ライフサイエンスコーパス: blood volume

1 on devices by increasing the processed whole blood volume.

2 ic shock by acutely withdrawing 50% of their blood volume.

3 as used as a surrogate of effective arterial blood volume.

4 imaging was used to measure tumor fractional blood volume.

5 ted with an impairment of effective arterial blood volume.

6 nancy to support rapid expansion of maternal blood volume.

7 SV (P = 0.021) and was proportional to total blood volume.

8 intraocular melanoma tumor area and relative blood volume.

9 corresponded with microvascular density and blood volume.

10 in SV during subsequent decreases in central blood volume.

11 ical role in reduction of blood pressure and blood volume.

12 utable to a small heart coupled with reduced blood volume.

13 erial pressures during reductions of central blood volume.

14 an emerging imaging biomarker of fractional blood volume.

15 kers for hypoxia and primarily determined by blood volume.

16 owed by (11)C-CO imaging to measure regional blood volume.

17 ciated with a decrease in effective arterial blood volume.

18 s an estimate of relative changes of central blood volume.

19 be required to replace more than the entire blood volume.

20 esponses to progressive reduction in central blood volume.

21 acers with high temporal resolution in small blood volume.

22 ging in small animals because of their small blood volume.

23 d errors, but it requires calibration of the blood volume.

24 associated with a parallel increase in tumor blood volume.

25 rrelated to regional differences in cerebral blood volume.

26 the paper filter extracted contains a fixed blood volume.

27 rho in inner retina was linked to changes in blood volume.

28 The assay was very sensitive with increased blood volumes.

29 mpaired venous return, and decreased central blood volumes.

30 n this study, we investigated the effects of blood volume (0.8, 1.0, and 1.2 ml), tube shaking (gentl

31 1) mainly as a result of decreased capillary blood volume (133.9+/-5.1 to 111.7+/-7.7 AU; P<0.05) wit

32 oanalytical HPLC-MS/MS assay requiring small blood volumes (15 microL) has been validated using aceta

33 vs 14 [range, 6-22] mL/100 mL/min), cerebral blood volume (2.4 [range, 1.6- 4.2] vs 3.9 [range, 3.4-4

34 . 1.45 g/kg [1.34, 1.57 g/kg]; p < 0.01) and blood volume (60 ml/kg [54, 64 ml/kg] vs. 71 ml/kg [65,

35 values were 0.93 for FMBV fractional moving blood volume (95% CI confidence interval : 0.82, 0.97) v

36 values were 0.95 for FMBV fractional moving blood volume (95% confidence interval [ CI confidence in

37 -e(-betat)) to quantify functional capillary blood volume (A), microvascular flow velocity (beta), an

38 criteria to select donors with an estimated blood volume above 3.5 l.

39 average 60% decrease in normalized cerebral blood volume (adults P < 0.05; children P < 0.001), whil

40 on, and assessed neurological deficit and HT blood volume after 24 hours.

41 ability to collect CTCs from a large patient blood volume allows this technique to be used experiment

42 logic (Gaussian normalized relative cerebral blood volume and apparent diffusion coefficient) paramet

43 mic properties of gliomas including cerebral blood volume and blood flow, vascular permeability, and

44 ntially linking together hormonal control of blood volume and blood glucose levels, and thus adding t

45 tational pooling excessively reduces central blood volume and cardiac output.

46 leeding either 35% or 25% of estimated total blood volume and compared with sham bled animals.

47 Blood volume and flow-extraction product derived at DCE

48 reased Gaussian-normalized relative cerebral blood volume and Gaussian-normalized relative cerebral b

49 olonged mean transit time, elevated cerebral blood volume and high mean transit time/cerebral blood f

50 as induced in rats by withdrawing 60% of the blood volume and maintaining a hypotensive state.

51 ulature rather than reduction in circulating blood volume and oxygen delivery.

52 However, the BOLD signal reflects changes in blood volume and oxygenation rather than neuronal activi

53 chnique that independently measures cerebral blood volume and oxygenation, continuously, in alert beh

54 ofluorescence and are affected by changes in blood volume and oxygenation.

55 E-MRI (n = 8) experienced decreases in tumor blood volume and permeability (P < .01).

56 There were negative correlations between blood volume and pimonidazole staining (r = -0.48, P = .

57 ovel class of loop diuretic that would lower blood volume and pressure without causing hypokalemia.

