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1 genes but not for the activation of MAPKs in bone marrow-derived macrophage.
2 in either mouse primary cortical neurons or bone marrow derived macrophages.
3 liensis infection of BALB/c (wild-type [WT]) bone marrow derived macrophages.
4 wever expression is increased over 2-fold in bone marrow derived macrophages.
5 Typhimurium, in a comparable manner to mouse bone marrow derived macrophages.
6 cytokines, which is attenuated in Mrp14(-/-) bone marrow-derived macrophages.
7 ec kinases and LPS in primary peritoneal and bone marrow-derived macrophages.
8 10, and GM-CSF, was also markedly reduced in bone marrow-derived macrophages.
9 viral infection on inflammasome responses in bone marrow-derived macrophages.
10 in murine RAW 264.7 macrophages and primary bone marrow-derived macrophages.
11 rease of RANKL-induced osteoclastogenesis in bone marrow-derived macrophages.
12 n monocytes, murine RAW 264.7, and wild-type bone marrow-derived macrophages.
13 emphasis on proteolytic efficiency in murine bone marrow-derived macrophages.
14 ulus for interleukin (IL)-1beta secretion by bone marrow-derived macrophages.
15 ed in MHV-infected Ifit2(-/-) relative to wt bone marrow-derived macrophages.
16 also activating ERK MAPK pathways in murine bone marrow-derived macrophages.
17 D88-NF-kappaB-dependent mode of induction in bone marrow-derived macrophages.
18 ory cytokines nor inflammasome activation in bone marrow-derived macrophages.
19 as aeruginosa are known to activate NLRC4 in bone marrow-derived macrophages.
20 ugh the alteration of potassium transport in bone marrow-derived macrophages.
21 r expression in RAW-264.7 cells and in mouse bone marrow-derived macrophages.
22 in cultured murine macrophages (B6-MCL) and bone marrow-derived macrophages.
23 2-like inflammatory response from ABCA1(+/+) bone marrow-derived macrophages.
24 lia virus infection of murine cell lines and bone marrow-derived macrophages.
25 rapidly down-regulated upon LPS exposure in bone marrow-derived macrophages.
26 B7-2, but not B7-1, on the surface of mouse bone marrow-derived macrophages.
27 nflammatory cytokine production by Cyld(-/-) bone marrow-derived macrophages.
28 human monocytic cell line (THP-1) and mouse bone marrow-derived macrophages.
29 r lipopolysaccharide-induced cytotoxicity in bone marrow-derived macrophages.
30 ts intracellular cycle within primary murine bone marrow-derived macrophages.
31 were recapitulated by hyperglycemia in mouse bone marrow-derived macrophages.
32 high-power field) of TAMs were GFP-positive, bone marrow-derived macrophages.
33 inflammasome is attenuated in Nrf2-deficient bone marrow-derived macrophages.
34 poorly and induced rapid cell death in mouse bone marrow-derived macrophages.
35 phage cell line RAW264.7, but not in primary bone marrow-derived macrophages.
36 abolished in Toll-like receptor (TLR2)(-/-) bone marrow-derived macrophages.
37 uced mRNA and protein expression of IL-18 in bone marrow-derived macrophages.
38 ta and interleukin 6 messenger RNAs in mouse bone marrow-derived macrophages.
39 potently inhibited Leishmania replication in bone marrow-derived macrophages.
40 uced IL-1beta secretion and NO production in bone marrow-derived macrophages.
41 phage phenotype was absent in peritoneal and bone marrow-derived macrophages.
42 re supported by experiments with Ifitm3(-/-) bone marrow-derived macrophages.
43 optotic cells and are better phagocytes than bone marrow-derived macrophages.
44 man monocyte-derived macrophages, and murine bone marrow-derived macrophages.
45 duced chemokine secretion by neutrophils and bone marrow-derived macrophages.
46 s, but GM-CSF has also been used to generate bone marrow-derived macrophages.
47 39 significantly regulated 105 probe sets in bone marrow-derived macrophages.
48 on of IL-1beta by murine dendritic cells and bone-marrow derived macrophages.
49 l production of NPY was observed in cultured bone marrow derived macrophage and dendritic cells (DCs)
50 as LVS clpB grew similarly to LVS in primary bone marrow-derived macrophages and a variety of cell li
51 strongly impairs many effector functions of bone marrow-derived macrophages and bone marrow-derived
52 Whereas TLR9-induced TNF-alpha secretion of bone marrow-derived macrophages and conventional dendrit
53 ts lytic replication of gammaHV68 in primary bone marrow-derived macrophages and decreases transcript
54 also observed by in vitro experiments, using bone marrow-derived macrophages and dendritic cells as r
55 TARM1 expression was also upregulated by bone marrow-derived macrophages and dendritic cells foll
56 osis and internalization of CpG-ODN by mouse bone marrow-derived macrophages and directly interacts w
57 A-induced macrophage cell death with primary bone marrow-derived macrophages and high-fat diet-induce
59 wild-type and T2S mutant bacteria in murine bone marrow-derived macrophages and human U937 cells.
