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1 , dermatologic conditions, or solid-organ or bone marrow transplantation.
2 sident beds that could not be transferred by bone marrow transplantation.
3 prevention of GVHD in preclinical models of bone marrow transplantation.
4 ant in many clinical applications, including bone marrow transplantation.
5 erapeutic responses of an NPC2 patient after bone marrow transplantation.
6 oughout life and are the functional units of bone marrow transplantation.
7 duals with diarrhea after they had undergone bone marrow transplantation.
8 en gained adaptive immunity after undergoing bone marrow transplantation.
9 gimens to improve the safety and efficacy of bone marrow transplantation.
10 e repopulated to a normal level by syngeneic bone marrow transplantation.
11 is a critical complication after allogeneic bone marrow transplantation.
12 iTregs) for the induction of tolerance after bone marrow transplantation.
13 GVHD) is the main complication of allogeneic bone marrow transplantation.
14 sease following sex-mismatched HLA-identical bone marrow transplantation.
15 reconstitution of hematopoiesis upon serial bone marrow transplantation.
16 as partially reduced and then recovered upon bone marrow transplantation.
17 r restrict alloreactivity after experimental bone marrow transplantation.
18 lls were severely compromised in competitive bone marrow transplantation.
19 Haploidentical, unmanipulated, G-CSF-primed bone marrow transplantation.
20 ecific P2X(7)-deficient animals generated by bone marrow transplantation.
21 rrected the T cell lymphopenia in mice after bone marrow transplantation.
22 er remission, and requirement for allogeneic bone marrow transplantation.
23 scence protein transgenic mice were used for bone marrow transplantation.
24 n this patient, who had undergone successful bone marrow transplantation.
25 r T cells after allogeneic but not syngeneic bone marrow transplantation.
26 (GVHD) is a major complication of allogeneic bone marrow transplantation.
27 HC class I-restricted T-cell responses after bone marrow transplantation.
28 the conditioning regimen, and declined after bone marrow transplantation.
29 th lentivirus expressing Hmga2 and performed bone marrow transplantation.
30 only, both, or neither were generated using bone marrow transplantation.
31 cally relevant murine models of experimental bone marrow transplantation.
32 smatched) and fully MHC-mismatched models of bone marrow transplantation.
33 regenerative capacity of HSCs in competitive bone marrow transplantation.
34 ophic epidermolysis bullosa after allogeneic bone marrow transplantation.
35 ally applied in clinical conditions, such as bone marrow transplantation.
36 to treat graft-versus-host disease following bone marrow transplantation.
37 eased resistance to MRSA was transferable by bone marrow transplantation.
38 ythroid short-term radioprotection following bone marrow transplantation.
39 changes during stress hematopoiesis, such as bone marrow transplantation.
40 ibuted to atherogenesis in a murine model of bone marrow transplantation.
41 requiring lifelong transfusion or allogeneic bone marrow transplantation.
42 f malignant cells, the therapeutic intent of bone marrow transplantation.
43 g recovery of the hematopoietic system after bone marrow transplantation.
44 ymphopenias and hinder T cell recovery after bone marrow transplantation.
45 ve oxygen species (ROS) following allogeneic bone marrow transplantation.
46 , both in physiological conditions and after bone marrow transplantation.
47 itical role of donor sleep in the success of bone marrow transplantation.
48 ponses in leukemia patients after allogeneic bone marrow transplantation.
49 her models, including whole-body irradiation/bone-marrow transplantation.
50 stion of long-term HSC function along serial bone marrow transplantations.
51 iology was 47.2% (95% CI, 34.3-59.1) and for bone marrow transplantation 22.8% (95% CI, 8.7-40.8).
53 (Tc1) or Tc17 cells combined with autologous bone marrow transplantation after total body irradiation
60 th a STAT6 inhibitor and IL-4(-/-)IL-13(-/-) bone marrow transplantation also protected against Schis
62 in vivo, we generated CD36 chimeric mice by bone marrow transplantation and evaluated the two models
63 an engraftment and the acquired tolerance of bone marrow transplantation and eventually clarified the
64 scle injury model combined with irradiation, bone marrow transplantation and in vivo imaging, we show
66 apeutic avenues, and some of them, including bone marrow transplantation and mesenchymal stem cell th
70 r human leukocyte antigen-matched kidney and bone marrow transplantation and the induction of mixed l
73 ents using pancreas-specific Perk knockouts, bone marrow transplantation, and cultured pancreatic isl
74 GvHD) is a common complication of allogeneic bone marrow transplantation, and has a major effect on t
75 be presented by the CD8(-) cDC subset after bone marrow transplantation, and inflammation during GVH
76 reatment, including chemotherapy, radiation, bone marrow transplantation, and newer modalities such a
77 also limits T lineage regeneration following bone marrow transplantation, and so contributes to the s
78 These data have implications for successful bone marrow transplantation, and suggest that tolerance
79 l irradiation, reconstitution with syngeneic bone marrow transplantation, and therapy with the immuno
80 ent of murine SGVHD, control and CsA-treated bone marrow transplantation animals were treated with br
82 alloreactive donor T cells after allogeneic bone marrow transplantation are limited by a concomitant
83 hieved in NOD mice receiving anti-CD154 with bone marrow transplantation as the means of tolerizing p
