ライフサイエンスコーパス: body temperature

1 liquid at room temperature to a hydrogel at body temperature.

2 ncluding heart rate, arterial pressures, and body temperature.

3 rotected mice from cold-induced decreases in body temperature.

4 temic anaphylaxis was measured by changes in body temperature.

5 logy, qPCR, HPLC, LC/MS and measures of core body temperature.

6 rience substantial and rapid fluctuations in body temperature.

7 rothermic and displayed lower 24-h minima of body temperature.

8 or mesalamine becomes more viscoelastic near body temperature.

9 g hidden Markov models of their movement and body temperature.

10 rgy conservation with reduced metabolism and body temperature.

11 omic functions, including blood pressure and body temperature.

12 adiotelemetry probes for measurement of core body temperature.

13 wering the self-healing temperature close to body temperature.

14 y to the power -0.19, an effect modulated by body temperature.

15 dhood, associated with a significant rise in body temperature.

16 ure but turns into a viscous gel on reaching body temperature.

17 the central circadian clock to help regulate body temperature.

18 had similar resting metabolic rate and core body temperature.

19 ta loggers to obtain direct measures of core body temperature.

20 to use preference behaviour to regulate its body temperature.

21 V1 ion channel, leading to its activation at body temperature.

22 of their simple use as drug carrier at human body temperature.

23 chinery to cause powerful heat activation at body temperature.

24 oth relative cool and stimuli exceeding host body temperature.

25 eduction in Ucp1-deficient mice reduces core body temperature.

26 PS, but also the associated falls in VO2 and body temperature.

27 e responses while increasing thirst and core-body temperature.

28 LCAD(-/-); VLCAD(+/+) mice compared with WT body temperature.

29 hibited an improved capacity to maintain the body temperature.

30 ir cells and calyces at room temperature and body temperature.

31 altering respiratory exchange ratio or core body temperature.

32 in the canonical triple-helical structure at body temperature.

33 tory proteins were more strongly affected by body temperature.

34 ls gel rapidly upon mixing and/or heating to body temperature.

35 he environment to a pathogenic yeast form at body temperature.

36 ic Et2 knockout mice displayed a normal core body temperature.

37 s the G-quadruplex to be metastable at human body temperature.

38 for the continuous recording of activity and body temperature.

39 controlling for confounding factors such as body temperature.

40 activating thermogenesis to maintain normal body temperature.

41 ral patterning of both underwater and aerial body temperature.

42 resent a target at which histamine modulates body temperature.

43 energy efficiency is due largely to a warmer body temperature.

44 nto mice to form a collagen-fibril matrix at body temperature.

45 l signature in the range equivalent to human body temperature.

46 hypothalamic nuclei involved in controlling body temperature.

47 ient rash and a short-lived elevation of the body temperature.

48 such as basal metabolic rate, body size, or body temperature.

49 lesion could produce two opposite effects on body temperature.

50 mice to regulate brown fat and maintain core body temperature.

51 wer from physiological fluids and operate at body temperature.

52 wild-type, and this defect is amplified near body-temperature.

53 d environmental conditions into estimates of body temperatures.

54 usceptible persons may develop lethally high body temperatures.

55 Warming to normal body temperature (37 degrees C) does not restore normal

56 fully recovered its original shape at human body temperature (37 degrees C), which indicated the gre

57 n pathogen is exquisitely responsive to host body temperature (37 degrees C), which induces a major m

58 nasal cavity (33-35 degrees C) than at core body temperature (37 degrees C).

59 a tunable release rate of the drugs at human body temperature (37 degrees C).

60 11.3 mM [95% CI, 9.0-14.1], p = 0.004), core body temperature (39.3 degrees C [95% CI, 39.0-39.5] vs

61 e QIV was associated with reduced risks of a body temperature above 39 degrees C and lower respirator

62 lic production and retention of heat to warm body temperature above ambient) enhances physiological f

63 to modern birds, but was able to elevate its body temperature above environmental temperatures.

