ライフサイエンスコーパス: bone marrow

1 ssue (cholangiocytes) and the immune system (bone marrow).

2 n the ratio of immature Gr1(lo) cells in the bone marrow.

3 C) originate from both the yolk sac (YS) and bone marrow.

4 ABIN1[D485N] mice transplanted with WT mouse bone marrow.

5 he early release of classical monocytes from bone marrow.

6 ind individual GMPs scattered throughout the bone marrow.

7 tical role in promoting ILC2 egress from the bone marrow.

8 lization and EMH but not HSC division in the bone marrow.

9 sion of early viral genes exclusively in the bone marrow.

10 ing homing and retention to the niche in the bone marrow.

11 the egress of multiple myeloma PCs from the bone marrow.

12 nate from common lymphoid progenitors in the bone marrow.

13 iates homing of prostate cancer cells to the bone marrow.

14 It commonly metastasizes to the bone marrow.

15 tumor, soft tissue and bone metastases, and bone marrow.

16 ading to ectopic platelet release within the bone marrow.

17 ery limited ability to repopulate from donor bone marrow.

18 of essential phenomena occurring within the bone marrow.

19 lenge mobilized ILC2 progenitors to exit the bone marrow.

20 chymal stromal cells and cancer cells in the bone marrow.

21 r in the presence of cytokeratin(+) cells in bone marrow.

22 ressor cells (MDSC) from precursors in mouse bone marrow.

23 ), liver (16.1%), brown adipose (29.7%), and bone marrow (32.9%)-and increases of 16.8-19.1% in seven

24 demonstrated increased [(18)F]-FDG uptake in bone marrow 4 days postinfection compared to surviving N

25 The ratio of mean tumor to bone marrow absorbed dose per unit administered activity

26 The tumor-to-dose-limiting-organ (bone marrow) absorbed dose ratio, that is, the therapeut

27 dalar activity was associated with increased bone-marrow activity (r=0.47; p<0.0001), arterial inflam

28 Amygdalar activity, bone-marrow activity, and arterial inflammation were ass

29 Besides osteoblasts, MMPs also give rise to bone marrow adipocytes and stromal cells in vivo.

30 ivo and in vitro models of AML, we show that bone marrow adipocytes from the tumor microenvironment s

31 ollowing withdrawal of teriparatide therapy, bone marrow adipocytes increased dramatically in number.

32 ort the first description of AML programming bone marrow adipocytes to generate a protumoral microenv

33 Bone marrow adipose tissue (MAT) is negatively associate

34 e aging characteristics, including increased bone marrow adiposity, decreased bone mass, and impaired

35 Meanwhile, it also inhibited bone marrow adiposity.

36 tation protocol relies on cell homing to the bone marrow after intravenous injection.

37 matched unrelated (MUD) donor T-cell-replete bone marrow allografting, obviating the need for additio

38 The ADC values of intact bone marrow and BMLs did not overlap.

39 lls promoted tumor cell dissemination in the bone marrow and enhanced osteolytic lesion formation, ir

40 diation damage to critical organs, including bone marrow and kidney.

41 In contrast, the bone marrow and lymph nodes of nonsurvivors showed incre

42 ADC values were measured in intact bone marrow and major BMLs.

43 stablishment of plasma cell niches in sorted bone marrow and rectal cell populations further supporte

44 ic stem cells (LSC) and their progenitors in bone marrow and relapse following treatment cessation.

45 We assessed differentiation of B cells in bone marrow and spleen and analyzed their endosomal morp

46 mice strongly decreased Samd14 expression in bone marrow and spleen.

47 However, transplantation of donor Cdk5(-/-C) bone marrow and T cells dramatically reduced the severit

48 Accumulation of erythrocytic parasites in bone marrow and the spleen has been reported in cases of

49 colorectal cancer cell dissemination in the bone marrow and they reveal a novel mechanism through wh

50 ion (TBI) damages hematopoietic cells in the bone marrow and thymus; however, the long-term effects o

51 hematopoietic stem or progenitor cells from bone marrow and to sensitize cancer cells to conventiona

52 semination of colorectal cancer cells in the bone marrow and tumor-driven osteolytic lesion formation

53 tricting effects of mutant SAMD9 proteins in bone marrow and was associated with increased length of

54 Levels of S1P in liver, bone marrow, and lymph fluid were measured using an enzy

55 effect of ENPP1 selectively on generation of bone marrow as well as splenic LLPCs.

