ライフサイエンスコーパス: bone marrow cell

1 ivo, and the same was observed with CD11b(-) bone marrow cells.

2 ent with an in vitro perturbation of Klf7 in bone marrow cells.

3 , and solute transporters but not markers of bone marrow cells.

4 tion of exosomes produced by GM-CSF-expanded bone marrow cells.

5 ral toxicity of shRNAs, we used normal mouse bone marrow cells.

6 2 cells on the gel as bone lining cells, and bone marrow cells.

7 wsh) mice and were labeled with transplanted bone marrow cells.

8 or successful in vitro osteoclastogenesis of bone marrow cells.

9 , were upregulated by GRO-gamma-primed mouse bone marrow cells.

10 markedly impaired as compared with wild-type bone marrow cells.

11 x-2 gene and activates beta-catenin in mouse bone marrow cells.

12 losuppression and by using moderate doses of bone marrow cells.

13 1-mutated patients but not of normal CD34(+) bone marrow cells.

14 number and function of fetal liver (FL) and bone marrow cells.

15 st, impaired hematopoietic reconstitution of bone marrow cells.

16 cant downregulation of miR-30c expression in bone marrow cells.

17 autoimmunity, and direct viral infection of bone marrow cells.

18 and osteoclasts were generated in vitro from bone marrow cells.

19 e receiving a transplant of Cited2 deficient bone marrow cells.

20 the CD11c(+) and CD11b(+) populations in the bone marrow cells.

21 nd 3) mixtures of 4T1 tumor cells and murine bone marrow cells.

22 performed whole-genome ChIP-Seq using mouse bone marrow cells.

23 es diminished the pro-metastatic behavior of bone marrow cells.

24 wild-type but not HIF-1alpha(fl/fl) LysM-Cre bone marrow cells.

25 production by Rag2(-/-)gamma(c)(-/-) myeloid bone marrow cells.

26 sis or growth arrest in normal MCs or normal bone marrow cells.

27 OCs, in primary cultures of RANKL-stimulated bone marrow cells.

28 controls inflammation via its expression in bone marrow cells.

29 myeloid leukemia (AML) compared with normal bone marrow cells.

30 meric mice that were reconstituted with GFP+ bone marrow cells.

31 mice receiving Sox4 virus-infected wild-type bone marrow cells.

32 clusion IRE can induce nanopore formation in bone marrow cells.

33 tion of myeloablated adult mice similarly to bone marrow cells.

34 d a growth advantage in the presence of host bone marrow cells.

35 chemotherapy-resistant MM, making up 90% of bone marrow cells.

36 arked mature peritoneal B-1a cells and adult bone marrow cells.

37 ted pit formation caused by RANKL-stimulated bone marrow cells.

38 production in MDSCs when compared to control bone marrow cells.

39 locus yeast artificial chromosome (beta-YAC) bone marrow cells.

40 on, thus promoting replenishment of depleted bone marrow cells.

41 /2 in mouse lineage marker negative (Lin(-)) bone marrow cells.

42 h Ldlr(-/-) mice transplanted with wild-type bone marrow cells.

43 e marrow proviral load 14-fold, and infected bone marrow cells 7-fold, but no inhibition was observed

44 Purpose To assess for nanopore formation in bone marrow cells after irreversible electroporation (IR

45 After elimination of pre-osteoclasts from bone marrow cells, alendronate did not affect osteoblast

46 The immense plasticity of bone marrow cells allows them to populate diverse tissue

47 experiments using FLVCR-deleted and control bone marrow cells, along with wild-type competitor cells

48 We studied primary MDS-enriched bone marrow cells and bone marrow sections by western bl

49 AF6 is expressed in the cytoplasm of healthy bone marrow cells and controls rat sarcoma viral oncogen

50 n, plasma cells in popliteal lymphoid nodes, bone marrow cells and granulocyte-macrophage progenitor

51 her SCL expression upregulates Kit in normal bone marrow cells and increases chimerism after bone mar

52 Ectopic expression of C/EBPalpha in mouse bone marrow cells and monocyte/macrophage cells, in the

53 Bone marrow cells and mouse embryonic fibroblasts derive

54 Despite decreases in ROS generation in bone marrow cells and p47(phox)-Nox2 signaling in osteob

55 omorphometry, and characteristics of primary bone marrow cells and preosteoblasts.

