ライフサイエンスコーパス: body fat

1 ons for both waist circumference and percent body fat.

2 and decreased BMI, waist circumference, and body fat.

3 sed EE, increased fat oxidation, and loss of body fat.

4 y measured weight and height) and percentage body fat.

5 may be involved in the development of excess body fat.

6 ings at follow-up compared to those who lost body fat.

7 o physiologically important effects on EE or body fat.

8 of 26.9 +/- 0.9 kg m(-2) , and 24.8 +/- 1.3% body fat.

9 olescents who showed stability of or loss of body fat.

10 to those who showed stability of or loss of body fat.

11 restriction, causing them to lose nearly all body fat.

12 aseline LTL, age, smoking, and percentage of body fat.

13 y associated with fat mass and percentage of body fat.

14 d satiety, resulting in leanness and reduced body fat.

15 e but were less consistent for percentage of body fat.

16 onships among body mass, height, and percent body fat.

17 dditional adjustments for visceral and total body fat.

18 7 g; 95% CI: 4.5, 10.9 g), and percentage of body fat (0.12%; 95% CI: 0.08%, 0.16%).

19 g; 95% CI: 21.4, 46.6 g), and percentage of body fat (0.55%; 95% CI: 0.37%, 0.72%).

20 1 (95% CI -0.00325--0.000969), percentage of body fat -0.00516 (95% CI -0.00761--0.0027), high densit

21 to -0.39), and 0.81 percentage points lower body fat (-1.03 to -0.59).

22 e-trained men [aged 21 +/- 1 y; 88 +/- 3 kg; body fat: 16% +/- 1% (means +/- SEMs)] received primed c

23 ning group had greater changes in percentage body fat (-2.4, 95% CI, -3.2 to -1.6) vs the aerobic gro

24 body composition when compared with peers (% body fat, 26.2 +/- 8.2 vs 22.7 +/- 7.1).

25 age, 65.5 years; mean BMI, 27.4 kg/m2; mean body fat, 29.5%).

26 age, 63.5 years; mean BMI, 27.0 kg/m2; mean body fat, 32.1%) and 4944 men (mean age, 65.5 years; mea

27 5 (-4.6, -2.0) cm; P = 0.02], and percentage body fat [-4.7 (-6.1, -3.5) compared with -2.6 (-3.9, -2

28 years), waist circumference (4-8 years), and body fat (8 years).

29 significant attenuation of the rate of total body fat accumulation, along with a decrease in hepatic

30 tor that markedly affects energy storage and body-fat accumulation in mammals, yet the underlying mec

31 ed by peak oxygen consumption, percentage of body fat (adiposity) by dual-energy x-ray absorptiometry

32 5% CI: -0.07, 0.13; P = 0.61); percentage of body fat adjusted mean difference: -0.26 (95% CI: -0.99,

33 5% CI: -0.08, 0.12; P = 0.66); percentage of body fat adjusted mean difference: 0.11 (95% CI: -0.60,

34 exposures were not associated with increased body fat among children 4-9 years of age, though high pr

35 during pregnancy are associated with higher body fat and abdominal fat in childhood.

36 The beta-DKO mice also had increased body fat and adipose tissue macrophage content, elevated

37 nificantly associated with the percentage of body fat and body mass index (both P < 0.05) while contr

38 GWG was associated with a higher percentage body fat and greater long-term PPWR in mothers with lowe

39 Differences in adiposity (high percentage of body fat and high proportion of deep subcutaneous and vi

40 and how the sensory nervous system regulates body fat and how food perception is coupled with the pro

41 Body fat and intrahepatic fat were detected by magnetic

42 rum cortisol were associated with changes in body fat and LBM, but did not explain much variance in e

43 e significantly leaner with prominently less body fat and lived significantly longer compared with wi

44 creased metabolic flexibility while reducing body fat and liver lipids, compared with untreated obese

45 ne (IOM) guidelines] to outcomes [percentage body fat and long-term PPWR (change in weight from prepr

46 used to derive estimates for the percentage body fat and PPWR associated with excessive GWG, was est

47 Total body fat and regional fat mass percentages of the child

48 Total percentage body fat and trunk fat tended to be associated with grea

49 ndex, waist circumference, and percentage of body fat) and the onset of depressive symptoms.

50 ity with age, accuracy in estimating percent body fat, and accuracy in classifying adolescents as ove

51 non-resting energy expenditure, one-half the body fat, and better glucose clearance.