58 ggest that Ang II, in addition to regulating blood volume and pressure, may be a master regulator of

59 le electrolyte and water transport and hence blood volume and pressure.

60 NaC is a central effector affecting systemic blood volume and pressure.

61 4 hours of hemorrhage (removal of 40% of the blood volume and subsequent blood removal/retransfusion

62 t 4 hrs of hemorrhage (removal of 40% of the blood volume and subsequent blood removal/retransfusion

63 agic shock (removal of 30% of the calculated blood volume and subsequent titration of mean arterial b

64 to construct a calibration curve between the blood volume and the extent of ion suppression.

65 dies in rodents because of their small total blood volume and the related difficulties in withdrawing

66 This study identified blood volume and tube shaking as novel preanalytical sou

67 Three-dimensional FMBV fractional moving blood volume and VFI vascularization flow index produced

68 ost optimizations (construction materials or blood volume) and optimization of efficient flow via min

69 lationship between the PA:A ratio, pulmonary blood volume, and cardiac function.

70 ayer-specific measurements of BOLD, cerebral blood volume, and cerebral blood flow in regions of posi

71 eceived a lower transfusion volume per liter blood volume, and experienced a smaller posttransfusion

72 Changes in body water, blood volume, and extravascular fluid volume were calcul

73 ion with moderate confidence for blood flow, blood volume, and hepatic arterial fraction in tumors an

74 ration, less focal increase in CBF, cerebral blood volume, and hyperoxygenation, the duration of whic

75 to assess spectrometric results, fractional blood volume, and immunohistochemical evaluation.

76 coefficients, cerebral blood flow, cerebral blood volume, and intratumoral susceptibility signals.

77 and 1.9%, for cerebral blood flow, cerebral blood volume, and mean transit time, respectively.

78 level of cerebral hemoglobin concentration, blood volume, and oxygen saturation.

79 rate of contamination was higher with lower blood volumes, and there was no significant difference i

80 e blood pressure regulation, reduced central blood volume appears to be an underlying mechanism respo

81 sculature by increasing muscle microvascular blood volume ( approximately 2-fold, P < 0.05) and incre

82 g platelet function and coagulation with low blood volumes ( approximately 100 mul) over a wide range

83 alterations in vessel calibre or fractional blood volume as assessed using susceptibility contrast M

84 etinotopically specific when imaged by using blood volume as when using the initial dip.

85 that harbors a large proportion of the total blood volume at any given time.

86 rformed to evaluate capillary blood flow and blood volume at rest and during low- or high-intensity c

87 average 80% decrease in normalized cerebral blood volume at the core of the lesion (P < 0.0001).

88 .001), suggesting an increase in unstressed blood volume at the lowest dose of norepinephrine.

89 cal imaging of light scattering and cerebral blood volume, autofluorescence flavoprotein imaging (AFI

90 r aortic arch) with intraoperative bleeding (blood volume between 60 and 250 mL suctioned from the th

91 em to hone in on depth information regarding blood volume, blood flow velocity and direction, vascula

92 Blood volume (BV) was measured using the optimized carbo

93 >0.91) for blood flow (BF), high (>0.84) for blood volume (BV), and lower (>0.30 and >0.39) for mean

94 arameters, including capillary permeability, blood volume (BV), blood flow (BF), and mean transit tim

95 Blood flow (BF), blood volume (BV), flow extraction product (K(trans)), a

96 Blood flow (BF), blood volume (BV), mean transit time (MTT), and capillar

97 Blood flow (BF), blood volume (BV), mean transit time (MTT), and permeabi

98 usion parameters [including Blood Flow (BF), Blood Volume (BV), Mean Transit Time (MTT)] and permeabi

99 MION functional MRI measured stimulus-evoked blood-volume (BV) changes.

100 training increased left ventricular mass and blood volume by approximately 12% and approximately 7% a

101 Aldosterone synthase (AS) regulates blood volume by synthesizing the mineralocorticoid aldos

102 t only the quantitative measurement of total blood volume can help identify the heterogeneity in plas

103 n level-dependent (BOLD) signal and cerebral blood volume (CBV) and blood flow (CBF), which in turn w

104 ng voluntary behaviors by measuring cerebral blood volume (CBV) and neural activity in the somatosens

105 ultaneously, we measured changes in cerebral blood volume (CBV) as a proxy of drug effects on neurona

106 th blood oxygen level-dependent and cerebral blood volume (CBV) contrasts at 9.4 tesla, as well as la

107 genation level-dependent (BOLD) and cerebral blood volume (CBV) fMRI signal.