64 cal exercise, menstruation, NSAIDs, alcohol, body temperature, acute infections, and antacids.

65 impaired ability to repel water and regulate body temperature after water immersion.

66 te functions like the perception of pain and body temperature and also have anti-inflammatory propert

67 hot temperatures is critical to maintaining body temperature and avoiding injury in diverse animals

68 ev-erbalpha also abolishes normal rhythms of body temperature and BAT activity.

69 as observed briefly, but only after the core body temperature and blood pressure began to decrease as

70 gy expenditure and food intake, which lowers body temperature and body weight.

71 was accompanied by an inability to maintain body temperature and by hyperglycaemia.

72 ation in 3-month-old LP offspring normalizes body temperature and causes postprandial hyperglycemia.

73 ssessed within 24 hours from reaching normal body temperature and compared with outcomes at 6 months:

74 tion rates due to a 6 degrees C reduction in body temperature and decreased protease activity; and (i

75 penic 3-month-old LP progeny shows increased body temperature and energy dissipation in association w

76 of iWAT even at room temperature, increasing body temperature and energy expenditure and preventing d

77 ABSTRACT: We sought to determine if body temperature and energy expenditure are influenced b

78 oes not result in arrhythmicity because core body temperature and exploratory activity rhythms persis

79 37 degrees C or 0.5 degrees C below the core body temperature and followed up for 1 year.

80 xperiments in rabbit ventricular myocytes at body temperature and found that 1) at a low pacing frequ

81 r LPS were both associated with reduced core body temperature and heat release.

82 Body temperature and immunity may be important but requi

83 er oral challenge, as evidenced by decreased body temperature and increased serum mouse mast cell pro

84 In experiment 2, METH-induced decreases in body temperature and locomotor activity at 23 degrees C

85 WS), as well as the circadian rhythmicity of body temperature and locomotor activity.

86 bipolar disorder show altered rhythmicity in body temperature and melatonin rhythms, high day-to-day

87 of care, but the inverse association between body temperature and mortality was robust and remained c

88 ed in the groups of WIN55, 212-2 with normal body temperature and placebo control.

89 essment of symptom scores and measurement of body temperature and plasma histamine levels.

90 GF-beta-neutralizing antibody improved their body temperature and prevented the increased adiposity p

91 or necrosis factor-alpha, sickness symptoms, body temperature and self-reported fatigue, and depressi

92 cellent outcome is possible in children with body temperature and serum potassium reaching the far li

93 mia, and have a markedly accelerated loss of body temperature and survival after cold exposure compar

94 Body temperature and survival were monitored closely, an

95 fluorodeoxyglucose PET to maintain a normal body temperature and to avoid fluorodeoxyglucose uptake

96 include survival, time-to-death, changes in body temperature and weight, and nasal wash samples cont

97 evices enabled real-time acquisition of core body temperatures and changes in heart rates and electro

98 vores both directly (e.g. through changes in body temperature) and indirectly (e.g. through changes i

99 2-2 hypothermia, 2) WIN55, 212-2 with normal body temperature, and 3) placebo control.

100 ated by lower symptom scores, less change in body temperature, and a lower increase of plasma histami

101 is regulated by the daily subtle rhythms in body temperature, and a new study by Gotic and colleague

102 ite C. neoformans being able to grow at bird body temperature, and are able to escape from bird macro

103 RO5263397 on sleep/wake, locomotor activity, body temperature, and cataplexy were assessed in two mou

104 likely being a physiologic response to lower body temperature, and has recently been associated with

105 O mice displayed reduced locomotor activity, body temperature, and hepatic triglyceride content in a

106 drive rhythms in activity and rest, feeding, body temperature, and hormones.