56 g misoprostol decreased M-CSFR expression in bone marrow cells and reduced the number of peritoneal m

57 PI3 kinase/Akt or Raf/MEK/ERK activation in bone marrow cells and transplanted them into recipient m

58 receptor family, is expressed in myeloid and bone marrow cells and was implicated as a checkpoint reg

59 CCR5-/- mice reconstituted with wild-type bone marrow cells and wild-type mice reconstituted with

60 Bone marrow cells and, alternatively, fetal liver cells

61 itors, increased long-term reconstitution of bone marrow cells, and a chronic myeloproliferative neop

62 e most abundant PGE(2) receptor expressed by bone marrow cells, and its expression further increased

63 cells represent a relatively small subset of bone marrow cells, and need to be enriched prior to anal

64 Here, we tested the hypothesis that bone marrow cell APOE genotype modulates pathological pr

65 romal-interacting molecule 1 (STIM1) gene in bone marrow cells are more susceptible to bacterial infe

66 When WT beta-YAC bone marrow cells are treated with the OGA inhibitor Thi

67 ke cell line, designated DC9, from Irf8(-/-) bone marrow cells as a model for DC development and func

68 5RA(-)CD123(-)CD71(+)CD41a(-)CD105(-)CD36(-) bone marrow cells as EEP giving rise to BFU-E, and Lin(-

69 clastogenesis assays using RAW264.7 cells or bone marrow cells as osteoclast precursors, addition of

70 Mechanistic studies define bone marrow cells as the source of the IL-10 signal and

71 e in telomere length in peripheral blood and bone marrow cells, as well as improved blood counts.

72 kemia-associated mutations in at least 5% of bone marrow cells at remission.

73 living donor or a deceased donor with frozen bone marrow cells available.

74 Clinical trials of bone marrow cell-based therapies after acute myocardial

75 A major obstacle to using bone marrow cell-based therapies for ischemic cardiovasc

76 d chemotherapy-resistant MM making up 80% of bone marrow cells before treatment.

77 These subsets also play a role in bone marrow cell (BMC) allograft rejection.

78 beta/Fc, or IL-2/Fc would enhance allogeneic bone marrow cell (BMC) engraftment and promote tolerance

79 ding the uncertainties about the outcomes of bone marrow cell (BMC) therapy for heart repair, further

80 In various organs, bone marrow cell (BMC) therapy has shown promising preli

81 nthusiasm, the therapeutic benefits of adult bone marrow cell (BMC) transplantation in patients with

82 Bone marrow cell (BMC)-based treatment for critical limb

83 -old 129SvJ mice and reconstituted them with bone marrow cells (BMC) from either 8-wk-old (young-to-o

84 asis, we found that a specific population of bone marrow cells (BMC) upregulated CSF-1R and secreted

85 monary CD11c+ cells and from GM-CSF-cultured bone marrow cells (BMCs(GM-CSF)).

86 CXCR4-positive bone marrow cells (BMCs) are critically involved in card

87 immunological tolerance using high doses of bone marrow cells (BMCs) has been demonstrated in mixed

88 n mice transplanted with Gfi-1(-/-);Id2(+/-) bone marrow cells (BMCs) in comparison with Gfi-1(-/-) B

89 c, effect of DG172 on the differentiation of bone marrow cells (BMCs).

90 RANTES production by Rag2(-/-)gamma(c)(-/-) bone marrow cells, but the presence of gamma(c) did not

91 rofiles for macrophages, derived from murine bone marrow cells by each CSF.