60 ion, infection experiments were performed in bone marrow-derived macrophages and in mice using threon
61 These strains were highly attenuated both in bone marrow-derived macrophages and in vivo compared wit
62 s isolated from Anxa2-deficient (Anxa2(-/-)) bone marrow-derived macrophages and lung parenchyma disp
66 transendothelial migration (TEM) activity of bone marrow-derived macrophages and Raw264.7 cells accor
68 TEM activity was decreased in Ninjurin1 KO bone marrow-derived macrophages and siNinj1 Raw264.7 cel
69 ted peritoneal macrophages) as compared with bone marrow-derived macrophages and the RAW264 cell line
71 function of anti-inflammatory intestinal and bone-marrow-derived macrophages and their ability to sec
72 tates its survival and replication in murine bone marrow-derived macrophages, and E. tarda infection
73 ity in vivo by tracking fluorescence-labeled bone marrow-derived macrophages, and found that tPA-defi
74 ly active, rescue replication of MHV(Mut) in bone marrow-derived macrophages, and inhibit RNase L-med
75 Murine macrophage-like cell line RAW264.7, bone marrow-derived macrophages, and primary peritoneal
76 more, after phagocytosis of myelin in vitro, bone marrow-derived macrophages are much more susceptibl
77 y lipoprotein (LDL)-mediated signaling using bone marrow-derived macrophage as well as an in vivo mod
78 uced increased migration of THP-1 and murine bone marrow-derived macrophages as well as activated mon
79 ymosan-induced peritonitis, M1- and M2a-like bone marrow derived macrophages, as well as by mesotheli
80 pro-inflammatory cytokines were increased in bone marrow derived macrophage (BMDM) from PLTP-/-, whil
81 ignificantly higher IL-6 secretion by murine bone marrow derived macrophages (BMDM) compared to cultu
83 d that treatment of RANKL-stimulated primary bone marrow-derived macrophage (BMDM) cultures with smal
84 d TBK1 activity was significantly reduced in bone marrow-derived macrophage (BMDM) from OPTN(D477N/D4
85 rom 129 mice is sufficient to enhance the B6 bone marrow-derived macrophage (BMDM) inflammasome respo
86 ulture of atrophied myotubes with or without bone marrow-derived macrophages (BMDM) and/or M-CSF reve
87 lations of wild-type mice with Nrp1-depleted bone marrow-derived macrophages (BMDM) confers resistanc
93 this study, we confirm that tolerized mouse bone marrow-derived macrophages (BMDM) selectively incre
94 of the eicosanoid metabolic network in mouse bone marrow-derived macrophages (BMDM) upon stimulation
96 e treated rat neonatal cardiomyocytes, mouse bone marrow-derived macrophages (BMDM), or mouse neutrop
102 3 or Pyrin inflammasome activation in murine bone marrow-derived macrophages (BMDMs) as an indicator
105 d NLRP3 was revealed when Nlrc4(S533A/S533A) bone marrow-derived macrophages (BMDMs) expressing phosp
107 aB pathway (IKKalpha/beta, NF-kappaB p65) in bone marrow-derived macrophages (BMDMs) from knockout mi
108 n of IFN regulatory factor 3 (IRF3), whereas bone marrow-derived macrophages (BMDMs) from mice carryi
110 human monocytic leukemia cell line THP-1 and bone marrow-derived macrophages (BMDMs) from wild-type a
111 Elavl1Mo KO), we show that HuR expression in bone marrow-derived macrophages (BMDMs) is needed to mai
116 and adoptive transfer studies revealed that bone marrow-derived macrophages (BMDMs) traffic to the l
117 S induced secretion of IL-6 and TNF-alpha by bone marrow-derived macrophages (BMDMs) was significantl
118 esser extent at Il1a) reaches high levels in bone marrow-derived macrophages (BMDMs), and the enhance
120 and is proteolytically processed in resting bone marrow-derived macrophages (BMDMs), dendritic cells
121 was transiently induced in LPS-activated WT bone marrow-derived macrophages (BMDMs), expression pers
124 fy H. capsulatum genes required for lysis of bone marrow-derived macrophages (BMDMs), we identified a