107 y rate, heart rate, systolic blood pressure, body temperature, and level of consciousness as previous

108 EEG, EMG, body temperature, and locomotor activity data were colle

109 d significantly reduced symptoms, changes in body temperature, and mast cell protease (MMCP-1) releas

110 ram, electromyogram, locomotor activity, and body temperature, and the efficacy of the TAAR1 agonist,

111 Changes in body mass, urine osmolality, body temperature, and thirst were monitored.

112 Drug dosing, body temperatures, and demographics were collected durin

113 T-ablated mice were able to maintain optimal body temperature ( approximately 35-37 degrees C) during

114 environmental temperature and regulation of body temperature are integral determinants of behaviour

115 KO(Nkx2.1) mice, although small increases in body temperature are present.

116 o power implantable devices, as they work at body temperature, are light and easy-to-miniaturise.

117 erms, which often experience fluctuations in body temperature as a result of their environment.

118 found that the Ppt1-KO mice had lower basal body temperature as they aged and developed hypothermia

119 ns, Trpa1(-/-) mice had the same dynamics of body temperature as Trpa1(+/+) mice and showed no weakne

120 rent OM, previous tympanocentesis, and lower body temperature, as well as during the winter, suggesti

121 determined home cage locomotor activity and body temperature at 23 degrees C ambient temperature.

122 nts a pathogen strictly adapted to the human body temperature, B. bronchiseptica causes infection in

123 lammatory response as assessed by changes in body temperature, basophil activation, and basophil depl

124 marked improvement in survival, body weight, body temperature, behavior, neuropathology, and disease

125 ity of anaphylaxis as measured by decline of body temperature, behavioral effects, serum IL-4, IgE, a

126 More than half of patients had a body temperature below 38.3 degrees C.

127 TRalpha and TRbeta, most notably heart rate, body temperature, blood glucose, and triglyceride concen

128 mine the effects of IV acetaminophen on core body temperature, blood pressure, and heart rate in febr

129 hypothalamic signaling pathways that control body temperature (BT), blood pressure (BP), and energy b

130 The preoptic area (POA) regulates body temperature, but is not considered a site for body

131 diac rhythm at baseline, but increasing core body temperature by as little as 3 degrees C causes cove

132 The preoptic area (POA) controls body temperature by modulating BAT activity, but its rol

133 Upon contact with serum and at body temperature, C. albicans performs a regulated switc

134 Body temperature can be reliably measured in the oesopha

135 Changes in body temperature can profoundly affect survival.

136 one to precipitation in poikilotherms (their body temperature can vary considerably) needs a companio

137 It is well documented that elevated body temperature causes tumors to regress upon radiother

138 Humans' core body temperature (CBT) is strictly controlled within a n

139 Core body temperature (CBT), salivary melatonin, subjective a

140 ce thermogenesis (primary outcome) and alter body temperature, cold and hunger sensations, plasma met

141 of both UCP1 and UCP3 accelerate the loss of body temperature compared to UCP1KO alone during acute c

142 ignificantly decreased allergic symptoms and body temperature decrease relative to placebo after chal

143 y temperature) increased, and 24-h minima of body temperature decreased, as the 24-h minimum ambient

144 y into two reciprocal patterns with a strong body temperature dependence.

145 pecies and characterized by a marked fall in body temperature due to an inhibition of brown adipose t

146 mplant recipients demonstrated elevated core body temperature during cold challenges, enhanced respir

147 Surprisingly, this inability to maintain body temperature during cold exposure in Ppt1-KO mice wa

148 exercise capacity, failure to maintain core body temperature during cold stress, and reduced ability

149 However, HFpEF mice failed to regulate body temperature during cold temperature exposure.

150 or reducing potentially lethal elevations in body temperature during excessive fever.

151 thus supports nervous tissue function at low body temperature during hibernation.

152 ures of organisms that cannot regulate their body temperature (ectotherms) depends on their ability t

153 e was closely associated with an increase in body temperature, energy expenditure, and whole-body fat

154 nts who dialyzed at 0.5 degrees C below core body temperature exhibited complete protection against w

155 llular growth combined with survival at bird body temperature explains the ability of birds to effici

156 Body weight, body temperature, food uptake, intestinal inflammation,

157 Body temperatures for these two species indicate that va

158 Endotoxemia increased body temperature from 36.9 +/- 0.4 degrees C to 38.6 +/-

159 o control group and WIN55, 212-2 with normal body temperature group.