92 Transplantation of MDSCs, generated from bone marrow cells by granulocyte-macrophage colony-stimu

93 otent stem cells such as C3H10T1/2 and human bone marrow cells can be committed to the osteogenic lin

94 osteoclastogenesis of C/EBPalpha(-/-) mouse bone marrow cells can be rescued by c-fos overexpression

95 estrogen-regulated form of Hoxb8 into mouse bone marrow cells can be used along with Flt3 ligand to

96 Previous studies have shown that mouse bone marrow cells can produce mast cells when stimulated

97 he demonstration that intravenously injected bone marrow cells can rescue irradiated mice from lethal

98 CCL2 needs to be expressed by bone marrow cells, circulating monocytes, and injured mu

99 nced differentiation into osteoclasts of the bone marrow cells co-cultured with galectin-8-treated os

100 tricted elimination of 5-HT(2B) receptors on bone marrow cells confers a complete resistance.

101 increased cytokine expression in mesenchymal bone marrow cells contributes to the inflammatory phenot

102 ed (i) a reduced spleen weight; (ii) reduced bone marrow cell counts and proliferation (BrdUrd flow c

103 In the bone marrow cell culture, the conditioned medium from ML

104 CD11b(+)GR1(+) MDSC derived from mouse bone marrow cells cultured in the presence of MT-RET-1 m

105 aP and TCDD enhanced osteoclast formation in bone marrow cell cultures and gavage with BaP stimulated

106 Studies using ex vivo bone marrow cell cultures showed that TSH inhibits and s

107 acrophage maturation was studied in vitro in bone marrow cell cultures, and in vivo in a model of per

108 mice in the aorta, draining lymph nodes, and bone marrow cell cultures, indicating that IRF5 maintain

109 In ex vivo bone marrow cell cultures, SCI increased the number of o

110 ene imaging in mice with tMCAO revealed that bone marrow cell cycling peaked 4 days after stroke (P<0

111 ylaxis, and 1.5 x 10(7) haploidentical donor bone marrow cells (day 0).

112 RF-4 also functions as a tumor suppressor in bone marrow cells deficient in MyD88, an IRF-4-interacti

113 reported that ApoeKO mice transplanted with bone marrow cells deficient in the Transient Receptor Po

114 chimeric mice implanted with P2X7R-deficient bone marrow cells, defining hematopoietic cells as a suf

115 cells as well as the use of Hsf1 null mouse bone marrow cells demonstrated that 17-AAG-enhanced oste

116 F and receptor activator of NF-kappaB ligand bone marrow cells derived from BLT1(+/+) and/or BLT1(-/-

117 abilities of FLT3 mutant transduced primary bone marrow cells derived from FL(-/-) mice.

118 press PML-RARA, RUNX1-RUNX1T1, or MLL-AF9 in bone marrow cells derived from WT or DNMT3A-deficient mi

119 Treatment with specific bone marrow cell-derived secreted proteins may provide a

120 proresolution mediator resolvin E1 (RvE1) on bone-marrow-cell-derived osteoclasts in an in vitro muri

121 imately 95% of animals receiving Nras G12D/+ bone marrow cells develop chronic myelomonocytic leukemi

122 recipients transplanted with Nras G12D/G12D bone marrow cells develop TALL (TALL-homo).

123 stricted expression of 5-HT(2B) receptors in bone marrow cells developed hypoxia or monocrotaline-ind

124 -type mice reconstituted with IL-6-deficient bone marrow cells developed significantly fewer behavior

125 stitution of CD47(-)/(-) mice with wild-type bone marrow cells did not restore leukocyte recruitment

126 use IM insensitivity, because primary murine bone marrow cells engineered to express low amounts of B

127 terozygous inactivation of Nf1 (Nf1(+/-)) in bone marrow cells enhances neointima formation following

128 drogen peroxide-induced DNA-strand breaks in bone marrow cells enriched for HSCs.

129 dlr(-/-)) mice transplanted with Sptlc2(+/-) bone marrow cells exhibited significantly fewer atherosc

130 ne methylation patterns occurred in cultured bone marrow cells exposed to mammary tumor-conditioned c

131 We have found that the adoptive transfer of bone marrow cells expressing constitutively active CARMA

132 tmentin vitro In this study, we transplanted bone marrow cells expressing green fluorescent protein,

133 d poorly in wild-type (WT) recipients and KO bone marrow cells failed to radioprotect lethally irradi

134 Cultivation of pig bone marrow cells for 5-7 d in presence of human rCSF-1

135 caerulein injections and isolated acinar and bone marrow cells for ex vivo studies.