125 nstituted mouse bone marrow neutrophils, and bone marrow-derived macrophages (BMDMs), we showed that
132 7BL/6, and CD1 mice models and C57BL/6 mouse bone-marrow-derived macrophages (BMDMs) were used as inf
133 uction in lipopolysaccharide (LPS)-activated bone marrow derived-macrophages (BMM) was detected in 6-
134 gated Nur77 in macrophage polarization using bone marrow-derived macrophages (BMM) from wild-type and
135 ation of ns2 mutant viruses is attenuated in bone marrow-derived macrophages (BMM) generated from wil
138 RNase L activation and replicates poorly in bone marrow-derived macrophages (BMM), while ns2(H126R)
141 ession of CTMP is also observed increased in bone marrow-derived macrophages (BMMPhi) from DIO mice a
142 ining 3 (NLRP3) inflammasome in primed mouse bone marrow-derived macrophages (BMMPhi), inducing a rob
145 irus replication was attenuated in wild-type bone marrow-derived macrophages (BMMs) and partially res
146 in LPS (1 ng/ml)-stimulated wild-type murine bone marrow-derived macrophages (BMMs) but failed to do
147 tion of miR-155 expression in primary murine bone marrow-derived macrophages (BMMs) during H. pylori
149 lysis, the bacterial modulation of miRNAs in bone marrow-derived macrophages (BMMs) in which activity
150 r ligation were severely reduced in Hck(-/-) bone marrow-derived macrophages (BMMs) or in RAW/LR5 mac
155 in the RAW 264.7 macrophage line and primary bone marrow-derived macrophages but did not affect LXR-d
156 n rates and mROS expression in mock-infected bone marrow-derived macrophages but reduced caspase-depe
157 deficiency adversely affected the ability of bone marrow-derived macrophages, but not dendritic cells
158 L-1beta secretion was impaired in Pgam5(-/-) bone marrow-derived macrophages, but not in Ripk3(-/-) b
159 se activation in TRIF-deficient immortalized bone-marrow-derived macrophage, but was fully inhibitory
160 RP3-dependent interleukin-1beta secretion by bone marrow-derived macrophages by activating nuclear fa
161 her, overexpression of NAIP1 in immortalized bone marrow-derived macrophages by retroviral transducti
162 significantly higher by wild-type (C57BL/6) bone marrow-derived macrophages compared with TLR2-defic
164 hemozoin was purified and added to in vitro bone marrow-derived macrophage cultures concurrently exp
167 kout of ASC in lipopolysaccharide-stimulated bone marrow-derived macrophages depressed HMGB1 activity
168 d J20/A7(-/-) mice, whereas the capacity for bone marrow-derived macrophages derived from A7(-/-) mic
172 e secretion of mature IL-1beta from unprimed bone marrow-derived macrophages during a productive infe
173 PTEN in lipopolysaccharide-stimulated mouse bone marrow-derived macrophages enhanced beta-catenin ac
176 man monocyte-derived macrophages, and murine bone marrow-derived macrophages following infection with
177 a lower parasite intake, parasite burden in bone marrow-derived macrophages from AnxA1(-/-) mice was
178 the sifA mutant was sensitive to killing by bone marrow-derived macrophages from BALB/c.D2 mice and
180 per 1 cells or T-helper 2 cells, we obtained bone marrow-derived macrophages from both strains and in
183 duction was recapitulated ex vivo in primary bone marrow-derived macrophages from Dusp1-deficient mic
185 ry cytokines and pro-inflammatory enzymes in bone marrow-derived macrophages from LKB1 KO than those
187 d M2 macrophage activation were confirmed in bone marrow-derived macrophages from mice with the myelo
190 The effects of estrogen are long-lasting; bone marrow-derived macrophages from ovariectomized mice
193 oviruses expressing WT or mutant PSGL-1 into bone marrow-derived macrophages from PSGL-1-deficient mi
195 hagic markers were constitutively induced in bone marrow-derived macrophages from Sphk dKO mice.
