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1 llowing: pocket infection; endocarditis; and bloodstream infection.
2 estinal malignancy, yet are also isolated in bloodstream infection.
3 rt-term mortality in patients with S. aureus bloodstream infection.
4 isional surgical site infection, and primary bloodstream infection.
5 There were no cases of cannula-related bloodstream infection.
6 stantial burden of arterial catheter-related bloodstream infection.
7 tor-associated pneumonia or catheter-related bloodstream infection.
8 the prevalence of arterial catheter-related bloodstream infection.
9 and S. marcescens occurred closer to time of bloodstream infection.
10 an underrecognized cause of catheter-related bloodstream infection.
11 reduce the risk of arterial catheter-related bloodstream infection.
12 i as the sole pathogen in a catheter-related bloodstream infection.
13 ing the empirical treatment of patients with bloodstream infection.
14 uitable treatment in patients with S. aureus bloodstream infection.
15 mia, but TLR2(-/-)mice could still resolve a bloodstream infection.
16 83 for intra-abdominal infection and 45 for bloodstream infection.
17 tant clone associated with urinary tract and bloodstream infections.
18 omising approach for combating P. aeruginosa bloodstream infections.
19 nd their role in the management of pediatric bloodstream infections.
20 ulatory system, capable of causing S. aureus bloodstream infections.
21 how this interaction impacts the outcome of bloodstream infections.
22 lin-susceptible Staphylococcus aureus (MSSA) bloodstream infections.
23 spective multicenter cohort of P. aeruginosa bloodstream infections.
24 portant and widely used diagnostic assay for bloodstream infections.
25 ol that offers a new paradigm for diagnosing bloodstream infections.
26 or-associated pneumonia and catheter-related bloodstream infections.
27 e for a high proportion of urinary tract and bloodstream infections.
28 on making for patients with life-threatening bloodstream infections.
29 identification of organisms responsible for bloodstream infections.
30 to the prevention of central line-associated bloodstream infections.
31 tion about the role of PhoQ in P. aeruginosa bloodstream infections.
32 ens associated with trauma-related wound and bloodstream infections.
33 studies providing data for catheter-related bloodstream infections.
34 ention decreased mortality for patients with bloodstream infections.
35 etecting microorganisms that cause pediatric bloodstream infections.
36 I-TOF with AST intervention in patients with bloodstream infections.
37 ; 85.7% were HACO infections, and 93.2% were bloodstream infections.
38 ylococcus isolates were not considered to be bloodstream infections.
39 pirical antibiotic therapy in the setting of bloodstream infections.
40 al therapy-related outcomes in patients with bloodstream infections.
41 cteria and yeasts in patients with suspected bloodstream infections.
42 s to reduce the risk for catheter-associated bloodstream infections.
43 nd outcomes of rapidly growing mycobacterial bloodstream infections.
44 f MDROs and development of hospital-acquired bloodstream infections.
45 or the most commonly identified organisms in bloodstream infections.
46 improved clinical outcomes for patients with bloodstream infections.
47 nts with less frequent Gram-negative bacilli bloodstream infections.
48 surface decolonisation reduced all-pathogen bloodstream infections.
49 t (ICU) patients may affect catheter-related bloodstream infections.
50 mine a worse outcome both in respiratory and bloodstream infections.
