ライフサイエンスコーパス: body wall

1 ing are likely to be located in the animal's body wall.

2 re appendicular derivations from the lateral body wall.

3 hin the developing nervous system and in the body wall.

4 ion of the dorsal part of the thorax and the body wall.

5 that connects the primitive gut tube to the body wall.

6 leading to bulging of the limb buds from the body wall.

7 vertebral fusion and closure of the ventral body wall.

8 iginating from the epithelium of the primary body wall.

9 thways by distorting the conformation of the body wall.

10 ic morphology with extensive coverage of the body wall.

11 on, for example, those that form the ventral body wall.

12 of appropriate attachment of the PHMP to the body wall.

13 e soft tissue sarcomas of the extremities or body wall.

14 roper development and closure of the ventral body wall.

15 g takes place continuously from the parental body wall.

16 spiratory trees, hemal system, tentacles and body wall.

17 ical ring, and the mesoderm of the secondary body wall.

18 t before it is stabbed through the partner's body wall.

19 ovide complete and independent tiling of the body wall.

20 he presumptive limb territory in the lateral body wall.

21 phic detachment of skeletal muscles from the body wall.

22 etains a vestigial attachment to the ventral body wall.

23 ed syncytium that connects the uterus to the body wall.

24 several region-specific aspects of the adult body wall.

25 bit notum identity to properly subdivide the body wall.

26 elayed separation of foregut from the dorsal body wall.

27 forms ventrolateral dermis of the interlimb body wall.

28 IV dendritic arborization neurons, tile the body wall.

29 uding the heart, neural tube, eyes, face and body wall.

30 derivative peptides) cause softening of the body wall.

31 e legs developed as direct outgrowths of the body wall, a condition retained in most insect species.

32 the herniation of organs through the ventral body wall, a decrease in the expression of Noggin, MyoD,

33 cripts enriched (two fold or greater) in the body wall and 56 transcripts enriched in the wing/hinge

34 ritic (md) sensory neurons tiling the larval body wall and a small number of bipolar neurons in the u

35 h dendritic arborization (da) neurons in the body wall and can detect vibration with chordotonal orga

36 esses across a characteristic portion of the body wall and has an axon that projects into the central

37 eurons has a soma associated with the dorsal body wall and hence is a putative stretch receptor in do

38 We examined MRP in embryonic body wall and intestinal muscles as well as in DFMs with

39 Os), which serve as stretch receptors in the body wall and joints and as auditory organs in the anten

40 lecules needed for touch sensation along the body wall and other regions of force sensitivity.

41 trale, an immense fascial pocket between the body wall and overlying blubber layer that reaches as fa

42 Abnormalities in body wall and sex muscles led to uncoordinated movement

43 merge from the neural tube, migrate into the body wall and siphon primordia, and subsequently differe

44 ns resulted in disappearance of MCs from the body wall and the accumulation of myofibroblasts in the

45 s rise to two distinct adult structures: the body wall and the wing.

46 ally be interpreted as a single touch to the body wall and used multiple suction electrodes to record

47 mpz-1 is expressed in about 60 neurons and body wall and vulval muscles.

48 les are more invasive, puncturing the female body-wall and inseminating directly into her body-cavity

49 : one directing UNC-89-A and -B primarily in body-wall and pharyngeal muscle, one internal promoter d

50 e a soluble LacZ reporter protein limited to body-wall and vulval muscles.

51 patterns, such as pannier, twist and Bar-H1 (body wall) and knot, nubbin and Distal-less (wing/hinge)

52 estricted motor columns that innervate limb, body wall, and neuronal targets, MMC neurons are generat

53 rvated completely overlapping regions of the body wall, and this finding suggests a lack of like-repe

54 (MASs), which connect somatic muscles to the body wall; and scolopale cells, which form an integral c

55 Human birth defects involving the ventral body wall are common, yet little is known about the mech

56 lpha functions in the closure of the ventral body wall, as well as offer insight into related human b

57 ect on the contractility of tube feet or the body wall-associated apical muscle, contrasting with the

58 digestive system (e.g., cardiac stomach) and body wall-associated muscles (e.g., apical muscle) and a