136 usly indicating the therapeutic potential of bone marrow cells for these patients.

137 CD19(low) bone marrow cells from 3H9;RS(-/-) mice were enriched in

138 HT7(-/-) mice and in mice reconstituted with bone marrow cells from 5-HT7(-/-) mice compared with con

139 Using a leukemic cell line and diagnostic bone marrow cells from 6 patients with B-progenitor cell

140 Towards this end, bone marrow cells from Axl(-/-) or wild-type mice were t

141 ice were irradiated and given transplants of bone marrow cells from C57BL6 mice, with or without the

142 Chimera mice reconstituted with bone marrow cells from CD11c-diphtheria toxin receptor (

143 Bone marrow cells from CLP-treated mice had normal OC pr

144 ctively inhibits colony formation of CD34(+) bone marrow cells from CML patients.

145 veloping in chimeric mice reconstituted with bone marrow cells from COL-EGFP mice very rarely showed

146 In addition, nonadherent bone marrow cells from Cx37(-/-) mice exhibit higher lev

147 We transplanted bone marrow cells from Fx-/- or control mice (Ly5.2) int

148 When mice, transplanted with bone marrow cells from green fluorescent protein (GFP) t

149 Analysis of blood and bone marrow cells from healthy and eosinophilic donors a

150 ansplant experiments in which we transferred bone marrow cells from Id cDKO mice into lethally irradi

151 es were not altered by the reconstitution of bone marrow cells from IDO(+/+) mice.

152 Furthermore, transplantation of bone marrow cells from integrin-beta3 KO mice into WT mi

153 17F mutation in hematopoiesis, we transduced bone marrow cells from Jak2(V617F) knockin mice with len

154 nimals is reconstituted with EGFP-expressing bone marrow cells from matched TgActb(EGFP) donors.

155 nd and a higher level of PARP3 expression in bone marrow cells from mice as compared with rats and hu

156 y in multiple myeloma cell lines or CD138(+) bone marrow cells from multiple myeloma patients comprom

157 -exposed mice promoted eosinophilopoiesis in bone marrow cells from naive mice, which was blocked by

158 myeloid leukemia (AML) compared with CD34(+) bone marrow cells from normal donors.

159 An analysis of bone marrow cells from patients and mice with X-CGD reve

160 formed a bioinformatic secretome analysis in bone marrow cells from patients with acute MI and discov

161 We found that bone marrow cells from patients with MM secreted higher

162 d in multiple myeloma cell lines and primary bone marrow cells from patients.

163 Bone marrow cells from preleukemic CD19-CreDeltaPB mice

164 Competitive transplantation of bone marrow cells from PTPsigma-deficient mice revealed

165 IL-33 cultures of bone marrow cells from Rag1 KO and Kit(W-sh/W-sh) mice a

166 Using bone marrow cells from Rc3h1(gt/gt) mice transferred int

167 Bone marrow cells from the cured mice displayed normal c

168 Moreover, isolated bone marrow cells from the sclerostin null mice show imp

169 decreased PTGER2 gene expression is valid in bone marrow cells from translocation t(7;12) positive pa

170 In this study, we report that bone marrow cells from wild-type C57BL/6 mice responded

171 Transfusion of c-kit(+) bone marrow cells from WT mice also restored endothelial

172 and microarray analysis in lineage-negative bone marrow cells from young and aged wild-type and apol

173 Dectin-1-dependent manner in GM-CSF-derived bone marrow cells (GM-BM).

174 ce reconstituted with MTMR9 shRNA-transduced bone marrow cells had an elevated proportion of T-box tr

175 that AKT3(-/-) mice receiving AKT3-deficient bone marrow cells had elevated clinical scores relative

176 Fx-/- mice receiving control bone marrow cells had intestinal inflammation and dyspla

177 irradiated wild-type mice reconstituted with bone marrow cells harvested from 5-HT7(-/-) mice.