53 lator-days; p < 0.001), and catheter-related bloodstream infections (1.0 vs 3.5 per 1,000 catheter-da
54 nd soft tissue infections than in nosocomial bloodstream infections (11.1% versus 5.6%, respectively;
55 group B, there were 53% fewer gram-positive bloodstream infections (15% [9 of 60] vs 32% [19 of 60];
57 were driveline infections (47%), followed by bloodstream infections (24% VAD related, and 22% non-VAD
58 = .03) and 64% fewer central line-associated bloodstream infections (3.4 vs 9.4 per 1000 central line
62 rrently the reference standard for detecting bloodstream infection, a multistep process which may tak
64 cloacae isolated between 2001 and 2011 from bloodstream infections across hospitals in the UK and Ir
66 ntion significantly reduced catheter-related bloodstream infection after large-scale implementation i
67 ly from the blood of patients with suspected bloodstream infections aids in diagnosis and guides trea
68 remain the leading cause of catheter-related bloodstream infection, although an increase in Gram-nega
70 n for empiric and definitive therapy of MSSA bloodstream infections among patients admitted to 122 ho
72 in patients at high risk of catheter-related bloodstream infection and central venous catheter or art
74 riking benefit for bacterial survival during bloodstream infection and dissemination to other tissues
75 al of 314 propensity score-matched S. aureus bloodstream infection and in 268 E. coli bloodstream inf
77 measure was a composite of catheter-related bloodstream infection and symptomatic deep-vein thrombos
78 rization was associated with a lower risk of bloodstream infection and symptomatic thrombosis and a h
79 ated vascular devices can act as a nidus for bloodstream infection and systemic pathogen disseminatio
80 tion and, more importantly, catheter-related bloodstream infection and warrants routine use in patien
81 cation of Gram-negative organisms that cause bloodstream infections and can significantly impact pati
82 (ICUs) resulted in greater reductions in all bloodstream infections and clinical isolates of methicil
83 ated clinical infection syndromes (including bloodstream infections and community-acquired pneumonia)
84 used as rescue therapy for complicated MRSA bloodstream infections and deserves further clinical eva
85 are divided into anatomic systems, including Bloodstream Infections and Infections of the Cardiovascu
86 ntact is associated with fewer gram-positive bloodstream infections and possible central line-associa
87 chlorhexidine may prevent hospital-acquired bloodstream infections and the acquisition of multidrug-
88 res ordered, and the cumulative incidence of bloodstream infections and ventilator-associated pneumon
90 ssociated pneumonia, central line-associated bloodstream infection, and catheter-associated urinary t
91 ality, lengths of hospitalization, recurrent bloodstream infections, and 30-day hospital readmissions
93 most common cause of central-line-associated bloodstream infections, and antibiotic resistance makes
94 ulture contamination, health care-associated bloodstream infections, and rates of the primary outcome
95 arge proportion of E. coli urinary tract and bloodstream infections, and they differ markedly in thei
99 Even with surgical and antibiotic therapy, bloodstream infections are associated with significant m
101 , 0.29 [95% CI, 0.10-0.82]; P = .02) and new bloodstream infection (ARR, 0.05 [95% CI, 0.00-0.09]; RR
102 e optimal preventive strategies for reducing bloodstream infections associated with arterial catheter
103 udies have shown that the occurrence rate of bloodstream infections associated with arterial catheter
104 median estimates of the incidence density of bloodstream infections associated with arterial catheter
106 wed records of all patients with C. glabrata bloodstream infection at Duke Hospital over the past dec
107 d susceptible K. pneumoniae isolates causing bloodstream infections at a tertiary care hospital in Ne
111 condary outcomes including hospital-acquired bloodstream infections, blood culture contamination, or
114 from hospital-admitted patients suspected of bloodstream infection (BSI) in 4 of 11 provinces in DRC
115 nce of beta-lactam resistance in VGS causing bloodstream infection (BSI) in neutropenic patients.