56 ls obtained from peripheral blood or through bone marrow aspirates, together with recent advances in

57 Bone marrow aspiration from the iliac crests and in vivo

58 een and a block in B cell development in the bone marrow at the small pre-B cell stage.

59 odies (n = 163) for the presence of abnormal bone marrow attenuation on VNCa images by using color-co

60 e reductions in the number and proportion of bone marrow B-lymphoid progenitors.

61 The exchange of bone marrow between wild-type and MAP3K14(aly/aly) mice

62 cible ischemia in Tc-99m SPECT who underwent bone marrow biopsy and were allocated to cells (n=16) or

63 After histologic analysis of the bone marrow biopsy specimen, diagnosis of Waldenstrom ma

64 prognostic meaning of different patterns of bone marrow (BM) (18)F-FDG uptake in HL.

65 rotein receptor null) mice transplanted with bone marrow (BM) cells from Ncr1(iCre)R26R(lsl-)(DTA) ,

66 Bone marrow (BM) chimera mice revealed that mucosal repa

67 Hematopoietic stem cells (HSCs) in the bone marrow (BM) form mature blood cells of all lineages

68 Here we report that bone marrow (BM) Gr-1(lo) immature myeloid cells are res

69 state following hematopoietic stress, e.g., bone marrow (BM) injury, transplantation, or systemic in

70 These mice showed increased bone marrow (BM) levels of the protein dickkopf-1 (Dkk1)

71 Asxl2(-/-) mice have an increased bone marrow (BM) long-term haematopoietic stem cells (HS

72 nced by the active crosstalk with an altered bone marrow (BM) microenvironment.

73 tic stem and progenitor cells (HSPCs) in the bone marrow (BM) microenvironment.

74 nds and/or paracrine Wnts emanating from the bone marrow (BM) niche.

75 (+) cells (n = 8 patients) or unfractionated bone marrow (BM) or peripheral blood mononuclear cells (

76 undamental microenvironmental determinant of bone marrow (BM) pathophysiology.

77 ells (HSCs) are mobilized from niches in the bone marrow (BM) to the blood circulation by the cytokin

78 nct distribution patterns, highest in blood, bone marrow (BM), or lymph nodes (LN), with the frequenc

79 cell types, including endothelial cells and bone marrow (BM)-derived cells.

80 ed postnatally in the prepuberty period from bone marrow (BM)-derived progenitors.

81 Evidence was presented that bone marrow (BM)-derived Sinusoidal endothelial cell PRO

82 Although the emergence of bone marrow (BM)-resident (p190)BCR-ABL-specific T lymph

83 follicular response and reducing the pool of bone marrow (BM)-resident long-lived PCs.

84 optimal compared with that seen in wild-type bone marrow (BM)-transplanted OS mice in peripheral bloo

85 ress of hematopoietic stem cells (HSCs) from bone marrow (BM).

86 (PRRT) may induce long-term toxicity to the bone marrow (BM).

87 lophosphamide was originally described using bone marrow (BM).

88 ranscriptome and cytometry analyses in mixed bone-marrow (BM) chimeras.

89 g pregnancy induced HSC proliferation in the bone marrow but not HSC mobilization.

90 more efficiently reduce the clonogenicity of bone marrow cancer cells from patients with acute myeloi

91 ity induce ectopic adipocyte accumulation in bone marrow cavities.

92 tem cells, with negligible effects on normal bone marrow CD34(+) progenitors from healthy donors.

93 ine effects of transplanted unmodified human bone marrow CD34+ (hBM34+) cells into symptomatic G93A m

94 beta/Fc, or IL-2/Fc would enhance allogeneic bone marrow cell (BMC) engraftment and promote tolerance

95 mice in the aorta, draining lymph nodes, and bone marrow cell cultures, indicating that IRF5 maintain

96 Clinical trials of bone marrow cell-based therapies after acute myocardial

97 receptor family, is expressed in myeloid and bone marrow cells and was implicated as a checkpoint reg

98 ice were irradiated and given transplants of bone marrow cells from C57BL6 mice, with or without the

99 Bone marrow cells from CLP-treated mice had normal OC pr

100 rs and that of inflammatory macrophages from bone marrow cells leads to macrophage heterogeneity.