178 in vivo labeling studies, where normal mouse bone marrow cells (i.e. high turnover) were evaluated po

179 In contrast, JAM-A deficiency in bone marrow cells impeded monocyte de-adhesion, thereby

180 he bcl-2/IgH translocation was determined on bone marrow cells in a centralized laboratory belonging

181 kemia-associated mutations in at least 5% of bone marrow cells in day 30 remission samples was associ

182 ed from RasGRP4-null mice by culturing their bone marrow cells in IL-3-enriched conditioned medium.

183 Nearly all the bone marrow cells in patients with myelodysplastic syndr

184 ent death of intestinal epithelial cells and bone marrow cells in response to double-strand DNA break

185 The contribution of bone marrow cells in the IGFBP-3-mediated response was e

186 stimulation increased IL-1beta expression in bone marrow cells in wild-type, Tie2Cre-IL-1R1(r/r), and

187 t the conditional abrogation of IRE1alpha in bone marrow cells increases bone mass as the result of d

188 es eosinophil differentiation of low-density bone marrow cells independent of further IL-5 stimulatio

189 ow bone mass phenotype, we demonstrated that bone marrow cell-induced osteoclastogenesis was reduced

190 uates the effect of repeated intramyocardial bone marrow cell injection in patients with residual or

191 Repeated bone marrow cell injection in previously responding pati

192 Intramyocardial bone marrow cell injection is associated with improvemen

193 Reconstitution of bone marrow cells into a TGF-alpha transgenic mouse mode

194 3(+/-); Apc(del/+); or Egr1(+/-), Apc(del/+) bone marrow cells into lethally irradiated Apc(del/+) re

195 nsdifferentiated human myeloid lineage CD34+ bone marrow cells into neural progenitors.

196 Transplantation of AEBP1(TG) bone marrow cells into non-transgenic (AEBP1(NT)) mice r

197 nsfer of Cx3cr1-proficient monocyte-enriched bone marrow cells into septic Cx3cr1-depleted mice preve

198 ction and perfusion with direct injection of bone marrow cells into the hearts of patients with ische

199 ued by adoptive transfer of affected HSCs or bone marrow cells into Treg-competent recipients.

200 Transplantation of Lrp5 null bone marrow cells into wild-type mice did not limit fibr

201 at the regulation of Bv8 expression in human bone marrow cells is also Stat3 signaling-dependent.

202 strated that CXCR3 and A(2B)AR expression on bone marrow cells is required for the antitumor effects

203 cated that blocking inflammasome activity in bone marrow cells is sufficient to improve healing.

204 Bone marrow cells isolated based solely on Fgd5 reporter

205 It is noteworthy that transplantation of bone marrow cells isolated from Akt2(-/-) mice to Ldlr(-

206 However, even when 95% of the donor bone marrow cells lacked FLVCR, all red cells in recipie

207 rs and that of inflammatory macrophages from bone marrow cells leads to macrophage heterogeneity.

208 Here, we show that non-(plastic)-adherent bone marrow cells (NABMCs) are more potent osteoprogenit

209 of soluble intracellular contents from whole bone marrow cells, named "Bone Marrow (BM) Soup", was re

210 howed negligible cytotoxic effects in normal bone marrow cells not expressing activated STAT5 protein

211 RANTES production by Rag2(-/-)gamma(c)(-/-) bone marrow cells occurred independently of the IL-15/IL

212 ecific PAP-iPSCs were generated from CD34(+) bone marrow cells of a CSF2RA-deficient patient with PAP

213 nificantly enhanced STAT3 phosphorylation in bone marrow cells of animals challenged with S. pneumoni

214 The bone marrow cells of Dnmt3a+/- mice had a subtle but sta

215 CLP did not promote OC formation from bone marrow cells of Itch-/- mice in vitro nor induce bo

216 ing and cause leukemia when expressed in the bone marrow cells of mice.

217 terminal erythropoiesis of lineage-negative bone marrow cells of Thra1 (PV/+) mice was rescued by th

218 lectron microscopy demonstrated nanopores in bone marrow cells only after IRE (P , .01).

