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1 ntributes to post-transplant CKD mineral and bone disorder.
2 n cherubism, a rare autosomal-dominant human bone disorder.
3 play a pivotal role in diagnosing metabolic bone disorders.
4 could lead to new treatments for age-related bone disorders.
5 jor drug target for osteoporosis and related-bone disorders.
6 es with risks for cancer, cardiovascular and bone disorders.
7 with great promise as therapeutic agents for bone disorders.
8 rs was an independent factor associated with bone disorders.
9 t role in the treatment of periodontitis and bone disorders.
10 ts further our knowledge of FGFR3-associated bone disorders.
11 al benefit and render BMT more applicable to bone disorders.
12 te human Opt as a candidate gene for brittle bone disorders.
13 therapeutic target for bone loss in various bone disorders.
14 d paralog of MAN1, an NE component linked to bone disorders.
15 ention, and treatment of pediatric metabolic bone disorders.
16 ct of mechanism-based anabolic therapies for bone disorders.
17 immunologic aspects of the autoinflammatory bone disorders.
18 ht yield novel therapeutics to treat typical bone disorders.
19 r bone remodeling and is altered in multiple bone disorders.
20 ions in CBF genes are found in leukemias and bone disorders.
21 , raising new directions in the treatment of bone disorders.
24 rteriosclerosis, vascular calcification, and bone disorders, all of which are also associated with ag
27 sk factor for periodontitis, an inflammatory bone disorder and the greatest cause of tooth loss in ad
30 phosphatemia in a mouse model of CKD-mineral bone disorder, and it reduced hyperphosphatemia and prev
33 nfections, vitamin D deficiency or metabolic bone disorders, anxiety disorders, and fractures; the in
39 -Engelmann disease, a nonmalignant metabolic bone disorder associated with increased TGF-beta activit
41 sents a summary of recent reports concerning bone disorders associated with disorders of the liver an
42 del, however, is complicated by the adynamic bone disorder; both calcitriol and paricalcitol stimulat
43 hosphonates (NBPs) are taken by millions for bone disorders but may cause serious inflammatory reacti
44 he pathophysiology of many acute and chronic bone disorders, but the specific inflammatory networks t
46 reatment of craniosynostosis or other severe bone disorders caused by mutations in FGFRs that current
48 esis imperfecta (OI) is an inherited brittle bone disorder characterized by bone fragility and low bo
49 O, MIM 174810) is a rare, autosomal dominant bone disorder characterized by focal areas of increased
50 sease of bone, or "osteitis deformans," is a bone disorder characterized by rapid bone remodeling res
51 Osteopetrosis is a heterogeneous group of bone disorders characterized by the failure of osteoclas
54 er small molecule leads for the treatment of bone disorders concluded with the discovery of a compoun
55 cancer diagnosis, cancer margin evaluation, bone disorder detection and glucose level determination.
56 anifestations of NF1, including learning and bone disorders emphasize the importance of dissecting th
60 en learned in the monogenic autoinflammatory bone disorders, IL-1 is emerging as an important pathway
62 cs and immunologic basis of autoinflammatory bone disorders including chronic recurrent multifocal os
63 TNF inhibits bone formation in inflammatory bone disorders is by promoting Runx2 proteasomal degrada
64 The chronic kidney disease (CKD)-mineral bone disorder (MBD) is a syndrome recently coined to emb
71 es in MSCs from individuals with the brittle bone disorder osteogenesis imperfecta, demonstrating suc
73 wing prevalence of metabolic and age-related bone disorders, our results demonstrate for the first ti
76 sent the first five cases of a new recessive bone disorder resulting from null LEPRE1 alleles; its ph
77 ures, gene identification in each 'vanishing bone' disorder should provide unique insights into genet
78 s an established concept in the treatment of bone disorders such as osteoporosis or certain forms of
85 een identified in monogenic autoinflammatory bone disorders that have allowed more detailed dissectio
88 ations of chronic kidney disease-mineral and bone disorder with a special focus on the incidence of f
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