108 LD), cerebral blood flow (CBF), and cerebral blood volume (CBV) HRF to ultrashort forelimb stimulatio

109 (18)F-FDG uptake, permeability, and cerebral blood volume (CBV) in children with pediatric brain tumo

110 ring neural activity and changes in cerebral blood volume (CBV) in the somatosensory cortex of awake,

111 d prospectively, with guidance from cerebral blood volume (CBV) MR imaging measurements.

112 nsulin to increase skeletal muscle capillary blood volume (CBV) reduces glucose uptake in insulin res

113 Cerebral blood volume (CBV), cerebral blood flow (CBF), and mean

114 hresholds for mean transit time and cerebral blood volume (CBV).

115 microvascular blood flow (MBF) and capillary blood volume (CBV).

116 eceptor occupancy and hemodynamics [cerebral blood volume (CBV)] in the domains of space, time, and d

117 d-oxygen-level-dependent (BOLD) and cerebral-blood-volume (CBV)-based laminar fMRI and used these to

118 lood [T/B] ratio), vascularization (cerebral blood volume [CBV]), and vascular permeability (contrast

119 Cerebral blood flow, cerebral blood volume, cerebral oxygen metabolism (CMRO2), oxygen

120 ransit time/cerebral blood flow and cerebral blood volume/cerebral blood flow ratios.

121 ely attributable to the significantly larger blood volume changes that occur in the perimysial space.

122 Blood volume changes were identical (P = 0.867), but the

123 a hemodynamic response of oxygen supply and blood volume closely coupled to the up-regulation of CCO

124 -based sampling procedure to enable accurate blood volume collection on commercially available DBS ca

125 We also studied the diagnostic value of blood volume contraction indices and B-type natriuretic

126 esis that a small heart coupled with reduced blood volume contributes to the postural orthostatic tac

127 fits, and the 2-tissue model with estimated blood volume correction (2Tv) performed best, particular

128 A fixed blood volume correction was applied.

129 e imaging weighted for blood oxygenation and blood volume, demonstrating increased signals in hippoca

130 nfluence the osmotic (plasma osmolality) and blood volume-dependent compensatory thresholds for antid

131 Blood volume did not significantly change the proportion

132 MLN9708 has a larger blood volume distribution at steady state, and analysis

133 of peripheral circulatory function, regional blood volume distribution by impedance plethysmography,

134 t does not mitigate the reduction in central blood volume during a simulated haemorrhagic challenge c

135 Artificial colloids substitute blood volume during surgery; with the administration of

136 stress attenuates the reduction in regional blood volumes during a simulated haemorrhagic challenge

137 Fifty percent blood volume exchange transfusion followed by a 60% hemo

138 There was no difference in the blood volume-expanding properties of the 2 preparations

139 Based on a combination of blood volume expansion and increasing platelet counts, t

140 ver the 6 hrs, the magnitude and duration of blood volume expansion by the two colloids were similar

141 70%), echocardiography led to the absence of blood volume expansion in the remaining 14 patients who

142 to determine the 2D tumor size and relative blood volume; eyes in group 2 (n = 6) were imaged by 3D

143 es to quantify and evaluate tumor fractional blood volume (fBV) as a noninvasive imaging biomarker of

144 iabetes mellitus (T2DM) patients using small blood volumes (fingerprick ~100 muL).

145 tely detected lower blood velocities and low blood volume flow in the carotid arteries after ligation

146 vides coregistered images of oxygenation and blood volume/flow with the underlying anatomy and concen

147 We also find that targeting blood volume fraction (a parameter of the model) through

148 in terms of bone-remodeling activity (BRA), blood volume fraction (BVF), and hypoxia.

149 Whole tumor and regional rate constants and blood volume fraction (VB) were computed by using compar

150 MR blood volume fraction and MR estimate of sO(2) parameter

151 ue oxygen saturation, mean transit time, and blood volume fraction in the cortex and caudoputamen; 2)

152 ospective clinical translation, we calculate blood volume fraction parameter values from in vivo cont

153 Mean transit time and blood volume fraction were comparable between the four g

154 in oxygen saturation, mean transit time, and blood volume fraction were subsequently measured using a

155 Brain perfusion (mean transit time and blood volume fraction) was comparable between the three

156 aps of the microvascular architecture (i.e., blood volume fraction, vessel diameter) and function (bl

157 a reversible 2-tissue-compartment model with blood volume fraction.