101 The bone marrow cells of Dnmt3a+/- mice had a subtle but sta

102 ent feedback mechanism through which myeloid bone marrow cells restore quiescence of myeloid-biased H

103 he murine context, silencing of AID in human bone marrow cells skews differentiation toward myelomono

104 ticated interaction network between multiple bone marrow cells that regulate different hematopoietic

105 id cell lineage displayed a dysregulation of bone marrow cells with a rapid decline in population at

106 We found that coculture of murine bone marrow cells with bladder tumor cells promoted stro

107 tion of myeloablated adult mice similarly to bone marrow cells.

108 ted pit formation caused by RANKL-stimulated bone marrow cells.

109 tion of exosomes produced by GM-CSF-expanded bone marrow cells.

110 , we used adoptive transfer, transgenic, and bone marrow chimera approaches to show increased infiltr

111 Bone marrow chimera experiments indicated that the obser

112 Consistent with this, bone marrow chimera studies show that aberrant Pkhd1 mus

113 iNKT thymic development in limited-dilution bone marrow chimeras and show that higher TCR avidity co

114 ellular MZ microenvironment, and analysis of bone marrow chimeras indicated that the MZ B cell develo

115 Bone marrow chimeras showed that bone marrow-derived cel

116 s, HLA-DR4-transgenic mice, MAIThighHLA-DR4+ bone marrow chimeras, and humanized NOD-scid IL-2Rgamman

117 al microscopy with DREAM-null mice and their bone marrow chimeras, we demonstrated that both hematopo

118 f infection, TLR2 knockout (TLR2KO)-->TLR2KO bone marrow chimeric mice exhibited increased bacterial

119 Bone marrow chimeric mice showed B cell intrinsic effect

120 g homeostasis, gammadeltaT17 cells emerge in bone marrow chimeric mice upon induction of skin inflamm

121 Using mixed bone marrow chimeric mice, we show that the impact of NK

122 REAM KO control mice, but not in WT or DREAM bone marrow chimeric mice.

123 neous tissue, muscle osseous structures, and bone marrow, consistent with advanced melanoma.

124 ecific Ab-secreting cells in lymph nodes and bone marrow, correlating with low Ab titers.

125 ve sampling from ileum, rectum, lymph nodes, bone marrow, CSF, circulating CD4+ T cell subsets, and p

126 T-cells are co-cultured with GM-CSF derived bone marrow dendritic cells (G-BMDCs).

127 enhanced maturation and cytokine release of bone marrow dendritic cells in vitro.

128 Bone marrow derived mesenchymal stem cells (MSCs) are re

129 in the chondrogenic differentiation of human bone marrow derived SSCs.

130 that eicosanoid production profiles between bone marrow-derived (BMDM) and peritoneal macrophages di

131 is critical for protection from IRI through bone marrow-derived adenosine 2a receptors.

132 Interestingly, whereas donor T cell- or bone marrow-derived CD70 plays no role in GVHD, host-der

133 Bone marrow chimeras showed that bone marrow-derived cells contributed to IL-1R-dependent

134 s mainly organized through the activation of bone marrow-derived cells in various tissues.

135 Bone marrow-derived circulating progenitor cells are inv

136 est that commonly used protocols to generate bone marrow-derived cultured dendritic cells yield a het

137 Bone marrow-derived cultured mast cells (BMCMCs) and per

138 onocyte-derived DCs and humanized TLR8 mouse bone marrow-derived DCs enabled benchmarking of the TLR8

139 3) showed lower expression in ES-DCs than in bone marrow-derived DCs.

140 hat PGE2 inhibits IL-27 production in murine bone marrow-derived DCs.

141 a, IL-6 and CD68), decreased accumulation of bone marrow-derived fibroblasts and TGF-beta expression.

142 ng the lineage-instructive signal in primary bone marrow-derived GM progenitors.

143 f autologous (auto) versus allogeneic (allo) bone marrow-derived hMSCs in NIDCM.

144 safety and efficacy of 2 doses of allogeneic bone marrow-derived human mesenchymal stem cells identic

145 Bone marrow-derived macrophage were polarized to an M2 p

146 lations of wild-type mice with Nrp1-depleted bone marrow-derived macrophages (BMDM) confers resistanc

147 eficient RAW cells and primary PHD2 knockout bone marrow-derived macrophages (BMDM).