160 ) with criteria for suspected enteric fever (body temperature >/=38.0 degrees C for >/=4 days without

161 /min, 2) respiratory rate>20 breaths/min, 3) body temperature>38 or <36 degrees C, or 4) leukocyte co

162 ndomly assigned 700 ICU patients with fever (body temperature, >/=38 degrees C) and known or suspecte

163 ion of nosocomial infections, and changes in body temperature, heart rate, and minute ventilation.

164 ed for monitoring human vital signs, such as body temperature, heart rate, respiration rate, blood pr

165 In mammals, rhythms in body temperature help to entrain and synchronize circadi

166 otherms and homeotherms live longer at lower body temperatures, highlighting a general role of temper

167 mer gel (NiPAAm-co-AAm) that contracts above body temperature (i.e. at 45 degrees C) to release compo

168 To maintain core body temperature in cold conditions, mammals activate a

169 ecreased circadian amplitude of activity and body temperature in mice, similar to findings in MDD pat

170 red that TRPV1 receptors also regulate basal body temperature in multiple species from mice to humans

171 mpounds disclosed herein do not elevate core body temperature in preclinical models and only partiall

172 on, eccrine sweating is a vital regulator of body temperature in response to thermal stress in humans

173 Increased body temperature in survivors was also associated with s

174 and F-IR/IGFRKO mice were unable to maintain body temperature in the cold and developed severe diabet

175 of critically ill septic patients, increased body temperature in the emergency department was strongl

176 especially mammals, which maintain constant body temperature in the face of environmental temperatur

177 for the WIN55, 212-2 hypothermia group, the body temperature in the other two groups was maintained

178 Both compounds reduced body temperature in the two narcolepsy models at the hig

179 nd Trpv1(-/-) mice, but only restored normal body temperature in Trpa1(-/-) and Trpa1(-/-)/Trpv1(-/-)

180 d hypothermia and APAP was without effect on body temperature in Trpa1(-/-) mice.

181 n addition, we found a 1.7 degrees C drop in body temperature in unfed LCAD(-/-); VLCAD(+/+) mice com

182 f APAP elicited a dose dependent decrease in body temperature in wildtype mice.

183 vity, including the prototypical decrease in body temperature in WPE-sensitized mice challenged with

184 n which radiotelemetry devices that recorded body temperatures in 24-hour increments were implanted.

185 We show that extreme operative body temperatures in exposed habitats match or exceed th

186 y is associated with lower environmental and body temperatures in lizards and amphisbaenians, but not

187 behavioral adjustments to maintain preferred body temperatures in the face of environmental variation

188 als maintain their high, relatively constant body temperatures in the face of wide variation in envir

189 ian dinosaurs and that not all dinosaurs had body temperatures in the range of that seen in modern bi

190 Body temperature increased 2 degrees C, and gut permeabi

191 E was 65.7 (13.5) minutes; and the mean core body temperature increased by 0.46 degrees C (0.20 degre

192 terothermy (as measured by 24-h amplitude of body temperature) increased, and 24-h minima of body tem

193 rrier, their inefficient growth at mammalian body temperatures indicates that the reservoir hosts of

194 TRPV1 KO mice have a normal basal body temperature, indicative of developmental compensati

195 Circadian oscillation of body temperature is a basic, evolutionarily conserved fe

196 Body temperature is commonly used to screen patients for

197 Maintenance of body temperature is essential for the survival of homeot

198 challenge to ectothermic organisms in which body temperature is largely dictated by the ambient temp

199 d to occur during the biological night, when body temperature is low and melatonin is synthesized.

200 Core body temperature is normally tightly regulated to within

201 risingly, despite the hypermetabolism, their body temperature is not elevated.