116 antifungal regimen for Candida parapsilosis bloodstream infection (BSI) in view of its reduced susce
121 nt Staphylococcus aureus (MRSA) carriage and bloodstream infection (BSI), which shows a male predomin
122 vancomycin dosing on patient outcome in MRSA bloodstream infection (BSI); (2) defining, testing, and
124 illin-resistant Staphylococcus aureus (MRSA) bloodstream infections (BSI) are classified epidemiologi
125 organism identification improves outcomes in bloodstream infections (BSI) but have not controlled for
128 ndida and multidrug-resistant (MDR) bacteria bloodstream infections (BSIs) and their crude death rate
129 in hospital-onset (HO) Staphylococcus aureus bloodstream infections (BSIs) and used whole-genome sequ
130 whether rising incidence rates of nosocomial bloodstream infections (BSIs) caused by antibiotic-resis
132 ta-lactamase inhibitors for the treatment of bloodstream infections (BSIs) due to extended-spectrum b
135 Information on community-acquired bacterial bloodstream infections (BSIs) in individuals infected wi
136 the treatment of gram-negative bacilli (GNB) bloodstream infections (BSIs) in patients presenting wit
145 theters are recommended for adults to reduce bloodstream infections but not for children because ther
146 tream infection were similar to those of all bloodstream infections, but the difference was not signi
149 ur knowledge, these are the first reports of bloodstream infections by Trichosporon inkin in patients
150 ering of blood cultures and the incidence of bloodstream infections calls into question the importanc
152 nfections (i.e., central-catheter-associated bloodstream infection, catheter-associated urinary tract
154 therapy failed in a neutropenic patient with bloodstream infection caused by a DAP-susceptible Entero
155 ort the case of a patient from Brazil with a bloodstream infection caused by a strain of methicillin-
156 taphylococci and protect mice against lethal bloodstream infections caused by a broad spectrum of MRS
157 patients with sepsis or septic shock due to bloodstream infections caused by GNB admitted between 20
158 ovel possibilities of treating or preventing bloodstream infections caused by pathogenic Gram negativ
159 acetylsalicylic acid therapy on mortality in bloodstream infections caused by S. aureus compared with
160 k for developing central catheter-associated bloodstream infections (CCABSIs) owing to children's chr
162 increase the risk of central line-associated bloodstream infections (CLABSIs) are not fully understoo
163 e was a composite of central line-associated bloodstream infections (CLABSIs), catheter-associated ur
164 ealthcare-associated central line-associated bloodstream infections (CLABSIs), using National Healthc
165 k therapy to prevent central line-associated bloodstream infections (CLABSIs), we performed a systema
166 catheters significantly reduced the risk of bloodstream infections compared with standard and hepari
167 percent of children experienced at least one bloodstream infection, corresponding to 5.11 (95% CI, 4.
168 in severe sepsis/septic shock, patients with bloodstream infection could be discriminated by a decrea
170 linically diagnosed sepsis, catheter-related bloodstream infection (CRBSI), and all-cause mortality.
171 r complications of HPN were catheter-related bloodstream infections (CRBSIs) (1.7/1000 d of PN) and i
172 e parenteral support (HPS), catheter-related bloodstream infections (CRBSIs) inflict health impairmen
174 receiver operating characteristic curve for bloodstream infection diagnosis was significantly greate
175 pid identification of microorganisms causing bloodstream infections directly from a positive blood cu
176 ed assay can rapidly detect F. tularensis in bloodstream infections directly in whole blood at the ea
180 ith increased incidence of enteric bacterial bloodstream infections (EB-BSI), this association has no
182 In two patients, an attenuated toxicity bloodstream infection evolved from an asymptomatically c
184 total hospital costs decreased by $2,439 per bloodstream infection, for an approximate annual cost sa
186 rmed a post hoc analysis of patients with PA bloodstream infections from a published prospective coho
188 By 24 months postimplementation, the rate of bloodstream infection had fallen 25.5% and was 15.1% low
190 Interventions to reduce hospital-acquired bloodstream infection have succeeded in reducing rates i
192 he primary endpoint in patients with E. coli bloodstream infection (hazard ratio, 0.78; 95% CI, 0.40-
193 scharged home (all p</=0.002); had decreased bloodstream infection, hospital-acquired pressure ulcer,
194 aureus (MRSA) is a frequent cause of lethal bloodstream infection; however, vaccines and antibody th
196 sfully treated A. oryzae catheter-associated bloodstream infection in an immunocompetent patient prio
198 associated with at least 400,000 episodes of bloodstream infection in patients with cancer every year
199 nvasive candidiasis is the third most common bloodstream infection in the intensive care unit (ICU) a
200 me sequencing of E. faecalis associated with bloodstream infection in the UK and Ireland over more th
201 lso available for E. faecium associated with bloodstream infections in 15 patients in neighboring hos
202 nin both have a promising role in predicting bloodstream infections in a manner more helpful than C-r
203 ent interventions on central line-associated bloodstream infections in adult intensive care units.