52 ite such a regain, weight, the percentage of body fat, and fat mass remained significantly reduced fr

53 et promoted a decrease in BMI, percentage of body fat, and leptin concentrations, which improved oocy

54 nding factors (age, body mass index, percent body fat, and systolic blood pressure), the prevalence o

55 sted for socioeconomic status, percentage of body fat, and total energy intake.

56 BMI, percent of body fat, and waist circumference were associated with i

57 BMI; in kg/m(2)), body weight, percentage of body fat, and waist circumference] and glucose and insul

58 per day, lung cancer, insulin resistance and body fat are most negatively correlated.

59 , pointing to qualitative characteristics of body fat as potential mediators.

60 comes were the BMI z score and percentage of body fat at 3 and 5 y of age.

61 ndex (BMI), waist circumference, and percent body fat at 8 y of age.

62 Every additional 1% of body fat at ages 8 to 10 years decreased insulin sensiti

63 BMI, fat mass, lean mass, and percentage of body fat at birth as well as at ages 1, 2 to 3, and 4 to

64 in postnatal life but had greater percentage body fat at term age (beta=5.73, p<0.001), but not at 3

65 s and bioelectrical impedance measurement of body fat) at age 11.5 years using the same data set in a

66 Further adjustment for percent body fat attenuated these associations.

67 e (beta -0.54 cm, -0.61 to -0.48), and whole body fat (beta -0.38 kg, -0.43 to -0.33), and with decre

68 ence (beta 0.41 cm, 0.28 to 0.54), and whole body fat (beta 0.40 kg, 0.30 to 0.50), and with increase

69 3.13, -0.50) at 4-8 years, and lower percent body fat (beta = -2.37%; 95% CI: -4.21, -0.53) at 8 year

70 ter stability with age and estimated percent body fat better than BMI (R2 = 0.64 vs 0.38 in boys and

71 Percent body fat (BF) was assessed via dual X-ray absorptiometry

72 ore (WZ), fat-free mass (FFM), percentage of body fat (%BF), and modifiable lifestyle factors for all

73 d 7 years, and fat mass index (FMI), percent body fat (%BF), and waist circumference (WC) at 7 years.

74 sulin sensitivity index (ISI), percentage of body fat (%body fat), visceral (VAT) and subcutaneous (S

75 nsor enables easily applicable and hand-held body fat burn monitoring for personalized and immediate

76 WO3 nanoparticles that can accurately follow body fat burn rates in real time.

77 Yet suitable biomedical sensors to monitor body fat burn rates in situ, to guide physical activity

78 ion on body weight, waist circumference, and body fat by conducting a systematic review and meta-anal

79 Body fat changes in the atazanavir/ritonavir arm were as

80 With higher %body fat, Chinese exhibited smaller increases in deep SA

81 wer serum insulin concentrations and percent body fat compared with all other groups.

82 wever, YAC128 mice were heavier and had more body fat compared with WT mice.

83 t circumference, and waist-to-hip ratio) and body fat composition (total body fat percentage and trun

84 oratory assessment.Nonlean individuals (high body fat) consumed most of their calories 1.1 h closer t

85 rowing evidence points to factors other than body fat content and fat distribution in determining a h

86 g BMI is associated with larger increases in body fat content in Asians, growing evidence points to f

87 had significantly greater body weight gain, body fat content, and glucose intolerance.

88 meters (body weight changes, hormone levels, body fat content, and glucose tolerance) in the exposed

89 ng a protective physiologic effect of higher body-fat content in later life.

90 fter adjustment for age, race, percentage of body fat, daily vigorous exercise, perceived stress, dep

91 y weight z-score (decrease of 3.1%), percent body fat (decrease of 2.4%), and percent trunk fat (decr

92 remained stable during the 14 d of cycling, body fat decreased (-2.2 +/- 0.7 kg; P = 0.02) and fat-f

93 upport to the premise that increased central body fat deposition is associated with body image dissat

94 Adolescents who gained body fat did not show a reduction in responsivity of rew

95 alth (mHealth) obesity prevention program on body fat, dietary habits, and physical activity in healt

96 Excess body fat diminishes muscle protein synthesis rates in re

97 effects on lipids, adverse events (AEs), and body fat distribution (dual-energy x-ray absorptiometry)

98 hology were unaffected, resulting in altered body fat distribution and a reduced VAT:SAT ratio in zeb

99 s in zebrafish to show that Plxnd1 regulates body fat distribution and insulin sensitivity.

