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1 t vector ability there of C. variipennis for bluetongue.
2 characterize thoroughly the pathogenesis of bluetongue.
3 % of clusters were unlikely to be related to Bluetongue.
4 hin a serotype affect the clinical course of bluetongue.
5 e been implicated in the recent epizootic of bluetongue.
6 n sheep, the animal species most affected by bluetongue.
7 heep, the animal species most susceptible to bluetongue.
10 tongue virus (BTV) is the causative agent of bluetongue, a major infectious disease of ruminants with
11 host, two-vector formulation for the R(0) of bluetongue, a vector-borne infection of ruminants that c
12 help identify geographic regions at risk for bluetongue and provide opportunities to prevent virus tr
13 m a veterinary perspective, the emergence of Bluetongue and Schmallenberg viruses show that northern
14 here are clear links between the severity of bluetongue and the mammalian host species infected, whil
17 ngue virus (BTV) is the etiological agent of bluetongue (BT), a hemorrhagic disease of ruminants that
20 umber of calvings among beef cows during the Bluetongue epizootic of 2007 and 2008, based on calving
23 g the calving season of primary infection by Bluetongue in 28% (n = 23) of the units first infected i
33 s of space and host, which is illustrated by bluetongue, Lyme disease, and West Nile virus, and it is
34 TV-8 strain isolated at the beginning of the bluetongue outbreak in 2006 was more virulent than a str
35 urves taken from two historical outbreaks: a bluetongue outbreak in cattle, and a whooping cough outb
36 a sheep that had been killed during a severe bluetongue outbreak that occurred in Rahuri, Maharashtra
37 highlight those areas of the epidemiology of bluetongue that are poorly understood, reflect upon why
39 C. variipennis genetic variation controlling bluetongue transmission will help identify geographic re
41 it pathogens of humans and livestock such as bluetongue virus (BTV) (Reoviridae), Oropouche virus (Bu
52 better define the molecular epidemiology of bluetongue virus (BTV) infection, the genetic characteri
63 and lentivirus-induced immunosuppression on bluetongue virus (BTV) pathogenesis as a mechanism for v
64 The entire genome of the reference strain of bluetongue virus (BTV) serotype 16 (strain RSArrrr/16) w
66 cally engineered the segmented RNA genome of bluetongue virus (BTV), a complex nonenveloped virus bel
69 t capsid structures of large viruses such as bluetongue virus (BTV), a member of the Orbivirus genus
71 tious diseases of ruminants and is caused by bluetongue virus (BTV), an arbovirus existing in nature
72 or infectious disease of ruminants caused by bluetongue virus (BTV), an arbovirus transmitted by Culi
73 ome penetration assay and cell biology, that bluetongue virus (BTV), an archetypal member of the Reov
74 tious diseases of ruminants and is caused by bluetongue virus (BTV), an arthropod-borne virus transmi
84 arried out during unprecedented outbreaks of bluetongue virus in Europe, an arbovirus of wild and dom
85 loped for the wind-borne spread by midges of bluetongue virus in NW Europe in 2006 is here modified a
91 full-genome sequence of an Indian isolate of bluetongue virus serotype 1 (BTV-1), strain IND1992/01.
92 enome sequence of an isolate (IND2004/01) of bluetongue virus serotype 10 (BTV-10) from Andhra Prades
94 f 19,188 bp) were sequenced from a strain of bluetongue virus serotype 3 (BTV-3) from India (strain I
96 odel based on the remotely related template, bluetongue virus VP7, improved from 8.7A to 6.0A, while
100 for epizootic hemorrhagic disease virus and bluetongue virus, failed to identify an etiologic agent.
102 ical vaccines that afford protection against bluetongue virus, the etiological agent, are not free fr
103 compared to the homologous protein (VP7) of bluetongue virus, which is also a member of the family o
106 rotavirus VP6 amplitudes were scaled to the bluetongue VP7 amplitudes derived from the atomic model
108 To compare VP6 and VP7, a projection map of bluetongue VP7 at 15 A resolution was generated using th
109 r their calving season of primary infection, Bluetongue was detected more rapidly after the start of
110 , although epizootic hemorrhagic disease and bluetongue were included in the differential diagnosis.
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