158 We use blood-volume functional MRI to monitor longitudinally th

159 g of whole blood, it is essential that large blood volumes (>or=3 ml) be efficiently lysed before bea

160 ms designed to prevent rapid fluctuations in blood volume have recently been shown to decrease the fr

161 By day 4, hepatic blood volume (HBV) in the responder group decreased by 2

162 h ISOI to simultaneously map oxygenation and blood volume [i.e., total hemoglobin (HbT)] in primary v

163 (i) is sensitive; (ii) requires only a small blood volume; (iii) is faster, less labor intensive, and

164 a progressive decline of the renal relative blood volume in all models.

165 Heat stress reduced blood volume in all regions (ranging from 7 to 16%), whi

166 While normothermic,LBNP reduced blood volume in all regions (torso: 22 +/- 8%; heart: 18

167 redictive timing, with increases of cerebral blood volume in anticipation of trial onsets even in dar

168 The loss of blood volume in distal pulmonary vessels is associated w

169 LBNP while heat stressed, the reductions in blood volume in each region were markedly greater when c

170 flow and greater expansion of microvascular blood volume in MAPC-treated mice than in controls.

171 Limited blood volume in mice precludes repeated sampling, render

172 An accurate prediction of blood volume in patients is physiologically and clinical

173 al maps of changes in tissue oxygenation and blood volume in response to mechanical whisker stimulati

174 e biomarker of subsequent reduction in tumor blood volume in response to sunitinib, and acquired resi

175 ng variables were calculated: VB (fractional blood volume in target area), K(1) and k(2) (kinetic mem

176 The model predicts that the blood volume in the endomysial space increases 24% and d

177 However, these significant changes in blood volume in the endomysium produce a change of only

178 (15)O-CO PET showed decreased blood volume in the femoral artery after the injury.

179 To calculate the net amount of blood volume in the liquids, colorimetric assay using ca

180 ient decreases in cell swelling and cerebral blood volume in the surround are consistent with early i

181 ded RBCs may be proposed as reporters of the blood volume in the tumor region.

182 ith arrested progression normalized cerebral blood volume in this region.

183 nown dopamine areas were consistent with the blood volume in those structures.

184 serve as a control reference to estimate the blood volume in various organs and species.

185 ) of a colloid solution) normalizes regional blood volumes in the torso, but does not mitigate the re

186 liliters per 100 milliliters per minute) and blood volume (in milliliters per 100 milliliters) were d

187 Response to hemorrhage (20% loss of blood volume), including plasma renin activity, was asse

188 ericardium restrains LV filling when central blood volume increases.

189 Restoring extracellular blood volume, increasing GFR and calcium excretion, and

190 ivity, 59% specificity), change in pulmonary blood volume index (77% sensitivity, 82% specificity), a

191 sensitivity, 98% specificity) and pulmonary blood volume index (92% sensitivity, 68% specificity), a

192 dex (r = 0.17; p = .001), baseline pulmonary blood volume index (r = 0.15; p = .001), change in pulmo

193 ex (r = 0.15; p = .001), change in pulmonary blood volume index (r = 0.16; p < .001), and change in P

194 Baseline cardiac index and pulmonary blood volume index were higher, whereas change in cardia

195 change in cardiac index, change in pulmonary blood volume index, and change in PaO2/FIO2 ratio indivi

196 change in cardiac index, change in pulmonary blood volume index, and change in PaO2/FIO2 ratio were l

197 ry dilution such as cardiac index, pulmonary blood volume index, and extravascular lung water.

198 s baseline cardiac index, baseline pulmonary blood volume index, the change in cardiac index, change

199 r premature neonates who have a very limited blood volume is a particular challenge.

200 These data suggest that blood volume loading during passive heat stress (via 11

201 e to earlier assessments of the magnitude of blood volume loss during hemorrhage.