59 Nidogen is localized to body wall basement membranes and is required to direct l

60 cell body of ddaE repositions itself on the body wall between 25 and 40 hr after puparium formation

61 downregulated had defects in the dorsalmost body wall, but did not appear to have been globally repa

62 class II neurons do not completely tile the body wall, but they nevertheless occupy nonoverlapping t

63 overlapping dendritic coverage of the larval body wall by Drosophila class IV dendrite arborization (

64 velops in two stages: initially touching the body wall causes circumferential indentation (CI), an em

65 Several tissues involved in normal ventral body wall closure are defective in the absence of AP-2al

66 otor-impaired NMII-B disrupts normal ventral body wall closure because of a dominant-negative effect.

67 since it has one of the most severe ventral body wall closure defects, thoracoabdominoschisis.

68 , yet little is known about the mechanism of body wall closure in mammals.

69 crest cell (NCC)-derived tissues, incomplete body wall closure, and abnormal skeletal patterning.

70 zation, linear heart tube formation, ventral body wall closure, and encasement of the fetus in extrae

71 caused perinatal lethality, edema, defective body wall closure, and skeletal abnormalities.

72 n external genital defects and disruption of body wall closure, as seen in the epispadias-extrophy co

73 During body wall closure, the left and right external genital p

74 complex morphological events responsible for body wall closure, we have studied this developmental pr

75 which these peptides affect the stiffness of body wall connective tissue are unknown.

76 n, causing a synergistic reduction of larval body wall contraction rate that is normally regulated th

77 After rapidly growing to establish body wall coverage, dendrites of Drosophila class IV den

78 on domain affected normal development of the body wall cuticle and appendages.

79 sly described Hoxb2(PolII) alleles that have body wall defects have been shown to disrupt the express

80 on of Vn/Egfr target genes and explains both body wall development and wing outgrowth.

81 ermic genitalia to penetrate their partner's body wall during copulation, frequently bypassing the fe

82 neurons establish dendritic coverage of the body wall early in Drosophila larval development and the

83 intercalation processes occur during ventral body wall elongation and closure.

84 encoded domains between the native embryonic body wall (EMB) and indirect flight muscle isoforms (IFI

85 roportion to their substrate, the underlying body wall epithelium, as the larva more than triples in

86 grow synchronously with their substrate, the body wall epithelium, providing a system to study how pr

87 he allantois, placental vasculature, ventral body wall, eye and heart.

88 f embryonic tissues such as the neural tube, body wall, face and eye lead to severe birth defects.

89 utant embryos fail to close the neural tube, body wall, face, and optic fissure, and they also displa

90 The ventral body wall fails to close in embryos homozygous for the n

91 Otx2 are expressed primarily in the anterior body wall, foregut and developing nervous system.

92 stage 10-11 embryos, Hro-hh is expressed in body wall, foregut, anterior and posterior midgut, repro

93 Here, we show that the process of ventral body wall formation in Xenopus laevis is similar to hypa

94 ent with a model in which defects in ventral body wall formation require the simultaneous loss of at

95 after birth, with a severe defect in ventral body wall formation.

96 o control animals at E11.5, before secondary body wall formation.

97 2 and Hoxb4 in cell types that contribute to body wall formation.

98 The ventral body wall forms in the tadpole, while limb (appendicular

99 t cell types, as these cells are part of the body wall fragment.

100 rization (da) neurons, which tile the larval body wall, grow their dendrites mainly in a 2D space on

101 sis results in human birth defects involving body wall, gut, and heart malformations and in mouse mis

102 ms have circumferential reinforcement of the body wall; however, no experimental characterization of

103 contribute to closure of the neural tube and body wall; however, potential molecular regulators of th

104 rocess and are required for specification of body wall identities in the thorax; however, contrasting

105 ated in the most ventral part of the primary body wall in Hoxb4(PolII) mutants.

106 bryos showed thinning of the primary ventral body wall in mutants relative to control animals at E11.