100 se our understanding of the genetic basis of body fat distribution and its molecular links to cardiom

101 tudies have implicated PLEXIN D1 (PLXND1) in body fat distribution and type 2 diabetes.

102 The genetic determinants of body fat distribution are poorly understood.

103 new insights into the underlying genetics of body fat distribution by conducting sample-size-weighted

104 The N[t]RTI regimen affected lipid and body fat distribution changes.

105 Variation in body fat distribution contributes to the metabolic seque

106 In conclusion, the genetic influence on body fat distribution could be mediated via several spec

107 Body fat distribution is a heritable trait and a well-es

108 We hypothesized that detailed body fat distribution measures might be more strongly as

109 Studying detailed body fat distribution measures might provide better insi

110 Body fat distribution was assessed based on anthropometr

111 d1 mutants, the effect on VAT morphology and body fat distribution was dependent on induction of the

112 e stress (Montreal Imaging Stress Task), and body fat distribution were measured using advanced magne

113 e-treated mice showed identical weight gain, body fat distribution, and insulin sensitivity compared

114 y Plxnd1 as a novel regulator of VAT growth, body fat distribution, and insulin sensitivity in both z

115 littermates displayed identical weight gain, body fat distribution, and macrophage infiltration into

116 dence-based research is discussed concerning body fat distribution, dyslipidemia, hypertension, diabe

117 We examined body fat distribution, independent of BMI and depressive

118 isease were also associated with a favorable body fat distribution, with a lower waist-to-hip ratio (

119 ave identified numerous loci associated with body fat distribution.

120 g to an even greater exacerbation of altered body fat distribution.

121 etic susceptibility to overall adiposity and body fat distribution.

122 etric measurements, blood pressure and total body fat distribution] of these adolescents were collect

123 ipocyte kinetics in individuals with varying body fat distributions and degrees of metabolic health a

124 At 96 weeks, body fat (estimated difference 2862.2 gr; 95% CI, 726.7

125 y associated with reduced BMI and percentage body fat for both sexes, with a graded pattern apparent

126 ysical activity, except in the case of whole body fat, for which the protective effects were stronger

127 Total body fat-free mass (175 +/- 96 vs 84 +/- 71, P < 0.001)

128 The primary outcome measure is a change in body fat from baseline to 12 weeks.

129 The primary outcome was change in percent body fat from baseline to 16 weeks.

130 and fat-free mass indices and percentage of body fat from bioimpedance; waist circumference; overwei

131 impending milkshake receipt predicted future body fat gain (r = 0.32), which is a novel finding that

132 abitually low calcium intakes would decrease body fat gain compared with girls who continued their lo

133 date response to milkshake receipt predicted body fat gain for adolescents with a genetic propensity

134 allele, but lower caudate response predicted body fat gain for adolescents with a genetic propensity

135 ed reward response to food receipt predicted body fat gain for adolescents with a TaqIA A2/A2 allele

136 2 allele and lower reward response predicted body fat gain for those with a TaqIA A1 allele.

137 t difference between groups in percentage of body fat gain over 12 mo (mean +/- SEM: dairy 0.40% +/-

138 palatable food and monetary reward predicted body fat gain over 3-year follow-up in healthy-weight ad

139 palatable food and monetary reward predicted body fat gain over a 3-year follow-up in healthy-weight

140 d receipt of monetary reward did not predict body fat gain, which has not been tested previously.

141 date response to milkshake receipt to future body fat gain, which is another novel finding.

142 on in response to food cues predicted future body fat gain.

143 weight, body mass index (BMI), percentage of body fat, glucose, insulin, homeostasis model assessment

144 for confounders and premenarcheal percentage body fat, greater consumption of caffeinated soft drinks

145 st that both females and males (i) increased body fat, (ii) decreased relative muscle mass, (iii) red

146 natal BPA exposure is associated with higher body fat, impaired glucose tolerance, and reduced insuli

147 sity, waist circumference, and percentage of body fat in 9-year-old children (n = 261) in the Center

148 ls of the effect of dairy food on weight and body fat in adolescents have been reported to our knowle

149 in childhood (beta = 0.079, P = 0.03) and % body fat in adulthood (beta = 3.4%, P = 3 x 10(-7)).

150 nergy surfeit, predicted future increases in body fat in both studies.

151 information on the neuroendocrine control of body fat in C. elegans.