148 aB pathway (IKKalpha/beta, NF-kappaB p65) in bone marrow-derived macrophages (BMDMs) from knockout mi

149 In LPS/ATP-stimulated bone marrow-derived macrophages (BMDMs), CLIC1 or CLIC4

150 Stimulation of bone marrow-derived macrophages (BMMs) with endotoxin re

151 also observed by in vitro experiments, using bone marrow-derived macrophages and dendritic cells as r

152 n rates and mROS expression in mock-infected bone marrow-derived macrophages but reduced caspase-depe

153 Bone marrow-derived macrophages did not release NE in re

154 The effects of estrogen are long-lasting; bone marrow-derived macrophages from ovariectomized mice

155 Our previous studies showed that bone marrow-derived macrophages from S100A4(-/-) mice ex

156 n of caspase-1 inhibitors or the infusion of bone marrow-derived macrophages genetically engineered t

157 CD44(+/+) murine bone marrow-derived macrophages produced higher TNF-alph

158 Ex vivo, TLR9(-/-) bone marrow-derived macrophages produced more A20 than W

159 oxide addition to CB3-infected NOD.Ncf1(m1J) bone marrow-derived macrophages rescued the inflammatory

160 licited in an analogous fashion using LPS in bone marrow-derived macrophages upon inhibition of caspa

161 In SIRT3 knock-out bone marrow-derived macrophages, NLRP3 activation promot

162 We have found that in murine bone marrow-derived macrophages, PGE2 via the cAMP/prote

163 Like bone marrow-derived macrophages, RNA editing in MG leads

164 Using bone marrow-derived mast cells (BMMCs), we tested the hy

165 Hierarchical clustering demonstrated that bone marrow-derived mast cells and BMBs shared specific

166 icant body of evidence has demonstrated that bone marrow-derived mesenchymal stem cells (BMSCs) showe

167 protein to responsive target cells, such as bone marrow-derived mesenchymal stem cells (BMSCs), rema

168 Bone marrow-derived mesenchymal stem cells (MSC) have be

169 UC-MSCs in vitro, compared with bone marrow-derived mesenchymal stem cells, displayed a

170 nce its down-regulation (DR) in both ex vivo bone marrow-derived mesenchymal stromal cells (MSC) and

171 were then used for the batch transduction of bone marrow-derived mesenchymal stromal cells ex vivo, f

172 Specifically, we demonstrate that bone marrow-derived monocytes and macrophages are essent

173 Bone marrow-derived monocytes and macrophages were the p

174 an monocyte-derived dendritic cells (moDCs), bone marrow-derived mouse dendritic cells (BMDCs), and m

175 erapies (such as hematopoietic stem cells or bone marrow-derived MSC or dendritic cells) for optimiza

176 cells promoted strong expression of PD-L1 in bone marrow-derived myeloid cells.

177 rable properties for T2 - weighted MRI, with bone marrow-derived primary human mesenchymal stem cells

178 ate that lung regeneration is facilitated by bone-marrow-derived myeloid cells that are recruited to

179 Patients with CXCR4 mutations have higher bone marrow disease burden, and those with nonsense CXCR

180 astic syndromes (MDS) are a diverse group of bone marrow disorders and clonal hematopoietic stem cell

181 both local expansion of metabolically active bone marrow documented by FDG uptake and with the number

182 dionuclide therapy by reducing the liver and bone marrow doses as well as the effective dose.

183 Additionally, jagged-2 expressed in bone marrow ECs regulated HSPC cell cycle and quiescence

184 magnetic resonance (MR) imaging of transient bone marrow edema syndrome (TBMES) and avascular osteone

185 stem/progenitor cells in the adult mammalian bone marrow ensure blood cell renewal.

186 Mast cell (MC) progenitors leave the bone marrow, enter the circulation, and settle in the sk

187 R (ASqPCR) for the KIT D816V mutation, and a bone marrow examination.

188 This defect was rescued by bone marrow exosomes from WT, but not miR-155(-/-), cell

189 cause of the short half-lives and suboptimal bone marrow exposure of the drugs.

190 es in 2 siblings presenting with progressive bone marrow failure (BMF), immunodeficiency, and develop

191 dult mice resulted in acute lethality due to bone marrow failure and intestinal atrophy featuring ste

192 ditary breast and ovarian cancers as well as bone marrow failure disorder Fanconi anemia (FA).

193 estriction (IUGR) with gonadal, adrenal, and bone marrow failure, predisposition to infections, and h

194 or older with thrombocytopenia secondary to bone marrow failure, requiring prophylactic platelet tra

195 Bone marrow fibrosis (BMF) develops in various hematolog

196 okines, reduced disease burden, and reversed bone marrow fibrosis in vivo.

197 Bone marrow fibrosis is the result of a complex and not

198 plasma cells (LLPCs), which persisted in the bone marrow for several months after vaccination.