107 through toughening pre-existing parts of the body wall; in contrast, the subcoxal theory suggests ple

108 s directing the morphogenesis of the ventral body wall, including cell migration, differentiation, an

109 The projections to the foot and body wall indicate that serotonin may also participate i

110 ep back muscles) and hypaxial muscles of the body wall (intercostal muscles, abdominal wall musculatu

111 thin two morphological classes partition the body wall into distinct, non-overlapping territorial dom

112 , PGCs migrate directionally from the dorsal body wall into the genital ridges.

113 suggest that widespread tiling of the larval body wall involves interactions between growing dendriti

114 hat a persistent somatopleure in the lateral body wall is a gnathostome synapomorphy, and the redistr

115 gest that separation of the foregut from the body wall is genetically controlled and that defects in

116 tudies showed that sensory feedback from the body wall is important and sometimes critical for genera

117 As a result, the body wall is independently tiled by the beta and gamma d

118 hanism against loss of coelomic fluid if the body wall is punctured, and it may also function in the

119 Caenorhabditis elegans, gentle touch to the body wall is sensed by six mechanosensory neurons that e

120 ristalsis; and (e) stretch of the gut and/or body wall is sufficient to produce peristalsis in the ab

121 sequently, the mesodermal compartment of the body wall is underdeveloped.

122 whereas exon 9b encodes one of the embryonic body wall isoform (EMB) relay domains.

123 the ventral ectoderm that protrudes from the body wall later covers only the paw.

124 gans of crawling caterpillars slide past the body walls like pistons in a new kind of legged locomoti

125 grating germ cells express CXCR4, whilst the body wall mesenchyme and genital ridges express the liga

126 During reverse crawls, abdominal body wall movements are powered by phase-shifted contrac

127 During forward crawls, abdominal body wall movements are powered by simultaneous contract

128 recognizing a key class of marker cells, the body wall muscle (BWM) cells, on a dataset of 175 C.eleg

129 redundantly with CeMyoD (HLH-1) in striated body wall muscle (BWM) fate specification in the C. eleg

130 intestine-specific SID-5 expression restored body wall muscle (bwm) target gene silencing in response

131 , normally containing exon 7d) and embryonic body wall muscle (EMB, normally containing exon 7a) isof

132 The results support the idea that body wall muscle activation contributes significantly to

133 ee novel UNC-97 interactors are expressed in body wall muscle and by antibodies localize to M-lines.

134 mple of this coupling is the interactions of body wall muscle and hypodermal cells in Caenorhabditis

135 a subset of neurons as well as in C. elegans body wall muscle and in male-specific diagonal muscles.

136 Descendants of MS contribute to body wall muscle and to the posterior half of the pharyn

137 We used body wall muscle as a test tissue because in conditions

138 f mammalian perlecan, that are important for body wall muscle assembly and attachment to basement mem

139 topaxin homolog in C. elegans, is located in body wall muscle attachments that are in vivo homologs o

140 The body wall muscle cells are a useful model for the study

141 ons suggest that gap junctions in C. elegans body wall muscle cells are responsible for synchronizing

142 fluorescent protein (GFP)-fusion proteins in body wall muscle cells causes discrete cytoplasmic aggre

143 The 81 embryonically born body wall muscle cells in C. elegans are comparable to t

144 jor components of the outward K+ currents in body wall muscle cells in culture.

145 in (GFP) secreted into the pseudocoelom from body wall muscle cells is endocytosed and degraded by co

146 roteins (Q29, Q33, Q35, Q40, and Q44) in the body wall muscle cells of Caenorhabditis elegans and sho

147 and large actin aggregates are formed in the body wall muscle cells, resulting in defects in their mo

148 quires synchronous activities of neighboring body wall muscle cells.

149 (APs) and Ca2+ transients among neighboring body wall muscle cells.

150 d function, fertility and rhythmic posterior body wall muscle contraction (pBoc) required for defecat

151 One class of DMP mutants, called anterior body wall muscle contraction and expulsion defective (ae

152 ptic activation of SER-2 facilitates ventral body wall muscle contraction, contributing to the tight

153 provide mechanical stability for supporting body wall muscle contraction.

154 r circuit capable of detecting and modifying body wall muscle contraction.

