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1 ntributes to post-transplant CKD mineral and bone disorder.
2 n cherubism, a rare autosomal-dominant human bone disorder.
3  play a pivotal role in diagnosing metabolic bone disorders.
4 could lead to new treatments for age-related bone disorders.
5 jor drug target for osteoporosis and related-bone disorders.
6 es with risks for cancer, cardiovascular and bone disorders.
7 with great promise as therapeutic agents for bone disorders.
8 rs was an independent factor associated with bone disorders.
9 t role in the treatment of periodontitis and bone disorders.
10 ts further our knowledge of FGFR3-associated bone disorders.
11 al benefit and render BMT more applicable to bone disorders.
12 te human Opt as a candidate gene for brittle bone disorders.
13  therapeutic target for bone loss in various bone disorders.
14 d paralog of MAN1, an NE component linked to bone disorders.
15 ention, and treatment of pediatric metabolic bone disorders.
16 ct of mechanism-based anabolic therapies for bone disorders.
17  immunologic aspects of the autoinflammatory bone disorders.
18 ht yield novel therapeutics to treat typical bone disorders.
19 r bone remodeling and is altered in multiple bone disorders.
20 ions in CBF genes are found in leukemias and bone disorders.
21 , raising new directions in the treatment of bone disorders.
22                                  An adynamic bone disorder (ABD) is an important complication of chro
23 n the Runt domain found in hematopoietic and bone disorders affect its affinity for DNA.
24 rteriosclerosis, vascular calcification, and bone disorders, all of which are also associated with ag
25 tudy revealed a high prevalence of metabolic bone disorders among viral cirrhotic patients.
26 phosphatemia in a mouse model of CKD-mineral bone disorder and alphaKL-null mice.
27 sk factor for periodontitis, an inflammatory bone disorder and the greatest cause of tooth loss in ad
28 d Amy, analogs of which are therapeutics for bone disorders and diabetes, respectively.
29                                     Systemic bone disorders and PAD are frequent in ESRD.
30 phosphatemia in a mouse model of CKD-mineral bone disorder, and it reduced hyperphosphatemia and prev
31 tations in CBF genes are found in leukemias, bone disorders, and gastric cancer.
32 tations in CBF genes are found in leukemias, bone disorders, and gastric cancers.
33 nfections, vitamin D deficiency or metabolic bone disorders, anxiety disorders, and fractures; the in
34               Finally, several cartilage and bone disorders are due to abnormalities in sulfate trans
35                                              Bone disorders are of significant concern due to increas
36                                    Metabolic Bone disorders are well-recognized extrahepatic complica
37          Findings from research into mineral bone disorder associated with CKD (CKD-MBD) could help t
38                  Osteoporosis is a metabolic bone disorder associated with compromised bone strength
39 -Engelmann disease, a nonmalignant metabolic bone disorder associated with increased TGF-beta activit
40                            Osteoporosis is a bone disorder associated with loss of bone mineral densi
41 sents a summary of recent reports concerning bone disorders associated with disorders of the liver an
42 del, however, is complicated by the adynamic bone disorder; both calcitriol and paricalcitol stimulat
43 hosphonates (NBPs) are taken by millions for bone disorders but may cause serious inflammatory reacti
44 he pathophysiology of many acute and chronic bone disorders, but the specific inflammatory networks t
45 e bone disease' has mainly been considered a bone disorder caused by collagen mutations.
46 reatment of craniosynostosis or other severe bone disorders caused by mutations in FGFRs that current
47 tegies and may inform clinical treatments of bone disorders causes by load-deficiency.
48 esis imperfecta (OI) is an inherited brittle bone disorder characterized by bone fragility and low bo
49 O, MIM 174810) is a rare, autosomal dominant bone disorder characterized by focal areas of increased
50 sease of bone, or "osteitis deformans," is a bone disorder characterized by rapid bone remodeling res
51    Osteopetrosis is a heterogeneous group of bone disorders characterized by the failure of osteoclas
52           Chronic kidney disease-mineral and bone disorder (CKD-MBD) is associated with secondary hyp
53 r risk and are components of CKD-mineral and bone disorder (CKD-MBD).
54 er small molecule leads for the treatment of bone disorders concluded with the discovery of a compoun
55  cancer diagnosis, cancer margin evaluation, bone disorder detection and glucose level determination.
56 anifestations of NF1, including learning and bone disorders emphasize the importance of dissecting th
57                                      Another bone disorder, familial expansile osteolysis (FEO), alth
58 CKD in this model is associated the adynamic bone disorder form of renal osteodystrophy.
59 gement of chronic kidney disease-mineral and bone disorders has recently changed.
60 en learned in the monogenic autoinflammatory bone disorders, IL-1 is emerging as an important pathway
61 es, which lead to the development of various bone disorders in both women and men.
62 cs and immunologic basis of autoinflammatory bone disorders including chronic recurrent multifocal os
63  TNF inhibits bone formation in inflammatory bone disorders is by promoting Runx2 proteasomal degrada
64     The chronic kidney disease (CKD)-mineral bone disorder (MBD) is a syndrome recently coined to emb
65                       Anemia and mineral and bone disorders (MBD) are both important and common compl
66               There are two primary types of bone disorders observed in patients with end-stage renal
67 e expression to osteoblasts for treatment of bone disorders or tumor metastasis to the skeleton.
68 ), certain types of cancer, and degenerative bone disorders (osteoarthritis).
69 ollagen triple helix, lead to the hereditary bone disorder osteogenesis imperfecta (OI).
70                               The hereditary bone disorder osteogenesis imperfecta is often caused by
71 es in MSCs from individuals with the brittle bone disorder osteogenesis imperfecta, demonstrating suc
72 ple helix results in the dominant hereditary bone disorder osteogenesis imperfecta.
73 wing prevalence of metabolic and age-related bone disorders, our results demonstrate for the first ti
74                                Human genetic bone disorders provide insight into bone regulatory proc
75 flammatory networks that regulate individual bone disorders remain to be elucidated.
76 sent the first five cases of a new recessive bone disorder resulting from null LEPRE1 alleles; its ph
77 ures, gene identification in each 'vanishing bone' disorder should provide unique insights into genet
78 s an established concept in the treatment of bone disorders such as osteoporosis or certain forms of
79 are potential therapies for the treatment of bone disorders such as osteoporosis.
80 process, and deregulation can lead to severe bone disorders such as osteoporosis.
81  represent potential therapeutic targets for bone disorders such as osteoporosis.
82 l, bone anabolic agents for the treatment of bone disorders such as osteoporosis.
83 ediator of joint destruction in inflammatory bone disorders, such as RA.
84         Cherubism is a rare autoinflammatory bone disorder that is associated with point mutations in
85 een identified in monogenic autoinflammatory bone disorders that have allowed more detailed dissectio
86                    Newly diagnosed metabolic bone disorders were seen in 58% of the breast cancer pop
87                                              Bone disorders were significantly more frequent in old p
88 ations of chronic kidney disease-mineral and bone disorder with a special focus on the incidence of f
89 e P392L SQSTM1 mutation in humans, develop a bone disorder with remarkable similarity to PDB.
90 ssure index (ABix) and evaluated mineral and bone disorders with bone histomorphometry.

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