204 standard central venous catheters to prevent bloodstream infections in children needing intensive car
207 bial-resistant S. marcescens associated with bloodstream infections in hospitals across the United Ki
208 eate the impact of propofol sedation on MRSA bloodstream infections in mice in the presence and absen
210 central venous catheters could help prevent bloodstream infections in paediatric intensive care unit
211 illin-resistant Staphylococcus aureus (MRSA) bloodstream infections in patients with impaired renal f
214 f catheter colonization and catheter-related bloodstream infection, including arterial catheters used
215 of discrete lineages caused the majority of bloodstream infections, including one subclone (ST131-H3
216 We found no difference in catheter-related bloodstream infection (internal jugular 1.0 vs. femoral
222 sion, these data suggest that S. epidermidis bloodstream infection is cleared in a highly efficient m
227 al jugular for the risks of catheter-related bloodstream infection, major catheter-related infection,
228 140 isolates of S. pneumoniae recovered from bloodstream infection (n = 70) and meningitis (n = 70) t
230 cal infections of >0.3/1,000 patient days or bloodstream infections of >0.03/1,000 patient days shoul
231 her catheter-related clinical sepsis without bloodstream infection or catheter-related bloodstream in
233 ections included pleuropneumonitis, isolated bloodstream infection, osteomyelitis, endocarditis, and
237 es in children should focus on prevention of bloodstream infections, particularly among neonates and
238 eus bloodstream infection and in 268 E. coli bloodstream infection patients, respectively (1:1 match
240 ma; any ICU-acquired infection; ICU-acquired bloodstream infection, pneumonia, and urinary tract infe
241 2013 and June 2014 with suspected or proven bloodstream infection, pneumonia, or sterile fluid and t
243 ich may suggest that the catheter-associated bloodstream infection prevention program was particularl
244 hlorhexidine bathing, 4) catheter-associated bloodstream infection program, and 5) daily goals sheets
246 in a reduced prevalence of catheter-related bloodstream infection (random effects relative risk, 0.6
252 is situations, decreases catheter-associated bloodstream infections, reduces blood product utilizatio
254 ty of America definition of catheter-related bloodstream infection remains the most precise definitio
255 inical S. aureus isolates from patients with bloodstream infections, representing two globally import
257 imens collected from patients with suspected bloodstream infections resulted in 35 PCR/ESI-MS-positiv
259 racteristics were excluded, catheter-related bloodstream infection risk was comparable between the si
261 an may, similarly, decrease catheter-related bloodstream infection risk, when compared with femoral.
263 icantly greater reduction in the rate of all bloodstream infections than either targeted decolonizati
264 Klebsiella pneumoniae causes severe lung and bloodstream infections that are difficult to treat due t
265 of MRSA, highlight the growing challenge of bloodstream infections that are effectively impossible t
266 , however, at high risk for catheter-related bloodstream infections that can result in substantial mo
267 , are responsible for the majority of fungal bloodstream infections that cause morbidity, especially
268 site infections, and 2 versus 0 for primary bloodstream infection; the effect was consistent across
269 o preventing central venous catheter-related bloodstream infection to arterial catheters as well.
270 methicillin-resistant Staphylococcus aureus bloodstream infections treated with vancomycin was perfo
273 omes (ICU mortality, central line-associated bloodstream infection, ventilator-associated pneumonia,
274 ile infection (CDI), central line-associated bloodstream infection, ventilator-associated pneumonia/e
277 the purposes of this study, catheter-related bloodstream infection was defined as positive blood cult
280 Low-dose acetylsalicylic acid at the time of bloodstream infection was strongly associated with a red
284 Trichosporon inkin, a novel organism causing bloodstream infection, was detected in 2 patients with p
285 lis is a natural heme auxotroph and cause of bloodstream infection, we explored whether restoration o
293 s activating protease in the pathogenesis of bloodstream infection, which indicates a greater complex
294 strategies for the pathogenesis of S. aureus bloodstream infections, which culminate in the establish
295 n versus baseline periods, hazard ratios for bloodstream infection with any pathogen in the three gro
299 ndidaemia is the fourth most common cause of bloodstream infection, with a high mortality rate of up
300 is the single most prevalent cause of fungal bloodstream infections worldwide causing significant mor
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