155 However, genetic studies have shown that body wall muscle development occurs in the absence of HL

156 lines expressing GFP-Htt fusion proteins in body wall muscle displayed a polyQ repeat length-depende

157 roles, allocating mesodermal cells into the body wall muscle fate and patterning a subset of these m

158 High Twist levels direct cells to the body wall muscle fate, whereas low levels are permissive

159 Mutant embryos have an abnormal body wall muscle fiber pattern arising from defects in m

160 holinergic receptor mediating contraction of body wall muscle in parasitic nematodes.

161 subunit composition for all three C. elegans body wall muscle ionotropic receptors provides a critica

162 ell as MgATPase V(max) compared to embryonic body wall muscle isoform (EMB) (expressed in a multitude

163 nical stress in polarized epithelia and axon-body wall muscle junctions.

164 We find that dorsal acute body wall muscle myosin (EMB-9c11d) shows a significant

165 The body wall muscle of a Drosophila larva is generated by f

166 focal-adhesion-like structures found in the body wall muscle of the nematode Caenorhabditis elegans.

167 The Wnt CWN-1 is expressed in the posterior body wall muscle of the worm as well as in the SMs, maki

168 When ectopically expressed in body wall muscle precursor cells, hyperactivated EGL-15

169 Histochemical staining of larval body wall muscle revealed that the mutant A-type lamin,

170 se body complex and a conserved regulator of body wall muscle sarcomere organization and organelle po

171 Further, we find that sexual modification of body wall muscle together with the nervous system is req

172 mak-1 is expressed in nematode body wall muscle, and antibodies to MAK-1 localize betwe

173 ous tissues including the nervous system and body wall muscle, and knockdown of smn-1 by RNA interfer

174 multiple tissues - the epidermis, intestine, body wall muscle, ciliated sensory neurons and touch rec

175 as expressed in pharyngeal and tail neurons, body wall muscle, spermatheca, and vulva.

176 l for excitation-contraction coupling in the body wall muscle, through the coordination of calcium si

177 By polarized light microscopy of body wall muscle, unc-96 mutants display reduced myofibr

178 ndrial localization to actin-rich I-bands in body wall muscle.

179 ssembly of sarcomeric actin filaments in the body wall muscle.

180 rganization of sarcomeric actin filaments in body wall muscle.

181 ls to distinct fates, such as heart, gut and body wall muscle.

182 eventually leading to an excess of hypaxial body wall muscle.

183 ak disorganization of the actin filaments in body wall muscle.

184 protein RNF-5 in the Caenorhabditis elegans body wall muscle.

185 quired for proper actin filament assembly in body wall muscle.

186 riments indicate that TWK-18 is expressed in body wall muscle.

187 fect and mild fiber degeneration in striated body wall muscle.

188 tal properties and molecular determinants of body-wall muscle APs.

189 tion (lf) mutants, SLO-1 was mislocalized in body-wall muscle but its transcription and protein level

190 wn that the focus of unc-52 action is not in body-wall muscle but most likely is in hypodermis.

191 arable to the junctional current (I(j)) into body-wall muscle cells caused significant depolarization

192 We found that body-wall muscle cells were electrically coupled in a hi

193 e UNC-9 expression in both motor neurons and body-wall muscle cells, analyses of miniature and evoked

194 In body-wall muscle cells, CTN-1 coclusters with SLO-1 at r

195 abditis elegans BK channel alpha-subunit, in body-wall muscle cells.

196 e made the first extracellular recordings of body-wall muscle electrophysiology inside an intact roun

197 rs are specified and there is a loss of most body-wall muscle fibers.

198 native indirect flight muscle and embryonic body-wall muscle isoforms only in the exon 7 region.

199 ment sites (M-lines and dense bodies) in the body-wall muscle of C. elegans.

200 Few body-wall muscle precursors are specified and there is a

201 increased frequency of Ca(2+) transients in body-wall muscle, and abnormal locomotion behavior.

