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1 and green cones, and prefer ultraviolet over blue cones.
2 toreceptor subtype, the S (short wavelength, blue) cones.
4 result in syndromes characterized by excess blue cones and loss of rods: enhanced S-cone syndrome (E
5 sed in human green/red cones but absent from blue cones and mediated ester hydrolysis for photopigmen
6 a are not detectably affected by ablation of blue cones, and are reduced twofold in mutant larval ret
7 d visual function, involving the minority S (blue) cones, and decreased rod and L/M (red/green) cone
8 ods and UV cones, RGB cones (red, green, and blue cones) are structurally similar and unite into mirr
9 he biocytin wide-field bipolar cell is an ON blue cone bipolar cell in the rabbit retina and is homol
11 olar type 6 (DB6; marked with anti-CD15) and blue cone bipolar cells (marked with anti-CCK precursor)
12 n the rabbit retina and is homologous to the blue cone bipolar cells that have been previously descri
14 ive cone terminals contact the dendrites of "blue-cone bipolar" cells instead, showing that they are
15 nd OFF midget bipolar cells as well as (ON) "blue-cone bipolar" cells, we examined 118 contiguous con
16 een rods share the same visual pigment, only blue cones but not green rods are able to dark-adapt in
17 ust sensitivity recovery in bleached red and blue cones but not in red and green rods, suggesting tha
19 despite sharing the same pigment, salamander blue cones, but not green rods, recovered their sensitiv
21 ast between color and background, and yellow-blue cone-contrast could account for dichromats' pattern
22 dependent patients had significantly reduced blue cone electroretinogram responses compared with matc
23 in transfected cells, which may explain why blue cone function is lost earlier than red/green-cone f
25 d not affect the ability of the red cone and blue cone/green rod pigments to activate transducin.
28 a rationale for why mammalian red, green and blue cones have comparable sensitivities, unlike their a
30 omatic resolution acuity under conditions of blue cone isolation (an indirect measure of the underlyi
33 X-linked cone dysfunction disorders such as Blue Cone Monochromacy and X-linked Cone Dystrophy are c
34 missense mutation presented with congenital blue cone monochromacy, with retinal lamination defects
37 nital stationary night blindness (CSNB), and blue-cone monochromatism (BCM)-in which nystagmus accomp
39 agglutinin, and then used antibodies against blue cone opsin and red-green cone opsin to identify the
40 llowed by regeneration, were hybridized with blue cone opsin cRNA for quantitative analysis of the bl
44 n, cyclic nucleotide-gated cation channel-3, blue-cone opsin, and beta-6-PDE) was evaluated by immuno
45 ic nucleotide-gated cation channel-3 [CNG3], blue-cone opsin, and cGMP phosphodiesterase [PDE]); were
46 that may play roles in the rod pathway, the blue cone pathway, and ganglion cell directional selecti
48 eurons in the short-wavelength cone (S-cone, blue cone) pedicle and to learn more concerning the uniq
50 xcess of the minority S (short wavelength or blue) cone photoreceptor type, but near absence of funct
51 tions in the outer segments of red/green and blue cone photoreceptors, B-type horizontal cells, sever
54 entified 12 amino acid residues in the human blue cone pigment that might induce the required green-t
56 ng shifts the spectral maxima of the red and blue cone pigments, but not that of the red rod pigment.
61 But, if regeneration is delayed or absent, blue-cone synaptogenesis increases and ectopic synapses
68 wild-type mouse ultraviolet (UV) and bovine blue cone visual pigments have absorption maxima of 358
71 , establishing synapses with ultraviolet and blue cones while eliminating red and green cone contacts
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