199 ion, LOX was expressed by tumor cells in the bone marrow from colorectal cancer patients with bone me

200 Dead cells accumulated in bone marrow from lupus patients but not from nonautoimmu

201 GSNOR KO animals receiving WT bone marrow had significantly reduced survival following

202 ization, and EMH during pregnancy but normal bone marrow hematopoiesis and EMH in response to bleedin

203 ted the origin of SF3B1 mutations within the bone marrow hematopoietic stem and progenitor cell compa

204 state cancer cells was sufficient to promote bone marrow homing within a short timeframe.

205 s blood parameters, leukocyte depletion, and bone marrow hypoplasia.

206 Increased bone marrow hypoxia is associated with increased recircu

207 ogenic effects of sorafenib led to increased bone marrow hypoxia, which contributed to HIF-dependent

208 ter infection and developed fewer spleen and bone marrow IgG plasma cells and memory B cells, compare

209 inducible NPs containing RA can engraft into bone marrow in vivo in the proximity of other leukaemic

210 Furthermore, transplantation of WT bone marrow into miR-155KO mice mitigated this phenotype

211 The TBI model better predicts the absence of bone marrow iron than SF concentration alone, and TBI ca

212 spin-echo (TSE) imaging in the detection of bone marrow lesions (BMLs) after knee trauma.

213 ng fibroblasts derived from hematopoietic or bone marrow lineages in hearts subjected to permanent le

214 in particular the potential contribution of bone marrow lineages to activated fibroblasts within the

215 re of epicardial origin and not derived from bone marrow lineages, endothelial-to-mesenchymal transit

216 e 1 (ENPP1) is preferentially upregulated in bone marrow LLPCs compared with their splenic short-live

217 oriasis, and mastocytosis skin as well as in bone marrow mast cells in patients with systemic mastocy

218 Thrombocytosis, bone marrow megakaryocytic proliferation, and presence o

219 By profiling mRNA expression in the bone marrow mesenchymal progenitor cell line ST2, we dis

220 decrease in bone formation and the number of bone marrow mesenchymal stem/stromal cells, likely due t

221 stem cell factor [SCF], ThPO, and IL-6) from bone marrow mesenchymal stromal cells (MSCs) in vitro.

222 imulation induces collagen deposition in the bone marrow mesenchymal stromal cells.

223 lated, at least in part, to signals from the bone marrow microenvironment (BMM).

224 nic cytokines and related alterations of the bone marrow microenvironment are commonly found in SM.

225 The bone marrow microenvironment influences malignant hemato

226 between human prostate cancer cells and the bone marrow microenvironment mediate bone metastasis dur

227 aintaining immunological homoeostasis in the bone marrow microenvironment, both in physiological cond

228 sults define novel epigenetic changes in the bone marrow microenvironment, which lead to beta-catenin

229 rimary MM cells alone and in the presence of bone marrow microenvironment.

230 omising target for the treatment of diabetic bone marrow mobilopathy and vascular disease.

231 Adult bone marrow monocytes can give rise to tissue-resident m

232 85), LV ejection fraction was similar in the bone marrow mononuclear cells (48.7%) and placebo groups

233 ST-segment-elevation myocardial infarctions, bone marrow mononuclear cells administration did not imp

234 nor chimerism at single-cell resolution from bone marrow mononuclear cells isolated from transplant p

235 acebo-controlled trial comparing 150 million bone marrow mononuclear cells versus placebo in 120 pati

236 The test group received allogeneic bone marrow MSCs by intradiscal injection of 25 x 10 cel

237 Conditional depletion of Foxp1 in bone marrow MSCs led to premature aging characteristics,

238 In bone marrow MSCs, FOXP1 expression levels declined with

239 ance and repair of our bones are formed from bone marrow myeloid progenitor cells by a complex differ

240 We propose that the bone marrow niche can be altered by anticancer therapeut

241 Thus, we show that WNT5A in the bone marrow niche is required to regenerate HSCs and leu

242 llenge, the clonal response of leukocytes in bone marrow of acute myeloid leukaemia (AML) patients, a

243 es that had increased CD64 expression in the bone marrow of BALB/c/By/J mice prior to L. monocytogene

244 tic differentiation and proliferation in the bone marrow of Jak2(V617F) mice, whereas TGF-beta1 or Cx

245 in vivo and were detectable in the blood and bone marrow of patients who had a response and patients

246 In contrast to PCs in the bone marrow, PCs in the gut have been considered short l

247 antigen-specific plasma cells and long-lived bone marrow plasma cells was detected in the MNP boosted