202 irecting expression of UNC-89-C primarily in body-wall muscle, and one internal promoter directing ex

203 ine in autophagic activity in the intestine, body-wall muscle, pharynx, and neurons of wild-type anim

204 lectrical coupling in Caenorhabditis elegans body-wall muscle.

205 ent and not in most late embryonic or larval body-wall muscle.

206 ity assays and histochemical staining of the body-wall muscle.

207 exible polymers that connect the pharynx and body-wall-muscle basement membranes.

208 The MS blastomere produces one-third of the body wall muscles (BWMs) in the C. elegans embryo.

209 rsors, while later expression is observed in body wall muscles and a subset of pharyngeal neurons.

210 L(2)efl] is specifically expressed in larval body wall muscles and accumulates at the level of Z-band

211 ts mutant phenotype, the complete absence of body wall muscles and corresponding presence of unfused

212 ning both remnants of specific larval dorsal body wall muscles and extrinsic myoblasts.

213 ress in the somatic mesodermal precursors to body wall muscles and fat body and together direct expre

214 kdown of lbx1 causes a specific reduction of body wall muscles and hypoglossal muscles originating fr

215 tes of MIF production are in the hypodermis, body wall muscles and in the nuclei of developing embryo

216 In contrast to many vertebrates, the ventral body wall muscles and limb muscles of Xenopus develop at

217 sion that leads to the formation of striated body wall muscles and non-muscle coelomocytes.

218 and for strong contractions of the posterior body wall muscles during defecation.

219 plementation is only slightly deleterious to body wall muscles during development or upon acute appli

220 been described in pharyngeal precursors and body wall muscles during embryogenesis, and amphid senso

221 neurons have defects in pathfinding and the body wall muscles have defective morphology.

222 number of identified motor neurons innervate body wall muscles in a highly stereotyped pattern.

223 s role of the affected pericardial cells and body wall muscles in developing and/or maintaining cardi

224 motoneurons innervate two slowly contracting body wall muscles in the larva.

225 y, CYK-1 and FHOD-1, are present in striated body wall muscles near or on sarcomere Z lines, where ba

226 The larval body wall muscles of Drosophila melanogaster arise by fu

227 The body wall muscles require ongoing NAD(+) salvage biosynt

228 Formation of the Drosophila larval body wall muscles requires the specification, coordinate

229 sis of the ionic currents from mutant larval body wall muscles showed a specific effect on delayed re

230 ignalling from the pharyngeal MC neurons and body wall muscles slows larval development.

231 e isoform (EMB) (expressed in a multitude of body wall muscles).

232 inkages from the myofibrillar lattice of the body wall muscles, across an intervening extracellular m

233 road range of tissues from the intestine, to body wall muscles, and neurons.

234 zes with epithelial hemidesmosomes overlying body wall muscles, beginning at the time of embryonic cu

235 is shell is comprised of the cuticle and the body wall muscles, either of which could contribute to t

236 the mutant synthetase using cell-selective (body wall muscles, intestinal epithelial cells, neurons,

237 re impaired in the hermaphrodites, including body wall muscles, pharyngeal muscles and vulval muscles

238 RP-3 was expressed primarily in the anterior body wall muscles, suggesting that it may play a role in

239 that attach to the hypodermis, specifically body wall muscles, vulval muscles, and mechanosensory ne

240 s apparent in the nerve ring, intestine, and body wall muscles.

241 channelrhodopsin-2 to induce contractions in body wall muscles.

242 usion and in myofibril assembly in embryonic body wall muscles.

243 e to particular pericardial cells and dorsal body wall muscles.

244 of aging Caenorhabditis elegans neurons and body wall muscles.

245 sor cells of the heart, visceral, and dorsal body wall muscles.

246 or excitation-contraction coupling in larval body wall muscles.

247 owth cones stall again when they contact the body wall muscles.

248 rgic motor neurons that synapse onto ventral body wall muscles.

249 e ZYX-1 protein is expressed in the striated body-wall muscles and localizes at dense bodies/Z-discs

250 Loss of clhm-1 in the body-wall muscles disrupts locomotory kinematics and bio

251 d by rhythmic and sequential contractions of body-wall muscles from the posterior to anterior segment

252 gle-copy transgenes that express rde-4(+) in body-wall muscles or hypodermis, however, enable silenci

253 Expressed in body-wall muscles, LECT-2 decorates neuronal processes a

254 y driving contraction waves of the abdominal body-wall muscles.