152 sociated with a preferential distribution of body fat in central regions, which does not, however, se

153 ower body mass index (BMI) and percentage of body fat in children.

154 and scFEM depots and was related to percent body fat in each depot.

155 urinary phthalate concentrations and percent body fat in models examining continuous exposures.

156 nsulin concentrations, and increased percent body fat in offspring.

157 tatus (indicated by weight, body mass index, body fat) in a cross-section, but longitudinal analyses

158 creased insulin, leptin, weight, and percent body fat) in the Long-Evans, but not Sprague-Dawley, str

159 osition, waist circumference, and percentage body fat] in children and adolescents.

160 sity, as revealed by reduced weight gain and body fat, increased lipid oxidation, attenuated hepatost

161 Body fat increases postmenopausal breast cancer risk.

162 regression as the way of finding the optimal body fat index.

163 To compare the accuracy of BMI vs other body fat indices of the form body mass divided by height

164 After 6 mo, total body fat, interleukin IL-6, and hepcidin were significan

165 In postmenopausal women, body fat is a major source of estradiol; therefore, chan

166 ecause the correct regression model (percent body fat is proportional to mass divided by heightn) sug

167 I; kg/m(2)), waist circumference (cm), whole body fat (kg), and obesity (WHO criteria of BMI >/=30 kg

168 and proton-spectroscopic imaging, and lower body fat (LBF) was imaged by dual-energy x-ray absorptio

169 Body fat, leptin, and insulin were increased in male, bu

170 m body mass divided by heightn in estimating body fat levels in adolescents.

171 replacing BMI z scores with TMI to estimate body fat levels in adolescents.

172 The tri-ponderal mass index estimates body fat levels more accurately than BMI in non-Hispanic

173 etric data were used to determine changes in body fat levels, body proportions, and the scaling relat

174 ogical night, than did lean individuals (low body fat) (log-rank P = 0.009).

175 ocaloric KD was not accompanied by increased body fat loss but was associated with relatively small i

176 ines the neuroendocrine axis of serotonergic body fat loss in Caenorhabditis elegans.

177 Body fat loss slowed during the KD and coincided with in

178 ever, causality of CLA-mediated responses to body fat loss, particularly the linkage between inflamma

179 feeding and metabolism, is a major driver of body fat loss.

180 hole-body lean mass (-1.0 +/- 0.2 kg), whole-body fat mass (-6.9 +/- 0.5 kg), appendicular lean mass

181 als were eradicated when normalised to whole-body fat mass (P = 0.416).

182 ent attenuated HFD-induced body weight gain, body fat mass accumulation, increased energy expenditure

183 mice) and effects on liver weight and total body fat mass being essentially independent of mERalpha

184 ring gained more body weight and had greater body fat mass compared to the control, and these differe

185 entified homeostat for body weight regulates body fat mass independently of fat-derived leptin, revea

186 Although whole-body fat mass was not affected, visceral adipose tissue

187 ; p = 0.01) following the confinement, whole body fat mass was only reduced in the Exercise group (-1

188 NX rats show increased food intake, enhanced body fat mass, and elevated plasma levels of triglycerid

189 ion patterns were observed for obesity risk, body fat mass, fat percentage, fat mass index, and waist

190 resented with a reduced total BW and overall body fat mass, smaller adipocytes, and reduced leptin le

191 obese individuals are proportional to whole-body fat mass, suggesting a compensatory down-regulation

192 GPRKO female mice is due to the reduction in body fat mass.

193 bolished when values are normalised to whole-body fat mass.

194 PPARgamma agonist, increases food intake and body/fat mass as side-effects.

195 Total body fat, measured from dual energy x-ray absorptiometry

196 ent for demographic, behavioral, and ectopic body fat measures did not explain racial/ethnic differen

197 commuters had significantly lower percentage body fat (men: -1.32% [95% CI -1.53 to -1.12], p<0.0001;

198 Most importantly, we found that the body fat metabolism was especially pronounced for most v

199 ath acetone levels that indicate intensified body fat metabolism, as validated by parallel venous blo

200 710), diabetic nephropathy (DN; n = 2452), % body fat (n = 555) and insulin secretion (n = 298).

201 BMI z scores and percentages of body fat of children in the DHA group did not differ fro

202 rt for dairy food as a stratagem to decrease body fat or weight gain in overweight adolescent girls.

203 139 men and 82 788 women for the percentage body fat outcome.