248 d publicly available GEP data from patients' bone marrow plasma cells, with long-term follow-up and c

249 rotection at sites of infection with that of bone marrow plasmablasts and plasma cells to control vir

250 smablast trafficking to and retention in the bone marrow play a previously unappreciated role in vira

251 epletion of LARP1 alone in human adult CD34+ bone marrow precursor cells leads to a reduction in 5'TO

252 Bone marrow progenitor analysis revealed depletion of he

253 Bone marrow reconstitutions showed that a lncRNA express

254 T imaging objectively identified subclinical bone-marrow recovery within 5 days of HSC infusion, whic

255 ling of vasculature in central and endosteal bone marrow regions.

256 xicity due to unwanted p53 activation in the bone marrow remains an unmet clinical challenge.

257 ganization of myeloid differentiation in the bone marrow remains unknown.

258 d quantification and tracking of subclinical bone-marrow repopulation in human beings and revealed ne

259 eared from the serum by liver-, spleen-, and bone marrow-resident phagocytic cells.

260 uced hematocrit, splenomegaly, and increased bone marrow reticulin.

261 es, we also used targeted gene sequencing on bone marrow samples and investigated clonal evolution fr

262 In bone marrow samples, the microfluidic-based plasma cell

263 Stromal cells in the bone marrow secrete a variety of cytokines that promote

264 B are required for B cell development in the bone marrow, Spi1 (encoding PU.1) was conditionally dele

265 e comparative hepatogenic potential of human bone marrow stem cells (BMSC) with stem cells derived fr

266 Rat bone marrow stem cells were mingled with silk hydrogels

267 In addition, the role of the bone marrow stroma in regulating clinical responses to D

268 xpression of osteopontin (OPN) in the murine bone marrow stroma is reduced.

269 ed endothelialized channels lined with human bone marrow stromal cells, which adopt a mural cell-like

270 ers (alginate and hyaluronic acid) and mouse bone marrow stromal cells.

271 ANG-(1-7) increased Slit3 levels in the bone marrow supernatants, which activated ROCK in LSK ce

272 the presence of cytokeratin(+) cells in the bone marrow, this MSC subpopulation could prove useful i

273 ogressively impairs their homeostasis in the bone marrow through an unidentified mechanism.

274 Comparison of adult bone marrow to fetal liver lysates demonstrated developm

275 of iron deficiency (ID) through sampling of bone marrow to identify the absence of body iron stores

276 Monocytes can mobilize from bone marrow, traffic to their required destination, and

277 The results prompt caution when bone marrow transfers from individuals potentially carry

278 that it is possible to estimate dd-cfDNA in bone marrow transplant patients that are unrelated or th

279 METHODS AND Using genetic lineage tracing or bone marrow transplant, we found no evidence for collage

280 and Functional Assessment of Cancer Therapy-Bone Marrow Transplant.

281 However, LT is not curative and only bone marrow transplantation (BMT) can correct the underl

282 scle injury model combined with irradiation, bone marrow transplantation and in vivo imaging, we show

283 The combination of bone marrow transplantation and local muscle radiation p

284 Reciprocal bone marrow transplantation between KitW/Wv and KitWsh/W

285 Bone marrow transplantation failed to rescue outgrowth.

286 Allogeneic bone marrow transplantation has been attempted in severe

287 Finally, bone marrow transplantation studies were performed to de

288 me polymerase chain reaction, and reciprocal bone marrow transplantation were used to evaluate the ef

289 After bone marrow transplantation, donor-derived immune cells

290 , both in physiological conditions and after bone marrow transplantation.

291 sident beds that could not be transferred by bone marrow transplantation.

292 , dermatologic conditions, or solid-organ or bone marrow transplantation.

293 y and resident monocytes are retained in the bone marrow vasculature, representing an important reser

294 d dose per unit administered activity to the bone marrow was 0.13, 0.086, 0.33, and 0.068 mGy/MBq aft

295 Surprisingly, the collapse of the Rev1Xpc bone marrow was associated with progressive mitochondria

296 Bone marrow was the predominant stem cell source (n = 83

297 ial AT, spleen, mediastinal lymph nodes, and bone marrow were quantified by flow cytometry.

298 ytes and B cells were also diminished in the bone marrow, whereas the number of CD8(+) T cells increa

299 Bone marrow will be assessed by histology or immunohisto

300 Bone marrow with </= 5% tumor involvement will be classi