202 The S-I model shows how body temperature is regulated by balancing the rates of

203 tress, and some changes (PS bouts, SWS time, body temperature, locomotor activity) persisted after th

204 veloped in 64 patients (14.4%) (defined as a body temperature<36.0 degrees C) within 24 hours of seps

205 In ectotherms, behaviors that elevate body temperature may decrease parasite performance or in

206 This sophisticated regulatory mechanism of body temperature may participate in the control of energ

207 Antipyretic therapy decreased body temperature (mean difference, -0.38 degrees C; 95%

208 , 3 h before (-3) or 3 h after (+3) the core body temperature minimum (CBTmin) measured on the baseli

209 se fall animals use torpor sporadically with body temperatures mirroring ambient temperatures between

210 bserved in torpid hibernators are defined by body temperature, not torpor per se.

211 phylaxis, the symptoms and decreases in core body temperature observed in wild-type mice were reduced

212 Four HCWs reached or exceeded a mean core body temperature of >/= 38.5 degrees C.

213 ts randomized to hypothermia achieved a core body temperature of 34.7 degrees C before reperfusion, w

214 lled at ~38-40 degrees C (slightly above the body temperature of 37 degrees C) for future in vivo USF

215 As humans have a body temperature of 37 degrees C, the bumpy form of the

216 orted severe fever in the previous 24 h or a body temperature of 37.8 degrees C or more at presentati

217 s can be very different from the equilibrium body temperature of an individual ectotherm.

218 t gradual, moderate declines in activity and body temperature of both shore and ice bears in summer,

219 the species barrier would be hindered by the body temperature of mammalian species.

220 ss to the core neural circuit regulating the body temperature of mammals.

221 The body temperature of small ectotherms, such as Drosophila

222 alysis of eggshells can be used to determine body temperatures of females during periods of ovulation

223 f the temperature of the environment and the body temperatures of the animals.

224 ody temperatures (theoretically equilibrated body temperatures) of small ectothermic animals during t

225 trointestinal motility, hormone release, and body temperature on a second-to-second basis.

226 d more symptoms (P = 0.003) and had a higher body temperature on presentation (P < 0.001) than patien

227 Plasma hyperosmolality delayed the mean body temperature onset of sweating (+1.24 +/- 0.18 vs. +

228 and pneumatic pressures and maintaining core body temperature, optimal patient outcomes can be achiev

229 lso exhibited no significant changes in core body temperature or cardiovascular rhythm, whereas sham-

230 in the short term and maintenance of optimal body temperatures over the long term, but its molecular

231 e and saliva formation, as well as decreased body temperature, parameters that are controlled by the

232 Body temperature peaked on day 6, pox lesions appeared o

233 To check the influence of subject-specific body temperature, pH, H2(18)O, and cellular produced CO2

234 n contrast to mammals that maintain constant body temperature, plant temperature oscillates on a dail

235 Anaphylactic symptom scores, core body temperatures, plasma histamine levels, basophil num

236 cantly increased blood pressure, heart rate, body temperature, pupil size, plasma cortisol, prolactin

237 my wherein metabolic rates are reduced, core body temperatures reach ambient levels, and key physiolo

238 bsequent Blo t 5 challenge induced decreased body temperature, reduction in movement, and a fall in s

239 c anaphylaxis, antigen-dependent decrease in body temperature, reflecting the anaphylactic reaction,

240 tor 1 (TRPV1), besides being involved in the body temperature regulation and in the response to pain,

241 r growth, wound healing, water repulsion and body temperature regulation.

242 Anaphylaxis was assessed by monitoring body temperature, release of mast cell mediators and/or

243 static control of breathing, heart rate, and body temperature relies on circuits within the brainstem

244 ogical processes and behaviors as diverse as body temperature, respiration, aggression, and mood.

245 mechanism responsible for coordinating daily body temperature rhythm and adaptability to environmenta

246 nt required for establishing and maintaining body temperature rhythm in a manner that is adaptable to

247 in relaying signals from the SCN controlling body temperature rhythms, while the ventral subdivision

248 Nontargeted screening measures, including body temperature screening among employees and visitors

249 racted to thermal stimuli approximating host body temperatures, seeking relative warmth while avoidin

250 e allergic skin response (ASR), anaphylaxis, body temperature, serum immunoglobulins, and mouse mast

251 racterized in pharmacokinetic, efficacy, and body temperature studies.