196 the lack of TTP expression in LPS-stimulated bone marrow-derived macrophages from the mice, whereas f
200 iments were performed in naive and polarized bone marrow-derived macrophages from wild-type (WT) and
202 but complementary approaches: 1) we compared bone marrow-derived macrophages from wild-type and TIM4(
203 acellular survival of the trpB mutant within bone marrow-derived macrophages from wild-type but not I
204 hoglucose enhanced the in vitro migration of bone marrow-derived macrophages from WT but not KO mice,
205 elin phagocytosis in vitro by LPS stimulated bone-marrow-derived macrophages from IL-10-null mice fai
208 n of caspase-1 inhibitors or the infusion of bone marrow-derived macrophages genetically engineered t
209 NAIP1 is poorly expressed in resting mouse bone marrow-derived macrophages; however, priming with p
211 as used to examine gene regulation in murine bone marrow-derived macrophages in response to 90-mum-di
212 ammatory gene programs by Gsk3 inhibition in bone marrow-derived macrophages in response to TLR4 stim
213 by nephritic glomeruli ex vivo and cultured bone marrow-derived macrophages in vitro, suggesting add
214 y, we identify a signaling pathway in murine bone marrow-derived macrophages in which activation of T
215 osome damage was observed in infected murine bone marrow-derived macrophages, increased with time, an
218 LLO was able to infect and replicate within bone marrow-derived macrophages indistinguishably from t
221 d AIM2 inflammasome activation and following bone marrow-derived macrophage infection with herpes sim
224 ased cathepsin activity determined in StB KO bone marrow-derived macrophages is not essential for inf
226 e very low levels of endogenous Cav-1 and in bone marrow-derived macrophages isolated from Cav-1(-/-)
227 s are confirmed by comparing Kir currents in bone marrow-derived macrophages isolated from Cav-1(-/-)
229 d pro-inflammatory enzymes were monitored in bone marrow-derived macrophages isolated from myeloid ce
230 itro; the latter responses were abolished in bone marrow-derived macrophages isolated from Nlrp3(-/-)
232 uced by dsDNA and other microbial ligands in bone marrow-derived macrophages lacking p205 revealed th
234 rtin messenger RNA and protein expression in bone marrow-derived macrophages, liver, and spleen of mi
235 on of human blood monocyte-derived or murine bone marrow-derived macrophages (Mos) increases their AC
237 s disrupted in macrophages using siRNA or in bone marrow-derived macrophages obtained from C57BL/6J-A
238 is mRNA stabilization response was absent in bone marrow-derived macrophages obtained from conditiona
240 or stable inhibition of HMGB1 expression in bone marrow-derived macrophages or fibroblasts resulted
242 to measure the mRNA and miRNA expression in bone marrow-derived macrophages over a time-series of 8
250 ll surface, but diminished TNF production by bone marrow-derived macrophages relative to wild type.
251 ine and paracrine proinflammatory effects on bone marrow-derived macrophages, renal endothelial cells
252 oxide addition to CB3-infected NOD.Ncf1(m1J) bone marrow-derived macrophages rescued the inflammatory
253 se-1 and -11 activation determined in StB KO bone marrow-derived macrophages resulted in enhanced IL-
254 Mechanistic analysis of superoxide-deficient bone marrow-derived macrophages revealed a marked diminu
259 ss proinflammatory cytokines than ABCA1(-/-) bone marrow-derived macrophages, similar to alternativel
260 lity of these cells to condition co-cultured bone marrow-derived macrophages so that the macrophages
261 mokine gene expression was examined in mouse bone marrow-derived macrophages stimulated through vario
262 f IFN-beta expression was also identified in bone marrow-derived macrophages stimulated with B. burgd
263 ases TNF, IL-6, and IL-12 secretion in mouse bone marrow-derived macrophages stimulated with LPS.
264 ions of Rapamycin in LC3B and ATG5 deficient bone marrow derived macrophages, suggesting that non-aut
265 specific for Sn, since they are taken up by bone marrow derived macrophages that are derived from wi
266 a production and pyroptosis in primed murine bone marrow-derived macrophages that is mediated by the
268 n both RAW264.7 macrophage cells and primary bone marrow-derived macrophages, the production of IFN-b
270 dulated the functional phenotype ascribed to bone marrow-derived macrophages: the B6 allele promoted
271 alpha was induced by strain Wakulla in mouse bone marrow-derived macrophages; this expression was dep
273 at in vitro, CTLA4Ig synergizes with NO from bone marrow-derived macrophages to inhibit T cell prolif
275 Fc receptor-mediated functions, because WKY bone marrow-derived macrophages transduced with Fcgr3-rs
279 licited in an analogous fashion using LPS in bone marrow-derived macrophages upon inhibition of caspa
280 ressed in immune cells and is upregulated in bone marrow-derived macrophages upon stimulation with in
281 F-1alpha, acting through CXCR4, expressed on bone marrow-derived macrophages, upregulated MMP-9 and s
291 kers CD 11b, F4/80 in fetal liver cells, and bone marrow-derived macrophages were dependent on functi
292 mpared with TLR2-deficient or CD14-deficient bone marrow-derived macrophages when stimulated with cur
295 can be reproduced in vitro by stimulation of bone marrow-derived macrophages with conditioned media f
296 In the current study, in vitro treatment of bone marrow-derived macrophages with EDPs induced M1 mac
298 d 4E-BP phosphorylation after stimulation of bone marrow-derived macrophages with lipopolysaccharide
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