255 s in response to varying tonus of C. elegans body-wall muscles.

256 egmental ectoderm developed normally, as did body wall musculature and some other mesodermal derivati

257 he basal vertebrates, separate ventrolateral body wall musculature of the trunk into two discrete lay

258 all of the myoblasts that contribute to the body wall musculature, as well as in a group of cells th

259 ult satellite cells associated with limb and body wall musculature, as well as the diaphragm and extr

260 interfere with the proper development of the body wall musculature.

261 ential development of gut musculature versus body wall musculature.

262 Caenorhabditis elegans, morphogenesis of the body-wall musculature involves short-range migrations of

263 hey show unique features such as an inverted body-wall musculature or a novel pharyngeal organ.

264 , like mammalian skeletal muscle, C. elegans body-wall myocytes generate all-or-none APs, which evoke

265 dy segment, have dendrites tiling the larval body wall nearly completely without redundancy.

266 Body wall neurons showed a variety of distinct response

267 in wts mutants, dendrites initially tile the body wall normally, but progressively lose branches at l

268 the perineum-the caudal portion of the trunk body wall not associated with the hindlimb.

269 entral (DV) compartments and limb (wing) and body wall (notum) primordia.

270 al) and distinguish the future wing from the body wall (notum).

271 iffening of mutable connective tissue in the body wall of A. japonicus, whilst holokinins (PLGYMFR an

272 The nerve terminals found in the body wall of Drosophila melanogaster larvae are readily

273 As a member of Cnidaria, the body wall of hydra is structurally reduced to an epithel

274 Pleurites are chitinous plates in the body wall of insects and myriapods.

275 ngs support an LPM contribution to the trunk body wall of these taxa, which is similar to published d

276 ressure waves (OPW), or shock wave, with the body wall or body armor produces two types of energy wav

277 h the neo gene has been removed did not have body wall or sternum defects.

278 evolutionary relationship between appendages/body-wall outgrowths in animals.

279 We elucidate how the four-layer body wall pattern, restricted to the non-mammal tetrapod

280 and cell apoptosis contributing to the open body wall phenotype.

281 the swimming rhythm expressed in nerve cord-body wall preparations and, at a different phase angle,

282 -distal axis into regions giving rise to the body wall (proximal), wing hinge (central) and wing blad

283 bstitutions in the turret (P90C, P89C/S97C), body wall (S65C/S190C, S65C/D315C) and the transmembrane

284 tome paired appendages form as outgrowths of body wall somatopleure, a tissue composed of somatic lat

285 y generate peptides (holokinins) that affect body wall stiffness in sea cucumbers, providing a novel

286 lial sheath, which are also expressed in the body-wall striated muscle.

287 and neurons receiving sensory input from the body wall synchronizes their growth to ensure proper den

288 aste organ, and three labeled neurons of the body wall that may be chemosensory.

289 including those associated with the primary body wall, the umbilical ring, and the mesoderm of the s

290 prevent the kidneys from detaching from the body wall, thus explaining their fusion and pelvic locat

291 ts of the pattern differentiating the future body wall tissue from the wing blade tissue.

292 failure of the lateral edges of the ventral body wall to fuse, leading to cardia bifida.

293 Ventral body wall (VBW) defects are among the most common congen

294 nimals revealed that unc-94a is expressed in body wall, vulval and uterine muscles, whereas unc-94b i

295 Second, the head and tail anchor while a body wall wave moves each abdominal segment in the direc

296 The anterior foot and body wall were densely innervated by 5HTli fibers but no

297 a neurons branch over defined regions of the body wall, which in some cases correspond to specific na

298 By mechanically stimulating the leech body wall while recording from mechanoreceptors, we show

299 into dorsoventral (DV) compartments and limb-body wall (wing-notum) primordia depends on Epidermal Gr

300 arbor and a smaller arbor in the peripheral body wall, with fewer terminal branches.