87 BO blood group mismatched solid organ and/or bone marrow transplantation between donor and recipient.
89 ostasis and thrombosis, we performed crossed bone marrow transplantations between C57BL/6J and Vwf(-/
90 in [ATG]) facilitates immune tolerance after bone marrow transplantation (BMT) across major histocomp
91 t donor-recipient immune tolerance following bone marrow transplantation (BMT) across MHC barriers vi
92 cause of late mortality following allogeneic bone marrow transplantation (BMT) and is characterized b
93 transplantation (VCA) with chimerism through bone marrow transplantation (BMT) are currently being pu
94 g hematopoietic stem cell transplantation or bone marrow transplantation (BMT) as therapy for various
96 ficantly improves survival (P < .0001) after bone marrow transplantation (BMT) by inhibiting the init
99 rineurial microenvironment using a series of bone marrow transplantation (BMT) experiments in transge
101 emia (ALL) persisting or relapsing following bone marrow transplantation (BMT) has a dismal prognosis
103 including alveolar macrophages (AMs), after bone marrow transplantation (BMT) have impaired host def
104 eir therapeutic potential following congenic bone marrow transplantation (BMT) in a proteoglycan-indu
106 ed virus (AAV)2/5-mediated gene therapy with bone marrow transplantation (BMT) in the INCL mouse.
107 nd to host alloantigens following allogeneic bone marrow transplantation (BMT) induce graft-versus-ho
109 curative potential of MHC-matched allogeneic bone marrow transplantation (BMT) is in part because of
112 However, simultaneous kidney or VCA and bone marrow transplantation (BMT) is problematic because
118 in murine and human recipients of allogeneic bone marrow transplantation (BMT) that intestinal inflam
119 vere and frequent complication of allogeneic bone marrow transplantation (BMT) that involves the gast
120 cGVHD) is a major complication of allogeneic bone marrow transplantation (BMT) the immunopathogenesis
121 n DNA-PKcs(3A/3A) mutant mice, which require bone marrow transplantation (BMT) to prevent early morta
123 nt study, chimeric mice were created through bone marrow transplantation (BMT) using wild-type and CX
125 recently achieved in the clinic by combining bone marrow transplantation (BMT) with kidney transplant
126 revents successful outcomes after allogeneic bone marrow transplantation (BMT), an effective therapy
127 the hematopoietic lineages to recover after bone marrow transplantation (BMT), but the reasons for t
128 is a major cause of mortality in allogeneic bone marrow transplantation (BMT), for which administrat
129 fe-threatening complication after allogeneic bone marrow transplantation (BMT), particularly in the p
148 rm-specific betaAR knockout (betaARKO) or WT bone-marrow transplantation (BMT) and after full reconst
150 invasive candidiasis in patients undergoing bone marrow transplantation but is not approved for use
151 lethal and morbid complication of allogeneic bone marrow transplantation, but GVHD is tightly linked
154 awa and Kabashima) address the issue whether bone marrow transplantation could be applied to patients
155 AB vector system when combined together with bone marrow transplantation could quickly knock down c-k
179 safely replaced with hydroxyurea therapy or bone marrow transplantation for a cohort of children wit
180 opical timolol for infantile hemangiomas and bone marrow transplantation for dystrophic epidermolysis
181 visable before commencing clinical trials of bone marrow transplantation for epidermolysis bullosa si
182 Specific tolerance after combined kidney and bone marrow transplantation for multiple myeloma with en
183 ylaxis for both acute and chronic GVHD after bone marrow transplantation from HLA-matched donors.
