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1 reduced food intake and body weight (mainly body fat mass).
2 ks ingested lipids to energy expenditure and body fat mass.
3 ame HF diet, despite comparable increases of body fat mass.
4 decrease in circulating IGF-I independent of body fat mass.
5 ) when controlled for the1-y change in total body fat mass.
6 weeks AQP7 null mice had 3.7-fold increased body fat mass.
7 bolished when values are normalised to whole-body fat mass.
8 d an MLS >2.0 for percentage of fat mass and body fat mass.
9 sis, atherosclerosis, onset of diabetes, and body fat mass.
10 oncentration is highly correlated with total body fat mass.
11 % more than controls due mainly to increased body fat mass.
12 ntrol rats and had large reductions in their body fat mass.
13 elevated plasma leptin levels independent of body fat mass.
14 GPRKO female mice is due to the reduction in body fat mass.
15 hole-body lean mass (-1.0 +/- 0.2 kg), whole-body fat mass (-6.9 +/- 0.5 kg), appendicular lean mass
16 ent attenuated HFD-induced body weight gain, body fat mass accumulation, increased energy expenditure
17 CCK-KO mice had reduced body weight gain and body fat mass and enlarged adipocytes, despite the same
19 d carbohydrate feeding on energy metabolism, body fat mass and muscle expression of fuel metabolism g
20 In conclusion, leptin levels reflect total body fat mass, and although visceral fat is known to pre
21 (compliant model) indicated that age, whole-body fat mass, and bone resorption were common predictor
22 NX rats show increased food intake, enhanced body fat mass, and elevated plasma levels of triglycerid
23 ots, bioelectrical impedance measurements of body fat mass, and measurements of body weight and girth
24 associations between body mass index (BMI), body fat mass, and percent body fat, measured by dual-en
25 neral content (BMC), lean tissue mass (LTM), body fat mass, and percentage fat are presented as funct
27 bdominal fat areas and negatively with lower-body fat mass as a percentage of total-body fat, after c
30 but the precise amount of calorie intake or body fat mass associated with optimal health and maximum
31 cknesses cannot be used to assess changes in body fat mass because of age-related fat redistribution.
32 mice) and effects on liver weight and total body fat mass being essentially independent of mERalpha
33 ring gained more body weight and had greater body fat mass compared to the control, and these differe
34 ight decreased by 10.2 +/- 5.5% (P < 0.001), body fat mass decreased by 18.7 +/- 11.3% (P < 0.001), a
35 e VF+ compared with the VF- group, but whole-body fat mass (determined using 3H2O) was not significan
36 ) [CI, -0.41 to 1.02 kg/m(2)]; P = 0.39), or body fat mass (difference, 0.39 kg [CI, -1.04 to 1.92 kg
39 different (P > 0.05) between machines, whole-body fat mass [DXAH-DXAL (DXAdiff) = 1152 +/- 1395 g], p
40 FHC and HF groups had increased body weight, body fat mass, fasting glucose, and were insulin-resista
41 ion patterns were observed for obesity risk, body fat mass, fat percentage, fat mass index, and waist
42 y expenditure (AEE), energy intake (EI), and body fat mass (FM) were measured longitudinally in 10 wo
45 we reported that S6K1(-/-) mice have reduced body fat mass, have elevated rates of lipolysis, have se
46 se dependently reduced body weight and whole-body fat mass in DIO mice due to marked increases in tot
47 entified homeostat for body weight regulates body fat mass independently of fat-derived leptin, revea
48 playing a critical role in the regulation of body fat mass, lipid metabolism, and insulin resistance.
49 icant differences in estimated percentage of body fat mass loss between treatment groups, and the fra
55 and increased production from expanded total body fat mass, rather than alterations in leptin clearan
56 resented with a reduced total BW and overall body fat mass, smaller adipocytes, and reduced leptin le
57 obese individuals are proportional to whole-body fat mass, suggesting a compensatory down-regulation
58 rum leptin levels are lowered by the loss of body fat mass that would accompany starvation and malnut
59 uglycemic clamp was increased, whereas total body fat mass, VAT, SSAT, and hemoglobin A1c were reduce
60 glucose tolerance test and minimal modeling, body fat mass was assessed by dual-energy X-ray absorpti
63 ; p = 0.01) following the confinement, whole body fat mass was only reduced in the Exercise group (-1
64 ntrast, at age 4 weeks, adipocyte volume and body fat mass were comparable in wild type and AQP7 null
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