219 mals in which iNOS or PARP1 was deleted from bone marrow cells only.

220 er obtained from in vitro differentiation of bone marrow cells or isolated from infarcted hearts alte

221 entiation without general toxicity to normal bone marrow cells or non-MLL cells.

222 -deficient mice reconstituted with wild-type bone marrow cells or wild-type mice reconstituted with I

223 physiological patterns of hematopoiesis and bone marrow cell outputs depend on the expression of ACK

224 The majority of WT recipients of Id cDKO bone marrow cells phenocopied Id cDKO cardiac fibrosis 4

225 1(+)c-Kit(-)CD25(+) (LSK(-)CD25(+)) cells, a bone marrow cell population of previously unknown functi

226 ential for the differentiation of alphaLP, a bone marrow cell population that gives rise to all known

227 the course of mast cell differentiation from bone marrow cells, procaspase-3 was present in cells of

228 Transplantation of Hb Null-derived bone marrow cells provides short-term radioprotection of

229 mice transplanted with Nras(G12D/+)p53(-/-) bone marrow cells rapidly develop a highly penetrant AML

230 xperiments indicated that CRAMP derived from bone marrow cells rather than structural cells was respo

231 Genetic deficiency of TF in bone marrow cells reduced coagulation in hypercholestero

232 NKL/M-CSF treatment of nonadherent Cx37(-/-) bone marrow cells rendered a 5-fold lower level of osteo

233 Transplanting KO mice with wild-type bone marrow cells rescued the angiogenic defect and amel

234 9-Tert treatment after telomere attrition in bone marrow cells rescues aplastic anemia and mouse surv

235 ic day 18.5 fetal liver and adult spleen and bone marrow cells, respectively.

236 ent feedback mechanism through which myeloid bone marrow cells restore quiescence of myeloid-biased H

237 nstitution of CEACAM1-deficient mice with WT bone marrow cells restored tumor infiltration of Gr1(+)C

238 IL-5 stimulation of low-density bone marrow cells resulted in expression of a panel of c

239 Sox4 transduction of mouse bone marrow cells results in increased expression of CRE

240 Induced deletion of P38alpha in bone marrow cells retards the maturation of mast cells i

241 ut the presence of gamma(c) did not increase bone marrow cell sensitivity to IL-15.

242 lantation experiments indicated that A(2b)AR bone marrow cell signals alone were not sufficient to el

243 he murine context, silencing of AID in human bone marrow cells skews differentiation toward myelomono

244 and PU.1 to the kappaE3' enhancer in primary bone marrow cells, STAT5 and PU.1 retrovirally transduce

245 nstituted with Adora2a(+/+) and Adora2a(-/-) bone marrow cells suggest that decreased IL-7Ralpha in n

246 xosome production, transfer and education of bone marrow cells supports tumor growth and metastasis,

247 Pig BMDM could be generated from bone marrow cells that had been stored frozen and thawed

248 cells give rise to megakaryocytes, the giant bone marrow cells that in turn break down to form blood

249 ticated interaction network between multiple bone marrow cells that regulate different hematopoietic

250 rine vomeronasal sensory neurons but also in bone marrow cells, the primary source for immune cell re

251 signals enhance the homing and attachment of bone marrow cells to bone surfaces and the commitment to

252 suppressed Flt3-L-induced differentiation of bone marrow cells to pDCs in wild-type but not HIF-1alph

253 r, the capacity of genetically B2R-deficient bone marrow cells to promote endothelial repair in vivo

254 TBI activates p53 to decrease the ability of bone marrow cells to suppress lymphoma development throu

255 strated significantly greater recruitment of bone marrow cells to the ischemic myocardium in the doub

256 IMS: There is growing interest in the use of bone marrow cells to treat liver fibrosis, however, litt

257 +)c-kit(+) cells from mice transplanted with bone marrow cells transduced with a MSCV-HOXB4-ires-YFP

