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1 ynthesis of type I collagen, the predominant bone matrix protein.
2 ding type I collagen, osteopontin, and other bone matrix proteins.
3 ssential cofactor for gamma-carboxylation of bone matrix proteins.
4 e phosphatase activity and the deposition of bone matrix proteins.
5 in and osteocalcin (osteoblast endochrondral bone matrix proteins), and proliferating cell nuclear an
9 associated with lysosomes in osteoclasts and bone matrix protein-containing vesicles in osteoblasts.
11 ion of the ruffled border in osteoclasts and bone matrix protein deposition in osteoblasts, without a
13 ERK and p38 inhibitors on the regulation of bone matrix protein expression and JunB and JunD levels
16 s of increased macrophages, PCNA levels, and bone matrix proteins in the aortic valve during experime
18 C-E1a was constructed using a noncollagenous bone matrix protein osteocalcin (OC) promoter to drive t
21 ity of Lrp5-/- osteoblasts to synthesize the bone matrix protein osteopontin after a mechanical stimu
22 ppresses bone formation in vivo and disrupts bone matrix protein synthesis by osteoblasts in vitro.
23 to expose osteoinductive or osteoconductive bone matrix proteins that should facilitate osteogenesis
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