204 healthy weight adolescent humans who gained body fat over a 2 or 3 year follow-up period show an inc

205 g study suggests that adolescents who gained body fat over our follow-up period experienced an increa

206 educes the blood LDL concentration and whole-body fat oxidation by modifying the saturation index of

207 LCHF) diet markedly increases rates of whole-body fat oxidation during exercise in race walkers over

208 Furthermore, whole-body fat oxidation was less (P < 0.001) and CHO oxidatio

209 iated with markedly increased rates of whole-body fat oxidation, attaining peak rates of 1.57 +/- 0.3

210 iver glycogen utilization, and reduced whole-body fat oxidation.

211 expenditure, driven by an increase in whole-body fat oxidation.

212 xcessive GWG was associated with 3.0% higher body fat (P < 0.001) and a 5.6-kg higher PPWR (P < 0.001

213 with NAFLD also had a higher amount of total body fat (p < 0.001) and subcutaneous fat (p < 0.001) th

214 cally obese sheep demonstrated greater total body fat (p < 0.001); LA volume (p < 0.001); LA pressure

215 cessive GWG was associated with 0.58% higher body fat (P = 0.55) and 2.06 kg PPWR (P = 0.24).

216 subcutaneous abdominal fat (P=0.47) or lower body fat (P=0.30).

217 Our study suggests that body fat partitioning does not explain interethnic diffe

218 We sought to determine whether differential body fat partitioning or abnormalities in muscle insulin

219 adipose tissue development and could explain body fat patterning variations in healthy and lipodystro

220 investigated any relation between changes in body fat patterns of patients with AN treated in a speci

221 ss (FM), fat-free mass (FFM), and percentage body fat (PBF) by gestational age (GA), with the use of

222 fter adjustment for age, race, percentage of body fat, perceived stress, pain-medication use, sexual

223 ith 1-year age increment after adjusting for body fat percent, fitness, resting heart rate, glucose l

224 mes after adjusting for covariates including body fat percent.

225 ficantly reduce body weight z-score, percent body fat, percent trunk fat, and serum level of interleu

226 [95% CI 0.066, 0.174]; P = 1E-5) and higher body fat percentage (0.301% [0.230, 0.372]; P = 1E-16) c

227 ary fitness (VO2peak; beta=-0.165, P<0.001), body fat percentage (beta=0.032, P<0.02), resting heart

228 d a genome-wide association meta-analysis of body fat percentage (BF%) in up to 100,716 individuals.

229 raction, P < 0.05), prevented an increase in body fat percentage (group x time interaction, P < 0.05)

230 8, 95%CI: 1.31-1.91; Ptrend < 0.0001), total body fat percentage (HR = 1.27, 95%CI: 1.06-1.53; Ptrend

231 [CI, 1.02 to 1.23] for quintile 2) and high body fat percentage (HR, 1.19 [CI, 1.08 to 1.32] for qui

232 [CI, 1.17 to 1.79] for quintile 1) and high body fat percentage (HR, 1.59 [CI, 1.28 to 1.96] for qui

233 er was not associated with BMI (P = 0.80) or body fat percentage (P = 0.38).

234 ch intake on BMI (P-interaction = 0.007) and body fat percentage (P-interaction = 0.03).

235 02), hip circumference (r=0.4, P=0.008), and body fat percentage (r=0.4, P=0.03) in HT.

236 ent molecular mechanisms that lead to higher body fat percentage (with greater subcutaneous storage c

237 were associated with a lower childhood total-body fat percentage and a lower android:gynoid fat mass

238 Body fat percentage and activity intensity appear to mod

239 o-hip ratio) and body fat composition (total body fat percentage and trunk fat percentage) measuremen

240 Low BMI and high body fat percentage are independently associated with in

241 waist circumference, hip circumference, and body fat percentage more than did the C group at both 12

242 Body mass index and DXA-derived body fat percentage were divided into quintiles, with qu

243 )]) and body composition (fat and lean mass, body fat percentage) between predominantly breastfed and

244 ference models), 401 435 (BMI), and 395 640 (body fat percentage).

245 ere associated with a higher childhood total-body fat percentage, android:gynoid fat mass ratio, and

246 ted mortality models containing both BMI and body fat percentage, low BMI (hazard ratio [HR], 1.44 [9

247 outcome of BMI, BMI Z score, BMI percentile, body fat percentage, skinfold thickness, waist circumfer

248 umption, body mass index, physical activity, body fat percentage, waist circumference, triglycerides,

249 ely related to age, with a similar trend for body fat percentage.

