may lower whole blood viscosity and partially reduce DBP without comprom
16 s a significant negative association between blood viscosity and the mean flow velocity in the middle
17 comes, including oxidative stress, increased blood viscosity, and impaired systemic response to infla
18 iscosity (P< or =.01), fibrinogen, corrected blood viscosity, and plasma viscosity (all P< or =.05).
19 uclear neutrophil; PMN) deformability, whole-blood viscosity, and platelet-neutrophil interactions on
21 r and tissue pressures and/or an increase in blood viscosity as a result of increased hematocrit.
22 an additional mechanism of increased in vivo blood viscosity as compared to that determined in vitro.
24 , we investigated the feasibility to monitor blood viscosity by electrical bioimpedance in 10 patient
26 data (immunoglobulin level, hematocrit, and blood viscosity), clinical examination results, and OCT
28 monstrates the proof of principle to monitor blood viscosity continuously in the human right atrium b
29 The M-protein and the related increase in blood viscosity could be a novel etiologic discovery for
32 bulk modulus of the ruptured wall material, blood viscosity, flow rate and mass density of the monoc
33 r the non-linear regression model estimating blood viscosity from its major determinants hematocrit a
34 quartiles, adults in the highest quartile of blood viscosity (hazard ratio = 1.68, 95% confidence int
37 Hemorheologic analyses revealed enhanced blood viscosity, increased aggregation, and disaggregati
39 of evidence support the notion that elevated blood viscosity may predispose to insulin resistance and
42 s distribution (1.05 mm) for SD increases in blood viscosity (P< or =.01), fibrinogen, corrected bloo
45 nse relationship with fibrinogen, plasma and blood viscosity, platelet count, coagulation factors VII
52 t than DM patients and had an elevated whole blood viscosity that correlated with plasma glucose (p =
56 n the following equation to estimate in-vivo blood viscosity (Viscosity(imp)) from plasma resistance
57 l configuration, the experimentally acquired blood viscosity was compared with a vacuum-driven capill
60 dynamics, myocardial blood flow (MBF), whole blood viscosity (WBeta), erythrocyte charge (EC) and mob
62 with reduced cell deformability and elevated blood viscosity, which contribute to impaired blood flow
63 in Experiment 1 is associated with increased blood viscosity, which is an important factor affecting
64 ry high hemoglobin concentrations cause high blood viscosity, which results in both compromised oxyge
65 pression by nitrate may thus act to decrease blood viscosity while matching oxygen supply to demand,
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