258 In mouse bone marrow cells transduced with MLL-AF9, we show that

259 ation of one such peptidomimetic, MM-102, in bone marrow cells transduced with MLL1-AF9 fusion constr

260 fted lethally irradiated recipient mice with bone marrow cells transduced with retroviruses expressin

261 ronchoalveolar lavage fluid cells, inhibited bone marrow cell transendothelial migration, and inhibit

262 initiate leukemia, mice were transduced with bone marrow cells transfected with a Prdm14 expression v

263 tor knockout mouse model in combination with bone marrow cell transplantation, MRI, and neurocognitiv

264 s the development of both pDCs and cDCs from bone marrow cells under flt3L stimulation, by acting on

265 l for the generation of methylomes from rare bone marrow cells using 160-300 ng of starting DNA.

266 CD8alpha(+)-equivalent cDCs from Nfil3(-/-) bone marrow cells was also impaired.

267 asophil differentiation from SHP-1 deficient bone marrow cells was significantly reduced.

268 on of oncogenic Kras(G12V) into p16/p19(-/-) bone marrow cells, we generated transplantable leukemia-

269 After 24 hours, whole bone marrow cells were analyzed in vitro: 1) colony-form

270 Approximately 85% of bone marrow cells were clonal in the myelodysplastic-syn

271 Splenocytes and bone marrow cells were exposed to nicotine ex vivo, and

272 peripheral blood mononuclear cells or mouse bone marrow cells were incubated with Toll-like receptor

273 In addition, donor bone marrow cells were infused on days 4 and 11 after tr

274 Bone marrow cells were isolated from wild-type (WT) or i

275 Approximately 0.3% of bone marrow cells were LepR(+), 10% of which were CFU-Fs

276 nd wild-type mice reconstituted with CCR5-/- bone marrow cells were protected against PH, suggesting

277 Mice lacking S1P3 on bone marrow cells were protected from IRI, and S1P3-defi

278 CD133-sorted HPCs and CD133-depleted bone marrow cells were purified from bone marrow specime

279 Both UT-7 cells and primary murine bone marrow cells were studied.

280 A total of 15 x 10(6) BALB/c bone marrow cells were transplanted after varying doses

281 In the present study, bone marrow cells were transplanted into P20 mice that e

282 In this study, wild-type (WT) bone marrow cells were transplanted into two lethally ir

283 MC3T3-E1 osteoblastic cells and bone marrow cells were used to verify if mechanical forc

284 nd toward greater hypomethylation in CD34(+) bone marrow cells when decitabine was delivered by a sho

285 We observe a more complex picture in primary bone marrow cells, where EVI1 suppresses Cebpa in stem c

286 n is required for Bv8 up-regulation in mouse bone marrow cells, whereas other Stat family members and

287 vivo assay, designed for peripheral blood or bone marrow cells, which can produce a clinical result w

288 marrow-derived mast cells (BMMCs) and mouse bone marrow cells, which contain hematopoietic progenito

289 ats induced osteoclast formation from normal bone marrow cells, which was attenuated by a TNF-alpha-n

290 id cell lineage displayed a dysregulation of bone marrow cells with a rapid decline in population at

291 at the transduction of CREB transgenic mouse bone marrow cells with a Sox4 retrovirus increases survi

292 ular similarities between megakaryocytes and bone marrow cells with an HSC cell-surface phenotype rem

293 We found that coculture of murine bone marrow cells with bladder tumor cells promoted stro

294 the frequency of MDSCs generated from mouse bone marrow cells with GM-CSF and IL-6 in vitro.

295 In vitro culture of mouse bone marrow cells with GM-CSF is a well-established meth

296 Finally, in vitro treatment of bone marrow cells with IL-33, but not IL-5, led to speci

297 Stimulation of cultured bone marrow cells with ligands to Toll-like receptor 2 (

298 recent observation of fusion of circulating bone marrow cells with, in particular, cerebellar Purkin

299 signature is robust and may be applicable to bone marrow cells without the need to isolate CD34+ cell

300 We characterized fluorescent bone marrow cells (YFP(+) BMCs) in the thy1-YFP mouse an