250 Humans also had the greatest body fat percentage.

251 ed that dietary pulse consumption may reduce body fat percentage.

252 (2) if of Asian ethnicity), and had a raised body fat percentage.

253 , or excess weight and for women with a high body fat percentage.

254 maller waist circumference and lower BMI and body fat percentage.

255 st circumference, body-mass index (BMI), and body fat percentage.

256 lly significant decreases in body weight and body fat percentage.

257 history method) on body mass index (BMI) and body fat percentage.Results:AMY1 copy number was not ass

258 ndependent of BMI, baseline total percentage body fat, percentage trunk fat, and percentage abdominal

259 ols, adults who were born preterm had higher body fat percentages (after adjustment for sex, age, and

260 for association with maximum BMI and percent body fat (PFAT) in 5,870 and 912 Pima Indians, respectiv

261 Outcomes were body mass index and body-fat phenotypes measured from anthropometry (ages 4,

262 c status, Tanner pubertal status, percentage body fat, physical activity, and total energy intake.

263 Analyses of body fat, plasma hormone levels, and visceral white adip

264 n of PFKFB2 further correlated with higher % body fat (r = -0.16, P = 0.02) and BMI (r = -0.17, P = 0

265 intervention were associated with changes in body fat (r = 0.39, P = 0.01) and LBM (r = -0.34, P = 0.

266 Overall primary outcome was body fat redistribution, and the main specific end point

267 o uncover the ancient and complex origins of body fat regulation.

268 mass and fat-free mass but not percentage of body fat relative to adequate GWG.

269 Adolescents who gained body fat reported significantly greater milkshake wantin

270 0.5] and 2.9% increase (95% CI: 0.3, 5.5) in body fat, respectively.

271 higher (P = 0.03) and 0.03% lower (P < 0.01) body fat, respectively.

272 core (rhoG = 0.45, P = 0.002), percentage of body fat (rhoG = 0.28, P = 0.04), fat mass (rhoG = 0.34,

273 s divided by heightn) suggested that percent body fat scales to height with an exponent closer to 3,

274 Body mass, body fat, sex, anogenital distance (AGD) (a proxy for an

275 ups.Our findings that the dairy group gained body fat similar to the control group provide no support

276 after adjustment for age, sex, percentage of body fat, sun exposure, physical activity, and dietary v

277 absorptiometry scans for estimation of total body fat (TBF) and lean body mass (LBM) (n = 10,525) wer

278 ssociations for smoking and alcohol, but not body fat, tended to be stronger for sessile serrated ade

279 penditure may be more effective for reducing body fat than caloric restriction, which is currently th

280 SGA were shorter, were thinner, and had less body fat than their non-SGA peers, irrespective of postn

281 sus those who showed stability of or loss of body fat, though these effects were partially driven by

282 Subjects lost weight and body fat throughout the study corresponding to an overal

283 en GWG and long-term PPWR and the percentage body fat varied by prepregnancy BMI (P-interaction </= 0

284 Percent body fat varied with both age and height during adolesce

285 tivity index (ISI), percentage of body fat (%body fat), visceral (VAT) and subcutaneous (SAT) adipose

286 reductions in body mass, BMI, percentage of body fat, waist:hip ratio, and leptin in the LGI-diet gr

287 aradigms assessing response to these events; body fat was assessed annually over follow-up.

288 Percentage of body fat was measured annually for 3 y of follow-up for

289 Body fat was not influenced by the diet assignment.Dairy

290 technical Commission body phantom simulating body fat/water composition and in vivo experiments on 2

291 ng body mass index, waist circumference, and body fat were associated with greater left ventricular (

292 etry, and increasing waist circumference and body fat were associated with worse global longitudinal

293 e blood as well as waist circumference and % body fat were lower post intervention in the RS4 group c

294 ight (m)2), waist circumference, and percent body fat were measured at annual or semiannual examinati

295 nflammation, hyperinsulinemia, and increased body fat, which are signatures of diet-induced diabetes

296 mass index, waist circumference, and percent body fat, while 2,5-dichlorophenol was positively associ

297 Adolescents who gained body fat, who largely remained in a healthy weight range

298 mass index, waist circumference, and percent body fat with conventional and advanced measures of card

299 al bone mass but also exhibit an increase in body fat with corresponding reductions in energy expendi

300 Despite similar percent body fat, youth with IGT versus NGT had higher GlyRa, lo