202 VLCBV was defined as cerebral blood volume<2.5th percentile of brain contralateral to

203 ntrast magnetic resonance perfusion cerebral blood volume maps were co-registered, segmented when cer

204 olume, whereas in vivo examination of larger blood volumes may be clinically restricted by the toxici

205 (2)R activity increases muscle microvascular blood volume (MBV) and glucose extraction, whereas unopp

206 ntraction each increase muscle microvascular blood volume (MBV) and glucose uptake.

207 d leg blood flow (LBF), muscle microvascular blood volume (MBV) and muscle protein turnover under pos

208 Microvascular blood flow (MBF) and blood volume (MBV) were quantified by contrast-enhanced

209 Muscle microvascular blood volume (MBV), microvascular blood flow velocity (M

210 Blood flow, blood volume, mean transit time (MTT), permeability-surf

211 ric modeling to determine tissue blood flow, blood volume, mean transit time, permeability, and hepat

212 sts, cardiac magnetic resonance imaging, and blood volume measurements.

213 etween Gaussian-normalized relative cerebral blood volume (nrCBV) and Gaussian-normalized relative ce

214 ely 10-fold more myeloid DCs than the entire blood volume of an average individual.

215 associated with increasing relative cerebral blood volume of NER (rCBVNER), which was higher with dee

216 on suppression zone in the chromatogram, the blood volume on the DBS cards can be calculated and furt

217 e calibration curve was used to estimate the blood volume on the DBS cards collected from 6 volunteer

218 hese peptides may not necessarily track with blood volume or invasive hemodynamic measurements in ind

219 ewed as a homeostatic response to changes in blood volume or tonicity.

220 Changes in blood volume (P = 0.867), strong ion difference (P = 0.2

221 ble 2-tissue model with 4 rate constants and blood volume parameter was preferred in 84% of cases.

222 ersible single-tissue-compartment model with blood volume parameter was the preferred plasma input mo

223 using an irreversible compartment model with blood volume parameter.

224 tissue-compartment models with and without a blood volume parameter.

225 cost, multiplexed assay requiring ultrasmall blood volumes, paving the way for the implementation of

226 We introduced dual-energy CT (DECT) perfused blood volume (PBV) as a PBF surrogate to evaluate whethe

227 FMBV fractional moving blood volume performed better than VFI vascularization f

228 Blood volume, permeability-surface area product, and v(e

229 g a potential link between the regulation of blood volume/pressure/osmolality and blood glucose.

230 sed on PRM analysis, a significantly reduced blood volume (PRM(rCBV)) at week 3 was noted in patients

231 ion flow index versus FMBV fractional moving blood volume produced an R(2) value of 0.211 and was sig

232 ion of Evans Blue Dye) and in renal relative blood volume quantified using in vivo microcomputed tomo

233 t effect is to decrease myocardial capillary blood volume rather than microvascular flow velocity, su

234 s highly accurate quantification of relative blood volume (rBV) and highly detailed three-dimensional

235 time-intensity curve, time to peak, relative blood volume (rBV), relative blood flow, and blood flow

236 ty curve [AUC], time to peak [TTP], relative blood volume [rBV], relative blood flow [rBF], and blood

237 Relative cerebral blood volume (rCBV) (maximum rCBV [rCBV(max)] and mean r

238 hesis was that a change in relative cerebral blood volume (rCBV) 1 month after RT-TMZ is predictive o

239 Relative cerebral blood volume (rCBV) and blood flow (rCBF) maps were acqu

240 Relative cerebral blood volume (rCBV) and flow (rCBF) maps were acquired b

241 tion caused an increase in regional cerebral blood volume (rCBV) around the lesion site after 6 h, to

242 imaging-based whole-tumor relative cerebral blood volume (rCBV) histograms.

243 erences were noted in age, relative cerebral blood volume (rCBV) in contrast-enhanced regions (cutoff

244 Relative cerebral blood volume (rCBV) maps were created from analysis of M

245 Relative cerebral blood volume (rCBV) ratio is one of the best noninvasive

246 oefficient (ADC) with high relative cerebral blood volume (rCBV) represented elevated choline (Cho)-t

247 Tumor relative cerebral blood volume (rCBV) was estimated from each DSC MR acqui

248 (PWI), especially maps of regional cerebral blood volume (rCBV), may provide similar diagnostic info

249 hanced perfusion-weighted (relative cerebral blood volume [rCBV]) imaging were evaluated in these 28

250 antly higher fMRI signals [relative cerebral blood volumes (rCBVs)] and atrophy were observed in both