252 ts of BIBDAV are likely species with a lower body temperature, such as snakes.

253 Measurements of circadian rhythms in body temperature suggest a biological mechanism through

254 To maintain body temperature, sweat glands develop from embryonic ec

255 We measured core body temperature (T(b)) daily rhythms of Cape ground squ

256 es (TPCs), which quantify how an ectotherm's body temperature (Tb ) affects its performance or fitnes

257 Rectal, mean skin and mean body temperature (Tb ), electromyographic activity (EMG)

258 In mammals, daily changes in body temperature (Tb) depend on the integrity of the sup

259 to investigate the potential causes of high body temperature (Tb) during lactation in mice as a puta

260 Mammals typically keep their body temperature (Tb) within a narrow limit with changin

261 EEG, EMG, body temperature (Tb), and locomotor activity (LMA) were

262 e scarce, endothermic animals can lower core body temperature (Tb).

263 Defense of core body temperature (Tc) can be energetically costly; thus,

264 f plasma uridine is required for the drop in body temperature that occurs during fasting.

265 thermia, as well as a late-phase decrease in body temperature that was abrogated by antibody-mediated

266 ore homoeothermic) and higher 24-h minima of body temperature (that is, became less hypothermic), tha

267 s respond to acute light by increasing their body temperature, the effect of acute light on temperatu

268 thermal limits both to air and to operative body temperatures (theoretically equilibrated body tempe

269 lly triggered inverse phase transition below body temperature, thereby forming an injectable depot.

270 hase transition between room temperature and body temperature, thereby forming an injectable depot.

271 t calorie restriction, IGF-1R signaling, and body temperature, three of the main regulators of metabo

272 als possess a unique ability to reduce their body temperature to ambient levels, which can be as low

273 s and ectotherms (e.g., fish) increase their body temperature to limit pathogen infection.

274 es C) resulted in decreases of skin and deep body temperatures to approximately 8 degrees C and 13 de

275 nisms, understanding the mechanisms by which body temperatures translate into major biogeographic pat

276 Mutant mice had normal body temperature, Ucp1 expression in brown adipose tissu

277 MPO deficiency caused high body temperature via upregulation of uncoupling protein-

278 Core body temperature was 1 masculineC higher after high fat

279 A higher initial body temperature was associated with reduced odds of a g

280 Among vital signs, body temperature was best at predicting mortality.

281 Core body temperature was controlled using an intravascular d

282 tivity (SWA) was significantly decreased and body temperature was increased with a delay of 5-6 h.

283 Body temperature was measured and classified according t

284 Body temperature was measured by means of telemetry, Il3

285 Body temperature was monitored, and serum mouse mast cel

286 Core body temperature was not significantly altered in the cu

287 The importance of body temperature was tested in animals from the fall; th

288 NDy neurons participate in the regulation of body temperature, we evaluated the thermoregulatory effe

289 AR agonists also influence the regulation of body temperature, we explored their potential as antipyr

290 /kg, i.v.) on food-maintained responding and body temperature were determined in these subjects prior

291 ylation in response to a 2 degrees C rise in body temperature were determined with the use of Caco-2

292 Odds ratios for mortality according to body temperature were estimated using multivariable logi

293 Changes in the body temperature were not correlated with changes in mea

294 Diurnal patterns of activity and core body temperature were progressively disrupted in R6/2 mi

295 or mechanical ventilation, or changes in the body temperature) were independently associated with ace

296 vital signs (heart rate, pulse oximetry, and body temperature) were monitored at regular intervals.

297 Mammals exhibit a higher body temperature when exposed to acute light compared to

298 also manifested a defective control of their body temperature, which was overly reduced.

299 gulation of appetite, reproductive function, body temperature, white fat mass, hepatic glucose output

300 uld not be used when accurate measurement of body temperature will influence clinical decisions.