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1 ology from embryonic life to fetal and adult bone marrow stem cells.
2 ral vector-mediated DAF gene transduction of bone marrow stem cells.
3 xpressed by ES cells and by adult neural and bone marrow stem cells.
4 the ischemically injured tubule derive from bone marrow stem cells.
5 ecretion in cultures of MM cells adherent to bone marrow stem cells.
6 d for the vascular regenerative potential of bone marrow stem cells.
7 ere expressed abundantly in the 5-FU-treated bone marrow stem cells.
8 aft with MHC-mismatched fetal liver or adult bone marrow stem cells.
9 iding a target for improving the function of bone marrow stem cells and their clinical utility in age
10 HIP is expressed in ES cells and in enriched bone marrow stem cells, and may be controlled by a promo
11 s, which may arise either locally or through bone marrow stem cells, and regulation of vascular endot
12 cribed in which unmobilized, highly purified bone marrow stem cells are transduced with a minimum amo
13 Mesenchymal stem cells (MSC) derived from bone marrow stem cells (BMSC) and adipose tissue stem ce
14 e comparative hepatogenic potential of human bone marrow stem cells (BMSC) with stem cells derived fr
17 that retroviral-mediated gene correction of bone marrow stem cells can lead to the development of no
18 that only the most primitive pluripotential bone marrow stem cells can support prolonged hematopoies
19 f this study was to test the hypothesis that bone marrow stem cells (CD34+) will improve histological
20 ble approach for corrective gene transfer to bone marrow stem cells, CD34+ cells were isolated from n
21 rovirus containing the Hmgb3 cDNA into mouse bone marrow stem cells demonstrated that enforced expres
27 pient mice with retrovirus-infected purified bone marrow stem cells from CD28(-/)- or wild-type donor
31 odologies for introducing foreign genes into bone marrow stem cells have prompted several groups to t
33 ral-mediated gene transfer (RMGT) into X-CGD bone marrow stem cells in preventing this abnormal infla
35 transfer of lineage-negative IL-13Ralpha1(+) bone marrow stem cells into IL-13Ralpha1-deficient mice
37 of these receptors, 5-HT(2B), is involved in bone marrow stem cell mobilization that participates in
38 n et al. demonstrate in mice that endogenous bone marrow stem cell mobilization, produced by a pharma
40 ogenic and hemopoietic colonies derived from bone marrow stem cells of aged and young adult male Spra
41 stic leukemia/lymphoma when expressed in the bone marrow stem cells of mice, the known involvement of
43 insic deficiency in the NZB Lin-Sca-1+c-kit+ bone marrow stem cell pool to differentiate into T cells
44 concentrations that inhibit committed human bone marrow stem cell proliferation, that is, granulocyt
46 tromal cells for growth, but, in contrast to bone marrow stem cells, required no additional growth fa
49 splant and higher APACHE III scores, but not bone marrow stem cell source, were associated with incre
53 e mechanisms that govern the capacity of the bone marrow stem cells to generate cardiac myocytes are
54 tants may also be valuable for expression in bone marrow stem cells to reduce myelosuppression brough
55 obilization, propagation, and/or delivery of bone marrow stem cells to the kidney hold potential as e
56 owing autologous transplantation of enriched bone marrow stem cells transduced with a retrovirus vect
58 e diagnosed with MM and underwent autologous bone marrow stem cell transplantation (BMT) were evaluat
59 , such as modulation of the immune system by bone marrow stem cell transplantation, pertinent to cond
60 the role of high-dose therapy and autologous bone marrow/stem cell transplantation (ASCT) during firs
62 ation and reduced expression of DOCK4 in MDS bone marrow stem cells was observed in two large indepen
65 differentiation of Gr1(+)CD11b(+) MDSC from bone marrow stem cells, whereas receptor antagonists blo
67 block the generation of dendritic cells from bone marrow stem cells, without affecting the generation
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