251 These include macronutrient metabolism, blood volume regulation, immune system support, endocrin

252 We used an in vitro technique to investigate blood volumes required to detect bacteremia and fungemia

253 To minimize the blood volume requirement and to precisely define shear s

254 Due to blood volume requirements as well as ethical and practic

255 ive RV flow, oxygen extraction fraction, and blood volume, respectively, from which RV MVO2 was calcu

256 ersible single-tissue-compartment model with blood volume seems to be a good candidate model for quan

257 he hemodynamic stress of increased effective blood volume, setting in motion untoward molecular and b

258 The blood volume signal, by contrast, is believed to be more

259 ulation of TH(VTA) neurons enhanced cerebral blood volume signals in striatal target regions in a dop

260 of hemorrhagic shock (removal of 30% of the blood volume, subsequent titration of mean arterial pres

261 ood loss, expressed as a percentage of total blood volume (TBV), mean arterial pressure, and heart ra

262 The reduction in central blood volume that accompanies heat stress may contribute

263 greatest interindividual variations are the blood volume (the clearance process is rapid, and early

264 After accounting for tumor blood volume, the extravasated nanoparticles were quanti

265 on of red blood cells is intended to restore blood volume, tissue perfusion, and oxygen-carrying capa

266 t alter the extent of the reduction in these blood volumes to LBNP relative to heat stress alone (tor

267 Patients with major burns have major (>1 blood volume) transfusion requirements.

268 findings suggest stimulus-evoked changes in blood volume underlie a component of the retinal intrins

269 reduce errors from variations in the spotted blood volume/unit area.

270 our of hypovolemia resuscitation with 35% of blood volume using a high-molecular-weight hydroxyethyl

271 3D three-dimensional FMBV fractional moving blood volume value +/- standard deviation was 11.78% +/-

272 nalysis showed higher FMBV fractional moving blood volume values than VFI vascularization flow index

273 lesion was present, and normalized cerebral blood volume values were analysed using a Food and Drug

274 IS method was used to estimate and calibrate blood volume variation and also to quantify the voricona

275 te method for estimating and calibrating the blood volume variation on DBS cards, which greatly facil

276 ry (LC-ESI-MS) for estimating and correcting blood volume variations on the DBS cards.

277 The total blood volumes varied from 25 to 94 mL, with a mean of 51

278 Renal dopamine regulates blood pressure and blood volume via a natriuretic effect.

279 Very low cerebral blood volume (VLCBV), diffusion, and hypoperfusion lesio

280 uantification of proliferating cells, and BM blood volume was estimated by measuring the changes in t

281 Three-dimensional FMBV fractional moving blood volume was measured on the vasculature from the ut

282 Cardiac fractional microvascular blood volume was not greater in anaemic fetuses, suggest

283 thermodilution) were obtained while central blood volume was reduced via lower-body negative pressur

284 Measurement of fractional blood volume was similar in all tumors (2.6 AU +/- 0.5 x

285 hemodynamic changes using BOLD and cerebral blood volume-weighted (CBVw) fMRI.

286 trating that tumors with a larger fractional blood volume were less hypoxic.

287 h-old infant, mean transit time and cerebral blood volume were low relative to cerebral blood flow, w

288 as normal but mean transit time and cerebral blood volume were low, consistent with perfusion in exce

289 t (Q(peak)), haemoglobin mass (Hb(mass)) and blood volumes were assessed prior to and following ET.

290 Relative changes in torso and regional blood volumes were determined by gamma camera imaging wi

291 scular blood flux rate whereas microvascular blood volumes were not different between groups at basel

292 During reductions in central blood volume while heat stressed, a greater decrease in

293 the use of dual-energy X-ray absorptiometry, blood volume with the use of a carbon monoxide (CO)-rebr

294 en a low-volume resuscitation (LVR) (10%-20% blood volume) with saline or various cell impermeants (s

295 salt losses and reduced "effective arterial blood volume." With no gold standard, the reported measu

296 ressure-targeted hemorrhagic shock, the mean blood volume withdrawn was significantly lower in the an

297 % of cardiac capillaries and halves perfused blood volume within the affected region.

298 The relative blood volume within the tumor demonstrated sonographical

299 ctin potently increased muscle microvascular blood volume without altering microvascular blood flow v

300 for leukocyte characterization using smaller blood volumes would thus be useful in diagnostic setting