184 tantly, in vivo thrombosis experiments after bone marrow transplantation from platelet-specific ERK5
187 cardiac hypertrophy or dysfunction, whereas bone marrow transplantation from wild-type mice into mic
188 after recipients were given T cell-depleted bone marrow transplantations from major histocompatibili
190 r-deficient, LDLr(-/-) chimeras, obtained by bone marrow transplantation, had smaller but, paradoxica
191 atched, or HLA-haploidentical, related donor bone marrow transplantation (haploBMT) has seen a reviva
193 apy and immunomagnetically purged autologous bone marrow transplantation has been shown to improve ou
194 tment or cure for epidermolysis bullosa, but bone marrow transplantation has been suggested to improv
198 ower after UCB than after 8/8 allele-matched bone-marrow transplantation (HR 0.63, 0.44-0.90; p=0.01)
199 ents (HR 1.62, 95% CI 1.18-2.23; p=0.003) or bone-marrow transplantation (HR 1.69, 95% CI 1.19-2.39;
203 rvival and other outcomes for UCB, PBPC, and bone marrow transplantation in patients aged 16 years or
204 use of high-dose chemotherapy and autologous bone marrow transplantation in patients with malignant d
206 lfan-based chemotherapy regimen was used for bone marrow transplantation in wild-type mice before sub
207 uential exposure to chemotherapy, and serial bone marrow transplantation increased senescence in anim
208 e marrow cells and increases chimerism after bone marrow transplantation, indicating that Scl is also
209 ditional Stat3 knockout strain and performed bone marrow transplantations into lethally irradiated re
216 tress, such as during anticancer therapy and bone marrow transplantation, is of clinical significance
218 this devastating disorder, and suggest that bone marrow transplantation might offer a feasible thera
221 s demonstrate efficacy in a JAK2V617F murine bone marrow transplantation model, the effects of JAK2 i
224 ry human hematopoietic cells and in a murine bone marrow-transplantation model using lentivirally med
230 85beta resulted in increased mast cells, and bone marrow transplantation of cells overexpressing p85b
231 atopoietic cell intrinsic activity of Itfg2, bone marrow transplantation of Itfg2-deficient cells was
237 mprovements in conventional chemotherapy and bone marrow transplantation, overall survival remains po
238 with wild-type (WT) recipients of allogeneic bone marrow transplantation, P-selectin(-/-) recipients
242 ation allowed the patient to be referred for bone marrow transplantation, potentially curative for hi
245 nockdown in macrophages using transgenic and bone marrow transplantation procedures to blunt HFD-indu
246 us cells might interfere with the outcome of bone marrow transplantation, protocols usually include c
247 marrow-derived cells migrate to the skin of bone marrow transplantation recipient mice, but these ce
248 lly lose function following transfer to male bone marrow transplantation recipients, we have explored
256 er intracranial injections of AAV2/5-PPT1 or bone marrow transplantation, separately as well as in co
257 t3-deficient mice, was less prominent in the bone marrow transplantation setting, possibly by limitin
260 a 1:1 ratio to peripheral-blood stem-cell or bone marrow transplantation, stratified according to tra
261 iciency did not affect immune responses, and bone marrow transplantation studies also indicated that
270 colony-forming/replating assays and in vivo bone marrow transplantation studies, we show that forced
271 s required on cells that are not replaced by bone marrow transplantation, such as vascular endothelia
272 findings, which may be relevant to clinical bone marrow transplantation, suggest that neither exposu
273 e development in vivo, and after competitive bone marrow transplantation, there was a nearly complete
274 LL-fusion-mediated leukemogenesis in primary bone marrow transplantation through suppressing Hoxa9/Me
278 approaches to augment host B7-H3 early after bone marrow transplantation to prevent GVHD and to devel
280 der, and patients are treated primarily with bone marrow transplantation to restore hematopoietic fun
282 ne therapy, substrate reduction therapy, and bone marrow transplantation to target the primary pathog
283 alcification of C57BL/6 LDLr(-/-) mice using bone marrow transplantation to trace ROSA26-LacZ-labeled
284 during a period of immunocompromise after a bone marrow transplantation to treat hypodiploid leukemi
285 ws efficacy in mouse models of recovery from bone marrow transplantation, ulcerative colitis, and par
287 il to reconstitute following irradiation and bone marrow transplantation unless the mice also receive
288 g lung allograft tolerance with tandem donor bone marrow transplantation using a short-duration nonmy
290 re model was more equivocal, so experimental bone marrow transplantation was used to examine hematopo
293 Through the use of mouse tumor models and bone marrow transplantations, we show that platelet-deri
294 me polymerase chain reaction, and reciprocal bone marrow transplantation were used to evaluate the ef
296 ppression is most profound during GVHD after bone marrow transplantation where an inflammatory cytoki
297 y from myeloablative challenge and following bone marrow transplantation, whereas BCL-XL was dispensa
298 sease (GVHD) is a complication of allogeneic bone marrow transplantation whereby transplanted naive a
299 iency virus or AIDS, or prior solid organ or bone marrow transplantation with receipt of chronic immu
300 dy of human leukocyte antigen (HLA) -matched bone marrow transplantation would provide low rates of s
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